lecture2 eyelid,orbit,lacrimal
TRANSCRIPT
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Eyelids, Orbit and Lacrimal System
Hernando L. Cruz Jr., EyeMD
Section of Ophthalmic Plastic, Reconstructive, Lacrimal & Orbital Surgery
Department of Ophthalmology
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Eyelids, Orbit and Lacrimal System
Eyelids Basic Anatomy and Physiology Eyelid Lesions Disorders of the Eyelashes Entropion Ectropion Ptosis
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Eyelids, Orbit and Lacrimal System
Orbit Applied Anatomy Clinical Evaluation of Orbital Diseases Diagnostic Modalities in Orbital Diseases Graves’ Ophthalmopathy Orbital Infections Orbital Tumors Orbital Fractures
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Eyelids, Orbit and Lacrimal System
Lacrimal System Applied Anatomy and Physiology Epiphora and Lacrimation Clinical Evaluations of Tearing Infections of the Lacrimal Passages Treatment of Lacrimal Obstructions Surgical Techniques
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Eyelids and Periorbital Structures
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Anatomy & Physiology
Eyelids Globe Protection
• 1. Screening and Sensing action of the Cilia
• 2. Secretion of the glands of the Eyelids
• 3. Movements of the Lids
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Anatomy & Physiology
Cilia “Eyelashes” first line of Defense 2 rows of about 100 - 150 in the upper and 50 -
75 in the lower lid nerve plexuses in each follicle glands in each follicle
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Anatomy & Physiology
Secretion of the Glands of the Eyelids Oily layer of the meibomian glands Forms the superficial element of the precorneal
tear film which prevents tear evaporation
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Eyelid Margin Anatomy
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Anatomy & Physiology
Movements of the Lids 3rd and most important element levator palpebrae superioris, orbicularis oculi
and Muller’s muscle
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Anatomy & Physiology
7 structural layers of the eyelid1. Skin and Subcutaneous Tissue
2.Muscle of Protraction
3.Orbital Septum
4. Orbital Fat
5. Muscle of retraction
6. Tarsus
7.Conjunctiva
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Upper Eyelid Anatomy
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Lower Eyelid Anatomy
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Anatomy & Physiology
I. Skin and Subcutaneous Tissue thinnest of the body no subcutaneous fat Upper lid crease
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Anatomy & Physiology
II. Muscles of protraction orbicularis oculi CN VII Pre-tarsal, Pre-septal, Orbital parts
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Orbicularis Oculi Muscle
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Anatomy & Physiology
III. Orbital Septum multilayered sheet of fibrous tissue fuses with the aponeurosis to form the lid
crease serves as a barrier between the eyelid and the
orbit
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Anatomy & Physiology
IV. Orbital Fat lies posterior the orbital septum and anterior the
levator aponeurosis with age-related attenuation - “eyebag”
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Anatomy & Physiology
V. Muscles of Retraction Upper Eyelid
• Levator Muscle and its Aponeurosis
• Muller’s Muscle Lower Eyelid
• Capsulopalberal Fascia
• Inferior Tarsal Muscle
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Anatomy & Physiology
Levator Palpebrae Superioris muscular portion 40 mm aponeurosis 14-20 mm whitnall’s ligament - functions as a suspensory
support of the upper eyelid innervated by CN III
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Whitnalls ligament
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Anatomy & Physiology
Muller’s Muscle originates at the undersurface of the
aponeurosis sympathetically innervated provides app. 2 mm of eyelid elevation
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Anatomy & Physiology
Lower lid retractors Capsulopalpebral Fascia - analogous to levator
aponeurosis Lockwood’s ligament - analogous to whitnall’s
ligament Inferior tarsal Muscle- analogous to Muller’s
muscle
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Lower Eyelid Anatomy
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Anatomy & Physiology
Tarsus firm, dense plate skeleton of the eyelid
Conjunctiva non-keratinizing squamous epithelium contains goblet cells & acc. Lacrimal glands
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Anatomy & Physiology
Vascular SupplyArterial Supply
ICA - supraorbital and lacrimal artery ECA - angular and temporal artery
Venous Drainage Pretarsal - angular vein (medially); superficial
temporal vein (laterally) Posttarsal - orbital vein
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Anatomy & Physiology
Nerve Supply Sensory
• Supraorbital Nerve (V1)- innervates the forehead and lateral periocular area
• Maxillary Nerve (V2)- innervates lower eyelid and Cheek
Motor• CN III
• CN VII
• Sympathetic Nerves
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Eyelid Lesions
Benign Eyelid Lesions Chalazion Hordeolum Miscellaneous
Malignant Lesions BCCa SCCa
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Cross section of the Eyelid Margin
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Benign Eyelid Lesions
Chalazion - chronic granulomatous inflammation of the meibomian glands.
