bojana gardijan 4th year march 16, 2010 mentor: a. Žmegač horvat
TRANSCRIPT
Bojana Gardijan4th year
March 16, 2010Mentor: A. Žmegač Horvat
destruction of heart tissue resulting from obstruction of the blood supply to the heart muscle
part of acute coronary syndrome, ACS non- STEMI
non ST-elevation MI STEMI
ST-elevation MI
diabetes hyperlipoproteinemia,
especially high LDL and low HDL
high blood pressure family history of
ischemic heart disease
obesity BMI>30 kg/m² age M>45, F>55 stress alcohol
a manifestation of coronary artery disease, also called ischemic heart disease
most common triggering event: disruption of an atherosclerotic plaque in an coronary artery clotting cascade sometimes results in total occlusion of the artery
chest pain (typically radiating to the left arm or left side of the neck)
shortness of breath (dyspnea) nausea, vomiting palpitations sweating anxiety
history and physical examination EKG CBC cardiac markers, preferably troponin T
(and complete biochemistry) differential diagnosis includes pulmonary
embolism, aortic dissection, pericardial effusion causing cardiac tamponade, tension pneumothorax, pancreatitis and esophageal rupture
MONA as soon as possiblemorphine, oxygen, nitrates, aspirin
clopidogrel, heparin, eptifibatide procedure of choice : PCI (percutaneous
coronary intervention) if unavailable: fibrinolysis
postinfarctial therapy: SAABstatins, ACEI, aspirin, beta blockers
varies greatly, depending on a person’s health, the extent of the heart damage and the treatment given
time to reperfusion is of great importance
a quick reaction saves lives
http://en.wikipedia.org/wiki/Myocardial_infarction
http://www.escardio.org