chapter 30: how animals move
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Chapter 30: How animals Move. NEW AIM: What types of motor systems have evolved?. I. Ciliates and Flagellates. A. Unicellular organisms w/o skeletal-muscular systems. B. Protozoans (unicellular heterotrophic protists) and primitive algae. ciliate. flagellate. Chapter 30: How animals Move. - PowerPoint PPT PresentationTRANSCRIPT
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
I. Ciliates and FlagellatesA. Unicellular organisms w/o skeletal-muscular systemsB. Protozoans (unicellular heterotrophic protists) and primitive algae
flagellate ciliate
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
I. Ciliates and FlagellatesA. Unicellular organisms w/o skeletal-muscular systemsB. Protozoans (unicellular heterotrophic protists) and primitive algae
Fig. 4.18 (9 + 2 arrangement)
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
II. PseudopodiaA. Used by amoeba to moveB. Cell extensions (no microtubules)
pseudopod
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
IIB. Chemotaxis vs. Phototaxis
A. Chemotaxis – process by which a cell directs their movement depending on a chemical in the environment – taxis = to move (hence the word taxi).
Ex. 1. Movement of sperm towards the egg (egg secretes chemicals that sperm are attracted to);
2.Movement of macrophages to a site of bacterial infection (broken cells release a chemical attractant)
3. Movement of bacteria to a high concentration of glucose
These are all examples of positive chemotaxis (move towards the chemical)
There can also be negative chemotaxis (move away from the chemical).
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
IIB. Chemotaxis vs. Phototaxis
b. Phototaxis – process by which an entire organism directs their movement depending the stimulus of light (this is NOT a plant moving towards light, which is called phototropism.
Ex. 1. Algal cell moves toward light (positive phototaxis)
Mov
emen
tEx. 2. Moths or fruit flies attracted to light
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
III. Hydrostatic skeletonsA. Fluid held under pressure in a closed body compartmentB. Helps protect other body parts (cushion from shocks)C. Gives body shapeD. Gives support for muscle actionE. Cnidarians (hydra) and annelids (earthworms)
Fig. 18.7
Fig. 30.1
Earthworms crawl by peristalsisEach segment expands and contracts independently
(setae)
http://www.biology.ualberta.ca/facilities/multimedia/uploads/zoology/oligochaete.swf
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
III. Hydrostatic skeletonsA. Fluid held under pressure in a closed body compartmentB. Helps protect other body parts (cushion from shocks)C. Gives body shapeD. Gives support for muscle actionE. Cnidarians (hydra) and annelids (earthworms)
Hydrostatic skeleton of hydra in 2 stages
Fig. 30.2
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
IV. ExoskeletonA. Hard external skeleton covering all the muscles and organs of some invertebratesB. Made of chitin in Arthropods
C. Calcium carbonate in mollusks
D. Protection
Fig. 30.2E. Limits growth (periodic molting and deposition of new exoskeleton necessary)F. Muscles attach to inside of exoskeleton
JOINTED APPENDAGES
Chitin is a polysaccharide of N-acetylglucosamine (glucose with an acetyl group)
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonA. Hard or leathery support tissue situated AMONG the soft tissues of animals
B. Sponges (Porifera)
i. Reinforce by tough protein fibers or hard calium salts/silica called spicules
Calcium salts
A rigid sponges thanks to spicules
Spongin - fibrous protein
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonA. Hard or leathery support tissue situated AMONG the soft tissues of animals
C. Echinoderms (sea stars, sea urchins, etc…)
i. Have hard plates beneath skin (the spikes are not an exoskeleton)
Dead sea urchin (endoskeleton)Sea Urchin
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
i. Site of muscle attachment
- permits movement when muscle contracts bringing bones closer together
ii. Protection and overall support
- bones enclose vital organs
- Ex. Rib cage surrounds thoracic organs (heart and lungs)
- Ex. Skull and vertebral column surround brain and spinal cord
iii. Contains both cartilage and bone
- Both connective tissue
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
iv. cartilage
- firm, but flexible
- makes up skeletons of lower vertebrates (rays and sharks)
- principle component of embryonic skeletons in higher vertebrates
- NO blood vessels (avascular) or nerves
- takes a longer time to heal than bone
Fig. 30.2EAmphibian endoskeleton (cartilage in blue)
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
iv. cartilage
- articular surface of the bones (sites where 2 bones contact each other), the rib cage, the ear, the nose, the bronchial tubes/trachea and the intervertebral discs.
