vertigo 1
TRANSCRIPT
Dr Zuraida ZainunMSc (Medical Audiology), MDMSc (Medical Audiology), MD
Senior lecturerSenior lecturerAudiology PrgrammeAudiology Prgramme
School of Helth SciencesSchool of Helth SciencesUniversiti Sains MalaysiaUniversiti Sains Malaysia
[email protected]://bal-exercise.blogspot.com/
Elicit history and evaluate dizziness
Understand vestibular testing
Knows differential diagnosis in dizziness
Understand management concepts
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‘’a disturbance that causes an individual to feel unsteady, giddy, woozy, or have a sensation of movement, spinning, or floating’’. http://www.nidcd.nih.gov/health/balance/balance_disorders.asp
Philip D. Sloane, MD, MPH; Remy R. Coeytaux, MD; Rainer S. Beck, MD; and John Dallara, MD Dizziness: State of the Science Ann Intern Med. 2001;134:823-832.
Dizziness subtype
Type of sensation
Temporal Characteristi
cs
Other Specification
Vertigo A feeling one that one or One’s surroundings are Moving (spinning)
Episodic vertigo (seconds to days) Continuous vertigo (most of the time for at least a week)
Characteristics, duration, and date of the first episode, length of episodes; and exacerbating factors.
Presyncope A lightheaded, faint feeling, as though one were about to pass out.
Typically occurs in episodes lasting seconds to hours.
1) Has syncope ever occurred during an episode2) Do episodes occur only when the patient is upright, or do they occur in other positions? 3) Are episodes associated with palpitations, medication meals, bathing, dyspnea, or chest discomfort?
Disequilibrium
Unsteadiness:- felt in lower limb- prominent when standing or walking- relieved by sitting or lying down
Usually present. Although it may fluctuate in intensity
Identify whether symptom occurs in isolation or accompanies another dizziness subtype; describe exacerbating factors.
Other dizziness; anxiety- related, ocular, tilting environment , other
A feeling not covered by the above definitions, may include swimming or floating sensations, vague lightheadedness, or feeling of dissociation.
Present all the time ~ days/weeks/years
-Is dizziness a/w anxiety or hyperventilation? - Was change in vision connected with dizziness onset? - Environment is tilting sideways (suggests an otolith problem?
http://www.aan.com/go/education/curricula/family/chapter5/section1
NatureNatureDurationDurationAssociated Associated symptomssymptomsPrecipitating Precipitating factorsfactors
OBJECTIVEOBJECTIVEVNGVNGVEMP (Ocul & Cer.)VEMP (Ocul & Cer.)V-HitV-HitEcohGEcohGPosturographyPosturographyRotating ChairRotating ChairSubjective vertical testSubjective vertical testSUBJECTIVESUBJECTIVEMalay Version VSSMalay Version VSSMalay version ModifiedMalay version Modified VSSVSS
Gen. exam.Gen. exam.Eye exam.Eye exam.Aural exam.Aural exam.Neurology Neurology exam.exam.Specific testSpecific test
*Chief complaints*Dizzy !! Lightheadacheness!! Headache!! Floating!!
Presyncope!!*Whirling !! Swaying!! Unsteadiness!!*True vertigo or not ? A) Nature*B) Duration of attack: BPPV-secondsTIA-minutesMeniere’s-hoursVestibular Neuronitis-Days
Ototoxins-years (See Hain, 1997) *C) Associated symptompositional related, hearing disturbance, headache, stress
D) Precipitating/ provoking factors
Spinning VestibularUnsteadiness Central lesionPresyncopal/ feeling faint Orthostatic Unspecific(dissociation) Psychology
•Otoconia exist within a part of the inner ear• crystals of calcium carbonate derived from a structure in the ear called the "utricle“
Duration of episode
Suggested diagnosis
Seconds Peripheral: unilateral loss of vestibular fx, late stage of acute vestibular neuronitis & MD
Seconds - minutes BPPV. perilymphatic fistula
Minutes – one hour
Posterior transient ischemic attack; perilymphatic fistula
Hours MD; perilymphatic; migraine. Acoustic neuroma
Days Early acute vestibular neuronitis*’stroke; migraine; Multiple sclerosis
Weeks Psychogenic (constant ~weeks w/o Improvement) *-Early acute vestibular neuritis can be two days or as long as one week or
more .
