perthes disease by dr.naveen rathor
TRANSCRIPT
PRESENTED BY-PRESENTED BY- DR.NAVEEN RATHORDR.NAVEEN RATHOR RESIDENT DOCTORRESIDENT DOCTOR
DEPT. OF ORTHOPAEDICSDEPT. OF ORTHOPAEDICS RNT MEDICAL RNT MEDICAL
COLLEGE,UDAIPURCOLLEGE,UDAIPUR
SynonymsSynonymsCoxa PlanaCoxa PlanaPseudocoxalgia (Calve)Pseudocoxalgia (Calve)Arthritis deformans juvenilis Arthritis deformans juvenilis
(Perthes)(Perthes)Osteochondroses of the hipOsteochondroses of the hipCoronary disease of the hipCoronary disease of the hipPRECOXALGIAPRECOXALGIA
Blood supply to femoral headBlood supply to femoral head Retinacular arteriesRetinacular arteries Metaphyseal Metaphyseal
arteriesarteries Artery of the teres Artery of the teres
ligamentligament
Blood supply to femoral headBlood supply to femoral head Infants Infants
1.1. Medial ascending cervical or inferior Medial ascending cervical or inferior metaphyseal arteries of trueta.metaphyseal arteries of trueta.
2.2. Lat epiphysealLat epiphyseal3.3. Lig teres – insignificantLig teres – insignificant
4 mts – 4 years4 mts – 4 years1.1. Lat epiphysealLat epiphyseal2.2. Med epiphyseal decrease in number.Med epiphyseal decrease in number.
Blood supply to femoral headBlood supply to femoral head 4 yrs to 7 years4 yrs to 7 years
Epiphyseal plate forms a barrier to Epiphyseal plate forms a barrier to metaphyseal vessels.metaphyseal vessels.
Pre-adolescentPre-adolescentAfter 7 yrs arteries of lig teres become After 7 yrs arteries of lig teres become
more prominent and anastomose with more prominent and anastomose with the lateral epiphyseal vessels.the lateral epiphyseal vessels.
IncidenceIncidence Male : Female = 4-5:1Male : Female = 4-5:1 2.5:1 in India2.5:1 in India Age of onset earlier in females.Age of onset earlier in females. Age – Age – Range – 2-13 years.Range – 2-13 years. Most common 4-8 years.Most common 4-8 years. Average – 6 years.Average – 6 years. Bilateral in 10-12 %Bilateral in 10-12 % Incidence more in Caucasians compared to Incidence more in Caucasians compared to
Negroid, mongoloid, Polynesians.Negroid, mongoloid, Polynesians. In India it is most prevalent in the west coast In India it is most prevalent in the west coast
especially in Udupi district.especially in Udupi district.
EtiologyEtiologyThe exact etiology of Legg Calve The exact etiology of Legg Calve
Perthes disease in not known but Perthes disease in not known but many factors related to etiology of many factors related to etiology of this disease have been mentioned.this disease have been mentioned.
1.1. Vascular supply:Vascular supply: - Angiograms and laser studies have shown - Angiograms and laser studies have shown
medial circumflex artery is missing or medial circumflex artery is missing or obliterated and obturator artery or the lateral obliterated and obturator artery or the lateral epiphyseal artery also affected.epiphyseal artery also affected.
2. 2. Increased intra-articular pressureIncreased intra-articular pressure3. 3. Intraosseous pressureIntraosseous pressure - - Patients has shown that the venous drainage in Patients has shown that the venous drainage in
the femoral head is impaired, causing an the femoral head is impaired, causing an increase in intraosseous pressure.increase in intraosseous pressure.
ETIOLOGYETIOLOGY
4. 4. Coagulation disorderCoagulation disorder - - Associated with absence of factor C or S.Associated with absence of factor C or S. - Increase in serum levels of lipoproteins,thrombogenic - Increase in serum levels of lipoproteins,thrombogenic
substance.substance.55. . Growth hormonesGrowth hormones - - Studies have shown reduced levels of growth hormones, Studies have shown reduced levels of growth hormones,
somatomedin A and C.somatomedin A and C.66. . Social conditionsSocial conditions - - Usually belong to lower socioeconomic status, reflects Usually belong to lower socioeconomic status, reflects
dietary and environmental factorsdietary and environmental factors..77. Trauma. Trauma
8.8.. . Abnormal growth and developmentAbnormal growth and development - - Bone age is lower than chronological age by 1-3 yrs,.Bone age is lower than chronological age by 1-3 yrs,. Ex: carpal bone age: 2 yrs (Triquetral and lunateEx: carpal bone age: 2 yrs (Triquetral and lunate)) - Usually shorter than their peers.- Usually shorter than their peers.9. 9. Genetic factorsGenetic factors - - Inheritance 2-20%;inconsistent pattern.Inheritance 2-20%;inconsistent pattern. - - More Incidence of low birth weight, abnormal birth More Incidence of low birth weight, abnormal birth
presentations.presentations. - First degree relatives have 35% more risk , 2- First degree relatives have 35% more risk , 2ndnd and and 33rdrd
degree relatives are 4 times more prone for degree relatives are 4 times more prone for perthes perthes disease.disease.
