viruses, artificial intelligence, comets and ccr5-delta 32 deletion

“Pandemics result from the emergence of an influenza strain to which large numbers of the population have not been exposed. A new virus that can be transmitted readily from person to

person and that can cause human disease can potentially lead to an influenza pandemic. Highly pathogenic avian influenza A

(H5N1) has now fulfilled two of these three criteria. Although a probable transmission of H5N1 avian influenza from an

infected child to two close contacts has been reported, sustained human-to-human transmission has not been documented. However, the H5N1 viruses that have now become endemic in Asian domestic fowl are being spread by wild birds and appear

unlikely to be eradicable.

H5N1 viruses are expanding their mammalian host range, and sporadic human infections with high fatality rateshigh fatality rates continue

to occur in Vietnam, Thailand, and Cambodia. These events increase the alarming likelihood that there will be ample

opportunity for further adaptation of H5N1 to human hosts”.

1"The mutations needed for the emergence of a potential pandemic virus are likely to originate and be selected within infected human tissues," said Professor Dr Prasert Auewarakul from

Mahidol University, Thailand. "We analyzed specific molecules called haemagglutinin on viruses derived from fatal human cases. Our results suggest new candidate mutations that may allow bird

flu to adapt to humans."

"This study shows that the H5N1 virus is adapting each time it infects a human," said Professor Dr Auewarakul. "Such adaptations

may lead to the emergence of a virus that can cause a pandemic. Our research highlights the need to control infection and transmission to humans to prevent further adaptations."

1Pandemic Mutations In Bird Flu Revealed - ScienceDaily, July 2008:

http://www.sciencedaily.com/releases/2008/07/080708200647.htm

Reuters, 1 May, 2013,

"This is a very, very serious disease in those who have been infected. So if this were to become more widespread it would be an extraordinarily devastating outbreak,"

Peter Openshaw, director of the centre for respiratory infection at Imperial college London, told the briefing.

2 glycoproteins, Haemagglutinin (HA) and Neuraminidase (NA) both recognise Sialic acid

The neuraminidase (NA) creates an enzyme-destroying enzyme itself and removes the substrate (surface layer) Sialic acid from infected cells so

that newly made viruses are released to infect more cells

To stop infection, the primary goal is to stop or halt the membrane fusion receptor-binding by the haemagglutinin (HA) to Sialic acid. Sialic acid is

bound in all HA, and receptor-binding is species-specific

During virus replication, Neuraminidase (NA) removes Sialic acid from cellular glycoproteins and glycolipids. As a result, newly-assembled viruses are

prevented from binding to the infected cell surface and from aggregating with each other through Sialic acid and HA reactions. Instead, they are released

from the cell to infect new cells and spread the infection

Anti-bodies against Neuraminidase (NA) block the receptor-destroying activity and as a consequence, limit infection

Haemagglutinin (HA) has 16 sub-variants and are in 2 groups:

Neuraminidase (NA) has 9 sub-variants:

H1, H2, __ , __ , H5, H6, __ , H8, H9, ___ , ___ , H12, H13, ___ , ___ , H16

Group 1:

Group 2:

H3, H4, __ , __ , H7, __ , __ ,H10, H11, ___ , ___, H14 , H15

Group 1:

Group 2:

N1, __ ,__,N4, N5, __, __, N8

N2, N3,__, __, N6, N7

1FAO-OIE-WHO Technical Update: Page1-6, Current evolution of avian influenza H5N1 viruses: 7 September 2011

Epidemiological analysis shows that the virus is still circulating in several countries in Asia and only in Egypt for Africa. Egypt and Indonesia have declared themselves endemic for H5N1 to the OIE

FAO considers H5N1 viruses endemic in poultry in five countries; Bangladesh, China, Egypt, Indonesia and Viet Nam

Globally, the number of reported outbreaks of H5N1 in poultry and wild birds increased since mid-2008. Between the 2009-2010 and the 2010-2011 time periods, the main increase occurs in Asia, with

a significant rise in number of events in some Asian countries and relatively large outbreaks in others

Obese people are more susceptible to complications with A[H1N1] and A[H5N1] including Pulmonary Emboli (blood clots) and DVT (deep vein

thrombosis) which can break off and move into the lungs. Also expresses in people with a history of cancer, childbirth, heart attack, heart surgery,

stroke, child birth control pills

Breathing difficulties linked to hypoxia and A[H3N2]. Cases of septic shock to diabetes and multi-organ failure such as renal or pulmonary

alveoli problems, and most activities that regulate breathing and oxygen regulation by the medula oblagata. Septic shock has high fatality rates

with young and old people, depending on the immune system, previous surgeries and “pre-existing conditions”.

AST (Aspartate Aminotransferase) tests can be requested for liver damage. CK levels will be high (males 14-20 Units Per Liter; females 10-36 Units Per/L)

As of 31 August 2011, 565 confirmed human cases of infection with avian influenza H5N1 virus from 15 countries were reported to WHO since 2003.

Of these, 331 died (case fatality rate: 58.6%).

In those Asian countries that FAO considers endemic for H5N1 in poultry, the reported incidence of human cases has remained stable or has

decreased since 2010

1918/1919 1957/1958 1967/1968 1977 2001 2009

A[H1N1]

A[H3N2]

A[H2N2]

*A[H5N1]

A[H1N1]

HA & NA similar

?

N2 Acquired

Higher morbidity/mortalityPandemic - Estimated 50 Million dead worldwide

Adamantanes Zanamivir (Relenza) ineffective and

Oseltamivir (Tamiflu) ineffective to A[H1N1] very early (2003-2004).

Deemed “unreliable” as Neuraminidase inhibitor. The *2004 avian H5N1 viruses are resistant to the adamantanes

2002-2007

Virus resistant to NAI Amantadine and

Rimantadine April 2009

B viruses transmission higher 2004-2005 in Japan

A[H5N1] Hong Kong 1997 outbreak

PorcineAvian

“Quadruple re-assortment”

Source InformationFrom the Departments of Pediatrics and Microbiology and Immunology, Weill Medical College of Cornell University, New York.

1Emergence of Avian Influenza A(H7N9) Virus Causing Severe Human Illness — China, February–April 2013 - Early Release May 1, 2013 / 62; 1-6

The genetic sequences of all viruses tested showed a known marker of resistance to the adamantanes, indicating that, although these drugs

(amantadine and rimantadine) are licensed for use in the United States, they should not be prescribed for patients with H7N9 virus infection.

1The genetic sequence of one of the publicly posted H7N9 viruses (A/Shanghai/1/2013) contains a known marker of NAI

(Neurominadase) resistance

The clinical relevance of this genetic change is under investigation but it serves as a reminder that resistance to antiviral drugs can occur

spontaneously through genetic mutations or emerge during antiviral treatment.

1 Emergence of Avian Influenza A(H7N9) Virus Causing Severe Human Illness — China, February–April 2013 - Early ReleaseMay 1, 2013 / 62; 1-6

1Viral Haemorrhagic Fevers (VHFs) include Lassa fever, Rift Valley fever, Marburg and Ebola haemorrhagic fevers, Crimean-Congo Haemorrhagic

Fever (CCHF) and Yellow Fever. Humans initially contract an infection with a haemorrhagic fever virus through exposure to rodents or insects (for

Ebola and Marburg VHFs, the natural reservoir is unknown). VHFs are often accompanied by bleeding, reflecting the widespread

presence of the virus throughout the patients body. As a result, the blood, urine, vomitus, pus, stool, semen and saliva from the VHF patient are

infectious.

SARS (Severe Acute Respiratory Syndrome) ARS (Acute Respiratory Syndrome) and nowMERS (Middle East Respiratory Syndrome)

NOROVIRUS/CORONAVIRUS

Avian Flu strains H1N1, H1N2, H5N1, H3N2 and H7N9 are possibly re-assorting and re-combining with other variants

1Infection Control for Viral Haemorrhagic Fevers, CDC (CENTERS FOR DISEASE CONTROL AND PREVENTION) U.S. DEPARTMENT OF HEALTH & HUMAN SERVICES, Public Health Service, World Health Organization, in the African Health Care Setting, p3 (PDF)

Middle Eastern Respiratory Syndrome Coronavirus – MERS-CoV

1She added that… it would be the first case registered in the State of Kuwait after Saudi Arabia, Qatar, Bahrain, Oman and the United Arab Emirates, knowing that the patient traveled to Saudi

Arabia in the recent period. (Updated: 12/11/2013 09:40:20 PM)

1http://www.araa.com/article/77104#sthash.S6llSyDg.dpuf

On 7 Nov 2013 the Saudi Ministry of Health reported a new case: a 43-year-old male from Jeddah, who developed symptoms on 27 Oct 2013. He sought medical treatment on 3 Nov 2013. He is currently in an intensive care unit. The patient does not have any underlying chronic disease. He has no recent travel history outside of Jeddah. He had significant contact with animals

but no contact with a known positive human case.

1Transmission of Avian Influenza Virus (H3N2) to DogsDaesub Song,*1 Bokyu Kang,*1 Chulseung Lee,* Kwonil Jung,† Gunwoo Ha,‡ Dongseok Kang,‡

Seongjun Park,§ Bongkyun Park,§ and Jinsik Oh‡ during May, August & September 2007

Canine influenza occurred in an animal clinic in which all 13 dogs housed in a shelter facility were found to be infected with the same virus; their clinical signs were nasal discharge, cough, and high fever. 47 (90%) of 52 were seropositive for canine influenza virus (CIV H3N2) at the first sampling and that 100% were

seropositive by the second sampling.

Discussion

“Because all genes of the canine isolates were of avian influenza virus origin, we concluded that the entire genome of the avian

influenza virus had been transmitted to the dogs. Transmission of avian influenza A virus to a new mammalian species is of great

concern, because it potentially allows the virus to adapt to a new mammalian host, cross new species barriers, and acquire pandemic

potential”. (ibid, p744)

1Emerging Infectious Diseases • www.cdc.gov/eid • Vol. 14, No. 5, May 2008

1Interestingly, recent reports have shown that the pandemic A/H1N1/09 human influenza virus can also infect dogs (Dubovi, 2010). In recent times, feline species, especially cats,

have become a noticeable host for influenza infections. They are susceptible to infections of avian influenza H5N1 virus and pandemic H1N1 influenza

Equine H3N8, avian H3N2, avian H5N1 and pandemic human H1N1 influenza viruses have been identified in dogs, while cats had been shown to be susceptible to avian H5N1 and pandemic human H1N1 influenza viruses (Harder & Vahlenkamp, 2010;

Songserm et al., 2006).

In this study, cats were susceptible to CIV H3N2 infection. Thus, cats may act as apotential intermediate host for the transmission of CIV H3N2 among feline and

canine species, possibly resulting in endemic establishment of the virus in companion animals. This raises a public health concern.

1Journal of General Virology (2011), Issue 92, 2350–2355


1Interspecies transmission of the canine influenza H3N2 virus to domestic cats in South Korea, 2010

Canine species were considered to be the new natural hosts for this virus. However, at the beginning of 2010, influenza-like respiratory signs, such as dyspnoea, were also observed among cats as well as in dogs in an animal shelter located in Seoul,

South Korea. The affected cats showed 100% morbidity and 40% mortality.

The eight viral genes isolated were almost identical to those of the canine influenza H3N2 virus, suggesting interspecies transmission of canine influenza H3N2 virus to the cat

(A/canine/Korea/GCVP01/07 and A/feline/Korea/01/2010 – both H3N2)

Our study shows, for the first time, that cats are susceptible to canine influenza H3N2 infection, suggesting that cats may play an intermediate host role in

transmitting the H3N2 virus among feline and canine species, which could lead to the endemic establishment of the virus in companion animals.

Canine influenza is caused by two subtypes of influenza A virus: H3N8 and H3N2. The H3N8 canine influenza virus (CIV) was first identified in dogs in the USA in

2005, and is known to originate from equine H3N8 (horse) influenza viruses.

*New Slide added March 28

As this presentation is being continually crafted and updated, it should be noted that the author was/is expecting a Tuberculosis

outbreak of some form and to some degree to appear, but not via transference from

domestic cats. As has been shown previously, although dogs and cats are now reservoirs and hosts of pathogenic viruses such as

avian flu (namely H3N2), Tuberculosis was not expected to be jumping into domestic

pets. The meat industry in ‘the UK’ has been allowing TB infected meat to pass through into consumer food chains. This finding will require more in-depth research on how this

may connect to Pseudotuberculosis and ancient pathogen suitability or like-attributes.

Several outbreaks of avian influenza infection have occurred in mammals.Influenza virus (H7N7) of avian origin was isolated from the lungs and brains of dead seals. In addition, it was replicated to high titers in ferrets, cats, and pigs and caused

conjunctivitis in humans (1981)

Avian origin influenza virus (H4N5) was reported as the cause of infection and death in harbor seals along the New England coastline

Avian origin influenza (H5N1) infection was identified in a dog after ingestion of a duck infected with subtype H5N1 during an outbreak in Thailand in 2004

Outbreaks of hemorrhagic pneumonia caused by equine influenza virus (H3N8) were noted in racing dogs, and a human influenza virus (H3N2) was isolated from

dogs (1976-1980)

In our study, virologic, serologic, pathologic, and phylogenetic analyses showed cross-species infection of an entire avian influenza A virus (H3N2) to another

mammalian species, dogs. Evidence of avian influenza virus infection in pet dogs raises the concern that dogs may be become a new source of transmission of

novel influenza viruses, especially where avian influenza viruses are circulating or have been detected. (ibid, p746)

RV antigen (glycoprotein G) is highly immunogenic, especially in the presence of adjuvant. Therefore, the DOI (Duration Of Immunity) in dogs vaccinated

with two initial doses, 12 months apart, is expected to be 3 yr (when using a 3 yr rabies vaccine) in dogs that are 1 yr of age.

The first modified live Canine Distemper Virus (CDV) vaccines were made in the 1950s and 1960s, some vaccines were highly virulent, causing

distemper-like disease, including encephalitis, in a high percentage of vaccinated dogs

Infectious vaccines must infect the host’s cells to immunize. These vaccines are the most effective because they can provide the same types of

immunity (cellular, humoral, systemic, and local) that are produced by natural exposure (i.e., immunity after recovery from infection or disease). However, the vaccine organisms are *attenuated and will not cause disease

*Live (attenuated):1. Having life; alive.2. Capable of replicating in a host's cells.3. Containing living microorganisms or viruses capable of replicating in a host's cells.(attenuated): Alive but weakened; an attenuated microorganism can no longer produce diseaseEtymology: L, attenuare, to make thin pertaining to the dilution of a solution or the reduction in virulence or toxicity of a microorganism or a drug by weakening it.

The first canine vaccine developed and licensed in 1997 using *recombinant DNA technology (*Genetic Engineering) was the canarypox-

vectored recombinant CDV(rCDV) vaccine.

