lbp
DESCRIPTION
lbp unpriTRANSCRIPT
Introduction
• Low Back Pain is a symptom• The pathologic basis for the pain may something
within the spine or lesion outside of the spine• Classified as :
– Causes within the spine :• Spondylogenic• Neurogenic
– Causes outside the spine :• Viscerogenic• Vascular• Psychogenic
Spondylogenic Back Pain
Derived from lesions involving
• Bony components spinal column
• Changes in the sacroiliac joints
• Changes in the soft tissues– Discs– Ligaments – muscles
Neurogenic Pain
• This pain can causes by :
– Tension, irritation or compression of lumbar
nerve root
– Lesion of central nerve system such as thalamic
tumors, arachnoid irritation
– Tumors involving the roots, like neurofibroma,
neurilemmoma, ependymoma and others cyste
Viscerogenic Back Pain
• Can cause by disorders of the kidneys,
pelvic viscera, lesions of lesser sac and
retroperitoneal tumors
• The pain is not aggravated by activity nor
its relieved by rest
Vascular Back Pain
• Abdominal aortic aneurysms, peripheral
vascular disease give rise to backache
• Insuff. A sup gluteal buttock pain, that
can ↑ by walking and ↓ by standing still
• Pure psychogenic back pain is rarely
seen in clinical
• Caused by emotional breakdown
Psychogenic Back Pain
Spondylogenic Back Pain : Osseous Lesions
• Pathological processes involving the vertebrae and intervertebral joints
• The pain will minor ↑ by activity, and it is not relieved by rest
• Divided into :– Trauma– Infection– Tumors– Metabolic bone disease (osteoporosis)
INFECTION• Relative rare, but potential source of back
pain
• Divide into :– Vertebral osteomyelitis :
• Pyogenic• Granulomatous (TB)• Miscellaneous
– Epidural abscess– Intervertebral disc infection– Intervertebral disc inflammation
TUMOR• Benign tumors and primary malignancies in the
vertebral column are rare, secondary lesions are common
• Primary tumors of spine can be seen on plain radiographs
• Older patient, more likely primary tumoris malignant
• Most benign tumor are in the posterior elements, except GCT, eosinophilic granuloma and hemangioma
• Most primary malignant tumors will be found in
the vertebral body
• The incidence of neurogical compromise is 75%
in malignant tumors and 40-50% in benign
tumors, due to direct tumor extension,
pathological fractures or skeletal deformity
• The prognosis for malignant tumors is not very
good
• Benign tumors :
– chordoma, osteoid osteoma, osteoblastoma, ABC, GCT,
osteochondroma, hemangioma, eosinophilic granuloma
• Malignant tumors :
– Primary : myeloma, chondrosarcoma, osteosarcoma,
Ewing’s sarcoma, fibrosarcoma and lymphoma
– Secondary : hypernephroma, thyroid, bowel ca (lytic
lesion), breast, prostate and lung tumor (blastic
lesion)
Spondylogenic Back Pain : Soft Tissue Lesions
• Lesions :
– Fibrositis
– Myofascial pain syndromes
– Sacralization of lumbar vertebra
– Disc degeneration
– Disc ruptures
Fibrositis (Fibromyalgia)
• Nonspecific inflammatory changes in fibrous tissue that felt for clinical syndromes of lumbago
• Chronic, widespread, nonarticular, musculoskeletal pain
Myofascial Pain Syndromes• If fibrositis is localized to one area of the
body, such as the low back
Sacralization
• Attachment between the transverse process
of L5 with the sacrum and associated with
back pain
Disc Degeneration
• The natural aging process, with or without
repeated minor episode of trauma, results
in :
– loss of nuclear jelly (because of failure to
reproduce the degradated proteoglycan)
– weakening of annulare support (because of
failure of collagen linking)
• The cascade process :
1. Fibroblasts fail to reproduce new collagen to replace
degradated collagen in the annulus
2. Chondrocytes fail to reproduce new proteoglycan to
replace degradated proteoglycan in the nucleus
3. Nutritional flow of glucose, O2 and sulfates to the
discs is decrease
4. These factors (number 3) change disc metabolism
negatively and likely decrease the pH within the disc
5. Decrease in pH gives upper hand to degrading
enzymes (protease), which further increases disc
degeneration
Disc Ruptures
• Divided into :– Contained :
• Protrusion
• Subannulare extrusions
– Noncontained :• Transligamentous extrusion
• Sequestered
Changes of the Sacroiliac Joints
Sacroiliac Joint syndrome
• Pain over SI joint
• Locally tenderness to palpation
• Referred to groin, trochanter and buttock
• ↑ by provocation test
• ↑ movement of asymmetry of the SI joint
OA degeneration post traumatic SI joint
• Tenderness over lower third SI joint
• Tenderness of pubic symphisis on palpation
• Symptoms reproduced by stressing the SI joint
• Accompanying degenerative changes in symphisis pubis
Treatment• Conservative
– Rest limited activity program, mattress, bedrest– Traction pelvic traction– Acute LBP therapeutic cold,superficial heat(infrared,whirlpool
bath,ultrasound)– Chronic LBP ultrasound, transcutaneous electrical nerve
stimulation (TENS) – Medication morphine, analgesic,AIDS,steroid– Occupational change– Activity of daily living and faulty posture– Low back exercise – Low back support and braces