TraumaTrauma In the developing femur , the major In the developing femur , the major
lateral epiphyseal artery must course lateral epiphyseal artery must course through a narrow passage ,which through a narrow passage ,which could make it susceptible to could make it susceptible to disruption in case of trauma.disruption in case of trauma.
Sequel to synovitisSequel to synovitisSynovitis of the hip occurs early in Synovitis of the hip occurs early in
LCPLCP
A controversial school of thought A controversial school of thought says that the increased pressure in says that the increased pressure in synovitis may cause a tamponade synovitis may cause a tamponade effect on the vasculatureeffect on the vasculature
Hypothesis for development Hypothesis for development of AVN of femoral headof AVN of femoral head
TRUETA’S HYPOTHESISTRUETA’S HYPOTHESIS - Age < 3 yrs: blood supply contributed by - Age < 3 yrs: blood supply contributed by
metaphyseal and retinacular arteries.metaphyseal and retinacular arteries. - Age 4-8 yrs: Retinacular arteries which enters - Age 4-8 yrs: Retinacular arteries which enters
head as lateral epiphyseal arteries gets head as lateral epiphyseal arteries gets compressed by lateral rotation muscles.compressed by lateral rotation muscles.
Thus trueta postulates that solitary blood supply Thus trueta postulates that solitary blood supply during 4-8 yrs makes vulnerable for AVN of head.during 4-8 yrs makes vulnerable for AVN of head.
After 8 yrs foveolar arteries of ligamentum teres After 8 yrs foveolar arteries of ligamentum teres contribute blood. contribute blood.
Caffey’s HypothesisCaffey’s Hypothesis Intraepiphyseal compression of blood Intraepiphyseal compression of blood
supply to ossification centersupply to ossification center
CausesCauses
PATHOGENESISPATHOGENESIS
LOSS OF BLOOD SUPPLY PRODUCES LOSS OF BLOOD SUPPLY PRODUCES AVN OF THE EPIPHYSEAL OSSCIFICATION AVN OF THE EPIPHYSEAL OSSCIFICATION CENTRE FOLLOWED BY RESORPTION OF CENTRE FOLLOWED BY RESORPTION OF DEAD BONE AND REPLACEMENT WITH DEAD BONE AND REPLACEMENT WITH NEWLY FORMED IMMATURE BONE.NEWLY FORMED IMMATURE BONE.
THE PROCESS IS DESCRIBED IN STAGESTHE PROCESS IS DESCRIBED IN STAGES
RADIOGRAPHIC STAGESRADIOGRAPHIC STAGES
FOUR WALDENSTROM STAGES:FOUR WALDENSTROM STAGES:1) INITIAL STAGE1) INITIAL STAGE2) FRAGMENTATION STAGE2) FRAGMENTATION STAGE3) REOSSIFICATION STAGE3) REOSSIFICATION STAGE4) HEALED STAGE4) HEALED STAGE
INITIAL STAGEINITIAL STAGE EARLY RADIOGRAPHIC EARLY RADIOGRAPHIC
SIGNS:SIGNS: FAILURE OF FEMORAL FAILURE OF FEMORAL
OSSIFIC OSSIFIC NUCLEUS TO GROWNUCLEUS TO GROW
WIDENING OF MEDIAL WIDENING OF MEDIAL JOINT SPACEJOINT SPACE
““CRESCENT SIGN”CRESCENT SIGN” IRREGULAR PHYSEAL IRREGULAR PHYSEAL
PLATEPLATE BLURRY/ RADIOLUCENT BLURRY/ RADIOLUCENT
METAPHYSISMETAPHYSIS
X-RayX-Ray Cresent Sign or Cresent Sign or
Salters sign or Salters sign or Caffey’s signCaffey’s sign
Caffey’s signCaffey’s sign As the disease progresses, a As the disease progresses, a
subchondral # may occur in subchondral # may occur in the anterolateral aspect of the anterolateral aspect of the femoral capital epiphysis.the femoral capital epiphysis.
Is an early radiographic Is an early radiographic feature best seen on the feature best seen on the frog-lateral projection. frog-lateral projection.
This produces a crescentic This produces a crescentic radiolucency known as the radiolucency known as the crescent, Salter’s or Caffey’s crescent, Salter’s or Caffey’s signsign
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Gage’s signGage’s sign Rarefaction in the Rarefaction in the
lateral part of the lateral part of the epiphysis and epiphysis and subjacent subjacent metaphysis.metaphysis.
‘‘Sagging Rope Sign’ Sagging Rope Sign’ This a curvilinear This a curvilinear
sclerotic line running sclerotic line running horizontally across the horizontally across the femoral neck.femoral neck.
It is confirmed by 3D CT It is confirmed by 3D CT studies.studies.
It is a finding in AP It is a finding in AP radiograph in a mature radiograph in a mature hip with Perthes’ hip with Perthes’ disease.disease.
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X-RayX-Ray Sagging rope sign in Sagging rope sign in
adults with history adults with history of perthes – radio of perthes – radio dense line overlying dense line overlying proximal femoral proximal femoral metaphysis, a result metaphysis, a result of growth plate of growth plate damage with damage with metaphysial metaphysial response.response.
