peripheral arteriography

1
276 aneurysms. A changing entity. Ann Surg 1992; 215: 435. Glenn, WWL, Geha AS, Hammond GL, Laks H, Naun- heim KS. Glenn's thoracic and cardiovascular sur- gery. 6th ed. Stamford: Appleton & Lange, 1997; 955-2325. Hamada S, Takamiya M, Kimura K, et al. Type A aortic dissection: Evaluation with ultrafast CT. Radiology 1992; 183:155. Hollier LH, Stanson AW, Glovicski P, et al. Arterio- megaly: Classification and morbid implications of diffuse aneurysmal disease. Surgery 1983; 93:700. ]ohnsrude IS, Jackson DC, Dunnick NR. A practical ap- proach to angiography. 2nd ed. Boston: Little, Brown and Company, 1987; 18:523-554. Kadir S. Diagnostic angiography. 1st ed. Baltimore: W.B. Saunders Company, 1986; 8:124-171. Kouchoukos NT. Aneurysms of the ascending aorta. In: Glenn WWL, ed. Glenn's thoracic and cardiovas- cular surgery. Vol II. Stamford: Appleton & Lange, 1997; 2225-2237. Lande A, Berkmen YM. Aortitis, pathologic, clinical and arteriographic review. Radiol Clin North An1 1976; 14:219. Lui RC, Menkis AH, McKenzie FN. Aortic dissection without intimal rupture: Diagnosis and manage- ment. Ann Thorac Surg 1992; 53:886. Minard G, Schurr M], Croce MA, Gavant ML, Kudsk KA, Taylor M], et al. A prospective analysis of trans- esophageal echocardiography in the diagnosis of traumatic disruption of the aorta. ] Trauma 1996; 40:225-30. Mirvis SE, Shanmuganathan K, Miller BH, White CS, Tur- ney SZ. Traumatic aortic injury: diagnosis with contrast-enhanced thoracic CT-five-year experi- ence at a major trauma center. Radiology 1996; 200: 413-22. Nienaber CA, Spielmann RP, von Kodolitsch Y, et al- Diagnosis of thoracic aortic dissection. Magnetic resonance imaging versus transesophageal echocar- diography. Circulation 1992; 85:434. Remy], et al. Treatment of hemoptysis by embolization of bronchial arteries. Radiology 1977; 122:33. Seizer SE, Orsi CD, Kirshner R, DeWeese ]A. Traumatic aortic rupture: Plain radiographic findings. AJR 1981; 137:1011-1014. Smith MD, Cassidy]M, Souther S, Morris E], Sapin PM, Johnson SB, et al. Transeophageal echocardiogra- phy in the diagnosis of traumatic rupture of the aorta. N Engl] Med 1995; 332:356. Warren RL, Hilgenberg AD, McCabe C]. Blunt and pen- etrating trauma to the great vessels. In: Glenn WWL, ed. Glenn's thoracic and cardiovascular surgery. Vol II. Stamford: Appleton & Lange, 1997; 2213-2224. Yamaguchi T, Guthaner DF, Wexler 1. Natural history of the false channel of type A aortic dissection after surgical repair: CT study. Radiology 1989; 170:743. Zotz R], Erbel R, Meyer]. Noncommunicating intramu- ral hematoma: An indication of developing aortic dissection: ] Am Soc Echocardiog 1991; 4:636. 4:00 pm Peripheral Arteriography Ernest j. Ring, MD 4:30 pm Venous and Pulmonary Arterial Imaging Steven C. Rose, MD Learning objectives: From this presentation, attendees should be able to: (1) list the different sonographic tech- niques used to evaluate each of the three major venous segments of the lower extremity (iliocaval, femoropopli- teal, and infrapopliteal) for deep venous thrombosis (DVT); (2) list the four fundamental items to evaluate sonographically in patients with lower extremity venous insufficiency; (3) list the sonographic criteria to diag- nose occlusion of (a) the sonographically accessible tho- racic veins (aXillary and internal jugular veins), and (b) the sonographically inaccessible conduit veins (cen- tral subclavian, innominate veins and superior vena cava [SVC)); (4) list six important anatomic variants of the inferior vena cava (IVC)/renal venous system, and three variants of the SVc,· and (5) list three sonographic imaging alternatives to pulmonary angiography for di- agnoses ofpulmonary embolism. BLOOD flow in normal lower extremity veins courses from the periphery toward the right atrium and from the subcutaneous superficial veins toward the deep venous system via perforating branches. Blood flow is largely unidirectional because of the bicuspid valves located principally distal to the inguinal ligament. Because of low intraluminal pressure (typically less than 15 mm Hg when supine), venous blood in the central portions of both the normal lower and upper extremity veins (eg, common femoral, axillary, or internal jugular veins) var- ies considerably with changes in intrathoracic and intra- abdominal pressure during respiration and right atrial pressure changes during the cardiac cycle. The pliant venous walls and relatively low intraluminal pressure permit ready distention with dependency or volume overload ("capacitance function") and compression with external pressure. Important anatomic variants include duplication of the popliteal, superficial femoral, and greater saphenous veins, and compression of the left common iliac vein by the overlying right common iliac artery. The latter con- dition may lead to synechiae formation and/or thrombo- sis (May-Thurner Syndrome). The most common indication for evaluation of lower extremity veins is diagnosis of suspected acute DVT. His- torically, the diagnostic gold standard is ascending con- trast venography that was assumed to have an accuracy approaching 100%. However, contrast venography en- tails patient discomfort, has risks associated with contrast injection (notably skin slough, induced thrombophlebi-

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Page 1: Peripheral Arteriography

276

aneurysms. A changing entity. Ann Surg 1992; 215:

435.Glenn, WWL, Geha AS, Hammond GL, Laks H, Naun­

heim KS. Glenn's thoracic and cardiovascular sur­gery. 6th ed. Stamford: Appleton & Lange, 1997;955-2325.