Current Canine ParvoVirus, type 2 (CPV-2) vaccines contain either CPV-2 or the CPV-2b variant. Vaccines from all the major manufacturers have been shown to provide sustained (several years) protection from all the

current CPV-2 variants (CPV-2a, b, and c)

The original Canine AdenoVirus, type 1 (CAV-1) vaccines, (no longer available in the US or Canada), caused allergic uveitis and other allergic reactions in a high percentage of dogs; therefore, CAV-1 vaccines were replaced in the US and Canada by the safer, but equally or more effective,

CAV-2 vaccines. CAV-2 vaccines are used to provide immunity to CAV-1 virus, the cause of canine infectious hepatitis. CAV-2 is a virus that causes and contributes to canine infectious

respiratory disease complex

Canine Parainfluenza Virus [CPiV]) could also be a MLV – Modified Live Virus

CANINES AND TRAN-SPECIES TRANSMISSION OF H3N2 & H3N8

Formalin: a clear solution of formaldehyde in water. A 37% solution is used for fixing and preserving biologic specimens for pathologic and histologic examination. (source: http://medical-dictionary.thefreedictionary.com/formalin)

As the name “noninfectious” implies, these vaccines do not infect the host to produce new antigen. Thus, they must contain adequate amounts of antigen to immunize. Because the antigen alone may not be adequate to immunize a dog, many of the noninfectious vaccines must also contain adjuvant. Adjuvants include a wide variety of substances that maintain or depot the antigen as well as

stimulate an inflammatory response to provide a more robust immune response to the vaccine antigens. This increased nonspecific stimulation of the immune system caused by adjuvants is required to induce a protective

response to antigens ..such as Formulin [sic]

Both infectious and noninfectious vaccines are important and required in every canine vaccination program. However, it is important not to mix

noninfectious vaccines with infectious vaccines in the same syringe, unless specified by the manufacturer

Administration of infectious vaccine to dogs, 6 wk of age, even in the absence of MDA (Maternally Derived Antibody) , is not recommended.

MDA is the most common reason early vaccination fails to immunize. In general, MDA is more effective at interfering with infectious vaccines

than noninfectious vaccines

1The World Organisation for Animal Health in support of the Biological and Toxins Weapon Convention and acts against bioterrorism, p1; 2 p2; 3 p3

1The World Organisation for Animal Health (OIE) has on more than one occasion stated its commitment to work within the framework of the Biological and Toxins Weapon Convention (BTWC).

The OIE has also with the support of the FAO (Food and Agricultural Organisation of the United Nations) and the WHO (World Health Organisation) .. mandated by the World Trade Organisation (WTO) ..to strengthen the ability and capacity of the national implementation of early detection by veterinary services as a “buffer” to early detect, diagnose and respond to incidental or deliberate disease incursions.

The OIE fully realizes and acknowledge the fact that 60% of human pathogens are zoonotic; that 80% of animal pathogens are multi-host; that 80% of pathogenic agents having a potential bioterrorist use are zoonotic

2 The OIE has, by linking this initiative to its overall aim to establish awareness and acceptance of the delivery of veterinary services as an international public good, elicited major financial support for this project from the World Bank and other donors, including some of the more financial affluent Member Countries of the OIE

The OIE as the worldwide observatory for animal health, has established an International Early Warning System

3 All [167] Member Countries of the OIE are obliged to inform the OIE Central Bureau within 24 hours of the occurrence of any unusual epidemiological event – such as for instance an act of bioterrorism involving the use of animal pathogens

3 The OIE also has a network of 180 Reference Laboratories and Collaborating Centres that in addition serves as the eyes and ears of the Central Bureau to compliment and verify information on animal diseases

1An important component of the system (GLEWS or GLobal Early Warning System) is the linkage with the human/animal pathogen interface by sharing complimentary information with the WHO on zoonotic diseases and animal diseases that could endanger human health for example in the unfortunate event of deliberate spread of animal pathogens

The OIE/FAO designated Reference Laboratories and Collaborating Centres will constitute an important contributor in the GLEWS for providing specific analysis and modeling trends.

ConclusionThe OIE shares with all its Member Countries the common concern about the potential devastating effects inherent to any act of deliberate misuse or spread of biological pathogens that can affect human and animal health and food and animal production. The OIE is committed to take hands with its colleagues in the FAO and WHO to enhance the aims and objects of the Biological and Toxins Weapon Convention by assisting countries to acquire the ability and expertise to apply the international standards, guidelines and recommendations of the OIE to cope with not only such unfortunate emergencies but also with any major animal and zoonotic disease that might affect animal or human health.

1The World Organisation for Animal Health in support of the Biological and Toxins Weapon Convention and acts against bioterrorism, p3

The BWC (Biological Weapons Convention) thus supplements the prohibition on use of biological weapons contained in the 1925 Geneva Protocol and to which the United Kingdom signed 10 April 1972 and ratified 26 March 1975 as 135th states party member signee.

(CONVENTION ON THE PROHIBITION OF THE DEVELOPMENT, PRODUCTION AND STOCKPILING OF BACTERIOLOGICAL (BIOLOGICAL) AND TOXIN WEAPONS AND ON THEIR DESTRUCTION) - Opened for signature: 10 April 1972, entered into force: 26 March 1975

ARTICLE I - CONVENTION ON THE PROHIBITION OF THE DEVELOPMENT, PRODUCTION AND STOCKPILING OF BACTERIOLOGICAL (BIOLOGICAL) AND TOXIN WEAPONS AND ON THEIR DESTRUCTION) - Opened for

signature: UK signed 10th April 1972, entered into force: 26th March 1975

Each State Party to this Convention undertakes never in any circumstance to develop, produce, stockpile or otherwise acquire or retain: (1) Microbial or other biological agents, or toxins whatever their origin or method of production, of types and in quantities that have no justification for prophylactic, protective or other peaceful purposes; (2) Weapons, equipment or means of delivery designed to use such agents or toxins for hostile purposes or in armed conflict.

ARTICLE VIIINothing in this Convention shall be interpreted as in any way limiting or detracting from the obligations assumed by any State under the Protocol for the Prohibition of the Use in War of Asphyxiating, Poisonous or Other Gases, and of Bacteriological Methods of Warfare, signed at Geneva on June 17, 1925.

ARTICLE XIII(1) This Convention shall be of unlimited duration. (2) Each State Party to this Convention shall in exercising its natural sovereignty have the right to withdraw from the Convention if it decides that extraordinary events, related to the subject matter of the Convention, have jeopardized the supreme interests of its country. It shall give notice of such withdrawal to all other States Parties to the Convention and to the United Nations Security Council three months in advance. Such notice shall include a statement of the extraordinary events it regards as having jeopardized its supreme interests .

ARTICLE XIV (6) This Convention shall be registered by the Depositary Governments pursuant to Article 102 of the Charter of the United Nations. The United Kingdom of Great Britain and Northern Ireland is the designated Depository Government for the Convention

1“We must form coalitions for change, often with partners well beyond the precincts of officialdom”.

1“We the People’s: The Role of the UN in the 21st Century” by Kofi A. Annan, Secretary-General of the United Nations,

Finally, the world desperately needs a vaccine against HIV. Of the $2 billion spent on research for the treatment of AIDS to date, only $250 million has been spent on creating vaccines, few of which are potentially useful for poor countries, where about 95 per cent of HIV infections occur. The Global Alliance for Vaccines and Immunization (GAVI, formed in 1999) serves as a model of what such partnerships (“coalitions”) can achieve

The Alliance is a creative coalition of national governments, development banks, business leaders, philanthropic foundations, the World Health Organization, the World Bank group and UNICEF

In January 2000, GAVI launched the Global Fund for Children’s Vaccines at the World Economic Forum in Davos. The Fund, assisted by a $750 million grant from the Bill and Melinda Gates Foundation, will provide resources to expand the reach of existing vaccines and to strengthen the infrastructures necessary to deliver vaccines in the poorest countries. The Fund will also support research for developing new vaccines.

[p.69] Therefore, I am asking all relevant United Nations entities to provide the necessary technical assistance that will make it possible for every willing state to participate fully in the emerging global legal order.

[p.74] Only by these means can we become a global public trust for all the world’s peoples

[p.79] I invite all governments that have not done so to sign and ratify the various conventions, covenants and treaties which form the central corpus of international law.

“The injection of antiviral drugs gives monkeys 100% protection” as a first line is most useful. Great for monkeys. But we, man, are not monkeys. As an opening line to convince the reader of its efficacy, it’s a detrimental one. The

information that has been collated and presented here is to allow you, the viewer, to discern and use common sense when reading ‘tabloid journalism’ when counter-balanced with the scientific opinions, data, results, and conclusions

which may (and usually does) conflict with the ‘news’, or the ‘expert’ who may be writing the articles.

Article 31. In all actions concerning children, whether undertaken by public or private social welfare institutions, courts of law, administrative authorities or legislative bodies, the best interests of the child shall be a primary consideration.

The United Nations Convention on the Rights of the ChildAdopted and opened for signature, ratification and accession by General Assembly

Resolution 44/25 of 20 November 1989entry into force 2 September 1990, in accordance with Article 49

Article 91. States Parties shall ensure that a child shall not be separated from his or her parents against their will,except when competent authorities subject to judicial review determine, in accordance with applicable law and procedures, that such separation is necessary for the best interests of the child.

Article 201. A child temporarily or permanently deprived of his or her family environment, or in whose own bestinterests cannot be allowed to remain in that environment, shall be entitled to special protection andassistance provided by the State.

Article 21States Parties that recognize and/or permit the system of adoption shall ensure that the best interests of the child shall be the paramount consideration..

The Articles clearly get worse and Inter-Country Adoptions are pushed more so than the actual “care” of the Child.

The UN is a different story.. and for another presentation!!

This presentation is purely to demonstrate that there is a larger Agenda at play. Most peoples don’t quite see “the dots” and the connections to the UN SYSTEM as an entity. But, as you’ll see with the

coming slides, there is other sub-agendas that folks should be made aware of.

So, let us continue….

“.. the best interests (of the child)” .. Hmm..

VACCINES

Recommended Immunization Schedulesfor Persons Aged 0 through 18 Years

UNITED STATES, 2012

This schedule includes recommendations in effect as of December 23, 2011. Any dose not administered at the recommended age should be administered at a

subsequent visit, when indicated and feasible.

The use of a combination vaccine generally is preferred over separate injections of its

equivalent component vaccines

The Recommended Immunization Schedules forPersons Aged 0 Through 18 Years are approved by

the

Advisory Committee on Immunization Practices(www.cdc.gov/vaccines/recs/acip)

American Academy of Pediatrics(http://www.aap.org)

American Academy of Family Physicians(http://www.aafp.org)

1Prevention and Control of Influenza with VaccinesRecommendations of the Advisory Committee on Immunization

Practices (ACIP), 2010 Dosing guidelines in the 2012 ACIP influenza vaccine recommendations

The 2010 influenza recommendations include new and updated information. Highlights of the 2010 recommendations include:

1) a recommendation that annual vaccination be administered to all persons aged 6 months for the 2010–11 influenza season;

2) a recommendation that children aged 6 months – 8 years whose vaccination status is unknown or who have never received seasonal influenza vaccine before (or who received seasonal vaccine for the first time in 2009–10 but received only 1 dose in their first year of vaccination) as well as children who did not receive at

least 1 dose of an influenza A (H1N1) 2009 monovalent vaccine regardless of previous influenza vaccine history should receive 2 doses of a 2010–11 seasonal

influenza vaccine (minimum interval: 4 weeks) during the 2010–11 season;

3) a recommendation that vaccines containing the 2010–11 trivalent vaccine virus strains A/California/7/2009 (H1N1)-like (the same strain as was used for 2009

H1N1 monovalent vaccines), A/Perth/16/2009 (H3N2)-like, and B/Brisbane/60/2008-like antigens be used;

1Morbidity and Mortality Weekly Report , August 6, 2010 / Vol. 59 / No. RR-8 http://www.cdc.gov/mmwr/pdf/rr/rr5908.pdf

AGRIFLU is a sterile clear aqueous suspension and is formulated to contain a total of 45 mcg hemagglutinin (HA) per 0.5-mL dose in the recommended ratio of 15 mcg HA of each of the following three influenza virus strains

recommended for the 2010/2011 influenza season:

Inactivated influenza vaccines are standardized to contain the haemagglutinin of influenza virus strains (typically two type A and one type B), representing the influenza viruses likely to be circulating in the

United States in the upcoming winter.

AGRIFLU - Novartis Vaccines and Diagnostics, Inc

A/California/7/2009 (H1N1) NYMC X-181 ; A/Victoria/210/2009 (H3N2) NYMC X-187 (an A/Perth/16/2009-like virus);

and B/Brisbane/60/2008

Influenza vaccines. (Minimum age: 6 months for trivalent inactivated influenza vaccine [TIV]; 2 years for live, attenuated influenza vaccine [LAIV])

For children aged 6 months through 8 years:*For the 2011–12 season, administer 2 doses (separated by at least 4 weeks) to those who did not receive at least 1 dose of the 2010–11 vaccine. Those who received at least 1 dose of the 2010–11 vaccine require 1 dose for the 2011–12 season.**For the 2012–13 season, follow dosing guidelines in the 2012 ACIP influenza vaccine recommendations

For most healthy children aged 2 years and older, either LAIV or TIV may be used. However, LAIV should not be administered to some children, including1) children with asthma, 2) children 2 through 4 years who had wheezing in the past 12 months, or 3) children who have any other underlying medical conditions that predispose them to influenza complications

For most healthy, non-pregnant persons, and aged between **2 & 49, either LAIV or TIV may be used, except LAIV should not be used for some persons, including those with asthma or any other underlying medical conditions that predispose them to influenza complications.

Source: The Recommended Immunization Schedules for Persons Aged 0 Through 18 Years PDF

WHO CHOOSES WHAT STRAIN IN MOST EFFICACIOUS?

1 The Vaccines and Related Biological Products Advisory Committee provides advice on vaccine strain selection to the Food and Drug

Administration (FDA), which selects the viral strains to be used in the annual trivalent influenza vaccines

1 Morbidity and Mortality Weekly Report , Page 5 & 6 http://www.cdc.gov/mmwr/pdf/rr/rr5908.pdf

The 2010–11 trivalent vaccines will contain: A/California/7/2009 (H1N1)-like, A/Perth/16/2009 (H3N2)-like &

B/Brisbane/60/2008-like antigens. The influenza A (H1N1) vaccine virus is derived from a 2009 pandemic influenza A (H1N1) virus

2 On February 23, 2012 the World Health Organization (WHO) recommended that the Northern Hemisphere's 2012-2013 seasonal influenza vaccine contain

the following three vaccine viruses:

A/California/7/2009 (H1N1) pdm09-like virus;A/Victoria/361/2011 (H3N2)-like virus;

B/Wisconsin/1/2010-like virus (from the B/Yamagata lineage of viruses).

2 http://www.cdc.gov/flu/about/qa/vaccine-selection.htm (PDF)

While the H1N1 virus is the same, the H3N2 and B vaccine viruses are different from those that were selected for the Northern Hemisphere for the

2011-2012 influenza vaccine.

Fluzone is standardized according to United States Public Health Service requirements and is formulated to contain HA of each of the following three influenza strains recommended for the 2012-2013

influenza season:

A/California/07/2009 (H1N1) NYMC X-179A A/Victoria/361/2011 (H3N2) IVR-165 and

B/Texas/6/2011 (a B/Wisconsin/1/2010-like virus)

FLUZONE (TIV) - SANOFI PASTEUR

VACCINE MANUFACTURER PRODUCT NAME AND TYPE

AGE TO ADMINISTER

INFLUENZAA/CALIFORNIA/7/2009pdm90v

GSK FLUARIX (TIV)

Minimum 3 years old


CHIRON FLUVIRIN (TIV)

Purified surface antigen. Minimum 4

years


SANOFI FLUZONE (TIV)

Subvirion. Minimum age (multidose vial) = 6 mo. Age

range (0.25mL prefilled syringe) = 6 through 35 mo.

Minimum age (0.5mL prefilled syringe) = 3 years.