FRAGMENTATION STAGEFRAGMENTATION STAGE
BONY EPIPHYSIS BONY EPIPHYSIS BEGINS TO BEGINS TO FRAGMENTFRAGMENT
AREAS OF AREAS OF INCREASED LUCENCY INCREASED LUCENCY AND DENSITYAND DENSITY
EVIDENCE OF REPAIR EVIDENCE OF REPAIR ASPECTS OF DISEASEASPECTS OF DISEASE
X-RayX-Ray Fragmentation of Fragmentation of
epiphysisepiphysis
X-RayX-Ray Metaphyseal Metaphyseal
widening and widening and cystic changes in cystic changes in femoral neckfemoral neck
X-RayX-Ray Lateral extrusion of Lateral extrusion of
femoral head and femoral head and changes in changes in acetabulum.acetabulum.
REOSSIFICATION STAGEREOSSIFICATION STAGE
NORMAL BONE NORMAL BONE DENSITY RETURNSDENSITY RETURNS
ALTERATIONS IN ALTERATIONS IN SHAPE OF FEMORAL SHAPE OF FEMORAL HEAD AND NECK HEAD AND NECK EVIDENTEVIDENT
ClassificationClassificationWaldenstroms classification.Waldenstroms classification.Catterall classification. Catterall classification. Salter classificationSalter classificationHerrings lateral pillar classification.Herrings lateral pillar classification.Modified Elizabethtown classification.Modified Elizabethtown classification.
In 1971 In 1971 used radiological findings of used radiological findings of
epiphyseal involvement to identify epiphyseal involvement to identify 4 groups4 groups
anterior femoral anterior femoral head head involvement involvement
no evidence of no evidence of sequestrum, sequestrum, subchondral subchondral fracture line, or fracture line, or metaphyseal metaphyseal abnormalitiesabnormalities
anterolateral anterolateral involvementinvolvement
Central sequestrum Central sequestrum
Well demarcatedWell demarcated
metaphyseal lesionsmetaphyseal lesions
Subchondral Subchondral fracture line fracture line – Ant ½– Ant ½
lateral column is lateral column is intact.intact.
large sequestrum - large sequestrum - 3/43/4thth of head. of head.
Junction is sclerotic. Junction is sclerotic.
Diffuse Metaphyseal Diffuse Metaphyseal lesionslesions , , anterolaterallyanterolaterally
Subchondral fracture Subchondral fracture lineline - post 1/2 - post 1/2
The lateral column is The lateral column is involved.involved.
Entire headEntire head
Diffuse or Diffuse or central central metaphyseal metaphyseal lesionslesions
posterior posterior
remodelingremodeling of of the epiphysisthe epiphysis
Catterall classificationCatterall classification Groups I and IIGroups I and II had a had a good prognosisgood prognosis
(in 90%) and required no intervention. (in 90%) and required no intervention. Groups III and IVGroups III and IV had a had a poor poor
prognosisprognosis (in 90 %) and required (in 90 %) and required treatment. treatment.
The classification is applied to the frog The classification is applied to the frog lateral and AP film during the lateral and AP film during the fragmentation phase fragmentation phase
CATERALL’S HEAD AT RISK SIGNSCATERALL’S HEAD AT RISK SIGNS
Extent of Extent of subchondral #subchondral # in both AP in both AP & lowenstein frog leg lateral xrays& lowenstein frog leg lateral xrays
reliable indicator in the group with reliable indicator in the group with fracturesfractures
extent of the fracture (line) is less than 50% of the superior dome of the femoral head› good results can be expected.
Extent of the fracture is more than 50% of the dome, › fair or poor results can
be expected
Based on radiographic changes in Based on radiographic changes in lateral portion of femoral headlateral portion of femoral head during during fragmentation stage on AP viewfragmentation stage on AP view
LATERAL PILLAR - lateral 15-30%LATERAL PILLAR - lateral 15-30% of of epiphysis on AP xrayepiphysis on AP xray
Group A Group A – – nono involvement involvement
Group B Group B – – at least 50 % at least 50 % of height of height maintained maintained
Group C Group C – – less than 50% less than 50% of height of height maintainedmaintained
AdvantageAdvantage
Easy application Easy application in active diseasein active disease
High correlation bet High correlation bet lat pillar height and lat pillar height and amount of head flattening amount of head flattening at skeletal at skeletal maturitymaturity
described in 1981described in 1981Used to predict the Used to predict the onset of onset of
degenerative joint diseasedegenerative joint disease following LCPDfollowing LCPD
I – I – Shape is normalShape is normal II – II – loss of head heightloss of head height
< 2 mm < 2 mm deviation of concentric deviation of concentric circlescircles
Group I & II – Group I & II – “Spherical Congruency”“Spherical Congruency”
III – III – Elliptical headElliptical head> 2 mm > 2 mm deviationdeviationContour matches Contour matches (“Incongrous/Aspherical (“Incongrous/Aspherical
congruency”)congruency”)
IV – IV – Flattened Flattened head, >1 cm of head, >1 cm of flatteningflattening
Contour Contour matches matches (“Incongrous/As(“Incongrous/Aspherical pherical congruency”)congruency”)
Resemblence Resemblence with with Cow’s hipCow’s hip
V – V – Collapsed head,Collapsed head,Contour mismatch Contour mismatch (“Incongrous/Aspherical (“Incongrous/Aspherical
Incongruency”)Incongruency”)
CLINICAL FEATURESCLINICAL FEATURESSYMPTOMSSYMPTOMS
MOST CHILDERN PRESENT WITH MILD AND MOST CHILDERN PRESENT WITH MILD AND INTERMITTENT PAIN IN THE THIGH OR A LIMP OR BOTH.INTERMITTENT PAIN IN THE THIGH OR A LIMP OR BOTH.
