epidemic modeling with cellular automata · 2018-03-16 · epidemic modeling with cellular automata...
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Epidemic Modeling with Cellular Automata
Bernadetta Stachura-Terlecka, Antoni Ligeza
07.03.2018
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Outline1 State of the art
2 Classical CA in use
3 History
4 Epidemic as conflict
5 Representation for MultiLayered Cell
6 Movement phase
7 LCCA rule
8 Modeling results
9 Problems and future work
10 Bibliography
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State of the art
z Modeling of epidemy is both of theoretical interest and practicalimportance,
z There exists several mathematical models of epidemyz Cellular Automata can serve as a basig modeling tool for such
spatial phenomena,z There exists several models of epidemy with CA in the literatur,
but only classical CA are in use.
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Modeling used classical CA
Figure: HIV modeling used classical CA
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Epidemic modeling
z Daniel Bernoulli - first matematical epidemic model - Structure ofBernoulli’s model - the population is divided into susceptibles, i.e.those who have not yet been infected, and immunes, i.e. thosewho have been immunized for the rest of their life after oneinfection.
z SIS, SIR, SEIR, SIRSz Threshold analysis - The model is based solely on the user’s
response to the drug, and it is shown that when a certaincombination of susceptible population size, individualsusceptibility, and infectiousness does not exceed a criticalthreshold value, there will be only few users.
z Reed–Frost model - is one of the simplest stochastic epidemicmodels. It was formulated by Lowell Reed and Wade Frost in 1928and describes the evolution of an infection in generations.
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Epidemic as conflict
parties to the conflictz virus expansion,z treatment, vaccine.
Epidemic is a spatial conflict with two actors (virus and human).
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LCCA
Figure: Graphical presentation for MultiLayered Cell use in LCCA
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Movement phase
z Select a random cell (infected peoples) from the countries,z For each of the individuals in the cell, randomly select a
neighbouring cell and move the individual into it (cell not beingfull),
z Repeat from step one until all the cells in the area have beenaccounted for,
z In every cells where was infected peoples set virus and time for24h in Layer 1,
z Every infected man heve set time = 216h in Layer 2,z Compute contact infections,z Repeat from step four for the next cell until all cells have been
accounted for.
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Rule
Virus Layer:z live time (max 24h)z A type flu - Z(C1n) = xy ∗ ZarA ∗ W
z B type flu - Z(C1n) = xy ∗ ZarB ∗ W
Medicine Layer:z Spread of virus (max 216h)z A type flu - vactine person - C2n = 0, 6 ∗ Z(C1n)
z B type flu - vactine person - C2n = 0, 4 ∗ Z(C1n)
z resistant person - C2n = 0 ∗ Z(C1n)
z C2n = Z(C1n)
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Modeling results
Figure: Modeling results
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Problems and future work
Problems under investigation:z modeling the rules of propagation and interaction (x, y
parameters),z validation of the rules,z validation of the final results,z model tuning,
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Bibliographyz 1. J. D. Murray: Wprowadzenie do biomatematyki [Introduction to
Biomathematics], Wydawnictwo Naukowe PWN, 2006z Polish National Institute for Hygiene – NIPH:
http://wwwold.pzh.gov.pl/oldpage/epimeld/grypa/index.htmz B. Stachura-Terlecka, A. Ligeza: Modeling and analysis of spatial
conflicts with Layered Competitive Cellular Automata in:Multimedia Communications, Services and Security , 2015,
z S. Ching Fu, G. Milne: Epidemic Modelling Using CellularAutomata
z S. Yan, D. He, J. Luo, W.Chen, X. Yang, M. Wei, X. Kong, Y. Li, X.Feng and Z. Zeng: Simulation of HIV/AIDS distribution using GISbased cellular automata model
z F.C.Hoppensteadt, J.D.Murray: Threshold analysis of a drug useepidemic model
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