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PRAMEHA – AN APPROACH BASED ON PATHOPHYSIOLOGICAL MECHANISMS
Dr K P Yadunandanan MD AyMedical OfficerGovt Ayurveda HospitalChelembra, Malappuram Dist
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Our SpecialitiesFrozen ShoulderOA KneeTennis ElbowCTSLeg PainFeet NumbnessErectile Dysfunction
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Ayurveda & DiabetesDiabetes = PramehaNisakathakadi or
KathakakhadiradiNirooryadiMehari powdersPaneeyams “Ayurvedic = Herbal Hypoglycemic
Drug”“Lack of a proper understanding of
disease & treatment approach”
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Disease Approach based on Pathophysioloical mechanismsAnalysis of the disease
MechanismsNidana
Genetics, lifestyle, season, locality, occupation etc
Poorvaroopa & LakshanaSpecific to diseases
SamprapthiPathophysiological mechanisms
BedaStages
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Patient ApproachSystematic Approach in Patient Examination & Evaluation
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Evaluation of VayuPrana
◦ JeevanaUdana
◦BalaVyana
◦Dhatu Parinama & DhatugatiSamana
◦Aharapaka Apana
◦Nishkramana
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Evaluation of AgniJatara
◦Aharapaka & MalanirmanamRanjaka
◦Raktha NirmanamDhatwagni
◦Dhatuparinama & Kleda Nirmanam
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Evaluation of Dhatu SaraTwakRakthaMamsa & MedasAsthi Majja & SandhiSukra
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Evaluation of MalaPureesha
◦Consistency Moothra
◦Colour etcSwedaOther mala
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Evaluation of ManasSatwaRajaTamas
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Type 2 Diabetes Mellitus
“A Vascular disease more than a true metabolic disease”
Macroangiopathy◦Stroke, CAD, Leg Gangrene
Microangiopathy◦Retinopathy, Nephropathy,
Neuropathy
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EpidemologyDiabetic epidemic
Between 2003 and 2025, the number of patients worldwide with diabetes is going to increase 72% to an estimated 333 million people
Epidemological profiles and trends loaded against India
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Predisposing FactorsGenetic predispositionObesityLifestyleLack of exerciseEnvironmentalInflammation
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DIABETES – PATHOPHYSIOLOGICAL MECHANISMS
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Beta Cell DysfunctionBlunting of the beta-cell response
with loss of first phase insulin secretion
With time, there is overall loss of insulin secretion
A decrease in beta-cell mass and death of these beta cells
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Insulin Resistance
“Defect in ability of insulin to stimulate glucose transport into the cell”
ObesityFree Fatty AcidsInflammation
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Alterations in Lipid MetabolismEctopic deposition of Lipids –
lipotoxicity◦Pancreas, Liver, muscle, heart
muscleInsulin ResistanceReduced TG synthesisEnhanced lipolysisIncrease in Free fatty Acids
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InflammationLow grade tissue InflammationInflammatory markers raised in
obese personsInflammation to endothelial
dysfunctionResults in Insulin ResistanceProgression of Disease
◦Death of fat cells & Beta cells
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Diabetes MellitusMultifactorial complex
mechanisms◦Pancreatic Dysfunction◦Insulin Resistance◦Altered Lipid Metabolism◦Inflammation
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Chronic HyperglycemiaHyperglycaemic pseudohypoxiaOxidative stressActivation of the coagulation
cascadeInflammation
“Endothelial Dysfunction of arteries”
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Diabetic AngiopathyMacroangiopathy
