toothwear

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Case 60 Toothwear SUMMARY A 35-year-old policeman presents having noticed that his anterior teeth are becoming shorter. Identify the cause and outline options for management. Examination Extraoral examination The patient is a fit-looking man and slightly overweight. No submandibular or cervical lymph nodes are palpable. The temporomandibular joints appear normal and there is no evidence of hypertrophy of the masseter muscles. Intraoral examination The appearance of the anterior teeth is shown in Figure 60.1. What do you see? The palatal surfaces and incisal edges of the upper incisor teeth are worn. The wear involves the enamel and dentine but not the pulp. The palatal surfaces of the teeth appear smooth and unstained. The incisal edges are rough, small chips of unsupported labial enamel having fractured away. If you were able to examine the patient, you would find that some of the upper and lower anterior teeth do not contact each other in the retruded contact and the intercuspal posi- tions. All other teeth appear normal and the palatal surfaces of the upper posterior teeth are unaffected. What does this appearance signify? This is toothwear, the loss of dental tissues through the processes of erosion, attrition and abrasion. Although each process may act alone, significant toothwear is usually the result of a combination of these processes and erosion is often dominant. The smooth surfaces suggest that erosion is a factor in this case and the distribution of enamel loss suggests that regurgitation of gastric acid may be the cause. Dietary acids are usually associated with erosion on the buccal or labial surfaces of the upper anterior teeth but if the patient rinses or swills acidic drinks in the palatal vault prior to swallowing, the pattern of erosion is very similar to that seen when gastric acid is regurgitated. Either source of acid might be the cause. Define erosion, abrasion and attrition. Erosion is the chemical dissolution of teeth by acids. Attrition is the wear of tooth against tooth. Mild degrees of attrition are normal. Abrasion is the wear of teeth by physical means other than the teeth. Differential diagnosis What is your differential diagnosis for this patient? 1. Dental erosion caused by gastric acid combined with attrition 2. Dental erosion caused by dietary acids combined with attrition 3. Attrition alone 4. Industrial erosion. History Complaint The patient has become increasingly aware of his shorten- ing front teeth. He is not greatly concerned about the appearance but feels that continued wear will eventually destroy the teeth completely. History of complaint He has noticed that his teeth have become worse over the last 3–5 years but cannot remember when he first noticed the signs. The patient has always attended a dentist regu- larly and has relatively few, small restorations and good oral hygiene. Medical history The patient is generally fit and well. He drinks about 10–20 units of alcohol each week. Fig. 60.1 Palatal view of the upper anterior teeth.

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  • GCase 60toothwear

    SUMMARYA 35-year-old policeman presents having noticed that his anterior teeth are becoming shorter. Identify the cause and outline options for management.

    Examination

    Extraoral examinationThe patient is a fit-looking man and slightly overweight. No submandibular or cervical lymph nodes are palpable. The temporomandibular joints appear normal and there is no evidence of hypertrophy of the masseter muscles.

    Intraoral examination

    The appearance of the anterior teeth is shown inFigure 60.1. What do you see?

    Thepalatalsurfacesandincisaledgesoftheupperincisorteethareworn.Thewearinvolvestheenamelanddentinebutnotthepulp.Thepalatalsurfacesoftheteethappearsmoothandunstained.Theincisaledgesarerough,smallchipsofunsupportedlabialenamelhavingfracturedaway.

    If you were able to examine the patient, you would find that some of the upper and lower anterior teeth do not contact each other in the retruded contact and the intercuspal posi-tions. All other teeth appear normal and the palatal surfaces of the upper posterior teeth are unaffected.

    What does this appearance signify?

    Thisistoothwear,thelossofdentaltissuesthroughtheprocessesoferosion,attritionandabrasion.Althougheachprocessmayactalone,significanttoothwearisusuallytheresultofacombinationoftheseprocessesanderosionisoftendominant.

    Thesmoothsurfacessuggestthaterosionisafactorinthiscaseandthedistributionofenamellosssuggeststhatregurgitationofgastricacidmaybethecause.Dietaryacidsareusuallyassociatedwitherosiononthebuccalorlabialsurfacesoftheupperanteriorteethbutifthepatientrinsesorswillsacidicdrinksinthepalatalvaultpriortoswallowing,thepatternoferosionisverysimilartothatseenwhengastricacidisregurgitated.Eithersourceofacidmightbethecause.