Symptom Suggested diagnosis
Aural fullness Acoustic neuroma;Meniere’s disease
Ear or mastoid pain
Acoustic neuroma; acute middle ear disease (e.g; otitis zoster oticus)
Facial weakness Acoustic neuroma; herpes zoster oticus
Facial neurologic
CPA tumour; CVA; MS
Headache Acoustic neuroma; migraine
Hearing loss MD; PLF; acoustic neuroma; cholesteatoma;otosclerosis;TIA or stroke involving anterior cerebella artery, herpes zoster oticus
Imbalance Acute vestibular neuronitis(usually moderate); CPA tumor (usually severe)
Nystagmus Peripheral or central vertigo
Phonophobia,photophobia
Migraine
Tinnitus Acute labyrinthitis; acoustic neuroma; Meniera’s disease
Provoking Factor Suggested diagnosis
Changes in head position
Acute labyrinthitis;BPPV; CPA Tumour ;multiple sclerosis (MS); PLF
Spontaneous episodes
AVN; CVA (stroke or TIA; MD ; migraine; MS
Recent URTI Acute vestibular neuronitis (AVN)
Stress Psychiatric or psychological causes; migraine
Changes in ear press., trauma, excess. straining, loud noises
Perilymphatic fistula (PLF)
Past medical history-vascular risk factors-ear surgery
Family History-Similar disorder ? -Migraine
Drug History-present and past exposures to ototoxins, antihypertensives.
Clinical Examination Clinical Examination
Aural Examination
otitis mediaear wax, perforated ear drumcholesteatoma
Eye ExaminationVisual acuityNystagmus
-saccadic, vestibular, pendular, congenital, alternating
Rebound nystagmus Saccades, pursuit,vergence, gaze
General Medical conditionBlood pressure (lying andsitting)Cardiac arrhythmias
Neurological Examinationcranial nerve palsies (Multiple sclerosis , acoustic neuroma, advanced brain stem tumor or basilar artery insufficiency
Neck examination
*Gait
*Cranial nerves
*Motor power and reflexes (e.g. Babinski)
*Sensory (proprioception)
Cerebellar sign ;a) Finger to noseb) Dysdiadokinesiac) Tandem gait (hell
to toe) with eye open and closed
*Romberg’s test Fall to one side:
- Posterior column lesion- Acute ipsilateral vestibular lesion
*Fukuda @ Unterberger test-Walk on the spot for 2 minutes with eye closed-Positive when patient turn > 45° -Ipsilateral peripheral lesion
l) Spontaneous nystagmus
MD, Vestibular Neuronitis, central disorders, to rule out Psychiatric (used Frenzel's goggles)
ii) Range of eye movements
Gaze paresis
Ocular paresis
iii) Cover test for strabismus : a deviation or misalignment eyes. strabism– eye muscle position ~ one or both
eyes may turn in (esotropia), out (exotropia), up
(hypertropia) or down (hypotropia). http://dewa-dony.blogspot.com/2008/10/strabismus.html
- to detect vestibular neuritis, acoustics, and to rule out psychiatric disturbance
Head-shake test - (Hain et al, 1987)75% sensitive but wrong side in 1/4 of the time.
Head Thrust test
http://cueflash.com/decks/CONTROL_OF_EYE_MOVEMENTS_-_57
Saccade when head turning toward lesion side
- to detect ototoxicity and other bilateral vestibulopathies
Dynamic illegible 'E' test or DIE
(Longridge, 87).
1. DIX-HALLPIKE TEST
-Rotatory upbeating; Post SCC
-Rotatory downbeating; Ant. SCC
video 1
video 2
video 3 cupulo
Treatment for Post. SCC- Epley’smenourve 2. ROLL TEST - horizontal nystagmus video 1
Treatment- Barbeque menourve
Video 1nystagmus
3) Fistula Test or Valsalva test- Occasionally helpful
4) Hyperventilation test – 30 seconds, look for nystagmus. Helpful when nystagmus changesdirection compared to vibration or head-shakingnystagmus.
5) Carotid Sinus Compression - for syncope patients.
6) Vertebral artery test - for persons with neck-
position induced vertigo (cervical vertigo).