Hamada S, Takamiya M, Kimura K, et al. Type A aorticdissection: Evaluation with ultrafast CT. Radiology

1992; 183:155.Hollier LH, Stanson AW, Glovicski P, et al. Arterio­

megaly: Classification and morbid implications of

diffuse aneurysmal disease. Surgery 1983; 93:700.

]ohnsrude IS, Jackson DC, Dunnick NR. A practical ap­proach to angiography. 2nd ed. Boston: Little,Brown and Company, 1987; 18:523-554.

Kadir S. Diagnostic angiography. 1st ed. Baltimore:

W.B. Saunders Company, 1986; 8:124-171.

Kouchoukos NT. Aneurysms of the ascending aorta.In: Glenn WWL, ed. Glenn's thoracic and cardiovas­cular surgery. Vol II. Stamford: Appleton & Lange,

1997; 2225-2237.

Lande A, Berkmen YM. Aortitis, pathologic, clinicaland arteriographic review. Radiol Clin North An1

1976; 14:219.

Lui RC, Menkis AH, McKenzie FN. Aortic dissectionwithout intimal rupture: Diagnosis and manage­

ment. Ann Thorac Surg 1992; 53:886.

Minard G, Schurr M], Croce MA, Gavant ML, Kudsk KA,

Taylor M], et al. A prospective analysis of trans­esophageal echocardiography in the diagnosis of

traumatic disruption of the aorta. ] Trauma 1996;40:225-30.

Mirvis SE, Shanmuganathan K, Miller BH, White CS, Tur­ney SZ. Traumatic aortic injury: diagnosis withcontrast-enhanced thoracic CT-five-year experi­

ence at a major trauma center. Radiology 1996; 200:413-22.

Nienaber CA, Spielmann RP, von Kodolitsch Y, et al­Diagnosis of thoracic aortic dissection. Magnetic

resonance imaging versus transesophageal echocar­diography. Circulation 1992; 85:434.

Remy], et al. Treatment of hemoptysis by embolizationof bronchial arteries. Radiology 1977; 122:33.

Seizer SE, Orsi CD, Kirshner R, DeWeese ]A. Traumatic

aortic rupture: Plain radiographic findings. AJR1981; 137:1011-1014.

Smith MD, Cassidy]M, Souther S, Morris E], Sapin PM,Johnson SB, et al. Transeophageal echocardiogra­phy in the diagnosis of traumatic rupture of the

aorta. N Engl] Med 1995; 332:356.

Warren RL, Hilgenberg AD, McCabe C]. Blunt and pen­etrating trauma to the great vessels. In: Glenn WWL,ed. Glenn's thoracic and cardiovascular surgery. VolII. Stamford: Appleton & Lange, 1997; 2213-2224.

Yamaguchi T, Guthaner DF, Wexler 1. Natural historyof the false channel of type A aortic dissection after

surgical repair: CT study. Radiology 1989; 170:743.

Zotz R], Erbel R, Meyer]. Noncommunicating intramu-

ral hematoma: An indication of developing aortic

dissection: ] Am Soc Echocardiog 1991; 4:636.

4:00 pm

Peripheral ArteriographyErnest j. Ring, MD

4:30 pm

Venous and Pulmonary Arterial Imaging

Steven C. Rose, MD

Learning objectives: From this presentation, attendeesshould be able to: (1) list the different sonographic tech­niques used to evaluate each of the three major venoussegments of the lower extremity (iliocaval, femoropopli­teal, and infrapopliteal) for deep venous thrombosis(DVT); (2) list the four fundamental items to evaluatesonographically in patients with lower extremity venousinsufficiency; (3) list the sonographic criteria to diag­nose occlusion of(a) the sonographically accessible tho­racic veins (aXillary and internal jugular veins), and(b) the sonographically inaccessible conduit veins (cen­tral subclavian, innominate veins and superior venacava [SVC)); (4) list six important anatomic variants ofthe inferior vena cava (IVC)/renal venous system, andthree variants of the SVc,· and (5) list three sonographicimaging alternatives to pulmonary angiography for di­agnoses ofpulmonary embolism.

BLOOD flow in normal lower extremity veins coursesfrom the periphery toward the right atrium and from thesubcutaneous superficial veins toward the deep venoussystem via perforating branches. Blood flow is largelyunidirectional because of the bicuspid valves locatedprincipally distal to the inguinal ligament. Because oflow intraluminal pressure (typically less than 15 mm Hgwhen supine), venous blood in the central portions ofboth the normal lower and upper extremity veins (eg,common femoral, axillary, or internal jugular veins) var­ies considerably with changes in intrathoracic and intra­abdominal pressure during respiration and right atrialpressure changes during the cardiac cycle. The pliantvenous walls and relatively low intraluminal pressurepermit ready distention with dependency or volumeoverload ("capacitance function") and compression with

external pressure.Important anatomic variants include duplication of

the popliteal, superficial femoral, and greater saphenousveins, and compression of the left common iliac vein bythe overlying right common iliac artery. The latter con­dition may lead to synechiae formation and/or thrombo­sis (May-Thurner Syndrome).

The most common indication for evaluation of lowerextremity veins is diagnosis of suspected acute DVT. His­torically, the diagnostic gold standard is ascending con­trast venography that was assumed to have an accuracyapproaching 100%. However, contrast venography en­

tails patient discomfort, has risks associated with contrastinjection (notably skin slough, induced thrombophlebi-