silicosis - dol6 chronic silicosis little respiratory impairment in early stages significant chest...
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Silicosis
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Silica Dust
Who may be affected: Sandblasters Pottery workers Mining, Quarrying, Tunneling, Drillers Foundry workers Stone Cutting, Polishing Brick workers
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Variations
Chronic Silicosis Accelerated Silicosis Acute - silicoproteinosis Si-tuberculosis
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Acute Silicosis
May develop after 1 to 3 years of exposure
Distinctive feature is a proteinaceous fluid that fills the alveoli “silicoproteinosis”
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Accelerated Silicosis
May develop after 5 to 15 years of exposure
Seen with blasters and silica flour workers May lead to progressive massive fibrosis
and death
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Chronic Silicosis
Little respiratory impairment in early stages
Significant chest x-ray changes usually after 10 to 20 years of exposure
Simple Complicated: Progressive massive fibrosis
Coalescence of fibrotic nodules
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Radiographic Findings Small (< 1 cm), rounded, nodular opacities “Diffuse fibronodular” disease Usually in mid/upper lung fields Coalescence, conglomeration with
progression Progressive Massive Fibrosis
“Egg-shell” calcification of lymph nodes
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Page 11 of
Handouts
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Grade 3/3
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Progression - Complicated Silicosis (6 years later)
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Further Progression – 6 yrs later
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Complications Larger nodules > 1 cm Cough, sputum, infection, dyspnea TB – hemoptysis, weight loss, fever Cor pulmonale Bronchitis Emphysema Lung cancer “Mixed dust” fibrosis – e.g. miners
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Diagnosis History of Exposure Characteristic Chest x-ray Pulmonary Function Testing
Normal/Restrictive/obstructive (mixed dusts) Decreased DLCO
May lead to several consequential conditions TB, scleroderma, rheumatoid arthritis
(Caplan’s Syndrome), lupus
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Program Guidance
PM 2-800: need 10 year latency and > or = 1/0
PM E-500 Exhibit 2
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What we covered
The 3 presentations of silicosis and their latency.
How the clinical picture of silicosis differs from asbestosis.
Possible complications (consequences) of silicosis.
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Questions