It is a painless round lesion within the tarsal plate
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Benign Eyelid Lesions
External Hordeolum- infection of the glands of Moll and Zeiss. Usually caused by staphylococcus.
Tender inflamed swelling in the lid margin
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Benign Eyelid Lesions
Internal Hordeolum- acute staphylococcal infection of the meibomian glands.
Tender inflamed swelling within the tarsal plate
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Benign Eyelid Lesions
Treatment Oral Antibiotics Topical Antibiotics Warm compress Surgical: I & C
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Benign Eyelid Lesions
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Miscellaneous Eyelid Lesions
Molluscum contagiosum - pox virus; painless umbilicated nodule
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Miscellaneous Eyelid Lesions
Strawberry Nevus – flat red lesion within 6 months of birth; involute spontaneously
Inc. in size during straining or crying but no pulsation and bruit
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Miscellaneous Eyelid Lesions
Port Wine Stain - nevus flammeus; well demarcated pink patch that darkens with age
45% incidence of glaucoma
5% sturge weber syndrome
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Miscellaneous Eyelid Lesions
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Miscellaneous Eyelid Lesions
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Xanthelasma
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Malignant Eyelid Lesions
Basal cell Carcinoma most common human malignancy 90% of cases occur in head and neck, 10% of
these involved the eyelid most common eyelid malignancy(90% of cases) predilection: lower lid, medial canthus, upper lid,
lateral canthus SLOW GROWING, LOCALLY INVASIVE
BUT NON-METASTASIZING
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Basal Cell Carcinoma
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Basal Cell Carcinoma
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Malignant Eyelid Tumors
Squamous Cell Carcinoma hard nodule or a scaly patch which develops
crusting erosions and fissures over a few months.
clinically, it may be indistinguishable from BCCa but it is important to differentiate the two in view of its metastatic potential of SCC
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Squamous Cell Carcinoma
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Malignant Eyelid Lesions
Treatment: complete excision is a must!