SANOFI FLUZONE (High dose)

Licensed for over 65’s


GSK *FLULAVAL (TIV)

Minimum 18 years old

INFLUENZAA/CALIFORNIA/7/2009pdm90

CSL *ALFLURIA (TIV)

Minimum 6 months old


NOVARTIS *AGRIFLU (TIV) Minimum 18 years old


MEDIMMUNE *FLUMIST (LAIV)

No thimerasol but formaldehyde used

in production

Website viewed and screenshot taken Friday, March 7, 2014

The National Harm Service, .. Sorry, the National (now Privatised) Health Service contradicts itself on its own front page, so one could assume that the NHS as an entity doesn’t care if it’s blatantly lying to the public when it

promotes AstraZeneca’s vaccines. The NHS information clearly opposes what the vaccine makers and the inserts declare. Yet, this information remains on the website for all to read and believe. They also hyperlink

to the WHO and the UN and to the Bill and Melinda Gates Foundation as references of ‘authority’ somehow. We urge you, the viewer, to confirm these facts, and investigate what these entities are driving towards. The UN is at the

heart of these problems. We supply website screen shots to exemplify the glaringly obvious contradictions and hypocrisy when researching these topics and by searching and referencing the source declarants to these ‘facts’ and

‘safety assurances’. Below are such examples of ineptitude…

http://www.sciencedirect.com/science/article/pii/S0264410X1201345X

It may seem curious to some, but it is worth noting that on the previous slide, when accounting of how safe the flu jab is, the article cites a hyperlink to ‘studies have shown that the flu vaccine is safe during any stage of pregnancy’. That hyperlink will take the viewer to

this biased article (scientific study it is not) involving Bill and Melinda Gates and the WHO, again.

As of today, FLUENZ, manufactured by GlaxoSmithCline is NOT to be administered to

pregnant women. Why would the NHS blatantly ignore that advisement, and why then link to a ‘study’ involving

Bill Gates which infers the opposite with the words,

“For the first time, SAGE recommended pregnant women should be made the highest priority for

inactivated seasonal influenza vaccination” when it’s clear the ingredient list and the side effects and dangers

associated with vaccinations oppose this statement.

ELSEVIER is also an entity that is affiliated with the UN and its organs which the NHS supplies a

hyperlink to from its home page.

Consider the issues regarding Genetically Modified Organisms and ask yourself a question – Do you, as mothers and fathers, really wish to inject your younglings with GMO’s too, along with adjuvants and toxins? If you love them as proclaimed, no one in their right mind could conceive injecting and infecting their

beloved ones with foreign substances, poisons and toxic chemicals, some of which have been cited, explored and disclosed within this presentation (and will be explored further later on) as the

correct decision to make. The adverse effects, both short term and long term should be considered always. These are ‘products’ and they can seriously compromise your health and those of loved

ones.

http://healthland.time.com/2010/05/14/male-birth-control-stopping-sperm-with-ultrasound/

The project is one of 10 to receive grants toward the goal of creating new technologies for contraception. Other projects

geared toward men include a male contraceptive pill that researchers say would work by limiting the maturation of

sperm, and research into the specific chemical compounds in the vagina that guide sperm to egg — which researchers hope to recreate in the lab and potentially use to “disrupt” sperm

navigation en route to the egg. (Earlier this year, researchers at the University of California, San Francisco uncovered clues

about how pH levels impact how sperm swim, and expressed hope that further research in this arena could yield possibilities for

male contraception as well.)

http://www.cdc.gov/vaccines/pubs/pinkbook/downloads/appendices/D/vacc_admin.pdf

- Vaccine Safety & Risk Communication -

There have been safety concerns about vaccines since the 18th century when the first smallpox vaccination campaigns began. Specific vaccine concerns have changed through time. An

increasing number of parents, even those who support vaccination, are beginning to raise vaccine safety concerns. Parents/guardians and patients are exposed to information about vaccines

through the media, internet, family members, and friends. Some of this information is inaccurate and misleading.

• displaying a positive attitude through facial expressions, body language, and comments;

[i.e. give the pretense you give a sh*t about anything you’re about to do, due to ignorance]

• using a soft and calm tone of voice;

[as the inoculator speaks softly, “this is not going to kill you at some future point!”]

• making eye contact, even with small children;

[Hypnotic “look into my eyes and trust me, even though I’ve never read the ingredients and have no idea what’s even in the vaccine that I’m going to intravenously inject into your body ”]

• explaining why vaccines are needed (e.g., “this medicine will protect you from getting

sick” or “this shot is a shield to protect your body against infection”); [“unlikely to”] ; and

• being honest and explaining what to expect (e.g., do not say that the injection will not hurt).

[or, “this may have toxins and known carcinogens in it which may harm you which I’m about to infect/inject you with. Fingers crossed you won’t faint!”]

Simple strategies that can be used by both parents and providers to make the process of receiving vaccines easier include:

Always recall to mind that any ‘ingredient’ that is contained within a vaccine is artificially synthesized and human beings are not animals (although, we are seemingly perceived as lower animals to some of the ‘expert’ scientific class). We are conscious beings and not mice, or rat, nor monkey. Mercury and aluminum (derived from alum) does not belong in the live body. We have a wonderful endocrine and immune system that deals with foreign invaders very well without the

aid of pharmacological interventions and their ‘wonder drugs’. All vaccines are ‘products’, saleable products, as a business sells their wares, and are usually endorsed by entities that have conflicts of interest (when researched). But vaccines are not a TV or a games console that you can return if you’re not fully satisfied with it, or the side effects caused by your willful consent and decision to purchase that ‘safe’ vaccine product, and the known risks that

accompany it. Nor can one force a product, especially an unsafe one, upon another, even if the seller convinces you their product is what you need. Buyer beware! As the fear pervades, some entities may insist that if you don’t buy (into) that ‘faith’, and don’t have a vaccination, you may somehow be deemed ‘irresponsible’. “Herd Immunity” ‘they’ call it. Beware

the propaganda on these matters. Vaccines are still drugs and do contain poisons, albeit it, they may currently hold the status of ‘legal’. No vaccine is ‘safe’. The list of side effects should obviate those contentions.

Concerning adjuvants such as aluminum. The sole reason GACVS was created in 1999 by the WHO was due to a side effect of a particular vaccine that expressed itself primarily in France called MMF (Macrophagic MyoFasciitis) and

held the qualities of a flesh-eating (necrotic) disease.

Review of the scientific evidence

1“The evidence presented demonstrated the existence of a distinctive histopathological entity called macrophagic myofasciitis, characterized by persistent focal accumulation in the deltoid

muscle of densely packed PAS – positive macrophages with osmiophilic crystal inclusions composed of aluminium, and evidence of a focal chronic inflammatory reaction. There are

supporting data showing a comparable transient lesion after intramuscular (IM) injection of aluminium- containing vaccines in experimental animal models.”

115 October 1999 About GAVCS; Macrophagic myofasciitis and aluminium-containing vaccines – WER Report (pdf)

In other words, the injection site for these particular vaccinations displayed an unusual immunological or inflammatory reaction to vaccines containing aluminium in the deltoid muscles where the needle was plunged intramuscularly.

VACCINE MANUFACTURER PRODUCT NAME AGE TO ADMINISTER

DTaPDiptheria Tetanus

Perussis

SANOFI DAPTACEL Tetanus & diphtheria toxoids and acellular Pertussis

(whooping cough)


Perussis

GSK INFARIX Tetanus & diphtheria toxoids and acellular Pertussis (wc)


Perussis

SANOFI TRIPEDIA Tetanus & diphtheria toxoids and acellular Pertussis (wc)

DTaP-IPVInactivated Polio Virus

GSK KINRIX Licensed for 5th (DTaP) and 4th (IPV) booster at 4-6 years

DTaP-HepB-IPV GSK PEDIARIX Licensed for doses at 2, 4, & 6 months (through 6 years of

age). Not licensed for boosters.

HPV4Human Papilloma Virus

MERCK GARDASIL Quadrivalent (types 6, 11, 16, 18). Licensed for males and females 9 through 26

years

HPV2Human Papilloma Virus

GSK CERVIRAX Bivalent (types 16, 18). Licensed for females 10

through 26 years

POLIO SANOFI POLIO TRIVALENT TYPE 1,2 & 3

1st vaccine: Hepatitis B (HepB) vaccine. (Minimum age: birth)At birth: • Administer monovalent HepB vaccine to all newborns before

hospital discharge

*The 2nd dose should be administered at age 1 to 2 months. Monovalent HepB vaccine should be used for doses administered before age 6 weeks.

*Infants who did not receive a birth dose should receive 3 doses of a HepB containing vaccine starting as soon as feasible. Administration of a total of 4 doses of HepB vaccine is permissible when a combination vaccine containing HepB is administered after the birth dose

2nd vaccine: Rotavirus (RV) vaccines. (Minimum age: 6 weeks for both RV-1 [Rotarix] and RV-5 [Rota Teq])

*The maximum age for the first dose in the series is 14 weeks, 6 days; and 8 months, 0 days for the final dose in the series. Vaccination should not be initiated for infants aged 15 weeks, 0 days or older

3rd vaccine: Diphtheria and tetanus toxoids and acellular pertussis (DTaP) vaccine (whooping cough). (Minimum age: 6 weeks)

*The fourth dose may be administered as early as age 12 months, provided at least 6 months have elapsed since the third dose.

Rotavirus vaccines (RV1/Rotarix, RV5/RotaTeq) and oral typhoid (TY21a/Vivotif) are the only U.S.-licensed vaccines that are administered by the oral route. RV1/Rotarix requires reconstitution prior to oral administration.

With these ‘medicines’ from manufacturers such as GlaxoSmithCline (GSK), the warnings are clearly stated above. The question will arise – does your ‘Health

Care Professional’ or doctor profess to know how dangerous a vaccine can be for

a pregnant woman when the vaccine insert states otherwise? Maybe it would

be prudent to change your doctor if they recommend you place toxins into your body to ‘protect’ an unborn baby

on its arrival into this plane of existence.

We could explore all the UK alternative products to that of the American vaccines, but the concerned viewer will do their own investigations on Engerix and Fendrix. We are simply pointing to the obvious

problems and side-effects of these “legal drugs” as the following slide elucidates…

Initial Hepatitis B risk

Engerix B contains:

• hepatitis B virus surface antigen• sodium chloride• disodium phosphate dihydrate• sodium dihydrogen phosphate• water for injections• aluminium hydroxide

Post-Vaccination

Post marketing surveillance:

ThrombocytopeniaEncephalitisEncephalopathyConvulsionsParalysisNeuritis (including Guillain-Barré syndrome, optic neuritis and multiple sclerosis)NeuropathyHypoaesthesiaErythema multiformeAngioneurotic oedemaLichen planusArthritisMuscular weaknessMeningitisVasculitisHypotensionAnaphylaxisAllergic reactions (including Anaphylactoid reactions and mimicking serum sickness)Apnoea in very premature infants (≤ 28 weeks of gestation)

“There is good evidence that pregnant women have a higher chance of developing complications if they get flu, particularly in the later stages of pregnancy. One of the most common complications of

flu is bronchitis, a chest infection that can become serious and develop into pneumonia.”

Septic Shock Syndrome Otitis Media (inner ear infection)

For the very minimum ailments one may get (bronchitis and/or pneumonia), the risks of obtaining something far worse (and possibly irreversible) are much more concerning (in the column to the right which shows the ‘after-sales complaints post-vaccination side-effects’ that didn’t show up in clinical trials before it was brought to market). A chesty cough, or inconvenient lung infection is a far cry from what can manifest within the body – maybe years later and unknown until then. But, here’s

the warning - You cannot take this product back to the store you bought it from. You cannot take this broken toy back to the toy shop

if things go wrong, or if worse ‘complications’ arise from it.

The ‘US’ and ‘UK’ (corporations) seem to follow the same

immunisation schedules for children and use the same vaccine manufacturers. Please do research the side effects of these vaccines

and compare them against the initial effects of contracting a virus

naturally, and the symptoms to be expected (see Rotarix vaccine)

http://www.nhs.uk/Conditions/vaccinations/Pages/childhood-vaccination-schedule.aspx

2 months

5-in-1 (DTaP/IPV/Hib) vaccine – this single jab contains vaccines to protect against five separate diseases:

diphtheria, tetanus, pertussis (whooping cough), polio and Haemophilus influenzae type b (Hib, a bacterial infection that can cause severe pneumonia or meningitis in young

children)

Pneumococcal (PCV) vaccine

Rotavirus vaccine

The NHS vaccination schedule

3 months

5-in-1 (DTaP/IPV/Hib) vaccine, second dose

Meningitis C

Rotavirus vaccine, second dose

4 months

5-in-1 (DTaP/IPV/Hib) vaccine, third dose

Pneumococcal (PCV) vaccine, second dose

In the manufacturing process, porcine-derived materials are used. Porcine circovirus type 1 (PCV-1) is present in ROTARIX. PCV-1 is not known to cause disease in humans.

2nd vaccine: ROTARIX (Rotavirus Vaccine, Live, Oral) - Oral Suspension Initial U.S. Approval: 2008 – GlaxoSmithKline [GSK]

**Lyophilize - to freeze-dry a substance under vacuum conditions.

The **lyophilized vaccine contains amino acids, dextran, Dulbecco’s Modified Eagle Medium (DMEM), sorbitol, and sucrose. DMEM contains the following ingredients: sodium chloride, potassium chloride, magnesium sulfate, ferric (III) nitrate, sodium phosphate, sodium pyruvate, D-glucose, concentrated vitamin solution, L-cystine, L-tyrosine, amino acids solution, L-glutamine, calcium chloride, sodium hydrogenocarbonate, and phenol red.

ROTARIX (Rotavirus Vaccine, Live, Oral), for oral administration, is a *live, attenuated rotavirus vaccine derived from the human 89-12 strain which belongs to G1P[8] type

3rd vaccine: DAPTACEL (Diphtheria and Tetanus Toxoids and Acellular Pertussis Vaccine Adsorbed) – Sanofi Pasteur

DAPTACEL vaccine is a sterile isotonic suspension of pertussis antigens and diphtheria and tetanus toxoids adsorbed on aluminum

phosphate, for intramuscular injection.

Each 0.5 mL dose contains 15 Lf diphtheria toxoid, 5 Lf tetanus toxoid and acellular pertussis antigens [10 mcg detoxified pertussis toxin (PT), 5 mcg filamentous hemagglutinin (FHA), 3 mcg pertactin (PRN), and 5 mcg

fimbriae types 2 and 3 (FIM)].

Other ingredients per 0.5 mL dose include 1.5 mg aluminum phosphate (0.33 mg of aluminum) as the adjuvant, ≤5 mcg residual formaldehyde, <50 ng residual glutaraldehyde and 3.3 mg (0.6% v/v) 2-phenoxyethanol (not

as a preservative).

aluminum adjuvant an aluminum-containing compound, such as aluminum hydroxide or alum, that by combining with soluble antigen forms a precipitate; slow release of the antigen from the precipitate on injection causes prolonged, strong antibody response.

adjuvant definition:1 a substance, especially a drug, added to a prescription to assist in the action of the main ingredient. 2 (in immunology) a substance added to an antigen that enhances or modifies the antibody response to the antigen. 3 an additional treatment or therapy.

http://www.nhs.uk/Conditions/vaccinations/Pages/childhood-vaccination-schedule.aspx

This *live, attenuated varicella vaccine is a lyophilized preparation containing sucrose, phosphate, glutamate, and processed gelatin as stabilizers

8th vaccine: VARIVAX - Varicella Virus Vaccine Live (ChickenPox)

The virus was initially obtained from a child with natural varicella

Each 0.5 mL dose contains the following: a minimum of 1350 PFU (plaque forming units) of Oka/Merck varicella virus when reconstituted and stored at room temperature for 30 minutes, approximately 25 mg of sucrose, 12.5 mg

hydrolyzed gelatin, 3.2 mg sodium chloride, 0.5 mg monosodium L-glutamate (MSG), 0.45 mg of sodium phosphate dibasic, 0.08 mg of

potassium phosphate monobasic, 0.08 mg of potassium chloride; residual components of MRC-5 cells including DNA and protein; and trace

quantities of sodium phosphate monobasic, EDTA, neomycin, and fetal bovine serum. The product contains no preservative.