    Define erosion, abrasion and attrition.

    Erosionisthechemicaldissolutionofteethbyacids.

    Attritionisthewearoftoothagainsttooth.Milddegreesofattritionarenormal.

    Abrasionisthewearofteethbyphysicalmeansotherthantheteeth.

    Differential diagnosis

    What is your differential diagnosis for this patient?

    1. Dentalerosioncausedbygastricacidcombinedwithattrition

    2. Dentalerosioncausedbydietaryacidscombinedwithattrition

    3. Attritionalone4. Industrialerosion.

    History

    ComplaintThe patient has become increasingly aware of his shorten-ing front teeth. He is not greatly concerned about the appearance but feels that continued wear will eventually destroy the teeth completely.

    History of complaintHe has noticed that his teeth have become worse over the last 35 years but cannot remember when he first noticed the signs. The patient has always attended a dentist regu-larly and has relatively few, small restorations and good oral hygiene.

    Medical historyThe patient is generally fit and well. He drinks about 1020 units of alcohol each week.

    Fig. 60.1 Palatalviewoftheupperanteriorteeth.

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    Investigations

    What investigations would you perform?

    Athoroughdietaryrecordshouldbetakenbythepatienttodeterminethetrueconsumptionofacidicfoodsanddrinks.Dietanalysissheetsneedtobefilledinfor4ormoredays,includingaweekend,anditisemphasizedthatthepatientshouldwritedownallthefoodsanddrinkstakenoverthattime,includingbetween-mealsnacks.Bothfrequencyandamountneedtobenotedandthepatientshouldbespecificallytoldtonotesuspectfoodssuchascarbonateddrinks,citrusfruitsanddrinks,vinegarandwhitewine,toensurethatnoneismissed.

    Studycastsofthepatientsteethshouldbetaken.Thesewillprovideabaselinerecordagainstwhichprogressionoferosioncanbedetected.Testsoftoothvitalityandintraoralradiographsmightbetakenifitissuspectedthatthewearhascompromisedthevitalityofthepulpinanyteeth.

    Diet analysis confirms the patients statement that he has a low consumption of acidic food and drink. This excludes dietary acid as a cause, leaving gastric acid as the only other source. Regurgitation erosion occurs when the stomach juice passes from the stomach into the mouth. The pH of stomach juice is around 1 or 2, and if regurgitation occurs frequently the damage to teeth can be catastrophic.

    Further differential diagnosis

    How might gastric acid enter the mouth?

    1. Gastro-oesophagealrefluxdisease2. Eatingdisorders3. Chronicalcoholism4. Rumination.

    What features of these conditions might aid definitive diagnosis?

    Gastro-oesophageal reflux diseaseisusuallyassociatedwithheartburn(intermittentretrosternalpainradiatingalongtheoesophagus,worsenedbylyingdownorarecentlargemeal),orepigastricpain(centredoverthexiphisternum).Whensymptomsarerelatedtomeals,thetermdyspepsiaissometimesused.Inmostpatientssymptomsofgastro-oesophagealrefluxareself-limitingandlittleornoacidentersthemouth.Inotherscompleteregurgitationintothemouthisfrequent,painbecomespersistentandpatientsseekmedicaladvice.Asmallproportionofpatientstreattheirpainwithover-the-counterantacidsandareunawareofthepotentialfordamagetotheirteethoroesophagus.Ahistoryoftakingantacidpreparationsisausefulindicatorfortheactivityofgastro-oesophagealrefluxdisease,andthispatienthasalreadyindicatedthathehasnoticedsomeregurgitation.Thisisthemostlikelycause.

    Eating disordersareacauseoferosioninyoungerpatients.Bothanorexianervosaandbulimianervosatendtoaffectyoung,adolescent,intelligentfemaleswithahistoryofoverprotectiveparents.Anorexiaisself-destructive.Suffererslosebodyweightbystarvingthemselvesand/orvomitingto

    Which of these causes would you exclude? Explain why.

    Attritionasasinglefactorismostunlikelytobethecause,becausesurfacesoftheteethdonotcontactintheintercuspalorretrudedcontactposition.Itwouldalsobemostunlikelythatocclusalwearcouldaffectthewholeofthepalatalsurfacesofallincisorsequally.Attritionisoftenassociatedwithmarkedbruxismbutthereisnoevidenceofmasseterichypertrophyonexamination.