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Malignant Eyelid Lesion
Treatment: Surgical Excision - complete removal of the entire
tumor• Fresh frozen section
• MOH’s technique
• Eyelid reconstruction Exenteration Radiotherapy Cryotherapy
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Disorders of Eyelashes
TrichiasisDistichiasis
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Disorders of Eyelashes
Trichiasis posterior misdirection of previously normal
lashes usually associated with trachoma and severe
chronic staph. Blepharitis
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Disorders of Eyelashes
Trichiasis
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Disorders of Eyelashes
Distichiasis - abnormal row of lashes
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Disorders of Eyelashes
Treatment Epilation Electrolysis Cryotherapy Laser thermoablation
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Entropion
Inversion of the Eyelid4 Types
Involutional
Cicatricial
Congenital
Acute Spastic
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Entropion
Involutional entropion most common and affects only the lower lid
Pathogenesis 1. Overriding of the orbicularis muscle 2. Horizontal lid laxity 3. Weakness of the lower lid retractors
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Entropion
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Involutional Entropion
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Entropion
Treatment1. Cautery 2. Transverse Lid-everting sutures3. Weiss procedure
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Entropion
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Entropion
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Entropion
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Entropion
Cicatricial entropion - usually caused by scarring of the palpebral
conjunctiva, which pulls the lid margin towards the globe
causes: cicatricial pemphigoid, SJ syndromes, trachoma, & chemical burns
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Cicatricial Entropion
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Entropion
Treatment contact lenses, epilation surgical correction
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Entropion
Congenital entropion due to improper development of the retractor
aponeurosis into the inferior border of the tarsal plate
inward turning of the entire lower eyelid and lashes
absence of lower lid crease DDX: Congenital epiblepharon
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Entropion
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Ectropion
outward turning of the eyelidusually associated with epiphora and
conjunctivitisTypes
Involutional Cicatricial Congenital Paralytic
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Ectropion
Pathogenesis Involutional (Senile) - excessive eyelid length;
weakness of the pretarsal orbicularis; laxity of the medial and canthal ligaments
Cicatricial - caused by scarring and contracture of skin and underlying tissues; e.g. trauma, burns, tumors
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Ectropion
Pathogenesis Paralytic Ectropion - facial nerve palsy
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Ectropion
TreatmentInvolutional Ectropion
determined by the position and amount of Horizontal lid Laxity.
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Ectropion
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Ectropion
TreatmentMild Medial Ectropion
Medial Canthoplasty
Severe Medial Ectropion Lazy T- procedure
Extensive Ectropion Bick procedure Kuhnt-Szymanowski procedure
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Ptosis
Drooping of the eyelidsTypes (My NAMe)
Neurogenic Aponeurotic
• Involutional
• Post-operative Mechanical Myogenic
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Ptosis
Neurogenic Ptosis - caused by acquired or congenital innervation defect.
Horner’s syndrome Marcus Gunn jaw winking syndrome Misdirection of CN III
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Neurogenic Ptosis
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Isolated CN III Paralysis
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Ptosis
Aponeurotic Ptosis - defect in the levator aponeurosis. It could be due to disinsertion or stretching.
Involutional Ptosis - degenerative changes in the levator aponeurosis
Post-operative Ptosis - occurs in 5% of patients following intraocular surgery (SR bridle)
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Involutional Ptosis
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Involutional Ptosis
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Ptosis
Mechanical Ptosis physical obstruction
impeding eyelid elevation in the presence of an otherwise normal levator muscle and CN III
E.g. Tumors, deramtochalasis, edema
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Ptosis
Myogenic ptosis congenital or acquired myopathy of the
Levator muscle 2 Types Simple congenital Ptosis Blepharophimosis Syndrome
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Ptosis
Simple Congenital Ptosis may be unilateral or bilateral during downgaze, the ptotic eyelid is higher
than the normal eyelid weakness of the superior rectus (some cases) head tilt with chin elevation high EOR and astigmatism
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Ptosis
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Ptosis
Blepharophimosis syndrome Telecanthus Epicanthus Other features: ectropion, poorly developed
nasal bridge, hypoplasia of the superior orbital rims
Amblyopia 50% of cases
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Ptosis
Blepharophimosis Syndrome
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Ptosis
Clinical Evaluation:
Excellent history taking
Is it a true ptosis or pseudoptosis ?