*Live (attenuated):1. Having life; alive.2. Capable of replicating in a host's cells.3. Containing living microorganisms or viruses capable of replicating in a host's cells.(attenuated): Alive but weakened; an attenuated microorganism can no longer produce diseaseEtymology: L, attenuare, to make thin pertaining to the dilution of a solution or the reduction in virulence or toxicity of a microorganism or a drug by weakening it.

What the system appears to be concocting is the idea that EVERY BABY BORN will have ADHD of some form (that ‘Spectrum’ they speak of) which automatically

‘qualifies’ and ‘entitles’ these babies to require mental health care by default. It would appear no normal

babies will be born in the future. They will be impaired, genetically compromised, or have ‘defective’

genes that IVF and gene manipulation will ‘correct’. It would appear too that this ‘re-wiring’ could lead to

psychopathy if one studies the medial frontal/frontal cortex (see next slide). It’s possible ingredients such as

aluminum hydroxide in vaccines could be the instigator. If this is occurring in the womb, then the

women are already carrying toxins to some degree

‘McMurphy’ would call it Electro-Shock Therapy

(or EFT)

It’s ok.. Science knows how to test these things. But it won’t be the Aspartame, fluoride, statins, TV, electromagnetic

radiation (SMART phones, Wi-Fi), Vaccines, E-Cigs, Climate Engineering,

Fukushima, Videogames, and most pharmaceutical products and

commercial food outlets. It won’t even be the terrible indoctrination/educational

system to blame…

No. it’s your genes.. And they need to address those defects.

Oh yes.. Just happens to be 30,000 children too. Do refer to the Occult Symbolism in media and the movies, part I

& II on this site. This ALL connects to a larger agenda.

You, the viewer, are encouraged to find and read ALL scholarly literature and

studies concerning toxicology on mice and rats with these two ‘products’.

Whatever the media discloses herein are deceptive and

misleading and should not be trusted as factually

accurate. Some people are overweight. This is true. But, certain cases of obesity could

be termed;

‘an observable sign that someone is suffering from

Acute Aspartame Poisoning’ or AAP

Meningococcal conjugate vaccines, quadrivalent (MCV4)

IngredientsThe active ingredients are:

50 mcg of Neisseria meningitidis Group B Neisseria Heparin Binding Antigen fusion protein (rbe); 50 mcg, of Neisseria meningitidis Group B Neisseria Adhesin A protein (rbe); 50 mcg of Neisseria meningitidis Group B Factor H Binding

Protein fusion protein (rbe); 25 mcg of Outer Membrane Vesicles from Neisseria meningitidis group B strain NZ98/254.

Antigens are adsorbed on aluminium hydroxide (0.5 mg aluminium). The other ingredients are: sodium chloride, histidine, sucrose and water for injections.

Multicomponent Meningococcal group B vaccine (recombinant, adsorbed)

Chart from JCVI interim position statement on use of Bexsero Meningococcal B vaccine in the UK, July 2013, pdf shows a declination of overall numbers of cases reported from years 2003 to 2012, so why the “rush to market”?

It was predicted long ago when the revelation came whereby Cervirax and Gardisil may be causing the rise in infertility and sterility; that IVF would be the new ‘market’ for surrogacy at

the “fall of man”. The IVF surrogacy connects with the biogenesis of the designer baby system, whereby a

“consumer” can order a child (a patented creation) to genetic specifications for the best chances of longevity, status, best job, preferable traits and the genetic removal of ‘unwanted genes’.

You shall be ‘blessed’.

The “fall of man” is a term for population reduction, or in its extreme condition, extinction at some future, quickened time, due to self-ignorance (vaccines which lead to sterility and the inability to conceive a baby naturally) through the belief of the health care

system. Man is falling, but man hasn’t fallen fully, yet.

The movie, “The Handmaid’s Tale” (right) exemplifies this philosophy quite clearly of what a surrogacy system can transform into in its most

perverse societal form. It’s also interesting to note the symbolism used in that movie and the subject matter and contexts the movie deals with. Do look to the other presentations about occult symbolism in movies and music videos for more information

concerning symbolism.

This past week, a British Court ruled that a man who does not have the capacity to give informed consentinformed consent

should have a vasectomy because it is in his “best interests.”

http://communities.washingtontimes.com/neighborhood/leading-edge-legal-advice-everyday-matters/2013/aug/18/forced-sterilization-new-wrinkle-and-continuing-co/#ixzz2dFrdYRIM

With regard to the first, in the current century, government forced

procedures have mostly been eliminated worldwide. Nonetheless, atrocities

beyond imagination are still going on in places like China and India in the name

of population control. China’s One-Child policy, in place since 1979,

accounts for approximately 336 million forced sterilizations.

Statins will create two things, primarily. Expect to see rises in – Type-2 Diabetes, and rises in Heart attacks. 2014 seems

to be the ‘year of the heart fail’. Take them as prescribed at your own risk. At least research the product and clinical

studies.

Viruses and CCR5-Delta 32 deletionViruses and CCR5-Delta 32 deletion

What Caused eye colour and skin colour to express a change?

Blue Eyes – What age is their origin?

A Naturally-acquired Immunity?

What does it reference?

1CCR5 Delta-32: a very beneficial mutation (PDF)

Cysteine-cysteine chemokine receptor 5 (CCR5) is found in the cell membranes of many types of mammalian cells, including nerve cells and white blood cells. The role of CCR5 is to allow entry of chemokines into the cell - chemokines are involved in signaling the body’s inflammation response to injuries.

The gene that codes for CCR5 is situated on human chromosome 3. Various mutations of the CCR5 gene are known that result in damage to the expressed receptor. One of the mutant forms of the gene is CCR5-delta32, which results from deletion of a particular sequence of 32 base-pairs. This mutant form of the gene results in a receptor so damaged that it no longer functions. But surprisingly, this does not appear to be harmful

Moreover, this mutation can be advantageous to those individuals who carry it. The virus HIV normally enters a cell via its CCR5 receptors, especially in the initial stage of a person becoming infected. But in people with receptors crippled by the CCR5-delta32 mutation, entry of HIV by this means is blocked, providing immunity to AIDS for homozygous carriers and greatly slowing progress of the disease in heterozygous carriers

Up to 20% of ethnic western Europeans carry this mutation, which is rare or absent in other ethnic groups. This suggests that the CCR5-delta32 mutation was strongly selected for sometime during European history . Some researchers have proposed that the plague epidemics that repeatedly swept Europe during the Middle Ages were responsible. However, recent experiments in mice suggest that Yersinia pestis, the cause of plague, can infect mammalian cells by other means and so some scientists have proposed that smallpox, which is caused by the variola virus, was the selection agent that historically caused CCR5-delta32 carriers to proliferate in Europe

To infect immune cells, HIV must first bind to chemokine receptors. Researchers discovered in 1996 that people who had a naturally occurring mutation in their genes for one of these CCR5, were strongly protected from developing AIDS — or even becoming infected in the first place — and suffered no ill effects from lacking the receptor.

It may be that infectious agents like HIV only became pathogenic after degeneration from their original ‘very good’ created state. Or it may be that humans did not live in the same environment as such pathogens and so were just not exposed to them. Perhaps both these scenarios apply.

CCR5-Delta 197-215 (84.8%) is the most frequent haplotype and is a single mutation step. It is also linked to 4 other chemokine receptors C1-5 and is the ancestral haplotype that other CCR5-Delta 32 haplotypes were derived

from it by 4-7 mutational or re-combinational events

The last change occurred 700 years ago in the caucasion population and had rapidly increased in its frequency. By strong, selective

pressure, possible an “ancient plague”, the nature of which is currently undetermined. Ancestry ranges from 127,000 years ago

1The CCR5 is a requisite co-receptor for both HIV-1 infection and progression to AIDS (Auto Immune Deficiency Syndrome)

1Dating the origin of the CCR5-Delta 32 AIDS-resistant allele by the coalescence of Haplotypes

The estimates derived here track the persistence of the 3-locus haplotype at 700 years; however, it is possible that the CCR5-Delta 32 mutation is

somewhat older. In spite of that uncertainty, the cumulative results point to a selective sweep and to one with enormous selective mortality within historic times, perhaps mediated by a widespread epidemic. The Bubonic Plague which claimed the lives of 25%-33% of Europeans during the Black Death from 1346-1352 (650 years ago) are which has multiple outbreaks in

Europe before and since, is an obvious candidate

1Since the beginning of the Acquired ImmunoDeficiency Syndrome (AIDS) epidemic, it was determined that sexual activity with multiple partners was the main risk factor to acquire human immunodeficiency virus (HIV)

infection with the subsequent development of Aids. However, there is strong evidence suggesting a natural resistance to infection in several individuals who have remained uninfected despite the fact that they

have had several expositions to HIV, particularly through sexual intercourse (Paxton et al. 1996). The biological basis for this resistance is just starting to be understood. To enter the target cell the HIV

requires the presence of the CD4 molecule that acts as receptor and of a second molecule or co receptor, that in the majority of cases is a chemokine receptor (Deng et al. 1996, Feng et al. 1996). Among the chemokine receptors, CCR5 and CXCR4 are the main co receptors for HIV entrance.

The Delta-32 allele is present mainly in Caucasian population (Liu et al. 1996, Martinson et al. 1997, Magierowska et al. 1998). In United States the frequency is 8% to 10% in white

population but less than 1% in Afro-American individuals. There is also a very low frequency of the mutation among Caucasians in Asia (Pakistan and India) and it has not been reported in

China, Japan or pure African population (Martinson et al. 1997).

In Latin America the frequencies have been barely studied. It was not detected among 32 individuals from Venezuela nor in Amerindian groups (Liu et al. 1996, Martinson et al. 1997).

From the literature one can conclude that ccr5-delta 32 genotype confers a high degree of protection to HIV infection, but it is not the main factor associated to resistance, since the majority

of exposed sero-negatives lack this genetic factor. Different mechanisms such as other co receptor mutations or immunological factors could account for the lack of infections in this high-risk group.

1Frequency of CCR5 D32 Mutation in Human Immunodeficiency Virus (HIV)-seropositive and HIV exposed seronegative Individuals and in General Population of Medellin, Colombia, Mem Inst Oswaldo Cruz, Rio de Janeiro, Vol. 95(2): 237-242, Mar./Apr. 2000

Repeated exposure to human immunodeficiency virus (HIV) does not always result in *seroconversion. Modifications in co receptors for HIV entrance to target cells are one of the factors that block the

infection.

1Partial protective effect of CCR5-Delta 32 heterozygosity in a cohort of heterosexual Italian HIV-1 exposed uninfected individuals

AIDS Research and Therapy1In conclusion, our data show a significantly higher frequency of CCR5-Delta 32 heterozygous

genotype among the Italian heterosexual ESN (Exposed SeroNegative) Individuals compared to HIV-1 seropositive patients, suggesting a partial protective role of CCR5-Delta 32 heterozygosity in this

cohort.Interestingly, a partial protection against HIV-1 infection has been observed in 2three different studies. In

particular, 3aHoffman et al described a significantly higher frequency of CCR5-Delta 32 heterozygous genotype among the uninfected partners of heterosexual discordant couples, but not in homosexual couples. Similar data were reported also by 3bPhilpott et al in a large cohort of women from different ethnic and racial background and with different transmission risk factors. In this study [15] the presence of the

Delta 32 allele was significantly associated with lower rates of HIV-1 infection among white individuals . 3cMarmor et al, analyzing a large sample of individuals, found a protective role of CCR5-Delta 32 allele in

uninfected subjects exposed to HIV-1 infection risk through homosexual intercourse.

Further data supporting the hypothesis of a partial protective effect of CCR5-Delta 32 heterozygous genotype have been recently described

To the best of our knowledge, the present is the first study investigating the allelic distribution of the genetic variants CCR5-Delta 32 and CD45-C77G in a cohort of Italian heterosexually HIV-1 exposed and uninfected individuals. Our data suggest a partial protective effect of CCR5-Delta 32 heterozygosis in

the Italian ESN cohort population.

3bCC chemokine receptor 5 genotype and susceptibility to transmission of human immunodeficiency virus type 1 in women. J

2,3aCCR5 genotypes in sexually active couples discordant for human immunodeficiency virus type 1 infection status.

3cHomozygous and heterozygous CCR5-Delta32 genotypes are associated with resistance to HIV infection.

You are unique because you have You are unique because you have your own perspective..your own perspective..

But what is the significance of blue eyes?But what is the significance of blue eyes?

Source: http://www.springerlink.com/content/2045q6234h66p744/?p=fd01cf5370de4697b60edcd139a48954&pi=2

http://www.thevarsity.ca/blog/96/entry/1880

Researchers from the Department of Cellular and Molecular Medicine at the University of Copenhagen have tracked blue eye colour to a common ancestor. The team examined mitochondrial

DNA from 800 blue-eyed individuals from countries which included Jordan, Denmark and Turkey. They found that blue

eyes are caused by a genetic mutation in the OCA2 gene, in the chromosome that is normally responsible for melanin

production.

“A genetic mutation affecting the OCA2 gene in our chromosomes resulted in the creation of a ‘switch,’ which

literally ‘turned off’ the ability to produce brown eyes," said Hans Eiberg, the study’s senior author. The ‘switch’ lowered melanin production in the iris, causing brown eyes to become lighter.

Eiberg and his team found genetic evidence that the mutation is relatively new and might have occurred around 6,000 to 10,000

years ago in a single ancestor.

“Originally, we all had brown eyes,” noted Eiberg.

The study can be found in the Journal of Human Genetics.

His remains were discovered 5,000ft up in the mountains of north-west Spain in 2006.Experts were astonished to find the ancient

hunter-gatherer, given the name La Brana 1, had a combination of African and

European genes.

Results from an analysis of DNA taken from a tooth show he had dark – possibly black –

hair and skin with deep blue eyesdeep blue eyes, the online edition of the journal Nature reports

The mixture of African and European traits implies that the racial transformation of modern humans was still in

progress long after they left Africa, with changes in eye colour coming before alterations in skin tone.

Study leader Professor Carles Lalueza-Fox, of the Institute of Evolutionary Biology in Barcelona, said: ‘The biggest surprise was to discover that this individual possessed African versions in the genes that determine the light

pigmentation of the current Europeans.

'Even more surprising was to find that he possessed the genetic variations that produce blue eyes.’

La Brana 1 shows genetic similarities to Scandinavians and also shared a common ancestor with people who lived

in Siberia more than 20,000 years ago.

La Brana 1's genome hints at some of the changes that occurred in humans as a result of switching from a hunter-

gatherer existence to farming.

Farming is thought to have driven changes in the human immune system as a result of exposure to bacteria and

viruses from animals.

But a number of DNA variants conferring resistance to infection in modern Europeans were already present in

the hunter-gatherer.

This suggests they did not arise as an adaptation to farming, but had a more ancient origin.

Writing in Nature, the scientists concluded: 'Our results indicate that the adaptive spread of light skin pigmentation

alleles (genetic variants) was not complete in some European populations by the Mesolithic, and that the spread of alleles

associated with light/blue eye colour may have preceded changes in skin pigmentation.'

Article (2005) archived and online (screen cap taken March 23, 2014) The media report this ‘discovery’ like it’s ‘new’ news 8 years later in 2014

Blue Eyes and Post-cataclysmic events – plausible genetic adaption of the homo sapien sapien species

"It's a major finding in a very sensitive area," said Stephen Oppenheimer, an expert in anthropological genetics at Oxford

University, who was not involved in the work. "Almost all the differences used to differentiate populations from around the

world really are skin deep.“

The work raises a raft of new questions – not least of which is why white skin caught on so thoroughly in northern climes once it

arose. Some scientists suggest that lighter skin offered a strong survival advantage for people who migrated out of

Africa by boosting their levels of bone-strengthening vitamin D; others have posited that its novelty and showiness simply made

it more attractive to those seeking mates.