◦Myocardial Infarction◦Stroke◦Leg gangrene
Microangiopathy◦Retinopathy◦End Stage Renal Failure◦Peripheral Neuropathy
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PRAMEHA
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BeejadushtiSahaja PramehaBeeja Avayava DushtiInherited Agni Vaigunya
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Kapha Krit Cha SarvamNavannaDadhiAnoopa MamsaAvyayamaAsuchitwamKapha Medo Moothra Vridhi
Physical & Mental Inertia
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AGNI & PRAMEHA
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Panchabuta AkashaVayuAgniApPrithvi
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Agnishomeeyam – Evolution of LifeLife – Dynamics
Of BhutasJala
◦ Dhatusara, OjasAgni
◦ Jatara, DhatwagniVayu
◦ Panchavayu
Vayu
JalaAgni
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Panchabuta & PramehaJalarogaJala Buta
Prasada – MalaA disease of Jaladushti progressing
to Dhatukshaya
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Agni & PramehaJataragni – Ahara Parinamam
◦Ahara to Rasa ◦Khara Mala - Pureesha
Dhatwagni – Dhatu Parinamam◦Annarasa to Dhatu Sara & Ojas ◦Drava Mala - Kleda – Moothra
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Agni & PramehaAgnidourbalyamAma formationSrotorodhaMedovridhiDhatwagnivaikalyaMedodushtiExcess KledaExcess Moothra formationDisintegration of DhatusMadhumeha
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Agnidourbalya & Ama – Beta cell Dysfunction & Insulin ResistanceAtyaharaAdhibojanaLack of proper exerciseAgnimandyamFormation of AmaMadhura Anna RasaSrotorodha
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Medovridhi - SthoulyamIncrease in Medas
◦Deposition of medas in abdomen,thighs
Dhatwagni VaikalyaFurthur increase in formation of
MedasSrotorodhaDhatwagni Dourbalya
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Medodushti - PrediabeticDhatwagnidourbalyaVridha Medas – Dushta MedasSthira Dhatu Sara – Shithila
DhatuDhatu Parinama VaikalyaProne to CAD, CVA
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Prameha – Vasthi RogaActivation of PithaDissolution of MedasFormation of excess Kleda Elimination of excess Kleda As
MoothraExcess formation of Moothra
‘Athimoothratha’
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Prameha – MadhumehaActivation Of Vyana & Apana
VayuWeakened Agni & Disintegration
Of Dhatu SaraDhatu & Ojas converted to KledaAvila Moothra - Increased
concentration of Urine‘Madhumeha – Asadhya’
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CLINICAL SCENARIO
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Agnimandya & Ama StageBeta cell stimulation & reducing
Insulin resistance Agnibalavardhana & Amahara
◦Panchakolam◦Indukantham
Eating Habit ModificationLife Style Modification
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Medoroga & Medodushti
Medohara & Atisthoulya ChikitsaDhatwagni Deepana
◦ Varadi Kashayam, Ayaskriti, Lodhrasavam◦ Loha Prayoga
Navayasam
◦ Mushta, Darvi, Thriphala, Devadaru◦ Vidangadi◦ Abyanga
Prabanjanam
◦ Udwarthanam Nalpamara, Eladi, Nisa
◦ Vyayama◦ Ahara Vihara
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Prameha - AtimehanaPitha SamanaKleda VishoshanaMoothra SthambanaMedo Doshahara & Agni DeepanaTiktha Rasa & Seetha Veerya
◦Ekanayakam◦Thettamparal◦Niroori◦Vanga & Abraka
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Moothra SthambanaKashaya
Nisakathakadi, KathakakhadiradiGulika
Nirooryadi, Swethagunjadi, Mehasamhari
Choornam◦Amritadi
Paneeya◦Ekanayaka Shadangam
‘Combine Medohara & Deepana Chikitsa’◦Chitrkathriphaladi, Navayasam
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Madhumeha - AvilamoothrathaAggressive Treatment Prevent progression to Vatha stagePitha Vatha samanam & intensive
SthambanaMedodoshahara &
