2. shock on trauma.ppt

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    Shock on trauma

    Tatat A. Agustian

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    ObjectivesDefine shockRecognize the shock state

    Determine the causeApply treatment principlesApply principles of fluid managementMonitor patients response Employ options for vasculer accessRecognize complications of vascular access

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    Key Issues : Shock Management

    Recognize inadequate organ perfusionIdentify the cause Hemorrhagic vs nonhemorrhagic

    Treatment

    Stop the bleeding! Restore volume

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    Cardiac Physiology

    CO = SV X HR

    Preload Contractility Afterload

    Venous

    Capacitance

    dp / dtVascular

    Tone

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    Cardiac PhysiologyCO = stroke volume x heart rate

    Stroke volume is determined by: Preload

    Volume of venous return to the heart Venous capacitance , volume status , differencebetween mean venous systemic pressure and right

    atrial pressure. Myocardial contractility Starlings Law

    Afterload Systemic vascular resistance

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    Pathophysiology

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    Cellular Alteration in shock

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    Recognition of Shock State

    1. Tachycardia2. Vasoconstriction

    2. Cardiac outputNarrow pulse pressure 3. Map

    3. Blood Flow

    Caution : Compensatory mechanisms

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    Pitfalls in shock Recognition

    Extremes of ageAthletesPregnancyMedications

    Hematocrit/hemoglobin concentration

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    Etiology of Shock

    HemorrhagicMost commonClinical clues History & Physical

    examination Selected

    diagnostic tests

    NonhermorrhagicTension

    pneumothoraxCardiogenic

    NeurogenicSeptic

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    Hemorrhagic ShockLoss of circulating blood volume

    Normal blood volume

    Adult 7% of ideal weight Child: 9 % of ideal weight

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    Classification of HemorrhageClass I-IV

    Not absolute

    Only A clinical guideSubsequent treatment determined by

    patient response

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    Class I Hemorrhage

    750 mL BVL

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    Class II Hemorrhage

    750 1500 ml BVL

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    Class III Hemorrhage1500 2000 ml BVL

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    Class IV Hemorrhage : 2000 ml 2000 ML BVL

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    Fluid Shifts : Soft tissue Injury

    Blood loss intoinjury site

    Compoundsintravascular loss.

    Tissueedema

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    Assessment and ManagementRecognize shockStop the bleeding !

    Replenish intravascular volumeRestore organ perfusion

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    Assessment and Management

    Airway and Breathing Oxygenate and ventilate Pao > 80 mm hg (10,6 kpa)

    C irculation Assess

    C ontrol T reat

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    Assessment and Management

    Disability cerebral perfusionE xposure/ E nvironment

    Associated injuries Prevent hypothermia

    Gastric and bladder decompression

    Urinary output

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    Management : Vascular Access

    2 large caliber, peripheral IV sCentral access Femoral Jugular Subclavian

    IntraosseousObtain blood for croossmatch

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    Management : Fluid Therapy

    Warmed crystalloid solutionRapid fluid bolus ringer,s lactate Adult: 2 Liters, Ringers Lactate Child :20 ml /kg Ringers lactate

    Monitor response to initial therapy

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    Reevaluate Organ perfusion

    MonitorVital signsCNS statusSkin perfusionUrinary outputPulse oximetry

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    Resuscitation Evaluation

    Hourly Urinary Output Inadequate output suggests

    inadequate resuscitation

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    Acid Base Abnormalities

    Monitor with ABGsUsual etiology

    Adult : Acidosis due to inadequateperfusion

    Child : Acidosis due to inadequateventilation

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    Therapeutic Decisions

    Patient response determinessubsequent therapyHemodynamically normal vshemodynamically stable

    Recognize need to resuscitate inoperating room

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    Therapeutic Decisions

    Rapid Response

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    Therapeutic Decisions

    Transient Response20% -40% blood lossDeteriorates after initial fluidsSurgical consultation evaluation

    Continued fluid plus bloodContinued hemorrhage : Operation

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    Therapeutic DecisionsMinimal to No Response

    > 40% Blood loss

    No Response to fluid resuscitationImmediate surgical consultation

    Exclude nonhemorrhagic ShockImmediate operation

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    Volume Replacement

    Warmed fluids Crossmatched PRBCs

    Type specificType O, Rh negativeAutotransfusionCoagulopathy

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    Pitfalls

    Equating Bp withcardiac output

    Extremes of ageHypothermia

    AthletesPregnancy

    MedicationsPacemaker

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    Avoiding Complications

    Continued hemorrhageFluid overload

    Invasive monitoring (ICU) CVP Pulmonary artery catheter

    Other problems

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    Keys to Successful Treatment

    Early control of hemorrhageEuvolemiaContinuous reevaluation

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    Questions

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    Summary

    Restore organ perfusion Early recognition of the shock stateOxygenate and ventilateStop the bleedingRestore volume

    Continuous monitoring of responseAnticipate pitfalls