lymph pathology
TRANSCRIPT
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PATOLOGI LIMPA
Kuliah PA
Fakultas Kedokteran
oleh
Noor Yazid AD
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KELAINAN KONGENITAL
♦ASPLENIA6APLASIA
♦A)NORMAL LO)ULASI
♦
A77ESSOR8 SPLEEN ditemuka# t.u. di 3auda &a#3reas
"i'.'astr(s&"e#i3us
9u#'si : ide#tik d'. Lim&a ' as"i Dam&ak#a : &d.tera&i s&"e#e3t(mi4 t;&uas
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SPLEENECTOMI
♦DE
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ETIOLOGISPLENOMEGALI
INFEKSI
GANGGUAN SIRKULASI
STORAGE DISEASE
NEOPLASIA
DISORDERD OF TE !LOOD
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AKUT SPLENI" TU#ORRada#' akut #(# s&esi9ik
MAKROSKOPIK MIKROSKOPIK
)erat : / 0,,$?,, 'r Si#us ditutu&i darah
Lu#ak@&u"&a seri#'$ Dia#tara#a tam&ak
di99"ue#t. "ek(sit PMN@makr(9a'
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ETIOLOGI SPLENITIS AKUT
PREDISPOSISI :
♠)AKTERIEMIA
.e#d(@Di&teri@Dse#t.basi"er@P#eum(#ia
♠ET.TISSUE DISTR.OF 7EMI7AL
♠SEERE TOEMI7 DISEASE
Stre&t(3(33us hem("ti3us 'r(u& A
♠P8EMIA
♠SEPTI7 EM)OLI
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SUBAKUT – CHRONIC
SPLENITIS♦ ETIOLOGI : .e#d(3arditis
♦ MAKROS. : Lim&a // → *,,, 'r.
&erabaa# ke#a"@ =.abu$abu
i#9ark
♦ MIKROS.: Pd.si#us B &u"&a tam&ak &adat
(.k.&e#uh makr(9a'Bse" &"asma
"ek(sit PMN Hu'a ba#ak@
mit(sis
&a'(sit
se" Retik.e#d(te" hi&ertr(9i B kub(id
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TBC LIMPA
7ARA MASUK : mi"ier
UMUR : t.u.a#ak
SPLENOMEGALI
)ERAT : +,, 4 !,, 'r.
MAKROS B MIKROS
TAMPAK TU)ERKEL
di : $ se"uruh ka&su"
$ 3.ma"&i'i
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T!" LI#PA #ILIAIR
Gambar makr(s :
Tam&ak
mu"ti&e" 6 di9us
tuberke"
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SPLENITIS SPESIFIK
T)7 SIFILIS SARKOIDOSIS
INF.MONO$ TIFOID )RU7ELLOSIS
NUKLEOSISMALARIA KALA AAR ISTOPLASMO$
SIS
S7ISTOSO$ ANTRAKOSIS AKTINOMIKO$
MIASIS SIS
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INFEKSI MONONUKLEOSIS
♦
ETIOLOGI : I#9eksi E)♦UMUR : *+ 4 *J th. &d.i'h 7"ass
♦KLINIK : miri& he&atitis &a#as@ he&at($
s&"e#(me'a"i."im9ade#(&ati 'e#e$ ra"@#eri te#''(r(ka#
♦MAKROS : besar 12$02N@"u#ak@s&t.da'i#'@
B hi&eremik.
♦MIKROS : &r("i9."im9(sit t&i36at&i3@
i#9i"tr.trabe3.B 3a&s
→ mudah ru&tur
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TYPHOID SPLENITIS
i Lim&a membesar 1+, 4 0+, 'r
iF(3a" #ekr(sis
i isti(sit ba#ak @ isi eritr(sit
i E#d(te" si#us a"ami hi&ertr(9i da# aktiv
mem9a'(sit eritr(sit da# se" debris.