MoothravisodhanaBalyam & RasayanamAgnideepanamVathasamanam
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AvilamoothramSilajathu Prayogam
◦RasayanamNisa & Amalaka in all stages
◦Nisa – Kledahara◦Amalaka – Rasayanam
Aveerabeejam◦Moothravisodhanam
Oushadham◦Akulyabdadi, Aakuyadi Gritam
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Prameha - AvilamoothramIntensive Sthambana ChikitsaDhara
◦Takradhara◦Special Dhara – 28 days
SirolepamMoordha Thailam
◦Nisoseeradi, ArukaladiAbyangam
◦Lakshadi KuzhambuPizhichil
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Krisha Meham Vata Pitha DoshaAgni VaishamyamStressSigns
◦Excess thirst & tiredness◦Karshyam
Sudden Progression to neuropathyVatha Pitha SamanamAgnideepanamMehahara & Balya
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Krisha Meham - ChikitsaOushadham
◦Indukantham, Nisakathakadi◦Nirooryadi◦Amritadi◦Abrakam
Dhara & SirolepaPizhichilRasayanam
◦Amritha, Amalaka
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PRAMEHA – COMPLICATIONS & ASSOCIATED SYMPTOMS
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Prameha – Complications in Skeletomuscular System Microangiopathy & tissue
ischemiaChronic Inflammation Excess Kleda & SodhaDathushosha & Disintegration of
Structures
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Approach – Management of Complications Aggressive Management of PramehaPrevent progressionEffective Management of ComplicationsKledahara & Sodha Hara
◦Kathakakadiradi with SilajathuVatha Samana
◦Rasnathamalakyadi, MaharasnadiBalyam
◦Bala, Amritha
“Treat Prameha effectively & complications will disappear”
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Frozen Shoulder ACKledahara & Sodhahara
◦Kathakakadiradi with ChandrapraVatha Samanam
◦Maharasnadi with Trayodasangaguggulu
Snehana◦Parinathakeree, Panchasneham
Balyam◦Ksheerabala Prayogam◦Navara
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Foot Abnormalities – Charcot footNeuropathic ArhtropathyManage PramehamVatha Samanam
◦Maharasnadi, GugguluBalyam
◦Navara
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Vascular NeuropathyPeripheral – DSPMicroangiopathy & Nerve
IschemiaNerve Damage – Arhtopathy
◦Pain, Numbness, muscle atrophyAutonomic
◦Urination problems◦Erectile Dysfunction
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Managemant - Vascular neuropathyAggressive Management of Prameha –
Vatha Pitha Meha◦ Nisakathakadi, Nirooryadi, Nisa & Amalaka◦ Amritadi
Dhara & SirolepaVatahara
◦ Rasnathamalakyadi, Ashtavargam,Balya & Rasayanam
◦ Bala – Ksheerabala◦ Amrita – Ksheeraguloochi◦ Abyangam & Pizhichil◦ Vasthi
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Vascular neuropathyAutonomic – ED Vata Pitha Meha Samana
◦Nisakathakadi, AmrithadiVrishya
◦Silajathu, Abrakam, Vangam◦Jeevaneeyaganam◦Pizhichil◦Vasthi
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Nephropathy - MadhimehamMadhumeha Chikitsa
◦Akulyabdadi Kashayam◦Silajathu
Rasayana – Saramabu Bavitha Silajathu
◦LajjaluSopha & Udara Chikitsa
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PAD & PramehaChronic Leg Pain
◦Macroangiopathy – Atherosclerosis of Tibial artery
◦Guggulutikthakam◦Rasasindooram◦Amrita & Guggulu◦Mahamanjishtadi◦Madhusnuhi
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PADLeg Gangrene
◦Guggulutikhakam◦Rasasindooram◦Mahamanjishtadi◦Amrita & Guggulu◦Madhusnuhi
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Hyperglycemia as symptom
Stroke◦Ama◦Amahara Chikitsa◦Shaddharanam yogam
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SummaryPrameha involves complicated
mechanisms which appear contradictory – Agnimandya, Ama, Medodushti, Dhatu Shaithilya & Ojakshaya.
Management requires understanding of different patho-physiology and treatment options should be adopted accordingly
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THANK YOU