surgery
DESCRIPTION
BowelTRANSCRIPT
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Intestinal ObstructionZeeshan Razzaq MRCS Ire, MRCS Ed, MRCS EngColorectal Registrar12-October-2015
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M.C – 80 yr Female
ED 1/7 History of: Abdominal pain
Intermittent, Colicky, Periumbilical Abdominal distension
Progressively worsening Vomiting
Non-Projectile, 2-3 times / day Constipation
Passing flatus
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PAST HISTORY
MEDICAL: Hypertension
SURGICAL: Open Appendicectomy at age of 30
Years Caesarean Sections x 2
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On Examination
Vitals Pulse:70/min, Regular BP: 130/80 Temp: 36.5 C RR: 15/min
Dehydrated CNS: GCS 15/15 CVS: S1 + S2 + 0 Chest: Bilateral NVB
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Abdominal Exam
Grid Iron + Pfennensteil incisions Distended No Guarding or Tenderness Non Peritonitic No Hernias Bowel Sounds: Hyperdynamic Per Rectum: Empty Rectum
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Investigations
FBC: Hb: 12 gm/dl WCC: 9000 Platelets: 299,000
U&E K: 4 meq/l
CRP: 17 Lactate: 1.3
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Investigations
PFA
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Investigations
CT Abdomen / Pelvis
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Investigations
CT Abdomen / Pelvis (With Oral & IV Contrast) Dilated proximal small bowel loops with
collapsed distal small bowel Transition point at Right Lower quadrant
at level of Mid to Terminal ileum No features suggesting bowel
Perforation / Ischaemia
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Management
Conservative Drip & Suck NG
Wide Bore Left on free drainage
Intake-Output Record IV Fluids Foleys Catheter to aim Urine output > 1ml/kg/hour
Analgesia Thrombo-Prophylaxis
TEDS Tinzaparin
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Management
Failed to respond to conservative management
High NG tube outputs Abdominal distension not settling No flatus or Bowel movements
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Management
Laparoscopy
Distended proximal small bowel loops with collapsed distal loops
Dense adhesions at right lower quadrant Free fluid Proximal small bowel viability
Questionable
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Management
Converted to Laparotomy
Band adhesion at terminal ileum: Divided Adhesions at Right lower quadrant: Adhesiolysis Hot packs for proximal segment of small bowel
Good peristalsis Good Mesenteric blood flow Colour changed to pink
Distended small bowel decompressed proximally via NG
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INTESTINAL OBSTRUCTION
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Classification Dynamic
Where peristalsis is working against a mechanical obstruction
In Small Bowel High Low
Large Bowel Intra Luminal Intra Mural Extra Mural
Adynamic
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Dynamic Obstruction
Causes Intraluminal
Impaction Foreign bodies Bezoars Gall stones Worms
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Dynamic Obstruction
Intramural Stricture Malignancy
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Dynamic Obstruction
Extramural Bands Adhesions Hernia Volvulus Intussusception
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Etiology
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Adynamic Obstruction
Absent Peristalsis Paralytic Ileus
Present but non-propulsive form Mesenteric vascular Occlusion Pseudo-obstruction
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Classification of Intestinal Obstruction
By Surgical Pathology Simple
Where blood supply is intact Strangulated
Where there is direct interference of the blood supply
By Nature of Presentation Acute Subacute Chronic Acute on Chronic
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Pathophysiology
Simple occlusion Peristalsis increases then
uncoordinated then absent Increase secretion and
decreased absorption leads to loss of fluids and electrolytes
Proximal distension is because of:
Gas Nitrogen 90 % H2S
Fluid collection Excessive fluid collection Retarded absorption
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Pathophysiology
Causes for dehydration and electrolyte loss
Reduce oral intake Defective intestinal absorption Losses due to vomiting Sequestration in bowel lumen
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Pathophysiology
In closed Loop Obstruction
Occlusion occurs at both ends of loop
Classic cause: tumour of Right Colon and competent Ilocaecal valve
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Pathophysiology
In strangulation End result of closed
loop obstruction Results in gangrene
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Clinical Features: Look for following questions
Is it obstruction and if so at what level?
Is strangulation present? Is dehydration present? What is the cause? What is the treatment of individual
cause?