THE ONSET OF PAIN MAY BE ACUTE OR INSIDIOUS THE ONSET OF PAIN MAY BE ACUTE OR INSIDIOUS THE CLASSICAL PRESENTATION IS DESCRIBED AS A THE CLASSICAL PRESENTATION IS DESCRIBED AS A
“PAINLES LIMP” THE CHILD LIMPS BUT DOES NOT “PAINLES LIMP” THE CHILD LIMPS BUT DOES NOT COMPLAINS OF DISCOMFORT.COMPLAINS OF DISCOMFORT.
PAIN IS AGRRAVATED BY MOVEMENT OF HIP AND PAIN IS AGRRAVATED BY MOVEMENT OF HIP AND RELIVED BY REST.RELIVED BY REST.
H/O OF TRAUMA USUALLY A MILD IS PRESENTH/O OF TRAUMA USUALLY A MILD IS PRESENT..
ClinicalClinicalPhysical:Physical: Painful gaitPainful gaitDecreased range of motionDecreased range of motion (ROM), (ROM),
particularly with internal rotation and particularly with internal rotation and abductionabduction
Atrophy of thigh musclesAtrophy of thigh muscles secondary secondary to disuseto disuse
Muscle spasmMuscle spasmLeg length inequalityLeg length inequality due to collapse due to collapse
ClinicalClinicalShort statureShort stature: Children with LCPD : Children with LCPD
often have delayed bone age.often have delayed bone age.Roll testRoll test
With patient lying in the supine position, With patient lying in the supine position, the examiner rolls the hip of the the examiner rolls the hip of the affected extremity into external and affected extremity into external and internal rotation.internal rotation.
This test should invoke guarding or This test should invoke guarding or spasm, especially with internal rotation.spasm, especially with internal rotation.
InvestigationInvestigationX-Ray –AP & Frog leg Lat viewX-Ray –AP & Frog leg Lat viewUSGUSGArthrographyArthrographyBone ScanBone ScanCTCTMRIMRIHAEMOGRAMHAEMOGRAM
Hematological parametersHematological parameters ESRESR CRPCRP Coagulability profile.Coagulability profile. X-raysX-rays USGUSG CT scanCT scan MRIMRI BONE SCANBONE SCAN ArthrographyArthrography Scintigraphy.Scintigraphy.
INVESTIGATIONSINVESTIGATIONS
Imaging – Radiographic Imaging – Radiographic FeatureFeature
Widening of the joint space and minor subluxationWidening of the joint space and minor subluxation Cresent sign/gaze sign/sagging rope signCresent sign/gaze sign/sagging rope sign Fragmentation and focal resorptionFragmentation and focal resorption Loss of sphericity of femoral headLoss of sphericity of femoral head Loss of height of lateral pillersLoss of height of lateral pillers Metaphyseal cyst formationMetaphyseal cyst formation Widening of the femoral neck & head (Coxa Magna)Widening of the femoral neck & head (Coxa Magna) Lateral uncovering &subluxation of the femoral headLateral uncovering &subluxation of the femoral head Head within head appearanceHead within head appearance Acetabular remodellingAcetabular remodelling
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Ultrasound features Ultrasound features Effusion, especially if persistentEffusion, especially if persistentSynovial thickeningSynovial thickeningCartilaginous thickeningCartilaginous thickeningAtrophy of the ipsilateral quadriceps Atrophy of the ipsilateral quadriceps
musclemuscleFlattening, fragmentation, irregularity Flattening, fragmentation, irregularity
of the femoral headof the femoral headNew bone formationNew bone formation
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Accurate imaging modality for early diagnosis Accurate imaging modality for early diagnosis of perthes disease.of perthes disease.
Evaluated congruity of articular surfaces, Evaluated congruity of articular surfaces, femoral head containment, joint effusion and femoral head containment, joint effusion and synovial hypertrophy.synovial hypertrophy.
Epiphyseal involvement clearly visualised on Epiphyseal involvement clearly visualised on MRI 3 to 8 months after first symptoms.MRI 3 to 8 months after first symptoms.
Diagnostic accuracy: 97-99%.Diagnostic accuracy: 97-99%. Also provides earlier and reliable information Also provides earlier and reliable information
on revascularisation and extent of femoral on revascularisation and extent of femoral head necrosis.head necrosis.
