stephen springer nolan wool. movement posture fluid propulsion generate heart beat
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Structure of a Muscle Fiber Cylindrical Sarcolemma
covering Sarcoplasm
cytoplasm Myofibrils Myosin Actin
Relationship:Neuromuscular Junction
Stimulus travels from: Brain Neuron Neuron Motor
Muscle Axon End Plate
Sliding Filament Theory
1. Heads of myosin attach to binding sites of actin
2. Myosin heads swivel3. ATP releases myosin heads4. Muscle relaxes
Muscle Contraction
Calcium binds to troponin Tropomyosin pulls aside Binding sites on actin
exposed
Calcium released from sarcoplasmic reticulum exposes the binding sites
Muscle Contraction (Cont.)
Myosin heads bind to actin Power stroke occurs (swivel of
heads) Muscle contraction occurs
Muscle Contraction (Cont.)
ATP binds to myosin Linkage is released
ATP splits into ADP and Phosphorous Causes the myosin head to cock back
Cycle continues* *If there are ATP & Calcium Ions
Fatigue
Glycolysis Fast-twitch muscle Oxygen Debt
Lactic acid build up +2 ATP
Cellular Respiration
Slow-twitch muscle Includes the process of
Glycolysis +34 ATP
Definition: When a muscle is exercised strenuously for a prolonged period and loses its ability to contract.
Cause: Low pH Accumulation of Lactic Acid
Cramping
Definition: A painful condition in which a muscle undergoes a sustained involuntary contraction
Causes: Lack of ATP Changes in extracellular fluid Uncontrolled stimulation
Superficial MusclesZygomaticus
Sternocleidomastoid
Rectus AbdominusExternal Oblique
Frontalis
Trapezius
Deltiod
Pectoralis Major
Biceps Bracii
Gracilis
Adductor Longus
Sartorius
Rectus Femoris
Vastus Lateralis
Gastrocnemius
Soleus
Fibularis Longus
Extensor Digitorum Longus
Tibialis Anterior
Expeliarmis*Harry Potter
Posterior Superficial Muscles
OccipitalisSternocleidomast
oidTrapezius
Infraspinatud
Deltoid
Teres MajorRhomboideus
Latissimus Dorsi
Gluteus Medius
Gluteus Maximus
Adductor Magnus
Semitendinosus
Semimembranosus
Biceps Femorous
Calcaneal Tendon
Fibularis Longus
Gostrocnemius
Soleus
Gluteus Maximus
Nobody Cares
Triceps Bracii
Brachialis
Prime Movers, Synergists, & Antagonists
Antagonists: Resist prime
mover
Prime Mover (agonist): Mainly responsible for
movement
Synergists: Helper muscles
Recruitment and Muscle Tone
Muscle Tone: Nerve impulses
continuously sent from spinal cord
Recruitment: Increases number
of active muscle fibers Summation or Tetanic
Increase of weight
Summation and Sustained Contraction
Summation: Combined individual
twitches Eventual relaxation
Sustained Contraction (tetanic): Combined individual
twitches Doesn’t relax Rigor mortis
DiseasesMyotonic Dystrophy—An Expanding Gene: Stronger and more threatening with each
generation RNA too large to leave nucleas, gets copied,
passed to next generation continuously Weakness of limbs
Hereditary Idiopathic Dilated Cardiomyopathy: Actin not anchored to
z-line properly Causes the heart
chambers to enlarge and eventually fail