Even study leader Keith Cheng said he was at first uncomfortable talking about the new work, fearing that the finding of such a

clear genetic difference between people of African and European ancestries might reawaken discredited assertions of

other purported inborn differences between races -- the most long-standing and inflammatory of those being “intelligence”.

Original Article (screen captured) by Rick Weiss, Washington Post Staff Writer Friday, December 16, 2005

A few genes have previously been associated with human pigment disorders -- most notably those that, when mutated, lead to albinism, an extreme form of pigment loss. But the newly found glitch is the first found to play a role in the formation of "normal"

white skin. The Penn State team calculates that the gene, known as slc24a5, is responsible for about one-third of the pigment loss that made black skin white. A

few other as-yet-unidentified mutated genes apparently account for the rest.

"I think human beings are extremely insecure and look to visual cues of sameness to feel better, and people will do bad things to people who look different," Cheng said.

Although precise dating is impossible, several scientists speculated on the basis of its spread and variation that the mutation arose between 20,000 and 50,000 years

ago. That would be *consistent with research showing that a wave of ancestral humans migrated northward and eastward out of Africa about 50,000 years ago.

Unlike most mutations, this one quickly overwhelmed its ancestral version, at least in Europe, suggesting it had a real benefit. Many scientists suspect that benefit has

to do with vitamin D, made in the body with the help of sunlight and critical to proper bone development.

*this research is ongoing and further discoveries continually offer contradictory the O.O.A (Out Of Africa) theory/evidence, including OOPARTS (Out Of Place Artifacts, ect

Middle Eastern Respiratory Syndrome Coronavirus – MERS-CoV

1 She added that… it would be the first case registered in the State of Kuwait after Saudi Arabia, Qatar, Bahrain, Oman and the United Arab Emirates, knowing that the patient traveled to Saudi Arabia in the recent period. (Updated: 12/11/2013 09:40:20 PM)

1 http://www.araa.com/article/77104#sthash.S6llSyDg.dpuf)

On 7 Nov 2013 the Saudi Ministry of Health reported a new case: a 43-year-old male from Jeddah, who developed symptoms on 27 Oct 2013. He sought medical treatment on 3 Nov 2013. He is currently in an intensive care unit. The patient does not have any underlying chronic disease. He has no recent travel history outside of Jeddah. He had significant contact with animals but

no contact with a known positive human case.

2nd confirmed MERS case - 52 years old

1http://www.araa.com/article/77104#sthash.S6llSyDg.dpuf)

England1_CoV - Whole genome sequence – added 13th November 2012

England1_CoV was highly similar to the coronavirus sequenced in the Netherlands – EMC/2012, JX869059

1England1_CoV (red) and EMC/2012 (blue) are closely related and that these viruses form a distinct clade that is related to HKU4 and HKU5 bat corona viruses.

Phylogenetic Analysis indicated that England 2_CoV clustered with England1_CoV and the virus identified in the Netherlands (EMC/2012, JX869059). The level of similarity within this

cluster of novel coronaviruses was greater than 99.6%.

1http://www.araa.com/article/77104#sthash.S6llSyDg.dpuf)

"(This) opens the way to being able to study all sorts of proteins from the past, and to study many physiological characteristics," Cooper said. "It's really paleobiologypaleobiology; you're studying how extinct

species function, and how they adapted to climate change and other past environmental

conditions that we can't get at in the fossil record."

This article (and parallel to tri-party human engineering being engendered in the state of England on the next slide) popped up in 2011 and it links to the idea of resurrection. The question is – the resurrection of what, and does that come with a hidden

package? What many may not be considering here is not just how the mighty mammoth lived and existed, or how the climate was at its demise – but, what pathogenic virus did (or ‘may do’ if resurrected) this ‘Giant/Beast’ carry? What is responsible for plague outbreaks, and was this an alternative explanation why the hunter-gatherer theory takes a rather different turn under that

conditions or understanding if they were being hunted due to being pathogen carriers and harbingers of disease, should they migrate from their ancestral lands due to an as-yet specific, identifiable cause. Could we be resurrecting an ancient plague by

resurrecting this Beast? Could plague victims found in (now opened) plague pits (and soil) in London be a cause for concern for airborne re-release of ancient pathogens, and are they causally-related through times past? We’ll explore the plague later in this thesis. Could today’s climate and/or environment assist in its voracity to transform and mutate today?

“The Kyoto University researchers are planning an expedition to the Siberian permafrost this summer in search

of a flash-frozen specimen still rich in DNA.”

In recent years, scientists have used samples of hair frozen in the Siberian ice for thousands of years to piece together the mammoth’s genetic code. And DNA preserved in bone

has been used to recreate the prehistoric giant’s blood.

Even if the scientists are successful, the problems don’t end with the birth. Project leader Professor Akira Iritani said: ‘If acloned embryo can be created, we need to discuss, before

transplanting it into the womb, how to breed (the mammoth) and

whether to display it to the public.’

Some experts hold that mammoths were hunted to extinction by the species that was to become the planet’s

dominant predator – humans.

Others argue that climate change was more to blame, leaving a species adapted for frozen climes ill-equipped to

cope with a warming world.

We’re treading a very dangerous path by genetically altering, re-arranging, genetically manipulating, re-assorting and re-wiring the very source code of life. And man. This led to massive catastrophe before, in times past and forgotten, and it may yet lead again to a future pandemonium if this ‘technology’ exits the genie’s bottle, too. Coupled to Artificial Intelligence, this could

evolve into a terrible monster that could be, virtually (pardon the pun), the demise of mankind as a species.

Mammoths and Bison are seemingly connected to pathogenic virus and the time frames or epochs are close

enough to make this contextual musing.

Both are found in regions in Russia (where there is much strive now with Crimea being annexed into the Russian

Federation); both are around or between the eras 10,000 BC – 7,000 BC – a period that is shrouded in darkness and seems to show very little in the historical accounts, but a major cataclysmic event (or more than one event) seems

to be connected to these dark times.

A few civilisations re-bounded in the 4th millennium (post-cataclysm), but the only observable findings at such sites have been pottery, nicer pottery, and then, even better pottery until, suddenly, around the 3100 BC time frame (specifically for Egypt and the historical unification of the North and south kingdoms under Narmer/Menes/Scorpion King after the

Naqqada I, II, & III periods), orthodox history would state that ‘empire-level buildings’ and civil constructions were occurring at this time, and the new birth of civilisations such as Sumer,

but with no conclusive historical accounting of how these ancient cultures acquired, developed and arrived at the level of sophisticated management of people, minerals, land, and

regions so effectively.

Law, education, administrative language, state religion, courts and legal matters, mathematics, astronomy, writing – all simply just arose without any previous historical lineage or accounts to extrapolate from, nor from ages prior to the 6 th and the 7th

millennium, except from locations such as Ugarit and Turkey (ancient anatolia) in the Levant.

Egypt and some meso-american cultures were demonstrating their building capabilities way ahead of orthodox

understanding of human accomplishments by those times.

The pioneering scientist who created Dolly the sheep has outlined how cells plucked from frozen woolly mammoth carcasses might one day help resurrect the ancient beasts. The notional procedure – bringing with it echoes of the Jurassic Park films – was spelled out by Sir Ian Wilmut, the Edinburgh-based stem-cell scientist, whose team unveiled Dolly as the

world's first cloned mammal in 1996.

"I've always been very skeptical about the whole idea, but it dawned on me that if you could clear the first hurdle of getting viable cells from mammoths, you

might be able to do something useful and interesting," Wilmut told the Guardian.

Woolly mammoths roamed the Earth tens of thousands of years ago in a period called the late Pleistocene. Their

numbers began to fall in North America and on mainland Eurasia about 10,000 years ago. Some lived on for a further 6,000 years. Their demise was likely

the result of hunting and environmental change.

"I think it should be done as long as we can provide great care for the animal. If there are reasonable prospects of them being healthy, we should do it. We can learn a lot

about them," he added.

If good-quality cells can be extracted from mammothremains – and that is a big if – they could be

reprogrammed into stem cells using modern procedures. These could then be turned into other kinds of cell,

including sperm and eggs. Mice have already been born from sperm and eggs made from stem cells

The mammoth carcass and blood were found on Maliy Lyakhovsky Island in the Novosibirsk archipelago in the

East Siberian Sea

"It has been remarkable to bring a complex protein from an extinct species back to life and discover important changes not found in any living species," said Alan Cooper of the University of Adelaide in

Australia, in a statement.

These 'legacy' genes have been linked to an increased risk from cancer and diabetes by new studies looking at our evolutionary

history.

However, it is not all bad news, as other genes we inherited from our species' early life could have improved our immunity to

diseases which were common at the time, helping us to survive.

Speaking to MailOnline, professor Chris Stringer, research leader in human origins at the Natural History Museum in London, said: “Neanderthals had been evolving outside of Africa of thousands of years and had been exposed to diseases which our species

had not come into contact with”.

1995 2007 2007

2008 2008 2009 2011

2002

So far, a lot of data and information has been thrown at you, the viewer. But it is hoped you will see multi-level connections within this thesis which may lead to seeking harder questions from making some uncomfortable conclusions by reason and logic, but not those two alone. Through inference to the numerous public articles

from the main-stream media and news outlets, and scholarly articles, here are some conclusions:

H1N1 pandemic flu is actually the source pathogen Spanish Flu that killed millions in 1913, unearthed via a frozen specimen in Siberia. No vaccine existed for it.

Vaccinations, of all kinds, are the primary cause of spread of virus and shedding virus amongst populations and are known to contain toxins such as thiomersal, formaldehyde and aluminum, causing the very infections the populace wishes to evade. Natural acquisition of virus outbreaks are known and

inherently-acquired genetic immunicity and protection (such as CCR5 delta-32 and FUT2 gene) have been derived from exposure to an ancient plague of which the origins are not known, and no vaccines

were invented to protect against.

Woolly mammoths that catastrophically “freeze” instantaneously due to ‘global warming’ and ‘environmental change’ needs to be explained. It is clear that other events must have occurred which set off an adaption within

most if not all species on the planet to make ‘adaptions’ to the new environment, albeit, inherited through violent or cataclysmic events. Within the last 30,000 years. The last manifest changes, such as blue eyes, and pigmentation of skin must have included the darkening of the visible Sun, or at the very least, obscuration by thick, dense clouds, possibly for a lengthy period of time, and pathogenic outbreaks may have also occurred due to cometary bodies interacting or even contacting with Earth directly. All life relies on the star in the sky for heat, light, and life. No sun; no life. Blue eyes seems to infer a “epoch marker” to this perception change

within man within these time frames.

Are these times when cannibalism emerged? Lack of sunlight would remove much life from the planet. No plants or foods would grow to sustain populations. Genetics seems to be releasing some answers…

There would seem to be a deliberate agenda to engineer some form of a pandemic viral outbreak , whether it be HPL (Highly Pathogenically Lethal) created pseudo-virus, or one synthesized in a laboratory from other materia, or whether it is resurrected via gene cloning technology in the form of a mammoth with its inherent pathogenic

viruses; man seems inherently purposeful in opening a Pandora's box they really shouldn’t open.

A strain of plague which has killed up to 100million people could strike again , scientists warn. They have linked the Black Death, which killed 50million

Europeans in the 1300s, and The Plague of Justinian, which struck 800 years earlier, suggesting they were caused by ‘distinct’ strains of the same pathogen .

Dave Wagner, professor in the Center for Microbial Genetics and Genomics at Northern Arizona University warned that it could return in the future.

He said 'We know the bacterium Yersinia pestis has jumped from rodents into humans throughout history and rodent reservoirs of plague still exist today in

many parts of the world. 'If the Justinian plague could erupt in the human population, cause a massive pandemic, and then die out, it suggests it could happen again.‘

Tiny samples of the plague bacteria were taken from skeletons belonging to two victims of the Justinian plague who were buried in Bavaria, Germany. Fragments of DNA were

found in their 1,500-year-old teeth and used to recreate the bacteria’s whole genetic code. Researchers compared it with a database of hundreds of modern plague

pathogens, some of which still kill thousands every year.

1However, by 1993, revelations about the dating of layers of volcanic acid in theGreenland ice in the vicinity of the AD 540 event - or rather the revelation that there were no acid layers dating to the years around AD 540 - meant that the volcano hypothesis was starting to look thin . This combination of factors allowed a new paradigm to be contemplated - was it possible that the serious global environmental event around AD 540 was not due to a volcano or volcanoes, but rather was due to the next most likely cause of a global environmental event i.e. some loading of the atmosphere from space?

1M G L Baillie, School of Archaeology and Palaeoecology, Queen’s University, Belfast, PDF, p.2

Three British astrophysicists had published a prior hypothesis, back in 1990 (Bailey, Clube and Napier 1990), in which they had proposed that the period between AD 400-600 had been a period of risk of bombardment by comet debris

However, for the purposes of this paper I am going to restrict the discussion mostly to the happenings around AD 540; an event that falls in a Dark Age and close to the date of the plague of Justinian (AD 542).

Hypothesis 1: Cometary instigators of pathogenic viruses on Earth and Ancient ‘Sun-Comet’ Gods and

returning/resurrecting Archetypes

1Yet, according to current knowledge, Tunguska-like impacts occur every 100 years or so - with devastating effects. It is, therefore, not far-fetched to hypothesise that a super-Tunguska (i.e. massive high level multi-megaton explosions and widespread atmospheric and/or oceanic impacts of fireballs over different locations of the globe) is capable of triggering ecological crises on a continental or even global scale and may occur every 2000, 3000 or 5000 years.

1Email Cambridge conference

2"No crater, no catastrophe". This is what could be called the motto of this school of thought. If we were to accept this restriction, Tunguska-like events or even super-Tunguskas would be automatically taken out of the equation. Due to their catastrophic detonation above ground (or in the oceans), they often leave no obvious fingerprints behind.

Russian comet February 15th, 2013A 600kg fragment of the Chelyabinsk Meteorite at the Chelyabinsk Regional HistoryMuseum. The chunk was recovered from the bottom of Chebarkul Lake, some 60km west of Chelyabinsk. The meteorite exploded over Chelyabinsk Region in February

2013

1Satellite data from NASA has revealed that that four days after the bolide explosion, the faster, higher portion of the plume

(red) had snaked its way entirely around the northern hemisphere and back to Chelyabinsk in Russia

1Mailonline, published: 11:16, 23 October 2013

1Upon commission of the pope in 1348, a group of learned men (and at that time only men were deemed capable of

being educated and hence, learned) of the medical faculty at Paris concluded that the disaster caused by Plague was a result of a conjunction of Saturn, Jupiter, and Mars in the 40th degree of Aquarius at 1:00 p.m. on March 20, 1345. This caused hot, moist conditions, which forced the

earth to exhale a virulent sulfurous miasma.

Curious Constellational Observations

1History of Epidemics and Plagues, p10

1“whether the dog days, as our astrologers pretended to express themselves, the influence of the Dog Star, had that malignant effect between the hours of 1 and 3 in the morning.. I know not,

but this was the time when it was reported that above 3,000 people died in one night”

1 Journal of the Plague 1665, Daniel Defoe, p172

2 “In the first place, a blazing star or comet appeared for several months before the plague, as there did the year after, a little before the Great Fire of London 1666. The old women,

and the hypochondriac older males remarked, especially afterward, though not till both these judgments were over, that those two comets passed directly over the city, and passed by close, so very near the houses, that it was plain they imported something peculiar to the

city alone”

“That the comet before the pestilence was of a faint, languid colour, and its motion very heavy, solemn and slow but that the comet before the fire was bright and sparkling, or, as others said, flaming and its motion swift and furious, and that, accordingly, one foretold a

heavy judgment slow but severe, terrible and frightful, as was the plague.