Feature Peripheral Vertigo Central Vertigo
Nystagmus Mix horizontal & tensional;inhib. by fixation of eyes;Fades after a few days; notchange direction with gazeto either side
Purely vertical , horizontal, ortorsional; not inhibited byfixation of eyes ; last weeksto months; change directionWith gaze towards fast phaseOf Nystagmus
Imbalance Mild to moderate; able towalk
Severe; unable to stand orwalk
Nausea, vomiting
May be severe Varies
Hearing loss,tinnitus
Common Rare
NeurologicSx
Rare Common
Latency(follow. pro-vocative)
Longer (up to 20 seconds) Shorter (up to 5 seconds)
Indication; Assess vestibular function Locate the lesion organ/part Causative factor/etiology Vestibular rehabilitation assessment
*Videonystagmography (VNG)/Electronystagmograpy (ENG)
*Video Head impulsetTest (V-HIT)
*Vestibular evoked myogenic potential (VEMP) - Ocular & cervical
*Electrocochleargraphy (EcohG)
*Rotating chair
*Computerized Posturography (CDP)
*Subjective vertical test
*VNG – Horizontal SCC
*Rotatory Chair – Horizontal SCC
file:///F:/LECTURE%202007/posturography/Posturography.htmfile:///F:/LECTURE%202007/posturography/Posturography.htm
*Evaluation of the inner ear (cochlea) has an excessive amount of fluid pressure.
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Iran Audiology Congress 26-28 May 2011 Dr Iran Audiology Congress 26-28 May 2011 Dr Zuraida ZainunZuraida Zainun
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http://www.unmc.edu/physiology/Mann/mann9.html
Video nystagmography (VNG)Video Head impulsetTest (V-HIT)
Ocular VEMP
Cervical VEMP
Rotating chair
Bone-conducted cVEMP
*Others investigations
*Audiological test* PTA* Tympanometry* ABR
*Radiological test* CT Scan*MRI Scan* Vascular studies
*Laboratory investigations:* FBC* Blood sugar* Lipid profile* Thyroid profile
*MALAY VERSION VERTIGO SYMPTOM SCALE QUESTIONNAIRES (MVSS) ~ 22 questions (34 items)
*MALAY VERSION MODIFIED VERTIGO SYMPTOM SCALE QUESTIONNAIRES (MMVVSS) ~ 14 items
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1. Investigation and diagnosis2. Explanation3. Rehabilitation plan
- correction of remediable problems-General medical condition
- general fitness programmed- physical exercise regimens (i.e. Vestibular rehabilitation by
physiotherapist/ homebased)
Cawthorne cookseey exercise (CCE) Customised CCE Epley’s Menourve Brandt Daroff exercise
- psychological assessment -Psychological intervention i.e. CBT, Relaxation Rx.
- medication- realistic family/social/occupational goals
- surgery
4. Monitoring/feedback/follow up
5. Discharge
Reproduced with permission from Luxon LM, Davies RA, eds. Handbook of vestibular medicine. London: Whurr Publishers, 1997.
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Cawthorne cookseey exercise (CCE)
Customised CCE
Bal Ex : Homebased video module for balance exercises = customised CCE +Prayer movement
1. Kroenke K, Lucas CA, Rosenberg ML, et al. Causes of persistent dizziness: a prospective study of 100 patients in ambulatory care. Ann Intern Med. 1992;117:898-904.
2. LM Nashner, FO Black, and C Wall, 3d Adaptation to altered support and visual conditions during stance: patients with vestibular deficits J. Neurosci. 1982 2: 536-544.
3. Shupert CL, Black FO, Horak FB & Nashner LM (1988) Coordination of head and body in response to support surface translations in normals and patients with bilaterally reduced vestibular function. In Amblard B, Berthoz A, Clarac F (eds) Posture and gait: Development, Adaptation and Modulation. New York: Elsevier Science Publishers.
4. Allum, J.H.J., Honegger, F. and Pfaltz, C.R. (1989) The role of stretch and vestibulo-spinal reflexes in the generation of human equilibriating reactions. Progress in Brain Research 80, 399-409
5. Bles W, de Jong JMBV. Uni- and bilateral loss of vestibular function. In: Disorders of posture and gait.—Bles W, Brandt T, eds. (1986) Amsterdam: Elsevier, 1986, PP 127-139
6. Fregly AR (1974) Vestibular ataxia and its measurement in man. In: Kornhuber HH (ed) Handbook of Sensory Physiology,. vol VI. Springer, New York, pp 321–360
7. Handbook of Balance Function Testing by Gary P. Jacobson
(Author), Craig W. Newman (Author), Jack M. Kartush Singular; 1 edition (October 1, 1997)
8. http://www.neuroanatomy.wisc.edu/virtualbrain/BrainStem/13VNAN.html
9. http://www.utmb.edu/otoref/Grnds/Vestibular-2004-0414/Vestibular-2004-0414.htm
10. http://www.bcdecker.com/SampleOfChapter/1550092634.pdf