nutritional diseases new.ppt

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    NUTRITIONAL DISEASES/DISORDERS

    MANIFESTATION

    * OVER NUTRITION

    * UNDER NUTRITION

    * MALNUTRITION

    CAUSES :COMPLEX

    * GENETICS

    * ENVIRONMENT* CULTURAL

    * PSYCHOLOGICAL

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    DASAR Tak langsung LangsungMISKIN Penghasilan

    PENDIDIKANRENDAH

    KesediaanPangan

    Pola Asuh

    Anak

    Akses thdPelayananKesehatan

    InfeksiPenyakit

    Asupan

    gizi/makanan

    Statusgizi

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    CLASSIFICATION OF NUTRITION DISORDERS

    1. PRIMARY

    Inadequate intake

    Low food consumption

    Defie of vit

    - IDD (GAKY)- Anemia

    Overnutrition due to over eating

    2. SECONDARY

    Intake good but

    DigestionAbsorption disturbed

    Utilization

    Excretion

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    WHAT HAPPENS IF INTAKE IS NOT ADEQUATE

    (Below Requ irement ? )

    STAGES :

    1. Body reserve is used

    eg. - Vit A Depo in Liver

    Can last for 3 months

    - Vit C in tissue reserve

    Lasts only for few days

    TISSUE DEPLETION

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    2 . IF TISSUE RESERVE IS ALL USED

    Biochemical changes

    OCCUR

    If not supplied, it will be followed with :3. Physio logical changes

    Due to metabolism disturbances

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    eg. B1in CHO Mtabolism

    * Zn in many Enzim activities* Vit A in adaptation process

    4. Anatomical changes

    * Tissue defect- Bitots spot

    - Xerrosis conjuction

    - Bleeding gUMS- (blood vessel fragility )

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    Inadequacy

    BCh Phych AnatchTissue depletion

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    FACTORS INFUENCING TISSUE DEPLETION

    A mount of tissue reserves High vs Low

    Degree of in adequacy mild vs moderate vs severe

    Degree of requirement

    Interact to produce

    Biochemical, and clinical sign & symptoms

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    Type o f Nutr i t ional Defic iences

    1. Acu te vs Ch ron ic

    - Depends on the VELOCITY of the inadequacy

    & pathologie lesions- Acute : easily treated

    - Chronic : improvement is slow

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    2. Mild vs Severe

    - depends on degree & intensity of tissue

    changes/destruction

    - Brings clinical implication

    - mild - acute :

    - acute- severe prognosis is different

    - mild chronic

    - severe chronic

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    Eg. Acute B1defie

    * head retraction in birds

    * Death in few days

    * Given B1quickly, recovered with no sequale

    3. DURATION/LENGTH of INADEQUACY

    Days, months, year

    * Long duration : worse response

    * Short duration : better response

    eg. - Mild poly neuropathy for years

    - Severe poly neuropathy for days/months

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    B iochem ical changes

    Detected through :

    Enzyme test precence of pathological substances in

    blood/urine

    Blood Markers- T3-T4(defie Iodine)

    - Serum ferritin (iron defie)

    - Serum vit A/retinol (vit A defie)

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    Phys iolog ical /Func t ional changes

    Found as :

    1. Subjective Complaints

    - Weakness

    - Low concentration

    - Low Peristaltic movement

    - Stomach gas increase

    2. Objective Findings

    - Low Physiological reflex

    - Phathological reflex present

    - Low dark adaptation ability

    - Low Hb

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    ANATOMICAL CHANGES

    1. Found as the farthest end of deficiency.

    2. Can be seen micros or macros copically in organs

    - Conjuctiva

    - Cornea

    - Gums

    - Tongue

    - Liver

    - Epithelial linings