But the other foretold a stroke, sudden, swift and fiery, as was the conflagration; nay, so particular were some people that they looked upon that comet preceding the fire of 1666, they fancied that they not only saw it pass swiftly and fiercely, and could perceive the motion with their eye, but they even heard it, that it made a rushing, mighty noise, fierce and terrible,

though at a distance, but just perceivable.”

2 Journal of the Plague 1665, Daniel Defoe, p23, 24

“I saw both these stars.. I was apt to look upon them as the forerunners and warnings of God’s judgments and especially after the plague had followed the first, I saw yet another I saw yet another

of the like kindof the like kind, I could not but say God had not yet sufficiently *scourged the city”

* Compared with the ancient biblical tale of Nineveh’s destruction as an ‘evil city’’ circa 7 th century BCE

Daniel Defoe, the author of the plague journal is a seemingly pious man, deciding to stay in London during the pandemic period of the outbreak when offered the opportunity to leave the city with a familial brother. It would be curious to

discover if Daniel had some immunity to this strain of the plague, and if he had any genetic of DNA corroboration which may support such a thesis as a possible related CCR5 Delta-32 deletion; perhaps facilitating this supernatural shield of protection to withstand the plague; or indeed, maybe it was truly ‘God’s hand” that was placed upon Daniels head

which allowed him, through piety and faith, to vestigiously document the ensuing pandemonium that occurred before his eyes, and for future posterity, doing ‘God’s will’. Here are a few lines from the bible which he may have overlooked.

1 Samuel 6: verses 4-6

[6:4] “Five (5) golden tumours and five (5) golden rats, according to the number of the Lords of the Philistines. For the same plague was on all of you, and on your lords”.

[6:5] “Therefore you shall make images of your tumours and images of your rats that ravage the land, and you shall give glory to the god of Israel: perhaps He will lighten his hand from

you, from your gods (canaanite Goddesses Ashtoreths and baals – 1 Samuel 7:4) and your land”

2 Samuel 24 verses 15-25

[24:15] “ So the LORD sent a plague upon Israel from the morning to the appointed time. From Dan to Beersheeba seventy thousand (70,000) men of the people died”

[24:16] “And when the Angel stretched out his hand over Jerusalem to destroy it, the LORD relented from the destruction and said to the Angel who was destroying the people, “ it is enough; now restrain your hand”.

2 Samuel 24: verses 1-9 (today’s equivalent of a Public Census and counting of the peoples), the number of inhabitants are counted before the accounting of souls lost to plague cited in 24:15 (below), which is 70,000

[24:1] “Again, the anger of the LORD was roused against Israel and he moved David against them to say, “Go, number Israel and Judah” … [24:2] go throughout all the tribes of Israel, from Dan to Beersheba, and count the people, that I may know the number of the people”

[24:9] “And there were in Israel eight hundred thousand (800,000) valiant men who drew the sword, and the men of Judah were five hundred thousand (500,000) men” (1.3 million total taken in 9 months and 20 days to count them all)

As a note, the next chapter is 1 Kings and Og of Bashan, the Giant King of the Philistines is named in 1 Kings 4:19. The time frame is circa 1000 BC

A medieval painting dated 1456 AD, depicting a pass of Halley's Comet

1“The manner of its first coming to London proves this also, viz, by goods brought over from Holland, and brought thither from the Levant; the first breaking out of it in a house in Long Acre,

where those goods were carried and first opened; its spreading from that house to other houses by the visible unwary conversing with those that were sick, and the infecting of the

parish offices who were employed about persons dead, and the like. These facts are known authorities for the great foundation point; that it went on and preceded from person-to-person,

and from house to house, and no otherwise’’

1,2,3,4Journal of the Plague 1665, Daniel Defoe, p191, p198, p201

2“by the well, I mean such as had received the contagion and had it really upon them and in their blood, yet did not show the consequences of it in their countenances (appearance/upon their faces); nay, even were not sensible of it themselves, as many were not for several (7) days”

3“There were the dangerous people, these were the people of whom the well people ought to have been afraid; but then, on the other side, it was impossible to know them’’

4 “but nobody appeared to be ill in the family for 16 to 18 days after, they were not so strict but they would connive at their going privately *abroad; nor would people be much afraid of them

afterwards, but rather think they were fortified [for] the better, having not been vulnerable when the enemy was in the house; but we sometimes found it had lain much longer concealed (longer

than 18 days)”

5 “the first person that died of the plague was December 20th, or thereabouts, 1664, and in, or about Long Acre whence the first person who had the infection was generally said to have

taken it from a parcel of silk imported from Holland, and first opened in that home’’

What type of Pathogen can survive a cold climate, and a long journey?

Can rat fleas really sustain themselves within Silk in a boxed parcel?

Can ancient pathogens and viruses ‘resurface’ or be ‘resurrected’?

QUESTIONS:

The Mammoth pathogen gene (if discovered) may have a property within it that gives it an ‘anti-freeze’ quality to it, aiding the mammoth to cope with adaption to

living within a colder climate than now, and possibly allowing further understanding for how the mammoth acclimatised to these adaptions over time and epochs; perhaps through warm and cold periods and differing locations via

migration. Samples of mammoth genetic material extends to 35,000 BC

Frenchman dies in Longacre, Drury Lane

Chronology, speed and spread of infection: Timelines and numbers of cases

November – December 1664: Frenchman dies in Longacre, Drury Lane; confirmed by two physicians and a surgeon. Suggested that the victim may have had some contact with Rotterdam and

Holland in 1663

February 1665 – St.Giles’s parish began to record higher death rates but were badly recorded to defer consciousness from the rising problem of the Plague to the general populace who were generally

the poor, the poor trade guilds and migrants seeking employ from the land of Eire (Ireland). These house rooms or ‘cellars’ were the dwelling places and accommodation of such peoples at the time.

December 20th-27th Buried 291 Increase +/-

December 27th -3rd Jan Buried 349 + 58

Jan 3rd - 10th Buried 394 + 45

Jan 10th – 17th Buried 415 +21

Jan 17th – 24th Buried 474 + 58

“the last bill was really frightful, being a higher number than had been known to have been buried in one week, since the preceding visitation of 1656” – Journal of the Plague, page 7

1“But after this we heard no more of any person dying of this plague, or of the distemper being in that place till the 9th February, 1665, which was about 7 weeks after .., and we were perfectly

easy as to the public for a great while for there were no more entered in the weekly bill to be dead of the plague till the 22nd April, then there were two more buried, not out of the same

house, but out of the same street (Drury lane); and as near as I can remember, it was out of the next house to the first (next door neighbour’s house) : this was 9 weeks asunder and after this we had no more till a fortnight (11 weeks total) and then it broke out in several streets

and spread everyway!”

1,2Journal of the Plague 1665, Daniel Defoe, p205, 206

2“It is true, there was, as I observed at first, and is well known to many yet living, a very cold winter, and a long frost, which continued 3 months, and this, the doctors say, might check

the infection …whereas the principal period when it was needed of this infection, which was from February to April was after the thaw was broken and the weather mild and warm

Defoe makes the claim that either monetary bribes to church and clergy officials were obliging the recording of false figures and finding other reasons for deaths by distempers other than from plague so as not to cause alarm to the populous. Typhus gave the same symptoms of plague and deaths by ‘spotted fever’ and fevers in general were usually over 400 cases in anyone week between

late July and later September. It would seem the inhabitants were also hoping the colder weather would extend a little longer; that it may halt or dispel the plague, but the record show it timed its assault perfectly when the warmer weather came. A lot of the wealthy who could afford to move

and relocate, even though way outside the city, did so very early on at the onset of the first death.

DECEMBER 20TH, 1664

FEBRUARY 9TH

APRIL 22ND

HOLLAND

DRURY, LANE LONGACRE

MAY 5TH

7 weeks

2 Dead neighbours

9 weeks11 weeks

”it was out of the next house to the first: this was 9 weeks asunder and after this we had no more till a fortnight (11 weeks total) and then it broke out in several streets and spread

everyway!”

“But after this we heard no more of any person dying of this plague, or of the distemper being in that place till the 9th February, 1665, which was

about 7 weeks after .., and we were perfectly easy as to the public for a great while..”

The weather at that time from December until late February 1665 was reported to be cold and severe frosts. Late February to early April, deaths decreased to some degree and the

population fell into an early false sense of abatement that the worst was over.

18th April – 25th April: Outbreak spreads to 3 surrounding parishes, mainly St. Andrews, Holborn, and St. Clement’s Danes

9th May – 16th May: 9 cases of plague; 6 cases of typhus and the frenchman from Longacre relocated to Bearbinder Lane near Stocks’ Market. Combined 50 cases of plague and typhus, or

‘spotted fever’.

23rd May – 30th May: 53 burials, 17 by plague.

“But those were trifling things to what followed immediately after, for now the weather was hot, and from the first week in June, this infection spread in a dreadful manner, and the bills [of

mortality] rose”

– Journal of the Plague, p9

Frenchman dies in Longacre, Drury Lane

Overview of Christopher and John Greenwood's map of London (1827)

Bearbinder Lane

Source full URL: http://www.oldbaileyonline.org/maps.jsp?map=green&mq_url=3&map_section_x=4&map_section_y=5&overview=yes

Source: http://www.oldbaileyonline.org/

Frenchman relocated

2nd week of July, 4 had died within the city: 1 in wood street, 1 in Fenchurch Street, and 2 in Crooked Lane

As the minor cases emerged, and, as if mirroring the same actions of the peoples during the ancient plague of Exodus, the nobility and gentry left the city in their droves for weeks. Due to this, during the months of May and mid-June, most of the horses were

removed and it became increasingly difficult to find a horse for hire or sale for up to 6 weeks during the frantic flight *abroad. But, with Health Certificates granted by the Lord Mayor in hand, they left, gaining free passage out from what were rumoured to

soon-be-tolls and policed gateways which would bar and forbid entrance to and from the city due to the outbreak, and in an effort to contain the affluval.

So, before any pretended or possible “lock-down” or quarantine measures could be legislated for, and as the city was relatively clear of the plague and typhus cases,

these Health Certificates were freely granted to the upper classes, and they abandoned the poor and the city.

Mid July – 1268 various ‘distempers’, more than 900 dying by plague, St. Giles’s and St. Martins-in-the-field accounting for around 421 of the deaths by plague

The route the plague took was parish to parish from Clerkenwell Cripplegate Shoreditch & Bishopsgate Aldgate, whitechapel Stepney

By the 18th July, an estimated 1,000 homes had been abandoned and over 200,000 individuals had abandoned London

*abroad meant domestically, as in another neighbouring county or borough, and not overseas as it is taken to infer in today's meaning

August 29th – Sept 5th 8252 6988

Sept 5th – Sept 12th 7690 6544

Sept 12th – Sept 19th 8297 7165

August 8th – 15th Of all diseases

5319

Of the Plague

3880

August 15th – 22nd 5668 4237

August 22nd – 29th 7496 6102

Sept 19th – Sept 30th 6460 5533

Sept 27th – October 3rd 5728 4929

October 3rd – October 10th 5068 4227

Totals 59918 49605

2“I have observed often, that in the parishes of Aldgate, Cripplegate, Whitechapel (Spitalfields) and Stepney, there were 500, 600, 700, 800 in a week in the bills; sometimes 2,000 a week in

those parishes; and I saw positively stated under hand (in writing) of one that made as strict an examination as he could, that there really died 100,000 people of the plague in that one year,

whereas in the bills, the article of the plague was but 68,590”.

The Parish clerks did the accounting and it’s understandable why true accounting figures could never be made as 116 sextons, grave-diggers, bell-men, bearers and the drivers of dead

carts died of the plague during their duties

1“the plague, which raged in a dreadful manner from the middle of August to the middle of October, carried off in that time 30,000 or 40,000 of these very people.. Even by account of the

weekly bills, which yet, I have reason to be assured, never gave a full account by many thousands, the confusion being such .. that in some places no account at all was kept, but they

worked on”.

1,2,3,4 Journal of the Plague 1665, Daniel Defoe, p92, 93, 123, 172

3“Five parishes only, there died over 5,000 in 20 days! I must be allowed to say that, if the bills of mortality said 5,000, I always believed it was twice as many in reality, there being no room

to believe that the account they gave was right; or that, indeed, they were, among such confusions as I saw them in, in any condition to keep an exact account”

4“..but now, on the contrary, most of the people who were taken (infected) during the last two weeks of August, and the first 3 weeks of September, generally died in two or three days time

at the farthest, and many the same day they were taken”

1 “..even just as a mad dog runs on and bites any man or woman, while the frenzy of the distemper was upon them, I mean the person so wounded, would have been incurably infected, as one that was sick before, and had the tokens upon him…

2 “.. For when the distemper was at its height, it generally made them raving and delirious, and when they were so, they would never be so persuaded to keep to

their beds by force; and many who were not tied, threw themselves out of windows when they found they could not get leave to go out of their doors…

3 “..They fell, as if touched by a stroke from Heaven, as men are killed by a flash of lightening; but they found reason to alter their opinion afterwards, for upon

examining the bodies of such after they were dead, they either had tokens (signs) upon them, or other evident proofs of the 4distemper having been longer upon them

than they had otherwise expected…

3 ibid, p166

2 ibid, p160

1 Journal of the Plague 1665, Daniel Defoe, p159

4 other ‘distempers’ would include scorbutic ailments or scurvy, and typhus or ‘spotted fever’ was also circulating simultaneously

1“One to six days after a human receives a flea-bite, the lymph nodes in the armpit (axilla) and groin become very tender and swollen (as large as an egg). These very painful swollen areas are called

buboes (from the Greek bubo, meaning groin). The buboes may suppurate, i.e., break and discharge a particularly fetid pus.

Each of the buboes shown below are on young children (to give you a perspective of size).

1History of Epidemics and Plagues

1“It is not rare for the area of the bite to become gangrenous and necrotic, i.e., the tissue dies. Other symptoms include restlessness, staggering gait, mental confusion, prostration, delirium, rapid pulse, nausea, aching of the extremities and back, and a high fever (at least 40° C = 104° F).


If the fever doesn‘t break, the infection spreads to the blood, causing blood poisoning and death. This is the course of bubonic Plague.

In some cases the microbe can proceed directly to the blood stream and this septicemic Plague can occur before the formation of buboes and results in death before a diagnosis can be made. Some scientists feel that this form of Plague can even be carried by either the common human flea or the body louse. In this infection of the blood, called septicemia, blood vessels break and leak under the skin causing a dark rash as the blood dries (hence the name Black Death, which was given in the 1500s). This blackening tends to begin at the extremities (as in the fingers of the hand shown below).

1 ”For both bubonic and septicemic Plague, there is hemorrhagic illness (internal bleeding), multiple system failure, and death. All of this occurs within three to seven days. The mortality rate for untreated bubonic Plague is about 50–75% and 100% for septicemic Plague.

The observed mortality rate for treated bubonic Plague is 14% and 22% for septicemic Plague.


Neither bubonic nor septicemic Plague is known to be transmitted from person-to-person”

Part of the "Hell" section of Bosch's Garden of Earthly Delights.

Outbreak began September 29th, on the feast of St. Michael (Michaelmas) in 1664, 7 (seven) months after a slow, heavy, sinister comet roamed over the City of London, placing it

around March, 1664

Beginning of August 1665 begat the start of the plague on the east side of the city. The mortality bill from the 11th – 18th July was 1761

By August 1665, large burial pits as large as 40ft in length, 15-16ft broad and in some places, 20ft deep or as close to the water table as possible, where some 50-60 corpses

were placed within. By mid to late August, dead carts were bringing from 200-400 corpses weekly in Aldgate and Whitechapel

6th September, after a huge pit was dug, and up to the 20th September, 1114 corpses had been thrown into it.