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Symptomatology
Pain First symptom Colicky in nature Centered around umbilicus --- Small
bowel Lower abdomen-----Large bowel With increasing distension, colicky is
replaced by diffuse pain Severe pain is indicative of strangulation Does not occur in Paralytic ileus
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Symptomatology
Vomiting The more distal the obstruction, the
longer the interval between symptoms and development of the nausea and vomiting
With progression, the vomitus alters from digested food to ---- Faeculent material
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Symptomatology
Distension In small bowel dependent on the Site of obstruction Visible peristalsis may be present Delayed in colonic obstruction Absent in mesenteric vascular occlusions
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Symptomatology
Constipation Absolute: Neither faeces nor flatus is passed Relative: Where flatus only is passed
Constipation is not present in Richter's Hernia Gallstone ileus Mesenteric vascular occlusion Obstruction associated with pelvic abscess Partial obstruction (faecal impaction/colonic neoplasm)
where diarrhoea may often occur
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Examination General
Dehydration Repeated vomiting Fluid sequestration Urea and haematocrit rise
Pyrexia Onset of ischemia Intestinal obstruction Inflammation associated with obstructing
diseases Hypothermia indicates septicemia
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Examination
Abdomen Inspection
Scars Site of distention Visible peristalsis Irreducible swellings
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Examination
Palpation Abdominal mass Tenderness
Indicates pending or established gangrene Peritonism indicates overt infarction or
perforation Rigidity Hernial Orifices
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Examination
Percussion Resonance
Auscultation Hyper-resonant Silence
DRE Impacted faeces Rectal tumor Blood on finger
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Features of Strangulation Presence of shock Pain Symptoms commence suddenly and recur
regularly Localised tenderness Rebound tenderness Rigidity Raised WCC & CRP Metabolic acidosis: Rising Lactate & Base
deficit
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Investigations
Supine Abdominal X-ray (PFA) Jejunum: Valvulae Conniventes Ileum: Feature less Caecum: Round gas Shadow in RIF Large bowel: Haustral Folds F.B and Gall stones could be seen
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PFA
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PFA
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CT Abdomen / Pelvis
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Management Principles
Gastrointestinal Drainage Fluid & Electrolyte replacement Relieve of Obstruction
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Management
Initial management Pass NG I/V fluids: Saline or Hartmanns Catheterise Antibiotic are not necessary but many
clinicians give because of overgrowth of the bacteria
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Management
Monitor Pulse Temp BP Respiratory rate Urine out put Abdominal girth Abdominal tenderness WCC, CRP & Lactate
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Conservative Management
Done in In 2-10 days of previous Surgery Multiple prior attacks of adhesive obstruction Poor general condition Patient unfit for Surgery
Look for signs to stop conservative treatment
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Surgical Management
The sun should not both rise and set on a case of unrelieved intestinal obstruction
Conservative management may be continued for 72 hours
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Surgical Management
Indication for early interventions Obstructed or strangulated external
hernia Internal intestinal strangulation Acute obstruction
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Surgical Management
Contra indications to Surgery Paralytic ileus Impacted faeces Volvulus No strangulation seen on previous
exploration
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Surgical Management
Operative assessment is directed to Site of obstruction Nature of obstruction Viability of the gut
Midline incision gives the best exposure
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Surgical Treatment
Principles of surgical intervention Management of the segment at the site
of obstruction The distended proximal loop The viability of the gut
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Differences between Viable and Nonviable
bowel Bowel Viable Nonviable
Circulation Dark color becomes lighter
Mesentery bleeds if pricked
Dark color remains
No bleeding if mesentery is pricked
Peritoneum Shiny Dull and lustless
Bowel musculature Firm
Pressure ring may or may not disappear
Peristalsis may be observed
Flabby, thin and friable
Pressure rings persist
No peristalsis
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Dilated Viable bowel
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Nonviable Gangrenous bowel
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Large Bowel Obstruction
Causes Carcinoma Diverticular disease Volvulus Pseudo-obstruction
Types Acute Chronic
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Management of Large bowel obstruction
Full resuscitation
Lesions on Right side Emergency right hemicolectomy If not Removable
Proximal stoma Ilio-transverse internal bypass
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Management of Large bowel obstruction
For left side Lesions Primary resection with one of following
Left hemicolectomy Double barreled colostomy (Paul-Mikulicz) Hartmann’s procedure On table lavage of colon with primary
anastomosis Primary anastomosis with proximal covering
stoma
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Rare Causes
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Rare Causes
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Rare Causes
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Rare Causes
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Rare Causes
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Rare Causes
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Thank You