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Ptosis
Causes of Pseudoptosis
1. Decrease vertical fissure height
2. Contralateral lid retraction
3. Ipsilateral hypotropia
4. Dermatochalasis
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Ptosis
Parameters1. Marginal Reflex distance
NV 4-5mm; Mild +3 Mod. +2 Severe 0 to -1
2. Vertical Fissure height NV male 7-10mm female 8-12mm
3. Levator Function good 12mm; fair 6-11mm poor 5mm or less
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Anatomy and Physiology
Orbit bony cavities : globes, EOM, nerves, fat and
blood vessels pyramidal or conical in shape consists of an apex, a base and 4 sides: roof
floor,medial wall and lateral wall 7 bones: frontal, zygomatic, maxillary,
sphenoid, ethmoid, lacrimal, & palatine
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Anatomy and Physiology
The Bony Orbit:
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Anatomy and Physiology
Roof of the Orbit frontal bone and lesser wing of the sphenoid located adjacent to anterior cranial fossa and
frontal sinus
Lateral wall of the Orbit zygomatic bone and greater wing of the
sphenoid
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Anatomy and Physiology
Orbital Roof
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Anatomy and Physiology
Medial Wall ethmoid, lacrimal, maxillary and sphenoid
bones forms the lateral wall of the sphenoid sinus
Floor of the Orbit maxillary, palatine,& zygomatic bones
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Anatomy and Physiology
Medial Wall
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Anatomy and Physiology
Orbital Apertures1. Optic Canal
Optic Nerve, Ophthalmic Artery, Sympathetic Nerves
2. Superior Orbital Fissure CN III,IV,VI, V1, Sympathetic Nerves
3. Inferior Orbital Fissure CN V2,
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Anatomy and Physiology
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Clinical Evaluation of Orbital Diseases
6 P’s Pain Proptosis Progression Palpation Pulsation Periorbital Changes
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Clinical Evaluation of Orbital Diseases
Proptosis Axial Displacement - retrobulbar lesions like
cavernous hemangioma, glioma, meningioma, AV mal, lesions with in the muscle cone
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Clinical Evaluation of Orbital Diseases
Non Axial Displacement - outside the muscle cone
Superior Displacement - maxillary tumor invading the floor of the orbit
Inferomedial displacement - dermoid cyst and lacrimal gland tumor
Bilateral proptosis Grave’s disease and lymphoma, pseudotumor
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Clinical Evaluation of Orbital Diseases
Progression Days to weeks - inflammatory diseases.
Infectious diseases, metastatic tumors
Months to years - dermoids, benign mixed tumors, lymphomas
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Clinical Evaluation of Orbital Diseases
Palpation superonasal - Mucoceles, neurofibromas dermoids superotemporal - lacrimal gland tumor pseudo
tumor
Pulsations with bruit - CCS Fistula without bruit - meningoencephalocoeles
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Diagnostic Modalities in Orbital Diseases
Primary Studies CT scan MRI Ultrasonography Histopathology
Secondary Studies Venography Arteriography
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Clinical Evaluation of Orbital Diseases
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Clinical Evaluation of Orbital Diseases
CT Scan Good for most orbital
conditions, esp fractures Good view of bone & Ca Degraded image of orbital
apex due to bony artifact Less soft tissue detail Good for metallic foreign
body Less expensive Shorter Scanning time
MRI Better for orbitocranial
lesions No view of bone & Ca Good view of Orbital Apex More soft tissue detail Contraindicated for Metallic
Foreign Body More expensive Longer Scanning time
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Graves’ Ophthalmopathy
Autoimmune disorder that is related to excess secretion of thyroid hormone
10-25% occurs in the absence of any thyroid dysfunction
Female/male ratio 8:14th to 5th decades of lifemost common cause of adult unilateral and
bilateral exophthalmos
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Graves’ Ophthalmopathy
Pathogenesis
1. Hypertrophy of Extraocular Muscles
2. Cellular Infiltration
3. Proliferation of orbital fat, connective tissue
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Graves’ Ophthalmopathy
Main Clinical Manifestation
1. Eyelid retraction
2. Soft Tissue involvement
3. Proptosis
4. Optic Neuropathy
5. Restrictive Myopathy
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Graves’ Ophthalmopathy
Eyelid Retraction
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Graves’ OphthalmopathySoft Tissue
Involvement
1. Conjunctival Injection
2. Chemosis
3. Eyelid Fullness
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Graves’ Ophthalmopathy
Proptosis
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Graves’ Ophthalmopathy
Restrictive Myopathy
IR>MR>SR>LR
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Graves’ Ophthalmopathy
CT Scan EOM
Hypertrophy with tendon sparing
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Key Points in Graves’ Ophthalmopathy
Eyelid retraction is the most common clinical feature; Graves’ ophthalmopathy is the most common cause of eyelid retraction.
