[ppt]jantung anatomi - frida magdalena | hidup itu … · web viewtitle jantung anatomi author...
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07/05/23 nn, JANTUNG 1
Elyana AsnarDepartemen Ilmu Faal
FK UNAIR
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07/05/23 3CVS-FF
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07/05/23 nn, JANTUNG 4
JANTUNGANATOMI
Elyana Asnar
• LETAK– RONGGA DADA KIRI– TERLINDUNG DINDING DADA– UKURAN 12-14 x 8-9 x 6 cm– BERAT 250-350 gm– BASIS : Superior-posterior : C-II– APEX : anterior-inferior ICS-V
• 2 jari di bawah papila mamae
• Bag ventrikel paling tebal
• Punctum maximum
• Point of maximal intensity
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07/05/23 nn, JANTUNG 5
JANTUNGANATOMI
• TERDIRI ATAS1. ATRIUM2. VENTRIKEL3. NODAL TISSUE & SERAT PENGHANTAR KHUSUS a. Sino Atrial Node
(SA node)b. Atrio Ventricular Node
(AV node)c. Bundle of Hisd. Sistem Purkinje
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07/05/23 nn, JANTUNG 6
JANTUNGANATOMI
1. SA node2. Internodal Pathway a. Bachman (anterior) b. Wenkebach (medial) c. Thorel (posterior)3. AV node4. Bundle of His5. Left Bundle Branche, Right Bundle Branche 6. Purkinje fibers
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07/05/23 nn, JANTUNG 7
1. Pericardium2. Epicardium3. Myocardium4. Endocardium
Lapisan jantung
Myocardium bagian kontraktil, mirip otot rangka
Antara pericardium dan epicardium terdapat rongga (cavum pericardii), berisi cairan pelicin
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07/05/23 nn, JANTUNG 8
Anatomi Jantung
Myocardium
bagian kontraktil,
mirip otot skelet.
Otot jantung bersifat:
otonom,
unvolunter,
berkontraksi ritmis
(autorhythmic cell).
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07/05/23 nn, JANTUNG 9
Anatomi Jantung
• Katub (valve) :- Mitral (bicuspidal): kiri- Tricuspidal: kanan- Aortic semilunar- Pulmonary semilunar
• Arah katub : searah. Stenosis membuka tak
sempurna Insufisiensi menutup
tak sempurna
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07/05/23 nn, JANTUNG 10
Fisiologi Sifat dasar otot jantung :
a. irritability (bathmotropic) = peka rangrang
b. conductivity (dromotropic) = hantar rangsang
c. contractility (inotropic) = dapat berkontraksi
d. rhythmicity ( chronotropic) = bersifat ritmis
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07/05/23 nn, JANTUNG 11
Irritability = peka rangrang
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07/05/23 nn, JANTUNG 12
CONDUCTIVITY = menghantarkan rangsang
Kecepatan hantar impuls :
• Otot atrium : 0.3 m/det
• AV node : 0.1 m/det
• Otot ventrikel : 0.4 – 0.5 m/det
• Purkinje fibres : 1.5 – 4 m/det
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07/05/23 nn, JANTUNG 13
…CONDUCTIVITY
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07/05/23 nn, JANTUNG 14
SA AV
radier His
Seluruh Purkinje DindingAtrium ke seluruh bagian ventrikel bersamaan
ventrikel berkontraksi sbg satu kesatuan
…CONDUCTIVITY
Internodal Pathway
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07/05/23 nn, JANTUNG 15
CONTRACTILITY = dapat berkontraksi
Rangsang membuka Ca channel, Ca masuk rangsang Ca keluar dari sarcoplasmic reti-culum dan berikat dengan troponin kontraksi otot
Bila Ca lepas dari troponin otot relaks, Ca dimasuk- kan ke dalam SR dan sebagian keluar
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07/05/23 nn, JANTUNG 16
Irama kontraksi jantung ikut irama SA node
SA memancarkan Potensial Aksi secara reguler ke seluruh atrium
AV node
sistem Purkinje
kontraksi ventrikel ritmis
Frekuensi impuls / kontraksi
SA node : 70-80 x/min
AV node : 50-60 x/min
Otot atrium : 20-40 x/min
Otot ventrikel : 10-30 x/min
RHYTHMICITY = bersifat ritmis
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07/05/23 nn, JANTUNG 17
…RHYTHMICITY
Normal : SA sebagai pace maker irama SA node irama sinus
Patologis : AV node sebagai reserve pace maker irama nodal lebih lambat dari irama sinus
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07/05/23 nn, JANTUNG 18
JANTUNG SEBAGAI POMPA
SYARAT POMPA JANTUNG YANG BAIK :
1. Katub berfungsi baik
2. Pengisian darah atrium dan ventrikel optimal
3. Kuat kontraksi optimal
4. Frekuensi jantung normal, kontraksi atrium dan ventrikel bergantian
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07/05/23 nn, JANTUNG 19
SIKLUS JANTUNG
1. Fase sistole (kontraksi)
2. Fase diastole (relaksasi)
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07/05/23 nn, FISIOLOGI JANTUNG 20
6 FASE SIKLUS JANTUNG
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07/05/23 nn, JANTUNG SIRKULASI-PSY 2107/05/23 21
PROYEKSI SUARA JANTUNG DI DADA
Suara I : Penutupan katup AV (“lub”)1. Katub mitral , ICS V midclav kiri2. Katub tricuspidal, ICS V
parasternal kiri
Suara II : Penutupan katup semilunar (“dub”)
1. Katup aorta, ICS II parasternal kanan
2. Katup pulmonal, ICS II parasternal kiri S II S II
S IS I
SISTOLE
DIASTOLE
Berasal dari suara katup jantung.