MRIMRI
Demonstrates actual contour of femoral head Demonstrates actual contour of femoral head and state of congruity of articular surfaces.and state of congruity of articular surfaces.
It provides reliable information regarding It provides reliable information regarding containment of femoral head within acetabulum.containment of femoral head within acetabulum.
Major advantage is that examiner can assess Major advantage is that examiner can assess congruity of hip in different positions.congruity of hip in different positions.
Often used in early diagnosis of hinge abduction Often used in early diagnosis of hinge abduction of hip.of hip.
Useful in fragmentation and reparative stages.Useful in fragmentation and reparative stages.
ARTHROGRAPHYARTHROGRAPHY
Bone ScanBone Scan Indicated to diagnose in early stages Indicated to diagnose in early stages
and to classify the severity.and to classify the severity.Diagnosis possible months before Diagnosis possible months before
signs appear on X-Ray.signs appear on X-Ray.Avascular areas show cold spots.Avascular areas show cold spots.Revascularisation can be detected Revascularisation can be detected
much before radiographic evidence.much before radiographic evidence.
Bone ScanBone Scan Convay et al Convay et al
classificationclassification Stage 1 is total Stage 1 is total
lack of uptakelack of uptake
Bone ScanBone Scan Revascularisation at Revascularisation at
lateral columnlateral column Failure to Failure to
revascularise at lat revascularise at lat column is a grave signcolumn is a grave sign
Also called Also called “scintigraphic head at “scintigraphic head at risk sign”risk sign”
Precedes radiographic Precedes radiographic head at risk sign by 2-head at risk sign by 2-3 mths3 mths
Bone ScanBone Scan Gradual filling of Gradual filling of
anterolateral partanterolateral part
Bone ScanBone Scan Return to normalReturn to normal
AVN caused by variety of conditionsAVN caused by variety of conditions - Sickle cell anemia- Sickle cell anemia - Other hemoglobinopathies- Other hemoglobinopathies - Thalassemia- Thalassemia - Steroid medication- Steroid medication - After traumatic hip dislocation- After traumatic hip dislocation - Treatment of developmental dysplasia of - Treatment of developmental dysplasia of
hiphip
DIFFERENTIAL DIAGNOSISDIFFERENTIAL DIAGNOSIS
1)1) Transient synovitisTransient synovitis2)2) Slipped femoral epiphysisSlipped femoral epiphysis3)3) Congenital dysplasia of hipCongenital dysplasia of hip4)4) Congenital coxa varaCongenital coxa vara5)5) Early TuberculosisEarly Tuberculosis6)6) Rheumatoid arthritisRheumatoid arthritis7)7) EPIPHYSEAL DYSPLASIASEPIPHYSEAL DYSPLASIAS
COMPARISON BETWEEN COMPARISON BETWEEN TRANSIENT SYNOVITIS &LCPDTRANSIENT SYNOVITIS &LCPD
LCP DISEASE TRANSIENT SYNOVITIS LCP DISEASE TRANSIENT SYNOVITIS -Average duration of - average duration of-Average duration of - average duration ofSymptom is 6-8 wk symptomes in days Symptom is 6-8 wk symptomes in days -synovial thickening -synovitis with -synovial thickening -synovitis with
capsularcapsular distension distension -bony changes & -no bony changes-bony changes & -no bony changes necrosisnecrosis
Comparison chartComparison chart
COMPARISON CHARTCOMPARISON CHARTPERTHES DISEASE EPIPHYSEAL DYSPLASIAUnilateral Bilateral involvement
If B/L, marked asymmetry, disease in differing stages and severity
Symmetrical findings
No involvement of other joints Involvement of other joints or spine.
Acetabulum not involved Involved
Sclerotic and cystic changes in femoral head and cystic changes in metaphysis
Few sclerotic changes in femoral head.
More tendency towards lateral calcification and subluxation
Little tendency.
Prognostic FactorsPrognostic Factors1.1. Age at diagnosisAge at diagnosis2.2. Extent of involvementExtent of involvement3.3. SexSex4.4. Catterall “head at risk” clinical signsCatterall “head at risk” clinical signs
ClinicalClinical1.1. Progressive loss of hip motionProgressive loss of hip motion2.2. Increasing abduction contractureIncreasing abduction contracture3.3. Obese childObese child
Prognostic featuresPrognostic features Age Age
<6yrs; good regardless of treatment<6yrs; good regardless of treatment 6-9years; not always satisfactory with containment6-9years; not always satisfactory with containment >10yrs; questionable benefit from containment, >10yrs; questionable benefit from containment,
poor prognosispoor prognosis GenderGender
Girls have worse prognosisGirls have worse prognosis Classification gradeClassification grade
Herrings lateral pillar classificationHerrings lateral pillar classification Salter and thompson grade B worse prognosisSalter and thompson grade B worse prognosis Caterral classification gradeCaterral classification grade
Caterral “head-at-risk” signsCaterral “head-at-risk” signs The five signs carry worse prognosisThe five signs carry worse prognosis
Others Others Body weight, decreased ROMBody weight, decreased ROM
Classification of PrognosisClassification of Prognosis
- CE(Center-edge)angle of Weiberg.- CE(Center-edge)angle of Weiberg. - Salters extrusion Index.- Salters extrusion Index. - Epiphyseal index.- Epiphyseal index. - Epiphyseal quotient. - Epiphyseal quotient. - Stulberg classification.- Stulberg classification.
