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8/7/2019 23369910-Pathophysiology-of-HCVD-DM2-CVD-Left-Basal-Ganglia
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PREDISPOSINGNon-Modifiable
AGE (loss of blood vesselelasticity)GENDERFAMILY (hereditary)Modifiable
LIFESTYLEDIET (high fat, sweet)
Increase fat deposit onthe walls of the blood
Atherosclesrosis
Pressure on the wall of blood
Some fragments joins in circulation
Goes to small vessels and clog (deeppenetrating arteries especially lacunar
Obstruction/ interruption of O2 supply dueto decreased blood supply
Lacunar infarct
Decrease oxygensupply to basal
INFARCTION
Insufficient insulinsecretion/ production of the
Glucose is not metabolizeby the body
Accumulation of glucose inthe blood stream
Increase viscosity of blood/sluggish flow of blood
Evaluate level ofblood glucose/increaseconcentration ofsugar in blood
Influences autonomic nervous
S/sx:CBGmonitoring:103-166mg/dL
Responsible for cerebellar
Also affects motor skills ofchewing & swallowing
Difficulty in chewing &swallowing could lead to
Causing weight loss
PRECIPITATINGDiabetes Mellitus
Narrowing of blood vessel
(coronary artery)
Decrease blood flowthrough the coronaryS/sx: BP: 140/90 mmHgDyspnea
Decrease blood flowto the heart
CORONARY ARTERY
Increase blood
Increase Peripheral Vascular
Severe elevation in BPw/o progressive target
HCVD
Hypertensive
Prognosis
If untreated:Furthercomplication
can occur: Ischemia
Anginapectoris
MI
Heartattack
CVA
If treated:
NursingManagement
DietManagement
PharmacologicMedication:(Aspirin, ACEinhibitor,Nitroglycerin forangina)
Recove
r
Death