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SPLENITIS LUETI7A
LUES KONGENITAL s&"e#itis
"im&a / @ 12N@ &erabaa# ke#a"
Mikr(s : Har.ikat / @ i#9i"trasi "im9(sit Bse" &"asma
LUES STADIUM III s&"e#(me'a"i
meru&aka# 3(#'estive 3ha#'e akibatsir(sis "ues
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HYALIN PERISPLENITIS
♦AKADEMIK INTEREST
♦7LINI7AL DISFUNGTION : $ #e'ati9
♦UNSUR : $ hia"i#
$ k("a'e# teba"
$ 9ibr(sis
♦ETIOLOGI : → mu#'ki# kr#.:
$ &eris&"e#itis
$ asites ' "ama
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8ALIN PERISPLENITIS
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SPLENITIS padaMALARIA♦STAD AKUT : membesar da# "u#ak
♦MIKROS : Parasit ditemuka# &ada :
&u"&a bebas eritr(sit
se" retiku"um
e#d(te" &e#uh isi &arasit B &i'me#
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SPLENITIS $ada #ALARIA
KRONIK : "im&a ///*,,,$?,,, 'r.
K(#siste#si : ke#a"
Ka&su" : me#eba" B me"ekat MIKROS : >ar.ikat tumbuh ///
ka"au sembuh .
→ "im&a me#'erut 6 3i3atri2 Bdisertai timbu#a# &i'me#
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ACUT VASCULAR DISEASE
♦PADA : $ &r(ses &e#3er#aa# mi"d $
hi&eremi
$ T(2emia
$ I#9eksi akut
♦MAKROS : Lim&a / %1+, 'r.@"u#ak
Q =ar#a merah
♦MIKROS : sembab &d si#us (k.er
→ 7.ma"&i'i me"ebar
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CHRONIC VASCULAR DISEASE( CHRONIC VENOUS CONGESTION )
♦ETIOLOGI : I#tra he&ati3 (bstru3ti(#
Obstruksi v.&(rta6v."ie#a"is
7(#'estiv ve#(us sstemi3 de3(m&.ka#a# @ 7P
E2tra he&ati3 (bstru3ti(#
tr(mb(sis v.&(rta s&(#ta# 6 (.k.sebab "ai#
♦)erat *,,, 'r. %on&esti'e
s$leno(e&ali
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INFARK LIMPA
♦ETIOLOGI : Pe#umbata# : $ emb("i
$ tr(mb(sis
♦
MAKROS : =.ke$&utiha# tertutu& 9ibri# di ka&su"
♦ARTI KLINIK : Pe#ti#' utk &drt tua d'#
Pe#.>a#tu#' 8' tiba #eri di dada kiri
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#AKROS)INFRAK LIEN
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INFARK LIMPA tr(mb(s
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HYPERSPLENISME
♦S8NDROME : 7(#'esti
Leuk(&e#i
A#emia♦ADA 1 TIPE : &rimer B seku#der
♦Seku#der (.k. "ek(&e#i@ LMN @ I#9eksi@
Fe"ts si#dr(m.♦ (polyar!r""#$ %&'&r$ a&)"a $ l&*op&"$#pl&o)&+al",
♦)ANTI S8NDR.: 'a#''.hem("itik &rimer@
diikuti ter"ibat#a "im&a
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RUPTURA LIENALIS
♦ KEKERASAN
♦ KLL
♦ PERKELAIAN
♦ >ATU ♦ SPONTAN
♦ INF.MONONUKL.
♦ MALARIA
♦ T8POID♦ LEUKEMIA
♦ AKUT SPLENITIS
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NEOPLASMA LIENALIS
♦ >INAK : haema#'i(ma B "m&ha#'i(ma
♦GANAS :
• Primer : LN B L Hara#'
• Seku#der : su"it
(.k.*. Tidak subur
1. Dd.arteri("e teba"
• Ka"au ada : $ &ertumbuha# retr('ade
$ &ertumbuha# di ka&su"