CE angle of WeibergCE angle of Weiberg Indicator of acetabular depth Indicator of acetabular depth
It is the angle formed by a It is the angle formed by a perpendicular lines through perpendicular lines through the midportion of the femoral the midportion of the femoral head and a line from the head and a line from the femoral head center to the femoral head center to the upper outer acetabular upper outer acetabular margin. margin.
Normal = 20 to 40 degrees, Normal = 20 to 40 degrees, with an average of 36 with an average of 36 degrees. degrees.
This angle may be slightly This angle may be slightly larger in women and in older larger in women and in older persons.persons.
Angle >25 = good, 20-25= Angle >25 = good, 20-25= fair, < 20 = poorfair, < 20 = poor
Salters extrusion IndexSalters extrusion Index If AB is more If AB is more
than 20% of than 20% of CD it CD it indicates a indicates a poor poor prognosis prognosis
TreatmentTreatmentObjectivesObjectives - To produce a normal femoral - To produce a normal femoral head and neckhead and neck - To produce a normal acetabulum- To produce a normal acetabulum - A congruous hip which is fully - A congruous hip which is fully mobilemobile - To prevent degenerative arthritis - To prevent degenerative arthritis of the hip later in lifeof the hip later in life
TreatmentTreatmentGOAL :GOAL :Treatment efforts are directed towardsTreatment efforts are directed towards - Restoration and maintenance of - Restoration and maintenance of full mobility of the hipfull mobility of the hip - Containment of the femoral- Containment of the femoral head.head. - Resumption of weight bearing- Resumption of weight bearing and full activity as soon as and full activity as soon as possiblepossible
Rational behind "containment"Rational behind "containment"
Containment of the head within the acetabulum is Containment of the head within the acetabulum is reported to reported to encourage spherical remodelling encourage spherical remodelling during the reossification and subsequent phases. during the reossification and subsequent phases.
However if there is However if there is total head involvementtotal head involvement and and the lateral pillar collapses then the effect of the lateral pillar collapses then the effect of containment is probably containment is probably lessless..
Therefore it seems that the extent of involvement Therefore it seems that the extent of involvement of the head is the of the head is the critical factorcritical factor and containment and containment simply optimizes the situation. simply optimizes the situation.
Methods of CONTAINMENT OF Methods of CONTAINMENT OF
HEADHEAD (a) Conservative methods (a) Conservative methods (b) Surgical methods(b) Surgical methods
TreatmentTreatmentTreatment is divided into 3 phasesTreatment is divided into 3 phases
Initial Phase – restore & maintain Initial Phase – restore & maintain mobilitymobility
Active Phase – Containment and Active Phase – Containment and maintainance of full mobility.maintainance of full mobility.
Reconstructive phase – correct Reconstructive phase – correct residual deformities.residual deformities.
Treatment ( Initial Phase )Treatment ( Initial Phase )Physiotherapy – active and passive Physiotherapy – active and passive range of motionrange of motion exercises to restoreexercises to restore motionmotionTraction – B/L skin traction and Traction – B/L skin traction and gradually abducting over 1-2 gradually abducting over 1-2 weeks till full abduction isweeks till full abduction is regained.regained.
Treatment ( Active Phase )Treatment ( Active Phase )Consists of containment of the Consists of containment of the
femoral head within the acetabulum. femoral head within the acetabulum. This can be achieved by This can be achieved by
orthosis orthosis or byor by surgerysurgery
Hip irritability with decrease of hip motion:Hip irritability with decrease of hip motion:
1-2 week period of bed rest with abduction traction1-2 week period of bed rest with abduction traction if recursif recurs 2-3 months period of surgical non containment to 2-3 months period of surgical non containment to
decrease risk of extrusion.decrease risk of extrusion.
X-ray taken bi-monthly for evaluation.X-ray taken bi-monthly for evaluation.
Treatment (Orthosis)Treatment (Orthosis) Non Ambulatory weight releivingNon Ambulatory weight releiving
1.1. Abduction broomstick plaster castAbduction broomstick plaster cast2.2. Hip spica castHip spica cast3.3. Milgram hip abduction orthosisMilgram hip abduction orthosis
Ambulatory Both limbs includedAmbulatory Both limbs included1.1. Petrie Abduction castPetrie Abduction cast2.2. Toronto orthosisToronto orthosis3.3. Newington orthosisNewington orthosis4.4. Birmingham braceBirmingham brace5.5. Atlanta Scotish Rite Brace Atlanta Scotish Rite Brace
Treatment (Orthosis)Treatment (Orthosis) Atlanta Scotish Rite Atlanta Scotish Rite
BraceBrace
Atlanta Scotish Rite BraceAtlanta Scotish Rite Brace
Newington orthosisNewington orthosis
Birmingham braceBirmingham brace
Toronto BraceToronto Brace
Treatment (Orthosis)Treatment (Orthosis) Orthotic treatment is discontinued when Orthotic treatment is discontinued when
the disease enters the reparative phase the disease enters the reparative phase and healing is established.and healing is established.
