8/7/2019 23369910-Pathophysiology-of-HCVD-DM2-CVD-Left-Basal-Ganglia

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PREDISPOSINGNon-Modifiable

AGE (loss of blood vesselelasticity)GENDERFAMILY (hereditary)Modifiable

LIFESTYLEDIET (high fat, sweet)

Increase fat deposit onthe walls of the blood

Atherosclesrosis

Pressure on the wall of blood

Some fragments joins in circulation

Goes to small vessels and clog (deeppenetrating arteries especially lacunar

Obstruction/ interruption of O2 supply dueto decreased blood supply

Lacunar infarct

Decrease oxygensupply to basal

INFARCTION

Insufficient insulinsecretion/ production of the

Glucose is not metabolizeby the body

Accumulation of glucose inthe blood stream

Increase viscosity of blood/sluggish flow of blood

Evaluate level ofblood glucose/increaseconcentration ofsugar in blood

Influences autonomic nervous

S/sx:CBGmonitoring:103-166mg/dL

Responsible for cerebellar

Also affects motor skills ofchewing & swallowing

Difficulty in chewing &swallowing could lead to

Causing weight loss

PRECIPITATINGDiabetes Mellitus

Narrowing of blood vessel

(coronary artery)

Decrease blood flowthrough the coronaryS/sx: BP: 140/90 mmHgDyspnea

Decrease blood flowto the heart

CORONARY ARTERY

Increase blood

Increase Peripheral Vascular

Severe elevation in BPw/o progressive target

HCVD

Hypertensive

Prognosis

If untreated:Furthercomplication

can occur: Ischemia

Anginapectoris

MI

Heartattack

CVA

If treated:

NursingManagement

DietManagement

PharmacologicMedication:(Aspirin, ACEinhibitor,Nitroglycerin forangina)

Recove

r

Death