1“and therefore, that in the beginning of the effluvia (infection), an order was published by the Lord Mayor and by the magistrates according to the advice

of the physicians and apothecary, that all of the dogs and cats should be immediately killed, and an officer was appointed for the execution.

It is incredible, if their account is to be depended upon, what a prodigious number of these creatures were destroyed. I think they talked about 40,000 dogs and five times as many (5x) cats. All possible endeavors were used also to destroy the mice and the rats, especially the latter, by laying rat’s bane and

other poisons for them, and a prodigious amount of them were also destroyed”

1,2,3,4 Journal of the Plague 1665, Daniel Defoe, p114, 115

Isoniazid and Rifampicin are both contenders for treatment of Mycobacterium Tuberculosis.

However, the viewer may not be aware that this strain of Tuberculosis is Mycobacterium Bovis

and is NOT the human variant which most may be susceptible to for contracting Tuberculosis. It

was initially a human-to-bovine strain

Isoniazid and Rifampicin are have both shown to be ineffective due to TB resistance (MDR – Multi-

Drug Resistance) and should never be given to the HIV-infected, or those who are immuno-

suppressed. There have also been other cases recently in the last few years in different nations where MDR has occurred, such as India. It’s also

been shown that TB can switch from latent TB infection to active TB disease via vaccination and virus shedding within 1 day after administration

makes vaccinated people more prone to spreading the virus amongst contactees. There is

also a long list of undesirable side effects.


1Interspecies transmission of the canine influenza H3N2 virus to domestic cats in South Korea, 2010

Canine species were considered to be the new natural hosts for this virus. However, at the beginning of 2010, influenza-like respiratory signs, such as dyspnoea, were also observed among cats as well as in dogs in an animal shelter located in Seoul,

South Korea. The affected cats showed 100% morbidity and 40% mortality.

The eight viral genes isolated were almost identical to those of the canine influenza H3N2 virus, suggesting interspecies transmission of canine influenza H3N2 virus to the cat

(A/canine/Korea/GCVP01/07 and A/feline/Korea/01/2010 – both H3N2)

Our study shows, for the first time, that cats are susceptible to canine influenza H3N2 infection, suggesting that cats may play an intermediate host role in

transmitting the H3N2 virus among feline and canine species, which could lead to the endemic establishment of the virus in companion animals.

Canine influenza is caused by two subtypes of influenza A virus: H3N8 and H3N2. The H3N8 canine influenza virus (CIV) was first identified in dogs in the USA in

2005, and is known to originate from equine H3N8 (horse) influenza viruses.

CONCLUSIONS

As the presentation continues to be compiled and updated as relevant articles come to light, it is important to remember that these ‘outbreaks’ have the potential to become zoonotic pandemics in a very short period of time.

The UN through its various organs and ‘partnerships’ seem to have a global monitoring system in place now for such an event. Some would argue it is prudent to have such a system in case of such a scenario. But, there is a difference between a possible outbreak and one that appears to be somewhat engineered to some degree, and a situation which is then

managed and controlled via the UN accordingly, through the framework of monitoring, sharing information and data collation.

The entire genome of H3N2 has transmitted to Dogs and Cats. H3N2 is probably the strain to monitor more closely now as it has potential to re-assort and re-combine with other strains (such as H1N1, H5N1, H3N8, or H7N9, or others) and

could potentially cause much distress and trauma, depending on how these strains acquire new DNA or RNA sequences whilst it moves around ethnically diverse locations and adapts. Now, cats and dogs (domestic pets the children love) are possible carriers of Mycobacterium Bovis. But this strain is not protection from human-to-human strains. Should

cats or dogs with H3N2 contract a strain of Mycobacterium Tuberculosis and it re-assorts or re-combines with a flu strain such as those mentioned about, or another unknown pathogen from the haemorrhagic family (dengue fever, ebola, Marburg, Crimean Congo Fever, ect), there is a potential for that strain to become highly pathogenic to both

domestic animals and humans. This is the worst case scenario.

Tuberculosis is highly contagious due to the nature of how it spreads to the lungs and spreads through the expulsion of droplets by way of coughing, sneezing and sputum. Although Mycobacterium is treatable, it is not the strain to be truly concerned about. Already, certain ‘treatments’ from pharmaceutical companies out on the market have shown to be

ineffective for treatment, as certain strains of Tuberculosis has adapted and gained resistance to these drugs (isonazid and rifampfin) in what’s knows as MDR or Multi-Drug resistant. Tuberculosis vaccines cannot be given to everyone due to the multi-drugs concoction used, and the toxicity of them, especially if patients have hypersensitivity (allergies),

HIV, diabetes or other major health issues.

One of the oldest pathogens on the planet is Pseudotuberculosis.

London has the highest (and growing) levels of TB infection in the country and the National Heath Service would struggle to cope with such an event. Air travel and other modes of transportation would also be affected to a major degree.

1A principal source on the origin of the *Black Death is a memoir by the Italian Gabriele de’ Mussi. This memoir has been published several times in its original Latin and has recently been translated into English (although brief

passages have been previously published in translation). This narrative contains some startling assertions:

If this account is correct, Caffa should be recognized as the site of themost spectacular incident of biological warfare ever, with the Black Death

as its disastrous consequence.

and that fleeing survivors of the siege spread plague from Caffa to the Mediterranean Basin.

that the Mongol army hurled plague-infected cadavers into the besieged Crimean city of Caffa, thereby transmitting the disease to the inhabitants;

Hypothesis 2: Deliberate release of a pathogen and deliberate population reduction as an act of Warfare

1Biological Warfare at the 1346 Siege of Caffa, authored by Mark Wheelis, History Review, Emerging Infectious Diseases • Vol. 8, No. 9, September 2002, p971

1Plague predated the Middle Ages by more than 1000 years. The first recorded epidemic erupted in Mongolia in 46 CE. Before it burned itself out, it had taken about two-thirds of the population with it. China was next to play host in 312 CE. Its northern and western provinces were decimated; more than 90% of the population was lost to the disease.

The next major Plague epidemics (pandemics?) occurred:

in 540 CE at Pelusium, Egypt and reached Constantinople in 542 CE and spread into Europe and Asia (the Plague of 2Justinian) in the following decade;

Smaller outbreaks continued for about 200 more years;

London in 1664–1666; Austria in 1711; The Balkans from 1770–1772.

The last major pandemic ran from 1855–1896 worldwide, but mostly in China and India, wherein more than 12 million died.

Manchuria in 1910–1911 lost about 60,000 to pneumonic Plague with a repeat in 1920–1921; A minor outbreak occurred as recently as the summer of 1994 in Surat, India closely following an earthquake in September 1993.

2Historians only recently have developed an interest in this particular pandemic that ranged from 541 to 767. Research indicates that its victims may have numbered as many as 100 million. Population losses of 50–60% in North Africa, Europe, and central and southern Asia have been estimated. The historian Procopius claimed 10,000 deaths each day. Whole provinces were wiped out. ― “All the inhabitants, like beautiful grapes, were trampled and squeezed dry without mercy”, wrote Bishop John of Ephesus of the Plague‘s destruction in Palestine


The ultimate origin of the Black Death is uncertain—China, Mongolia, India, central Asia, and southern Russia have all been suggested

Tentative chronology of the initial spread of plague in the mid-14th century

Spring 1347

The de’ Mussi account is presumed to have been written in 1348 or early 1349 because of its immediacy and the narrow time period described. The original is lost, but a copy is included in a compilation of historical and geographic accounts by various authors, dating from approximately 1367. The account begins with an introductory comment by the scribe who copied the documents: “In the name of God, Amen. Here begins an account of the disease or mortalitywhich occurred in 1348, put together by Gabrielem de Mussis of Piacenza.”

“It was as though arrows were raining down from heaven to strike and crush the Tartars’ arrogance. All medical advice and attention was useless; the Tartars died as soon as the signs of disease appeared on their bodies: swellings in the armpit or groin caused by coagulating humours, followed by a putrid fever”.

1“The dying Tartars, stunned and stupefied by the immensityof the disaster brought about by the disease, and realizing

that they had no hope of escape, lost interest in the siege. Butthey ordered corpses to be placed in 2catapults and lobbed into the city in the hope that the intolerable stench would

kill everyone inside.

1Biological Warfare at the 1346 Siege of Caffa, authored by Mark Wheelis, History Review, Emerging Infectious Diseases • Vol. 8, No. 9, September 2002, p9732Historical texts describe their great machines of war as being capable of hurling a large stone (several hundred kilos!) some 300 meters, History of Pandemics, p6

What seemed like mountains of dead were thrown into the city, and the Christians could not hide or flee or escape

from them, although they dumped as many of the bodies as they could in the sea. And soon the rotting corpses tainted the air and poisoned the water supply, and the

stench was so overwhelming that hardly one in several thousand was in a position to flee the remains of the Tartar army. Moreover one infected man could carry the poison to others, and infect people and places with the disease by look alone. No one knew, or could discover, a means

of defense”.

Flesh and Blood, 1985

1”From historical accounts we know that the Black Death was imported into southern Europe from Asia, and reached Marseilles (southeast France) by November 1347. Plague then spread to

western France by land and sea, reaching Narbonne and Carcassonne at the beginning of 1348”

1Citation: Haensch S, Bianucci R, Signoli M, Rajerison M, Schultz M, et al. (2010) Distinct Clones of Yersinia pestis Caused the Black Death. PLoS Pathog 6(10): e1001134. doi:10.1371/journal.ppat.1001134: Received May 28, 2010; Accepted September 7, 2010; Published October 7, 2010, p4

3 years

The Silk Road route

114th century Europe, following the caravan routes, it was in the lower Volga River basin in 1345, the Caucasus and Crimea by 1346, Constantinople by 1347, Alexandria in the autumn of 1347,

Cyprus and Sicily in that year, Italy by winter 1347, Marseilles by January of 1348, Paris in spring 1348, followed by Germany and the Low Countries in that year, Norway in May 1349,

eastern Europe by 1350, and finally Russia in 1351, but smaller outbreaks continued for about 200 more years;


Caspian S

ea

1Distinct Clones of Yersinia pestis Caused the Black Death


“we show that at least two variants of Yersinia pestis spread over Europe during the second pandemic. The analysis of up to 20 diagnostic markers reveals that the two variants evolved near

the time that phylogenetic branches 1 and 2 separated and may no longer exist. Our results thus resolve a long-standing debate about the etiology of the Black Death and provide key

information about the evolution of the plague bacillus and the spread of the disease during the Middle Ages.

There is no doubt that the ongoing third pandemic was caused by biovar Orientalis, but an attribution of the first and second pandemics to Antiqua and Medievalis (branch 2 – next slide),

respectively, is questionable. Unlike Devignat’s hypothesis (each plague pandemic was caused by a different biovar), recent aDNA (ancient DNA) analyses of samples from the 7th –9th and 18th centuries

yielded Orientalis-specific microsatellites and the characteristic 93 bp glpD deletion, thus suggesting that the Orientalis biovar also caused Justinian’s plague and the second pandemic”.

Medievalis biovar is unable to reduce nitrates due to a G to T mutation that results in a stop codon in the napA gene

Orientalis biovar cannot ferment glycerol because of a 93 bp deletion in the glpD gene

“Y. pestis evolved from its parent species Yersinia pseudotuberculosis within the last 20,000 years”

The samples from France and England also have the ancestral version of SNP s12, which places them near the split between branches 0, 1 and 2. In contrast, the samples from the Netherlands have the derived version of SNP s12, which maps them to the beginning of branch 1

UK and France

MEDIEVALIS BIOVAROREINTALIS BIOVAR

ANTIQUA BIOVAR

ANTIQUA BIOVAR

“Y. pestis evolved from its parent species Yersinia pseudotuberculosis within the last 20,000 years”

“Together with prior analyses from the south of France and Germany, our data from widely distributed mass plague pits ends the debate about the etiology of the Black Death, and unambiguously demonstrates that Yersinia pestis was the causative agent of the epidemic plague that devastated Europe during the Middle Ages.

However, there is an apparent discrepancy between the genotypes identified in this study and those reported elsewhere on aDNA from Justinian’s plague (542 CE) and the second pandemic”


In the first of these analyses, aDNA from Justinian’s plague and the second pandemic was concluded to correspond to biovar Orientalis (now designated 1.ORI) on the basis of microsatellites.

The second analysis demonstrated the presence of the 96 bp glpD deletion that is characteristic of 1.ORI in samples from the 7th to 9th century (Justinian’s plague)and about 1720 (the end of the second pandemic). In our study we did not find the glpD deletion in Yersinia pestis aDNA from the Black Death period (1348-50).

“Our finding of identical genotypes (based on 20 markers) in Saint-Laurent-de-la-Cabrerisse and Hereford thus lends support to historical evidence which suggest that plague spread from France to England in the second half of the 14th century”.

However, the Yersinia pestis genotype identified in our skeletons from Bergen op Zoom differed from those found in Hereford and Saint-Laurent-de-la-Cabrerisse, implying that Bergen op Zoom (and possibly other parts of the southern Netherlands) was not directly infected from England or France in AD 1349”.

“Instead, our results are more consistent with the idea that the genotype in Bergen op Zoom represents a different route of

plague spread, possibly from the northern to the southern Low Countries in

AD 1350”.

“After analysing the teeth of the corpses, scientists believe the bubonic plague – spread by the bites of infected fleas living on black rats – mutated into a

more virulent strain that passed easily from human to human”

They were found close to Smithfield Market last year, in neat rows on two levels sealed under a layer of clay. Thousands of bodies are thought to have been interred at an emergency burial site there.

Teeth from 12 of the skeletons were sent for analysis and four tested positive for Yersinia

pestis - the deadly bacterium is responsible for both bubonic and pneumonic plague. But the researchers concluded that the bubonic strain

could not have had the devastating impact seen during the Black Death.

Dr Tim Brooks, an expert in infectious diseases at Public Health England, said: 'As an explanation for the Black Death in its own right, [bubonic plague

is] simply not good enough.

“Neither bubonic nor septicemic Plague is known to be transmitted from person-to-person”

City Year Mortality

Dantzig 1427 80,000

Paris 1466 40,000

Moscow 1570 200,000

Lyon 1572 50,000

Venice 1576 70,000

London 1603 38,000

Egypt 1603 1,000,000

London 1625 35,417

Naples 1656 300,000

London 1665 *68,596*”and I saw positively stated under hand (in writing) of one that made as strict an examination as he could, that there really died 100,000 people of the plague in that one year, whereas in the bills, the article of the plague was but 68,590”. – Journal of the Plague, Daniel Defoe, p93

1427 1466 1570 1572 1576 1603 1603 1625 1656 1665

170110,000

20,000

30,000

75,000

50,000

100,000

200,000

1.000,000

500,000

300,000

2,000,000

Protestant/Germanic Monarchy

Moscow

Naples

London

Egypt

1345

Re-installation of Feudal/Neo-Feudal System

London

London

Post-Great Fire of London Map, 1666

Year

Deaths

2014

John Evelyn’s new map of London post-AD 1666

Does Yersinia pseudotuberculosis originate from the soil itself and Earth, and if cometary debris falls to Earth after a fly-by, could this fallen mixture of space bacteria (such as ammonium and iridium) lead to food

contamination and the spread of ancient plague, and does yersinia pseudotuberculosis have an age of around 20,000 BC - would this transmit into the food chain and then into other animal hosts (and/or fleas) to acquire new

DNA/RNA gene coding? Would Earthquakes permit re-release of ancient pathogens from the soil/earth?