The radiographic evidence of healing areThe radiographic evidence of healing are1.1. Appearance of regular ossification in the Appearance of regular ossification in the
femoral head.femoral head.2.2. Increased density of femoral head should Increased density of femoral head should
disappear.disappear.3.3. Metaphyseal rarefaction involving the Metaphyseal rarefaction involving the
lateral cortex of the metaphysis should lateral cortex of the metaphysis should ossify.ossify.
4.4. There should be intact lateral column.There should be intact lateral column.5.5. There should be normal trabecular bone There should be normal trabecular bone
in the epiphysis.in the epiphysis.
So finally…. before planning So finally…. before planning surgery, first think of atleast 4 surgery, first think of atleast 4
things …..things …..Pathological stagePathological stageAge Age Range of motionRange of motionCatterall & herrings stagingCatterall & herrings staging
Treatment ( Surgical)Treatment ( Surgical)Femoral varus osteotomy.Femoral varus osteotomy. Inominate osteotomy.Inominate osteotomy.Combined femoral and inominate Combined femoral and inominate
osteotomyosteotomyValgus osteotomyValgus osteotomyShelf arthroplastyShelf arthroplastyChiari osteotomyChiari osteotomyCheilectomy.Cheilectomy.Trochanteric advancement or arrest.Trochanteric advancement or arrest.
PELVIC OSTEOTOMYPELVIC OSTEOTOMY Redirectional OsteotomyRedirectional Osteotomy
Salter’s osteotomy to Salter’s osteotomy to reorient the acetabulumreorient the acetabulum
Shelf OperationShelf Operation To create a bony shelf to To create a bony shelf to
cover the extruded part of cover the extruded part of the epiphysis.the epiphysis.
Displacement OsteotomyDisplacement Osteotomy Chiari osteotomy is Chiari osteotomy is
another way to improve the another way to improve the coveragecoverage..
Inominate osteotomyInominate osteotomy IndicationsIndications::1.>6 yrs1.>6 yrs2.mod. Or severely affected head with loss of 2.mod. Or severely affected head with loss of
containment(Catterall grade 3containment(Catterall grade 3rdrd & 4 & 4thth))
RequirementRequirement::Able to abduct 45 deg. And femoral head to be Able to abduct 45 deg. And femoral head to be
contained in positioncontained in positionADVANTAGE :ADVANTAGE :1)Anterolateral coverage of frmoral head1)Anterolateral coverage of frmoral head2)Lengthening of extremity2)Lengthening of extremity3)Avoidance of second surgery for implant removal3)Avoidance of second surgery for implant removal
COMPLICATIONS:-COMPLICATIONS:-1)Inability to achieve containment in 1)Inability to achieve containment in
older childolder child2)Sometimes limb lenghtening2)Sometimes limb lenghtening3)Increase hip pressure can cause 3)Increase hip pressure can cause
further AVNfurther AVN
Shelf ArthroplastyShelf ArthroplastyPerformed to enlarge the volume of Performed to enlarge the volume of
acetabulum.acetabulum.
Indication:A deficient acetabulum cannot Indication:A deficient acetabulum cannot be corrected by pelvic osteotomybe corrected by pelvic osteotomy
Contra indication: Dysplastic hip with Contra indication: Dysplastic hip with spherical congruity spherical congruity
ADVANTAGE:-ADVANTAGE:-1)LATERAL ACETABULAR GROWTH 1)LATERAL ACETABULAR GROWTH
STIMULATIONSTIMULATION2)SHELF RESOLUTION AFTER FEMORAL 2)SHELF RESOLUTION AFTER FEMORAL
EPIPHYSEAL OSSIFICATIONEPIPHYSEAL OSSIFICATION3)PREVENTION FROM SUBLUXATION3)PREVENTION FROM SUBLUXATIONCOMPLECATIONS:-COMPLECATIONS:-1)LOSS OF HIP FLEXION1)LOSS OF HIP FLEXION2)LATERAL CUTANEOUS NERVE AT RISK2)LATERAL CUTANEOUS NERVE AT RISK
i)Curved incision below iliac crest, strip glutei.ii) Mobilize and divide reflected head of rectus femoris
iii) Trough in bone above insertion of capsule.iv) Strips of cancellous bone inserted into trough so that they form a
canopy on superior surface of hip joint.v) Pack web space between flap and graft canopy with gratft
vi)Repair rectus and lose the wound.
Chiari osteotomyChiari osteotomy Capsular interpositional Capsular interpositional
arthroplasty(capsule is arthroplasty(capsule is interposed b/w newly interposed b/w newly formed acetabulum roof formed acetabulum roof & femoral head)& femoral head)
Usually after 10 yr of ageUsually after 10 yr of age Deepens the deficient Deepens the deficient
acetabulum by medial acetabulum by medial displacement of distal displacement of distal pelvic fragment and pelvic fragment and improves sup.lat.femoral improves sup.lat.femoral coverage.coverage.