    Industrial erosionisnowveryuncommon.Acidpresentintheairoftheworkingenvironmentcausesdentalerosionbutimprovementsinhealthandsafetyatworkhavealmosteradicatedthiscondition.Carbatteryacidworkersusedtosuffererosion,particularlyaffectingthebuccalsurfacesoftheteeth.Thepatientsprofessionaspolicemanmeansheisunlikelytobeexposedtoanacidicenvironmentthoughsomepatientsmaybeexposedtovolatileacidsthroughhobbyactivities.

    What specific questions would you ask? Explain why.

    Toothwearisoftenmultifactorialandthepatientmustbequestionedaboutallcauses.

    Do you suffer acid regurgitation from the stomach?Regurgitationofstomachacidcanbenoticedbythepatientbecauseofthetaste.However,itmaybeunnoticedifithappensatnight,andmayormaynotbeassociatedwithsymptomsofgastricdisease.Occasionalmildrefluxintotheoesophagusorpharynxisrelativelycommon.

    What is your alcohol intake and what is the pattern of consumption?Thepatienthasindicatedanintakeof1020unitsofalcoholeachweek.Patientsoftenunder-representtheirintakeanditwouldbeworthcheckingthiswiththepatient.Manyalcoholicdrinksareacid,contributingtodietaryacid(below),andbingepatternsofdrinkingareoftenassociatedwithvomiting.Thepossibilityofahistoryofchronicalcoholismshouldbeconsidered.

    Do you have a high consumption of acidic foods or drinks?Thisisacommoncauseofdentalerosion.Theintakeofbothacidfoodsanddrinksmustbeascertained,togetherwiththewayinwhichtheyareconsumed.

    Do you grind or clench your teeth during the day or at night?Bruxismorotherparafunctionalhabitsarecommoncausesofincreasedwear.

    Have you ever suffered from an eating disorder such as anorexia or bulimia nervosa?Thesecausesofgastricregurgitationneedtobeexcluded.Sucheatingdisordersareuncommoninmalesbuttheirincidenceisincreasing.

    In response to your questioning, the patient denies frequent acid intake, vomiting or bruxism. However, he indicates that he does suffer some acid regurgitation associated with his dyspepsia (heartburn) and that alcohol is sometimes associated with the attacks. He has had heartburn and regurgitation for 20 years but is not taking any regular medication to relieve the symptoms. He had not considered this significant enough to mention on his medical history questionnaire.

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    SE

    symptoms.Refluxmaybecausedbyareductioninpressurearoundtheloweroesophagealsphincter(asforinstanceinhiatushernia)orabnormaloesophagealmotility.Referralisnecessarytodetectsuchassociatedconditionsand,ifsymptomsmerit,toconsidertreatmentwithdrugswhichblockacidsecretion.

    Aconservativeapproachshouldbetakentotreatmentoftheerosion.Noimmediatetreatmentisrequirediftheerosionisrelativelyminor,asthepatientishappywiththeappearanceoftheteeth.Ifthecauseisidentifiedandtreated,erosionwillceaseorprogressmoreslowly.Studymodelstakenatyearlyintervalsmaybecomparedtothosetakenattheinitialvisittoassessprogression,andifthetoothwearprogresses,restorationsmaybeconsidered.

    Intheearlystagesoferosionprovisionalplasticrestorationswillprotectthepalatalsurfacesfromfurtherdamage.Howevertheshortlifespanofsuchrestorationscommitsthepatienttofurthertreatment.Alternativelypalatalveneersofporcelain,compositeormetalprovidealongertermrestorationifthereissufficientspaceocclusallyfortherestorations.

    What precautions must be taken to prevent iatrogenic damage when restoring worn teeth?