High frequency of reactive arthritis in adults after Yersinia pseudotuberculosis O:1 outbreak caused by contaminated grated carrots - Ann Rheum Dis (2013)

Transmission of Yersinia pseudotuberculosis in the Pork Production Chain from Farm to Slaughterhouse - Appl. Environ. Microbiol. (2008)

Multiple Outbreaks of Yersinia pseudotuberculosis Infections in Finland - J. Clin. Microbiol. (2004)

A Widespread Outbreak of Yersinia pseudotuberculosis O:3 Infection from Iceberg Lettuce - The Journal of Infectious Disease (2004) - The present study is the first report of a Yersinia pseudotuberculosis outbreak in

which a specific food has been implicated in a population-based, controlled epidemiologic study and traced back to be the source of contamination. Also noted was roe deer (Capreolus capreolus) fecal matter

Salad and Pseudoappendicitis: Yersinia pseudotuberculosis as a Foodborne Pathogen - The Journal of Infectious Disease (2004)

Reactive arthritis after an outbreak of Yersinia pseudotuberculosis serotype O:3 infection - Ann Rheum Dis (2003)

virF-Positive Yersinia pseudotuberculosis and Yersinia enterocolitica Found in Migratory Birds in Sweden - Appl. Environ. Microbiol. (2003)

The Superantigen Gene ypm Is Located in an Unstable Chromosomal Locus of Yersinia pseudotuberculosis - J. Bacteriol.

(2002)Reactive Arthritis and Reiter's Syndrome Following an Outbreak of Gastroenteritis

Caused by Salmonella enteritidis - Clinical Infectious Diseases (2001)

ARTIFICIAL INTELLIGENCE IN THE MOVIES AND TRANSGENICS

1927 1940

GENETICS, ARTIFICIAL INTELLIGENCE AND THE MEDIA

“That we are in process of developing a whole series of techniques which will enable the controlling oligarchy who have always existed and

presumably will always exist to get people to love their servitude. This is the, it seems to me, the ultimate in malevolent revolutions shall we say,

and this is a problem which has interested me many years and about which I wrote thirty years ago, a fable, Brave New World, which is an account of society making use of all the devices available and some of the devices

which I imagined to be possible making use of them in order to, first of all, to standardize the population, to iron out inconvenient human differences, to create, to say, mass produced models of human beings arranged in some sort of scientific caste system. Since then, I have continued to be

extremely interested in this problem and I have noticed with increasing dismay a number of the predictions which were purely fantastic when I made

them thirty years ago have come true or seem in process of coming true.”

1 “If you are going to control any population for any length of time, you must have some

measure of consent, it’s exceedingly difficult to see how pure terrorism can function indefinitely.

It can function for a fairly long time, but I think sooner or later you have to bring in an element of

persuasion; an element of getting people to consent to what is happening to them”

“I’m inclined to think that the scientific dictatorships of the future, and I think there are going to be scientific

dictatorships in many parts of the world, will be probably a good deal nearer to the Brave New World pattern than to the 1984 pattern, they will a good deal nearer not because of any humanitarian qualms of the scientific dictators but simply because the Brave New World pattern is probably a good deal more

efficient than the other.”

1 Aldous Huxley, Berkley speech, 1962

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“You’ll find for example that the experienced hypnotist will tell one that the number of people, the percentage of people who can be hypnotized with the

utmost facility (snaps), just like that. is about 20%, and about a corresponding number at the other end of the scale are very, very difficult or almost

impossible to hypnotize.But in between lies a large mass of people who can with more or less

difficulty be hypnotized,..”

So that’s about 60%!

“But, once given the fact that there are these 20% of highly suggestible people, it becomes quite clear that this is a matter of enormous political

importance, for example, any demagogue who is able to get hold of a large number of these 20% of suggestible people and to organize them is really

in a position to overthrow any government in any country.”

To Huxley, the fact that all citizens aren’t fully and completely 100% brainwashed is why absolute tyranny hasn’t succeeded, whereas, the gradualism of self-consented

slavery and domination using science is preferable

“I mean it’s very fortunate that we have people who are moderately suggestible in themajority and who therefore preserve us from dictatorship but do permit organized

society to be formed.”

If this were a dictatorship, it would be a heck of a lot easier, just so long as I'm the dictator," Bush joked.-- CNN.com, December 18, 2000

How bad are Bush's relations with the Hill? "A dictatorship would be a heck of a lot easier, there's no question about it," he recently joked. Congressional leaders aren't laughing. http://www.businessweek.com/stories/2001-07-29/a-gentlemans-c-for-w

The “creeping red weed” analogy of H.G Wells’ science fiction novel (and soundtrack), “War of the Worlds”


IBM - SyNAPSE: a cognitive computing project from IBM Research - United States

Meeting of the minds

Researchers at IBM have been working on a cognitive computing project called Systems of Neuromorphic Adaptive Plastic Scalable Electronics (SyNAPSE).

IBM is combining principles from nanoscience, neuroscience and supercomputing as part of a multi-year cognitive computing initiative. The Defense Advanced Research Projects Agency (DARPA) has awarded approximately US$21 million in new funding from for phase 2 of the SyNAPSE project.

http://www.ibm.com/smarterplanet/us/en/business_analytics/article/cognitive_computing.html

Published on Aug 15, 2011

Few robots can run, and the researchers say no machine but MABEL can do it with such a human-like gait. Its weight is distributed like a person's. It has a heavier torso and light, flexible legs with springs that act like tendons. MABEL is in the air for 40 percent of each stride, "like a real runner," Grizzle said.

"It's stunning," said Jessy Grizzle, a professor in the Department of Electrical Engineering and Computer Science. "I have never seen a machine doing a motion like this."

With a peak pace of 6.8 miles per hour (with the ability to sprint up to a speed of over 10mph), MABEL is believed to be the world's fastest bipedal robot with knees.

“And yeah, PETMANPETMAN can walk the walk — but he doesn’t stop there either. Scientists have programmed the robot to “simulate human physiology,” so that when being

exposed to chemical agents, researchers can send signals to the robot that forces it to mimic human sweating and switch its body temperature like a real-life soldier might do

while on the field.”

A breakthrough bionic ear that can 'hear' radio frequencies beyond the range of normal human ears has been created by scientists at Princeton University.The researchers used a radical 3D printing technique to create the ear with the electronics of a hearing aid inside it.

They say it is a major step towards creating 'cybermen' such as those seen in the Terminator films, which combine living cells and electronic circuits.

'This field has the potential to generate customized replacement parts for the human body, or even create organs containing capabilities beyond what human biology ordinarily provides.'

“We propose a complete process for designing, simulating, and fabricating synthetic skin for an

animatronics character that mimics the face of a given subject and its expressions. The process starts with

measuring the elastic properties of a material used to manufacture synthetic soft tissue. Given these

measurements we use physics-based simulation to predict the behavior of a face when it is driven by the

underlying robotic actuation.”

http://www.disneyresearch.com/research-areas/robotics/

Dr Marek Urban has developed a unique type of plastic material that turns red or “bleeds” when

damaged, and self-repairs like human skin when exposed to sunlight

‘Our new plastic tries to mimic nature, issuing a red signal when damaged and then

renewing itself when exposed to visible light, temperature or pH changes.’

1 Recently, the Air Force Bioeffects Division announced it’s exploring how radiation from non-lethal energy weapons can

cause changes to the body at the molecular level. The Air Force is pretty broad about what kind of weapons it wants to test: “directed energy, riot control agents, broadband light, acoustic sounds,

and blunt impact materials.”

1 http://www.wired.com/dangerroom/2013/04/air-force-directed-energy/


Here’s how it works: Inferno uses four frequencies spread out over 2 to 5 kHz. The idea behind it is that unlike a regular siren, these particular frequencies have a uniquely disturbing effect on people (and presumably cats, dogs and any other living thing). At 123 dB, it’s loud, but not significantly louder than any other alarm system. The advantage, according to Dr. Goldman, is the combination of frequencies. The human ear just doesn’t like it. I agree, I really didn’t like it.

Summary verdict: Being a guinea pig for a sonic ray was truly a whole lot worse than being a guinea pig for the pain ray. I would happily volunteer again to be hit by the Pentagon’s pain ray. It was fun, like being Bugs Bunny dancing around when Elmer Fudd tries to shoot him. I never, ever again want to be hit by the Inferno.

http://www.wired.com/dangerroom/2008/02/i-was-a-puke-ra/ - I Was a Sonic Blaster Guinea Pig By Sharon Weinberger 02.13.08

Dave, as always, says it exactly as he (and I) see it in 1 image…

1 In close collaboration with the Department of Homeland Security, Defense Advanced Research Projects Agency (DARPA), and the Technical Support Working Group (TSWG), IOS is developing a family of light-based, non-lethal devices for law enforcement and, potentially, military applications.

IOS’ devices produce extremely bright, strobe sequences of light in different colors. The different colors of light are programmed to flash in a predefined pattern, to which the brain cannot adjust. The effect is a “wall of light,”

which blinds the adversary and conceals the user's location, giving the user at least a momentary advantage during which the adversary can be subdued.

Light-based, non-lethal devices currently under development at IOS include an electronic reusable flash grenade, an electronic reusable flash-bang grenade,

and a Light Emitting Diode Incapacitator (LEDI).

IOS LED Incapacitator named byTIME Magazine as one of the Best

Inventions of 2007!

1 http://www.intopsys.com/nonlethal.html

1 The LED Incapacitator (LEDI) uses a range-finder to measure the distance to a

target’s eyes and then unleashes continually changing, multi-color light

pulses that both blind and disorient the person. Intelligent Optical Systems, a

small company in Torrance, CA, is developing the weapon with money from

Homeland Security’s Science and Technology division, which thinks its

possible to have the weapon deployed to cops, National Guard troops and

border agents by 2010.

But first the company’s chief scientist Vladimir Rubtsov wants to discover more

powerful patterns and colors, which he’ll get this fall with tests on volunteers at Penn State University’s Institute for

Nonlethal Defense Technology. ”There’s one wavelength that gets everybody,”

Lieberman said, according to the newsletter. “Vlad calls it the evil color.”

1 http://www.wired.com/threatlevel/2007/07/homeland-secu-3/


http://nldt2.arl.psu.edu/

“And yeah, PETMANPETMAN can walk the walk — but he doesn’t stop there either. Scientists have programmed the robot to “simulate human physiology,” so that when being

exposed to chemical agents, researchers can send signals to the robot that forces it to mimic human sweating and switch its body temperature like a real-life soldier might do

while on the field.”

1 But when Vulcan goes fully online in early 2013, businesses will have one mammoth supercomputer to play with. Vulcan should do about 5 petaflops, or 5 quadrillion calculations per second. That would put it at number four on today’s list of the world’s most powerful computers.

1 http://www.wired.com/wiredenterprise/2012/06/vulcan/

Source http://www.tandfonline.com/doi/abs/10.1080/21550085.2012.685574#preview

http://www.smatthewliao.com/2012/02/09/human-engineering-and-climate-change/

V. Rio Summit - 5. We reaffirm our commitment to make every effort to accelerate the achievement of the internationally agreed development goals, including the

Millennium Development Goals by 2015

Christoff Heyns, UN Special Rapporteur, Unofficial Reports Concerning Legal Matters in the United Nations, Drones, Volume 46, 1st June, 2012, Number 10, p161

16. We reaffirm our commitment to fully implement the Rio Declaration on Environment and Development, Agenda 21, the Programme for the Further

Implementation of Agenda 21,..

“Decisions over life and death in armed conflict may require compassion and intuition. Humans - while they

are fallible - at least might possess these qualities, whereas robots definitely do not. [Robots] should not have the power of life and death over human beings,”

Heyns stated.

Itskov says he wants to work with DARPA - the Defense Advance Research Projects Agency in the U.S military.

DARPA is already researching ways for its troops to use their minds to remotely control androids who will take human soldiers' place on the

battlefield. The Pentagon's hi-tech research arm, has earmarked $7million for research into the project, also nicknamed Avatar.

According to the DARPA's 2013 budget: 'The Avatar program will develop interfaces and algorithms to enable a soldier to effectively partner with a

semi-autonomous bi-pedal machine and allow it to act as the soldier’s surrogate.

2009

Technology could indeed be perceived as a ‘foreign, alien invader’. It invades the living room, privacy, social interaction (and common civility), it

distracts; it vampires time and mental energy. But, on the flip side (and playing devils advocate here), it no

doubt has its uses. Some do treat the internet as a digital incarnation of the library of Alexandria of sorts. It

allows some to create in order to manifest ideas and concepts. It’s a fine balance…

But, like any addiction, it can cause behavioural issues, lack of concentration, delusional perceptions (virtual vs

the real world of matter and substance), it can also bring out all the darker aspects of the mind too.

As this is being created, it has been noted that subtle ‘plague language’ is being used. It is unknown why this meme has surfaced at this time, and as it is a curious sub-harmonic from the number 3 theme that is observed, so the following

have been included for context. Is this a public notice (predictive programming output) for a future pandemic or viral outbreak? Fortunately, the Winter months are passing, the floods are receding, and the Spring is not far off.

If drones can hack wi-fi and passwords, and there are some that have been dumb enough to make their dwelling ‘SMART’, one had best reconsider what one has done for the sake of ‘efficiency’ or ‘ease’.

Wi-fi is an untested and experimental technology and there is no way to know what intracellular damage passive microwave radio

frequencies are doing. Drone may also hack your devices at home and turn your lights (or heating, or water) OFF without

consent

If your entire dwelling is a microwave-saturated environment with wi-fi connected devices to make your life ‘easier’, you may actually

be shortening your life immeasurably, or compromising it drastically.

SMART phones and SMART technology also allows the emptying of said SMART WALLET

without the users knowledge. It’s that easy with digital. No need for anymore ‘real’ bank robbers.

They can just simply hack the software and take it. It could happen as quickly as passing by a shop

mannequin and the SMART phone is zapped and the account is laid bare and empty. It’s that fast.

As more and more victims jump into the Artificial Fairyland and the ‘cloud’ (cloud cuckoo land), and while Google glass or

Oculus Rift is being worn in the home; in a [virtual] world of their own, they are, paradoxically, destroying the very real realm of matter and substance that surrounds them, as machines and Artificial Systems replace natural man and man-power (with

commercial delivery systems such as apple drones and parcels).

There was a phrase heard in times past, “get your head out of the clouds’. Now it seems it’s actively promoted as the

future and a safe realm to be (digitally stored). We’ve come such a long way as a species, only to fall yet again, at the end, as we transform to become digital persons/avatars with a digital ID (most probably an Internet ID) and digital (artificial)

friends.

“Computer Love” – it’s the new ‘craze’ that some would hope you’d become addicted to.. it’s not quite the same ‘artificial love’ as washing that car; the ‘pride and joy’, on a

Sunday afternoon.

This is effectively a Public Notice. Continue to embrace the digital, artificial, UN-human, Borg system, and this is what will be. And do take notice of the possessive “our children”. That should be written as ‘the children’, or at the very

least, ‘YOUR children’. Again, another subtle warning where the medes inform the public who’s property is who’s.

This topic may seem galaxies apart. But, come 2021, the reality we have today will not be the same as tomorrow

For a little more context to why this slide is here, do consult the Occult Symbolism in Movies and Music parts I & II to see how this connects to the Artificial SMART grid system (The “Digital Venus Fly trap”) and why this seems to resemble the subject matter shown in the TV series, ‘Caprica’; the pre-story of

Battlestar Galactica; of how man fell when it created Artificial Life, and before the ‘Created’ turned on its

‘Creators’.

“All of this has happened before, and will all happen again” was a tagline in Battlestar Galactica. History

repeats.

Pop by again soon…

Yes. We got to the mammoths!

More slides were added and more will be as it is collated, if it appears relevant to the overall thesis

We hope this presentation was enlightening

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Thank you, stay safe and stay sharp

(updated and re-uploaded March 31, 2014 14:00hrs)

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