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Aorta dan arteria Pulmonalis saling menyilang, sehingga pada auskultasi suara katup Aorta di ICS 2 parasternal kanan, sedang
katup a.Pulmonalis di ICS 2 parasternal kiri.
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07/05/23 nn, JANTUNG 23
PROYEKSI SUARA JANTUNG
1. Katub mitral , ICS V midclav kiri2. Katub tricuspidal, ICS V parasternal kiri3. Katup aorta, ICS II parasternal kanan4. Katup pulmonal, ICS II parasternal kiri
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07/05/23 nn, JANTUNG 24
SATU SIKLUS JANTUNGTERDIRI :Sistole = fase kontraksi = 0,3 detDiastole = fase relaksasi = 0,6 det
--------------- 0,9 det
Heart Rate = 60 : 0,9 = 66,6 67 x / menit Normal 60 – 100 x / min 72 x / min Bradikardi < 60 x / min Takikardi > 100 x / min
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07/05/23 nn, JANTUNG 25
STROKE VOLUME( ISI SEKUNCUP )
= Jumlah darah yang dipompa satu kali oleh ventr kiri / kanan
Pada manusia dewasa sehat SV + 70 ml
SV mengikuti hukum Frank-Starling :Jantung beradaptasi terhadap beban yang diberikan“ Semakin kuat jantung diregang (semakin besar volume
ventrikel) semakin kuat pula jantung berkontraksi”
SV = EDV ESV
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07/05/23 nn, JANTUNG 26
CARDIAC OUTPUT (CURAH JANTUNG)
CO = Jumlah darah yang dipompa oleh jantung dalam 1 menitCO = SV x HR = 70 ml x 72x / menit = 5040 ml/menit
( 5000 ml/menit )Dipengaruhi oleh :1. Pre load : EDV2. After load : Tekanan darah,
Viskositas darah
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07/05/23 nn, JANTUNG 27
CARDIAC OUTPUT MENINGKAT
Kompensasi agar transport oksigen mencukupi :
1. Anemia : Hb rendah
2. Kehamilan : sirkulasi placenta = arterio-venous shunt
3. Berdiri : 20 % > duduk
4. Latihan jasmani :
5. Lain-lain : demam, hipertiroidi, emosi
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07/05/23 nn, JANTUNG 28
Pengaruh suhu
1. Peningkatan suhu sd. 42o C permeabilitas membran self excitation process frek
2. Peningkatan suhu > 42o C frek
3. Penurunan suhu frek
hal ini berhubungan dengan metabolisme otot jantung
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07/05/23 nn, JANTUNG 29
PENGATURAN FUNGSI JANTUNG
Pengaturan oleh syaraf otonom1. Simpatetik
Inotropik positif ( kekuatan kontraksi )Chronotropik positif ( frek , 170-230 x / min) CO
2. ParasimpatetikInotropik negatif ( kekuatan kontraksi )Chronotropik negatif (frek , maksimal menurun sampai 20 –30 x / min CO
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07/05/23 nn, JANTUNG 30
DETAK JANTUNG (HR)
Rangsang saraf simpatik atau epinefrin me-
ningkatkan HR.Rangsang saraf
parasimpatik atau acetilcholin me-nurunkan HR.
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07/05/23 nn, sirkulasi FK 31
• Pada kelainan katub mitral hambatan aliran di jantung
timbunan darah di atrium tekanan v.Pulmonalis meningkat (back flow darah) edema paru sesak nafas.