    Accuratediagnosisisessential.Ifattritionratherthanerosionisdiagnosed,occlusalsplintsmightbeprescribed.ThesewouldworsenerosioncausedbygastricregurgitationbecauseacidwouldbetrappedbeneaththesplintawayfromthepH-neutralizingeffectofsaliva.Thereisalsothepotentialforunglazedorunpolishedporcelaintoweartheenameloftheopposingteeth.Thismightbecomesignificantifthereisanelementofattritioncausingthewearoriftherestorationwereallowedtooccludeagainstdentine.

    loseweightinanattempttoimprovetheirbodyself-image.Asmallproportionofpatientswithsevereanorexiadiefromthedisorder.Unlikeanorectics,bulimicpatientsusuallyhaveastablebodyweight.Theyeatanddrinkinbingesandvomittocontroltheirbodyweight.Theremaybeanaccompanyinghistoryofdrugandalcoholabuse.

    Alcoholism.Asnotedabove,alcoholismisassociatedwithdentalerosion,eitherthroughvomitingorthelowpHofsomealcoholicdrinks.

    Ruminationisanunusualpractice,beingthehabitualchewingoffood,swallowingandthenregurgitatingitmixedwithstomachacidtobechewedandswallowedagain.Itisconsideredrarebutthereisnoaccurateinformationonitsprevalenceanditisthoughttoaffectyoung,healthyandmainlyprofessionalpeople.Ifthehabitiscontinueditcancausesignificantdamagetoteeth.

    The patient gives a clear history of regular heartburn and symptomatic regurgitation. He denies rumination and alco-holism and there is no suggestion of an eating disorder, an unlikely possibility in this age group.

    Diagnosis

    What is your diagnosis?

    Thediagnosisistoothwearcausedprimarilybyerosion.Thecauseoftheerosionisgastricacidrefluxsecondarytogastro-oesophagealrefluxdisease.

    TreatmentHow will you manage the patient?

    Thepatientshouldbereferredtoagastroenterologistorhisgeneralmedicalpractitionerforfurtherinvestigationofhis

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    Case 4: Gingival recessionSUMMARYHistoryExaminationInvestigationsDiagnosis

    Case 5: A missing incisorSUMMARYHistoryExaminationInvestigationsTreatment

    Case 6: Downs syndromeSUMMARYHistoryExaminationInvestigationsDiagnosisTreatmentLong-term managementAcknowledgements

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    Case 8: Painful trismusSUMMARYHistoryExaminationDifferential diagnosisInvestigationsTreatmentAnother possibility

    Case 9: A large carious lesionSUMMARYHistoryExaminationInvestigationsDiagnosisTreatmentOperative treatment

    Case 10: A lump on the gingivaSUMMARYHistoryExaminationFurther examination and investigationsDifferential diagnosisTreatmentDiagnosisOther possibilities

    Case 11: Pain on bitingSUMMARYHistoryExaminationDifferential diagnosisInvestigationsTreatment

    Case 12: A defective denture baseSUMMARYAcrylic complete dentureCobaltchromium casting

    Case 13: Sudden collapseSUMMARYHistoryExaminationPrognosis

    Case 14: A difficult childSUMMARYHistoryExamination

    Case 15: Pain after extractionSUMMARYHistoryExaminationDiagnosis

    Case 16: A numb lipSUMMARYHistoryExaminationInvestigationsDifferential diagnosisDiagnosis

    Case 17: A loose toothSUMMARYExaminationInvestigationsDiagnosisTreatment

    Case 18: Oroantral fistulaSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatmentOther possibilities

    Case 19: Troublesome mouth ulcersSUMMARYHistoryExaminationDiagnosisTreatment

    Case 20: A lump in the neckSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatmentAnother possibility

    Case 21: Trauma to an immature incisorSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatment

    Case 22: HypoglycaemiaSUMMARYHistoryExaminationTreatmentOther possibilitiesFurther points

    Case 23: A tooth lost at teatimeSUMMARYHistoryExaminationDiagnosisInvestigationsTreatment

    Case 24: A problem overdentureSUMMARYHistoryExaminationDiagnosis

    Case 25: Impacted lower third molarsSUMMARYHistoryExaminationInvestigationsAnother case

    Case 26: A phone call from schoolSUMMARYHistoryExaminationTreatmentFollow upOther possibilities

    Case 27: Discoloured anterior teethSUMMARYHistoryExaminationInvestigationDifferential diagnosisTreatmentPrognosisPrevention

    Case 28: A very painful mouthSUMMARYHistoryExaminationDiagnosisInvestigationsTreatmentPrognosisFinal outcome