FEMORAL OSTEOTOMYFEMORAL OSTEOTOMY
Technically less demanding than innominate osteotomyTechnically less demanding than innominate osteotomy
Usually 20Usually 200 0 varus angulation & 20varus angulation & 200 0 IR appears sufficient.IR appears sufficient.
Good to decide abduction, internal rotation or flexion on a Good to decide abduction, internal rotation or flexion on a pre-operative arthrogram.pre-operative arthrogram.
Femoral varus osteotomyFemoral varus osteotomyIndicationsIndications: 1.>6yrs of age: 1.>6yrs of age 2.excessive femoral anteversion 2.excessive femoral anteversion 3.catterall grade 33.catterall grade 3rdrd & 4th & 4thComplicationsComplications:1.excessive post op varus:1.excessive post op varus2.Persistant ext.rotation2.Persistant ext.rotation3.Shortening of extremity3.Shortening of extremity4.Incresed abductor lurch4.Incresed abductor lurch5.Trochanteric over growth5.Trochanteric over growth6.Delayed or non union6.Delayed or non union
varus should not be varus should not be exceeds more than 110 exceeds more than 110 degree.degree.
Level of osteotomy
Insertion of guide pin and reaming of femur
First depth marking flush with lateral cortex
Removal of wedge to customize it
Uuuu uUuuu u
usually we use varususually we use varus (medial closing wedge)(medial closing wedge) osteotomy fixed with an osteotomy fixed with an pediatric hip screwpediatric hip screw
Plate and compression screw application
Insertion of bone screws.
Valgus OsteotomyValgus Osteotomy
Indication:hinged Indication:hinged abduction of hipabduction of hip
CHEILECTOMYCHEILECTOMYLarge malformed femoral head Large malformed femoral head
outside the acetabulum causing outside the acetabulum causing painful sensation on abduction/lack painful sensation on abduction/lack of abductionof abduction
Removal of malformed femoral head Removal of malformed femoral head protruding outside acetabulumprotruding outside acetabulum
Result after short term follow up are Result after short term follow up are good but detoriates with timegood but detoriates with time
Widening of the jointWidening of the jointUnloads the joint Unloads the joint
spacespaceReduces pressure Reduces pressure
over headover headArticular cartilage Articular cartilage
repairrepairMaintain congruencyMaintain congruencyAllows 50 degree Allows 50 degree
flexionflexion
TTROCHANTRIC OVERGROWTHROCHANTRIC OVERGROWTH -due to premature closure of femoral epiphysis-due to premature closure of femoral epiphysis - Elevation of trochanter decreases tension - Elevation of trochanter decreases tension
and mechanical efficiency of pelvic and and mechanical efficiency of pelvic and trochantric muscles.trochantric muscles.
- Shortened femoral neck moves trochanter - Shortened femoral neck moves trochanter closer to centre of rotation of hip, line of pull closer to centre of rotation of hip, line of pull of muscles becomes more vertical.of muscles becomes more vertical.
- Impingement of head to the roof limiting - Impingement of head to the roof limiting abduction.abduction.
Normal growth pattern
Long. Growth arrested, greater trochanter continues
GEAR STICK SIGNGEAR STICK SIGNDifferentiation b/w trochrntric Differentiation b/w trochrntric
impingment & other cause of impingment & other cause of restricted hip abductionrestricted hip abduction
In case if trochentric impingment-In case if trochentric impingment-hip extension-impingmented during hip extension-impingmented during
abductionabductionHip flexion-no impingment during Hip flexion-no impingment during
abductionabduction
Trochanteric advancementTrochanteric advancement IndicationsIndications::
- Trochanteric over - Trochanteric over growthgrowth
- Capital femoral - Capital femoral physeal growth physeal growth arrestarrest
Recommended Indications for Recommended Indications for diff. surgeriesdiff. surgeries
Hinged abduction - valgus Hinged abduction - valgus subtrochanteric osteotomysubtrochanteric osteotomy
Severly Mal formed femoral head – Severly Mal formed femoral head – cheilectomycheilectomy
Coxa magna – shelf augmentationCoxa magna – shelf augmentationA large malformed femoral head with A large malformed femoral head with
lat.subluxation – Chiari’s pelvic lat.subluxation – Chiari’s pelvic osteotomyosteotomy
Capital femoral physeal arrest – Capital femoral physeal arrest – Trochanteric advancementTrochanteric advancement
Recent AdvancesRecent AdvancesAnticoagulantAnticoagulantBotulinum toxinBotulinum toxin Ibadronate :this has shown there Ibadronate :this has shown there
importance in rat model by increase importance in rat model by increase spericity of femoral headspericity of femoral head
Still lot more work to do in this fieldsStill lot more work to do in this fields
References References Campbell’s operative Orthopaedics – Campbell’s operative Orthopaedics –
1010thth edition editionTachdjian’s paediatric orthopaedics – Tachdjian’s paediatric orthopaedics –
33rdrd edition editionMercer’s Orthopaedic surgery – 9Mercer’s Orthopaedic surgery – 9thth
editionedition Journals of bone and joint surgeryJournals of bone and joint surgery InternetInternet
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