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07/05/23 nn, sirkulasi FK 32
GAGAL JANTUNG KANAN & KIRI
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07/05/23 nn, JANTUNG 33
VASKULARISASI JANTUNG
Sistem arteri
1. A.coronaria sinistrar. descendens ant.r. circumflexusr. marginalis
otot jantung kiri
2. A.coronaria dextrar. descendens post.r. marginalis otot jantung
kanan + kiri
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07/05/23 nn, JANTUNG 34
Diagram of the epicardial, intramuscular, and subendocardial coronary vasculature.
Epicardial coronary arteries
Subendocardial arterial plexus
Cardiac muscle
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07/05/23 nn, JANTUNG 35
PENGATURAN ALIRAN DARAH KORONERA. Swadaya lokal
1. Kebutuhan oksigen (O2)2. CO2 ↑3. Laktat ↑4. Adenosin ↑ 5. Ion Kalium ↑ dan Hidrogen ↑ menyebabkan dilatasi ok.1. O2 otot jantung O2 koroner lemah, dilatasi
2. O2 otot jantung menghasilkan adenosin dilatasi
ATP P + ADP P + AMP P + ADENOSIN vasodilatasi koroner
Terjadi vasodilatasi
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07/05/23 nn, JANTUNG 36
PENGATURAN ALIRAN DARAH KORONERB. SARAF1. Parasimpatis pengaruhnya sangat kecil diabaikan2. Simpatis : extensif
zat-zat transmitter simpatis :epinefrin dan norepinefrin berpengaruh pada pembuluh koroner, tgtreseptor α konstriksireseptor β dilatasi
a. coronaria epicardial : res α > banyaka. coronaria intramuscular : res β > banyak
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07/05/23 nn, JANTUNG 37
PENGATURAN ALIRAN DARAH KORONER
RANGSANGAN SIMPATIS
Konstriksi a.epikardial (res.α)Dilatasi a. intramuskuler (res.β )
BAGIAN TERBESAR aliran darah terjadi dalam
a. intramuskuler
EFFEK NETTO : dilatasi koroner
Konstriksi a.epikardial res.
Pada beberapa keadaan timbul: ISKEMIA MIOKARDIUM
NYERI ANGINA (Nyeri dada iskemik)
Dengan adanya: faktor metabo-lit dan kadar O2 (sebagai vaso dilator) maka beberapa detik akan kembali normal
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07/05/23 nn, JANTUNG 38
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07/05/23 nn, JANTUNG 39
PENYAKIT JANTUNG KORONER & INFARK MIOKARD
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07/05/23 nn, JANTUNG 40
Angioplasti Koroner Transluminal Perkutan
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07/05/23 nn, JANTUNG 41
Stents and bypass surgery are common treatments for blocked arteries.
Stents are tiny, expandable mesh tubes that hold arteries open.
Bypass surgery involves rerouting the blood flow around the blocked artery by using a healthy artery from somewhere else in your body.
4107/05/23 nn, JANTUNG SIRKULASI-BIDAN
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07/05/23 nn, JANTUNG 4207/05/23 nn, JANTUNG SIRKULASI-BIDAN
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07/05/23
STENT
Stents are tiny, compared with fingers4307/05/23 nn, JANTUNG SIRKULASI-BIDAN
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07/05/23 nn, JANTUNG 44
TQ
Buah Semangka ….…,
Berhidung Belang ….!
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07/05/23 nn, JANTUNG 45
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07/05/23 nn, FISIOLOGI JANTUNG 46
CONDUCTIVITY
internodal pathway (3)SA AV junction AV
(junctional fibres)radier His
Seluruh Purkinje DindingAtrium ke seluruh bagian ventrikel bersamaan
ventrikel berkontraksi sbg satu kesatuan
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07/05/23 nn, JANTUNG 47
Potensial Aksi SA node
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07/05/23 nn, FISIOLOGI JANTUNG 49
PENGATURAN FUNGSI JANTUNG
Intrinsik 1. Heterometrik autoregulation
pengaturan bila ada perubahan ukuran panjang serabut otot jantung
terkait dengan Hukum Frank-Starling
terkait dengan EDV
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07/05/23 nn, FISIOLOGI JANTUNG 50
PENGATURAN FUNGSI JANTUNG
2. Homeometric autoregulation
Pengaturan tanpa ada perubahan panjang serabut otot jantung
terkait dengan perubahan metabolisme otot jantung,metabolisme kekuatan elemen kontraksi (mis: demam, hipertiroidi)