- Location
Cartilage in yellow
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
v. bone
- composed of calcium phosphate salts and strands of collagen protein- skeleton of mature higher vertebrates
- cells in bone
1. Osteoblasts - build bone (bbb)2. Osteoclasts - break down bone
You should know which hormones interact with each cell type…
- Osteoporosis - imbalance between osteoblasts and osteoclasts leading to weak bones
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
v. bone- spongy vs. compact bone- yellow marrow vs. red marrow
Fig. 30.5 Fig. 30.3
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
v. bone- highly vascular unlike cartilage
Fig. 30.5
Each osteon consists of concentric layers, or lamellae, of compact bone tissue that surround a central canal, the Haversian canal through which blood vessels and nerves run.
Lacuna = the region where osteocytes reside.
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
v. bone- highly vascular unlike cartilage
Fig. 30.5
Each osteon consists of concentric layers, or lamellae, of compact bone tissue that surround a central canal, the Haversian canal through which blood vessels and nerves run.
Lacuna = the region where osteocytes reside.
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
v. bone- connected at joints- can be movable (your elbow) or immovable (bones of the skull)
Fig. 30.3
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
V. EndoskeletonD. Vertebrates
v. bone- axial (blue - skull, ribs, vertebrae) vs. appendicular (yellow - appendages) skeleton
Fig. 30.3
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementA. The skeleton and muscles interact in movementB. Muscle system is an EFFECTOR of the nervous system
Fig. 30.7
D. Insertion of a muscle
i. Portion attached to bone that moves Insertion of bicep
Insertion of tricep
C. MUSCLES CAN ONLY CONTRACT (SHORTEN)
E. The ORIGIN is the attachment to the non-moving bone
Origin of bicep
Origin of tricep
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementE. Extensor
Fig. 30.7Extensor
i. Muscle that extends or straightens the bones at a jointEx. Tricep is an extensor - it contracts and straightens arm at elbow
F. Flexori. Muscle that bends a joint to an acute angle
Ex. Bicep is a flexor - it contracts and bends arm at elbow
Flexor
Bicep and Tricep are antagonistic muscles
ALL animals have pairs of antagonistic muscles
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementG. Tendons (dense connective tissue)
i. Connect muscles to bonesEx. Achilles tendon
Fig. 30.7
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementH. Ligaments
i. Connect bones to bones
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementI. Three types of muscles
i. smoothii. cardiac
iii. skeletal
- movement caused by CONTRACTION in ALL 3 types
- Contraction caused by sliding of actin and myosin filaments past each other inside cells…
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementI. Three types of muscles
i. Smooth muscle
a. involuntary muscles (autonomic NS) in arteries and veins, gastrointestinal tract, bladder, uterus
b. nonstriated- simply means that actin and myosin do not have clear organized arrays
c. smooth muscle cells connected by gap junctions in tissues (allow action potential to pass from one cell to next) - electrical synapsed. Single nucleus per cell
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementI. Three types of muscles
ii. Cardiac muscle
a. Single nucleus per cellb. striated
- actin and myosin have clear organized arrays
c. connected by gap junctions in tissues (allow action potential to pass from one cell to next) - electrical synapsed. Involuntary (autonomic NS)
Chapter 30: How animals Move
NEW AIM: What types of motor systems have evolved?
VI. Muscle contraction and movementI. Three types of muscles
iii. Skeletal muscle
a. Voluntary (intentional physical movement; somatic NS)
c. striated- actin and myosin have clear organized arrays
d. Stimulated by nerves at neuromuscular synapses
b. Muscle cell = single, large, multinucleated fiber
e. Action potential in cell stimulates calcium release into cytoplasm, which in turn causes contraction
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
VIII. Neuromuscular junction
Fig. 30.10
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
IX. How does a motor neuron make a muscle fiber contract?Fig. 30.10
1. Action potential (AP) reaches synaptic knob 2. Acetylcholine released into synaptic cleft3. Sodium moves through muscle fiber (just like a neuron)
4. AP travels along T-tubules (membranous tubules that fold in through cells) deep into the fiber
Neuromuscular junction video on website
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
IX. How does a motor neuron make a muscle fiber contract?
Fig. 30.10
1. Action potential (AP) reaches synaptic knob 2. Acetylcholine released into synaptic cleft3. Sodium moves through muscle fiber (just like a neuron)
4. AP travels along T-tubules (membranous tubules that fold in through cells) deep into the fiber
5. AP causes Ca++ to be released from sarcoplasmic reticulum (SR = ER) of fiber into cytoplasm
Muscle action potential video on website
Chapter 30: How animals Move
NEW AIM: How do muscle fibers contract?
