vascular assessment.pdf
TRANSCRIPT
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Vascular Assessment
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Normal Physiology of theCardiovascular System (CVS)
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Heart contracts approximately every 0.8s
Divided into 2 phases
Cardiac output is about 5 litres
Myocardium has the ability to contractwithout nerve impulses. Myogenicity.
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The most important of these is the
sinoatrial (SA) node, situated in the rightatrium.
according to demands placed upon it.
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Sympathetic nerve acts on the heart via
beta-receptors.
Increasing stoke volume and heart rate.
arasympat e c vagus nerve s ows eartrate
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Overall purpose of all vessels is to deliver
blood with its nutrients etc and to removewaste products.
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Capillary Function
Exchange of substances
Pressure drop
Capacitance vessels
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Arterial, Venous and CapillaryStructure
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Assessment of the Lower Limb. In order to maintain tissue integrity and
viability, it is essential that blood flow tothe lower limbs is maintained and
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It is important that the Podiatrist is aware
of what to look for in a healthy vascularsystem, and is clear regarding what
relation to blood supply
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Beginning the assessment Initially, you will have made sure you have
collected information regarding patientdetails, including medical and surgical
, ,and also an indication of any relevantfamily history, such as ischaemic heart
disease, hypertension, diabetes, stroke, etc.
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What indications would encourage you to
perform a vascular assessment on yourpatient?
A good approach to vascular assessmentis to follow a simple structure, so that no
aspect is missed,
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One thing often forgotten whencarrying out any clinical examination isthat you should use the information
collected to make a diagnosis. Withoutthis, your examination is meaningless.Additionally, should you become
nvo ve n t gat on, t e ac o a diagnosis may impair your defence asit could be implied that you were
unable to interpret the resultsof your tests.
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A further point to note aboutassessment is that no single test resultor observation should be used to
formulate your diagnosis. Instead, youshould be able to combine the results
working diagnosis, on which you thenbase your management plan, or yourreferral to another
health professional.
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Arterial Insufficiency Pain
Pallor
Pulselessness
Paraesthesia Paralysis
Perishing cold.
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Causes Thrombosis,
Embolus,
Aneurysms,
Oedema Transient ischaemia
Chronic ischaemia
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Arterial in-sufficiency is usually associated
with pain Site
ature Duration
Aggravating Factors
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This stage can be recognised by mottling,
muscle tenderness, motor or sensorydeficit and necrosis.
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Intermittent claudication
Night cramps
Rest pain
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Observations. Skin may appear
Pink
White
Blue Red
Dusky pink.
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Tissue Viability A poor blood supply will mean that the skin
and other soft tissues will be receivinginadequate nourishment
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Clinical Tests Temperature gradient
Sub-papillary plexus filling time (CRT)
Buergers test
Allens test
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Pedal Pulses
Posterior Tibial
DorsalisPedis
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Peroneal
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Anterior Tibial
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Doppler Ultrasound Allows pulses to be heard more clearly
Sound waves bounce of objects (usuallyred blood cells)
an a so e e to a pr nter
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ABPI This was first described by Yao in the
1960s Calculated by recording the systolic
posterior tibial artery (ankle), using thefollowing method;
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Lay patient flat for 10 mins
Measure systolic brachial pressures (L+R)
Measure systolic pressures in PT
Measure systolic pressure in DP Divide the highest pressure on left foot
with highest overall
brachial pressure
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Venous Insufficiency Arises in the superficial and/or deep veins
Affects the return of blood to the heart
Pooling of blood in the ankles
Exacerbated by gravity More evident around the malleoli
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Past History Venous problems? DVT Pregnancy?
Varicose veins Familial
Telangiectases around the medial malleoli
can n cate poor ra nage Mottled cyanosis to lower third of limb
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Symptoms Varicose related pain
Ankle oedema
Alleviated by elevation
Varicosities- especially on standing Unilateral PVD
Bilateral - CHF
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Temperature In venous insufficiency the skin often feels
warm, which suggests that the arterialsupply is satisfactory. However, it should
result in inflam-mation of the veins(phlebitis) due to the presence of thethrombosis
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Varicose Veins Presence of these may be due to valvular
incompetence in the superficial division ofthe venous system in isolation, or in
created by back-pressure due to anobstruction in the veins.
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Tissue Viability Poor venous return results in the
accumulation of waste products andtherefore the viability of the surrounding
lipodermatosclerosis may develop
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Oedema Associated with venous problems
Hydrostatic pressure causes leakage into
t e t ssues
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Leg Shape Patients with chronic venous ulceration
and oedema may develop characteristic'champagne legs',
' ',this is characteristic oflipodermatosclerosis.
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Pulling all the informationtogether.
Once the vascular assessment is
complete, the next step is to decide on amanagement plan suitable for the patient.
results of the assessment may indicatethat it is better to refer the patient to aspecialist for
further investigation.
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