Graves’ Ophthalmopathy is the most common cause of unilateral and bilateral proptosis.
Graves’ Ophthalmopathy is 6 more times more common in female than male.
This condition is associated with hyperthyroidism in 90% of cases, but 6% are Euthyroid.
Severity of Ophthalmopathy may not parallel serum levels of T3 or T4. Ophthalmopathy may be asymmetric. Urgent care may be required for optic Neuropathy or severe proptosis If surgery is needed the usual order of surgery is DECOMPRESSION
followed by SQUINT SURGERY followed by EYELID SURGERY
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Orbital Infections
Preseptal Cellulitis Infection confined to the eyelids and periorbital
tissues anterior to the orbital septum Globe is uninvolved, Pupillary rxn, VA, & EOM’s are NORMAL no chemosis, no pain
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Orbital Infections
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Orbital Infections
Orbital Cellulitis active infection posterior to the septum 90% occurs as a 2ndary extension of bacterial
sinusitis fever, proptosis,chemosis, EOM restrictions,
pain on eye movement decrease VA, pupillary abnormalities
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Orbital Infections
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Orbital Tumors
Vascular capillary hemangioma cavernous
hemangioma lymphangioma
Lacrimal Gland Benign Mixed Tumor Malignant Tumor
Rhabdomyosarcoma
Cystic Lesions dermoid cyst mucocele
Neural optic nerve glioma
MetastaticTumor invasion from
adjacent structures
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Capillary Hemangioma
Most common tumor of the orbit in childhood
increase in tumor size during crying and straining
absent bruit and pulsation
involute spontaneously
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Cavernous Hemangioma
Most common benign orbital lesion in adults
middle-aged women commonly affected
enhanced well-encapsulated mass on CT scan
Tx: Surgical Excision
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Rhabdomyosarcoma
Most common primary orbital malignancy of childhood
age-onset is 7-8 y/o rapid onset of proptosis Tx: Exenteration,
Radiation Therapy combined with systemic chemotherapy
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Pleomorphic Adenoma
Most common epithelial tumor of the lacrimal gland
4th -5th decades of life, mostly men
progresssive, painless, downward & inward displacement
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Epidermoid / Dermoid Cyst
Dermoid is a benign cystic teratoma
well-encapsulated lined by stratified squamous & contain dermal appendages
Epidermoid - does not contain dermal appendages
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Fractures of the Orbit
Orbital floor Fracture Most frequently
involve wall Usually along the
infraorbital canal
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Orbital Floor Fracture
Clinical Features Periocular Changes – ecchymosis, edema,
subcutaneous emphysema Enophthalmos Infraorbital nerve anesthesia Diplopia
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Fractures of the Orbit
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Fractures of the Orbit
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Fractures of the Orbit
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Fractures of the Orbit
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Fractures of the Orbit
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Lacrimal System
PunctaAmpullaecanaliculilacrimal sacnasolacrimal duct
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Tear Flow Physiology
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Evaluation of Tearing
Lacrimation vs EpiphoraLacrimation - reflex over production of
tears from stimulation of CN V by irritation of the cornea and conjunctiva
Epiphora - normal tear production but there is physical obstruction on the drainage system
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Infections of Lacrimal Passages
Canaliculits - unilateral epiphora with mucopurulent discharge. “Pouting of the punctum” on slit lamp exam.
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Infections of Lacrimal Passages
Dacryocystitis infection of the lacrimal sac. Presents as a painful swelling at the medial canthal area.
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Surgical Techniques
External DCREndoscopic Laser-Assisted DCRTranscanalicular Endoscopic DCR
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Thank you for your kind attention!