    Case 29: Caution X-raysSUMMARYHistory

    Case 30: Whose fault this time?SUMMARYFirst patientDiagnosisSolutions What can you do to save the patient a further appointment and yourself the additional cost of extra trays? What you can do about the upper tray? There are a number of options but all have problems (Table 30.1). The only alternative would be The next patientSUMMARYHistoryExaminationSolutionTreatmentAcknowledgement

    Case 31: Ouch!SUMMARYHistory

    Case 32: A swollen face and pericoronitisSUMMARYHistoryExaminationDiagnosisTreatment

    Case 33: First permanent molarsSUMMARYHistoryExaminationInvestigationsDiagnosisFurther investigationTreatment planningFurther information

    Case 34: A sore mouthSUMMARYHistoryExaminationInvestigationsDiagnosis

    Case 35: A failed bridgeSUMMARYHistoryExaminationInvestigationsTreatment

    Case 36: Skateboarding accident?SUMMARYHistoryExaminationInvestigationsDiagnosisTreatment

    Case 37: An adverse reactionSUMMARYHistoryDifferential diagnosisAnother possibility

    Case 38: Advanced periodontitisSUMMARYHistoryExaminationInvestigationsDiagnosisTreatment

    Case 39: Fractured incisorsSUMMARYHistoryExaminationInvestigations

    Case 40: An anxious patientSUMMARYHistoryInvestigation and diagnosisAnxiety managementTreatmentPrognosis

    Case 41: A blister on the cheekSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatment

    Case 42: Will you see my son?SUMMARYHistoryExamination

    Case 43: Bridge designSUMMARYHistoryExaminationDiagnosis

    Case 44: Management of anticoagulationSUMMARYHistoryExaminationInvestigationsDiagnosisTreatmentPostoperative

    Case 45: A white patch on the tongueSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatmentPrognosis

    Case 46: Another white patch on the tongueSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatment

    Case 47: Molar endodontic treatmentSUMMARYHistoryExaminationInvestigationsDiagnosisTreatment

    Case 48: An endodontic problemSUMMARYHistoryExaminationInvestigationsTreatmentAcknowledgement

    Case 49: A swollen faceSUMMARYHistoryExaminationInvestigationsDiagnosisTreatment

    Case 50: Missing upper lateral incisorsSUMMARYHistoryExaminationInvestigationsTreatment

    Case 51: Anterior crossbiteSUMMARYHistoryExaminationDiagnosisInvestigationsTreatment

    Case 52: Refractory periodontitis?SUMMARYHistoryExaminationInvestigationsDifferential diagnosisFurther investigationsTreatment

    Case 53: Unexpected findingsSUMMARYHistoryExaminationInvestigationsDifferential diagnosisTreatmentFurther investigations

    Case 54: A gap between the front teethSUMMARYHistoryExaminationInvestigationsDiagnosisTreatment

    Case 55: A lump in the palateSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatmentPrognosis

    Case 56: Rapid breakdown of first permanent molarsSUMMARYHistoryExaminationDifferential diagnosisInvestigationsDiagnosisTreatment

    Case 57: Oral cancerSUMMARYHistoryExaminationDiagnosisPrinciples of treatmentFurther investigationsTreatment planningTreatment

    Case 58: A complicated extractionSUMMARYHistoryExaminationDiagnosisTreatment

    Case 59: Difficulty in opening the mouthSUMMARYHistoryExaminationDiagnosisInvestigationsTreatment

    Case 60: ToothwearSUMMARYHistoryExaminationDifferential diagnosisInvestigationsFurther differential diagnosisDiagnosisTreatment

    Case 61: Worn front teethSUMMARYHistoryExaminationInvestigationsDiagnosisTreatment planningTreatment

    Case 62: A case of toothacheSUMMARYHistoryExaminationInvestigationsDiagnosis

    Case 63: A child with a swollen faceSUMMARYHistoryExaminationInvestigationsDifferential diagnosisDiagnosisAetiologyTreatment

    Case 64: A pain in the neckSUMMARYHistoryExaminationDiagnosisInvestigationsTreatmentAnother possibility

    Case 65: Failed endodontic treatmentSUMMARYHistoryExaminationInvestigationsDiagnosisTreatment

    Case 66: A pain in the headSUMMARYHistoryDifferential diagnosis

    Index