trauma from occlusion - subharti dental collegedental.subharti.org/periodonotics/tfo.pdf · 2019....
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TRAUMA FROM OCCLUSION
Dr. Amit Wadhawan
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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ADAPTIVE CAPACITY OF THE PERIODONTIUM TO OCCLUSAL
FORCES • Whenever a force is exerted on the crown of
a tooth the periodontium attempts to accommodate it.
• Varies in different persons and in the same person at different times.
• The effect depends on the magnitude, direction, duration and frequency of the forces.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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3 Dr. Amit Wadhawan, Subharti Dental College, SVSU
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TFO • When occlusal forces exceed the
adaptive capacity of the tissues, tissue injury results. The resultant injury is termed trauma from occlusion.
• TFO refers to the tissue injury, not the occlusal force.
• An occlusion that produces such injury is called a traumatic occlusion.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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ACUTE AND CHRONIC TRAUMA
• Acute: results from an abrupt occlusal impact, e.g. biting on a hard object.
• Chronic: develops from gradual changes in occlusion produced by wear, drifting movement and extrusion of teeth along with parafunctional habits such as bruxism and clenching.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• The criterion that determines if an occlusion is traumatic is whether it produces periodontal injury, not how the teeth occlude.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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PRIMARY & SECONDARY TFO
• When TFO is the result of alterations in occlusal forces it is called primary TFO.
• When it results from reduced ability of the tissues to resist the occlusal forces it is known as secondary TFO.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Primary TFO
• Only alteration to which a tooth is subjected is occlusion. – insertion of a high filling
– insertion of a prosthetic replacement that creates excessive forces on abutment and antagonistic teeth
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Secondary TFO
• Occurs when adaptive capacity of tissues to withstand occlusal forces is impaired by bone loss.
• Previously well tolerated forces become traumatic.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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STAGES OF TISSUE RESPONSE TO INCREASED OCCLUSAL
FORCES
• STAGE I: Injury
• STAGE II: Repair
• STAGE III: Adaptive remodeling of the periodontium
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Stage I: Injury
• Tissue injury is produced by excessive occlusal forces.
• The body attempts to repair the injury and restore the periodontium.
• This can occur if the forces are diminished or if the tooth drifts away from them.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• Chronic force – periodontium is remodeled to cushion its impact.
• Ligament is widened at the expense of bone, resulting in angular bone defects without periodontal pockets, and the tooth becomes loose.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• Axis of rotation / fulcrum
• Single rooted teeth – located in the junction between the middle third and the apical third of the clinical root.
• Forces of occlusion create areas of pressure and tension on opposite sides of the fulcrum.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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14 Dr. Amit Wadhawan, Subharti Dental College, SVSU
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15 Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• Areas of periodontium most susceptible to injury from excessive occlusal forces are :
furcation areas
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• Slightly excessive pressure : – stimulates resorption of alv. bone
– Widening of pdl lig. space
• Slightly excessive tension : – Elongation of pdl lig. Fibers
– Apposition of alv. bone
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• Areas of increased pressure – blood vessels are numerous and reduced in size.
• Areas of increased tension – blood vessels are enlarged.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• GREATER PRESSURE
• Compression of fibers – areas of hyalinization
• Subsequent injury – necrosis of areas of ligament.
• Vascular changes – within 30 min- impairment and stasis of blood flow
• at 2-3 hrs blood vessels packed with erythocytes, which start to fragment
• between 1-7 days disintegration of blood vessel walls
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• SEVERE TENSION
• Widening of periodontal ligament
• Thrombosis
• Hemorrhage
• Tearing of pdl lig.
• Resorption of alv. bone
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• Pressure severe enough to force the root against the bone causes necrosis of the pdl lig and bone.
• The bone is resorbed from viable pdl lig adjacent to necrotic areas and from marrow spaces - undermining resorption.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Stage II: Repair
• Repair is constantly occurring in the normal periodontium and TFO stimulates reparative activity.
• When bone is resorbed by excessive occlusal forces , body attempts to reinforce the thinned bony trabeculae with new bone : buttressing bone formation.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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TYPES OF BUTTRESSING
• Central buttressing – occurs within the jaw
• Peripheral buttressing – on the bone surface
• Peripheral buttressing produces a shelf like thickening on the alv margin termed as lipping.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Stage III: Adaptive Remodeling
• If the repair process cannot keep pace with the destruction, the periodontium is remodeled in an effort to create a structural relationship in which the forces are no longer injurious to the tissues.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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• This results in a thickened pdl lig , which is funnel shaped at the crest, and angular defects in the bone with no pocket formation.
• The involved teeth become loose.
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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STAGES
• Injury phase – increase in areas of resorption and decrease in bone formation
• Repair phase – decreased resorption and increased bone formation
• Adaptive remodeling – resorption and formation return to normal
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Insufficient Occlusal Force
• Thinning of pdl lig.
• Atrophy of the fibers
• Osteoporosis of alv. Bone
• Reduction in bone height
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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CAUSES
• Open-bite relationship
• Absence of functional antagonists
• Unilateral chewing habits
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Reversibility of traumatic lesions
• TFO is reversible
• Injurious force must be removed for repair to occur
• Persistent trauma and inflammation impair the reversibility of traumatic lesions
Dr. Amit Wadhawan, Subharti Dental College, SVSU
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Influence of TFO on progression of marginal
periodontitis
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HISTORICAL BACKGROUND
• 1901: Karolyi postulated that interaction may exist b/w TFO & alveolar pyrrohea.
• Box (1935) & Stones (1938) reported experiments in sheep & monkeys.
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CONCEPTS
• GLICKMAN CONCEPT (1965)
• WAERHAUG CONCEPT (1979)
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GLICKMAN CONCEPT
• Progressive tissue destruction of the periodontium at a "traumatized tooth" will be different from that characterizing a "non-traumatized" tooth.
• Such teeth develop angular bony defects & infrabony pockets.
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ZONES OF PERIODONTIUM
• Zone of irritation : marginal & interdental gingiva.
• Zone of co-destruction : periodontal ligament, the root cementum and the alveolar bone
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• The spread of an inflammatory lesion from the zone of irritation directly down into the periodontal ligament (i.e. not via the interdental bone) may hereby be facilitated
• This alteration of the "normal“ pathway of spread of the plaque-associated inflammatory lesion results in the development of angular bony defects.
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WAERHAUG’S CONCEPT
• Examined autopsy specimens.
• Concluded that angular bony defects and infrabony pockets occur equally at periodontal sites of teeth which are not affected by TFO as in traumatized teeth.
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CONCLUSIONS
• TFO cannot cause pocket formation.
• Causes bone resorption – s/be regarded as a physiological adaptation to the forces.
• In teeth with periodontitis, TFO may enhance the rate of progression of the disease, i.e. act as a co-factor in the destructive process.
39 Dr. Amit Wadhawan, Subharti Dental College, SVSU