VII. Muscle contractionA. Skeletal muscle
i. Muscle composed of bundles of fibers (cells)
- striation = alternating light and dark band of myofibrils
iii. Sarcomere - repeating unit of the myofibril (region b/w two Z lines) – you see sarco- you think muscle
ii. Muscle fibers (cells) contain numerous myofibril (contractile protein structures)
- thin filament: two strands of actin polymers and one strand of regulatory protein- thick filament: staggered array of multiple myosins
- dark band vs. light band
Fig. 30.8
Chapter 30: How animals Move
NEW AIM: How do muscle fibers contract?
VII. Muscle contractionB. Sliding-filament model
Fig. 30.9
Sarcomere contraction video on website
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
VII. Muscle contractionB. Sliding-filament model
Fig. 30.9
1. ATP binds to myosin head (causes detachment from actin)
2. ATP hydrolyzes to ADP and Pi- energy used ratchet back the head
- head is now in an unstable (high energy) state
3. Head binds to actin
4. ADP and Pi are released resulting in the power stroke
5. ATP binds, head releases, repeat again, but grab the next actin closer to Z-line
Sliding filament video on website
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
VII. Muscle contractionB. Sliding-filament model
Fig. 30.8
1. ATP binds to myosin head (causes detachment from actin)
2. ATP hydrolyzes to ADP and Pi- energy used ratchet back the head
- head is now in an unstable (high energy) state
3. Head binds to actin
4. ADP and Pi are released resulting in the power stroke
5. ATP binds, head releases, repeat again, but grab the next actin closer to Z-line
Aside: Rigor Mortis– when an animal dies, it becomes stiff (hence why we call dead people stiffs). This is because ATP is needed to release the myosin head from the actin filaments. No ATP, no release, muscle can’t relax.
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
IX. How does a motor neuron make a muscle fiber contract?
Fig. 30.10
6. Myosin binding sites on actin usually blocked by regulatory strand (troponin and tropomyosin)
7. Ca++ binds to part of regulatory strand (troponin) of thin filament, which causes tropomyosin to move off myosin binding site so myosin can bind.
Muscle action potential video on website
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
IX. How does a motor neuron make a muscle fiber contract?
Fig. 30.10
http://www.tvermilye.com/pmwiki/pmwiki.php?n=Animation.Video12
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
X. Vocabulary for a skeletal muscle cellA. Sarcolemma: plasma membrane
B. Sarcoplasmic reticulum (SR): endoplasmic reticulum
C. Sarcomere: single unit of the myofibril
D. Sarcoplasm: cytoplasm
Chapter 30: How animals Move
AIM: How do muscle fibers contract?
XI. MalfunctionsA. Arthritis
- inflammation of joints causing swelling and severe pain
- inflammation of tendon usually at site of attachment to bone caused by physical stress and irritation (common in athletes)
B. Tendonitis
- causes: autoimmune, infection (septic), gouty arthritis, etc…
EXTRAS
I. Heart Attack:A. Coronary Thrombosis
B. Angina pectoris- narrowing of coronary arteries resulting in an inadequate supply of blood (oxygen) to heart muscle and intense pain in chest/shoulder/arm (referred pain).- typically caused by arthrosclerosis
EXTRAS
II. oxyhemoglobinA. Hemoglobin with oxygen bound
A. Inflammation of bronchi, caused by bacteria, virus or other irritant (i.g. tobacco smoke)
III. Bronchitis
EXTRAS
IV. Asthma- chronic disease where airways constrict, become inflamed, and are lined with excessive mucus.- triggered by exposure to an allergen, tobacco smoke, cold or warm air, perfume, pet dander, moist air, exercise or exertion, emotional stress.
- genetic / environmental- allergic response
EXTRAS
IV. AsthmaAlbuterol (Salbutamol)
- typical used in inhalers- stimulates -2 adrenergic (adrenalin) receptors, which relaxes airway smooth muscle
EXTRAS
V. CO2 carried mostly in blood as bicarbonate (carbonic acid), which acts as a buffer in the blood (pH 7.4)
VI. Habits
- acquired by repetition, which established pathways for nerved impulse transmission, which permit rapid automatic responses to various stimuli.- performed without thinking- DO NOT confuse with habituation
EXTRAS
VII. Filament
EXTRAS
VIII. Photolysis
EXTRAS
VIII. Enzyme/Substrate relationship
EXTRAS
IX. Enzyme/Temperature and Enzyme/pH Relationship