large bowelobstruction m k alam al maarefa college
TRANSCRIPT
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Large BowelObstruction
M K Alam
Al Maarefa College
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Anatomy
Distal end of Ileum anus (about 1.5 m)
Smallest diameter: Sigmoid colon
(diverticulosisis form here due to the high pressure)
Primary function of the large intestine
Completion of absorption, esp. final absorption of water
Normal flora manufacture certain vitamins- B complex, K
Formation, storage and expulsion of feces
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Large bowel
General characteristics
larger internal diameter
Presence of epiploic appendices
Presence of taeniae coli
Presence of the haustra
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Large bowel obstruction
An emergent condition requires early
identification and prompt surgical
intervention
Possibility of perforation of the distended
colon with risk of fecal peritonitis.
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Pathophysiology of Mechanical LBO
A. Interruption of the flow of the intestinal contents
bowel dilatation above the obstruction mucosal
edema + impaired venous drainage and arterial blood
flow to the bowel (ischemia) :
Mucosal permeability bacterial translocation
+systemic toxicity + dehydration + electrolyte
abnormalities.
Perforation and fecal peritonitis
The process is accelerated in closed loop obstruction
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Pseudo-obstruction (Ogilvie’s syndrome)
Colonic dilatation without anatomical lesion
Multiple medical and surgical illness
PSY and SY activity loss of peristalsis,
distention by gas and fluid
Maximum in the caecum
perforation and fecal peritonitis (3-15%)
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Etiology of LBO
Prevalence increase with age as does its main causes
Neoplasm (benign or malignant) 60%
Stricture (diverticular or ischemic) 20%
Volvulus (colonic, sigmoid or coecal) 10%
Intussusceptions
Adynamic ileus, Ogilvie’s syndrome
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Etiology
Fecal impaction, foreign body
Adhesions
Hernia
IBD
Ped: Hirschsprung’s, meconium ileus, imperforate anus
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Etiology
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Neoplasm and diverticular disease:
Tumor growth luminal narrowing gradual onset of obstruction.
Diverticular disease muscular hypertrophy of the colonic wall with repeated inflammation and fibrosis luminal narrowing
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Colonic volvulus: Twisting of the bowel on its mesentery ischemia
perforation. 20% of the causes of LBO.
A. Sigmoid volvulus:• Common in elderly and frail with long h/ o
constipation and laxatives• Younger Pt- association with high fiber diet.• The twist is ante-clockwise
B. Coecal volvulus: Less common Clockwise twist
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Intussusception
Primarily pediatric disease, usually with no leading point.
Two third of adult intussusception are caused by tumor
Entero-colic Or Colo-colic types
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Acute colonic pseudo obstruction (Ogilvie’s syndrome):
Functional obstruction.
Elderly debilitated patients
Medical (infections, Cardiac disease)
Trauma (operative, non operative).
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Clinical presentation
Complaints suggesting LBOCrampy abdominal painConstipationAbdominal distention Nausea and Vomiting Symptoms suggestive of peritonitis Fistula (passage of air, mucus or feces in
the urine)
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Assessment of symptoms should attempt to distinguish the following:
Acute Vs acute on chronic obstructiononset, H/o bowel movement, stool caliber,
recurrent LLQ pain and weight loss
Complete Vs partial obstruction By symptoms and rectal examination
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Mechanical Vs functional obstruction (illeus or Ogilvie’s syndrome)
In ACPO, symptoms develop over 1-2 days up to 1 week, distention is early sign, fever is a bad sign
Intussusception Recurrent, intermittent colicky pain relevied by
fetal position with weight loss and fatigability
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Physical examination
Complete examination is necessary:
Abdomen: Distension, ? Asymmetrical,
tenderness, ↑ BS, mass
Inguinal and femoral region
Rectum: empty, blood, mass
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Laboratory investigation
CBC
Serum chemistry
Serum lactate
Coagulation profile
Stool for Occult blood
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Plain radiograph
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Contrast radiography with enema
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Contrast enhanced CT
Can distinguish between partial and
complete obstruction and site of obstruction
Gastrographine (water soluble contrast) is
used if bowel perforation is suspected.
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Ca right colon Intussusception
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Management
Initial therapy include: Correction of fluid and electrolytes imbalance with
fluid monitoring Bowel rest NGT Appropriate preoperative antibiotic
Specific management:
Illeus: Tx of the underlying disorder Cessation of drugs slowing colonic motility
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ACPO (Ogilvie’s syndrome) If no perforation conservative Tx, management of the
underlying disorder for 24hr. If failed, consider neostigmine or colonic decompression (success rate 80%)
If perforation or if conservative Tx failed surgical intervention with high mortality and morbidity.
Volvulus: Sigmoiod volvulus:
Sigmoidoscopy and deflation, flatus tube, later elective surgery.
Sig. failed, urgent laparotomy, un-twisting of the loop, per anal decompression followed by either fixation of the sigmoid loop “if viable” or sigmoid colectomy with anastomosis or Hartmann’s procedure.
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Coecal or transverse colon volvulus:
Volvuls should be reduced followed by either fixation (caecopexy) and, or caecostomy.
If the cecum is ischemic or gangrenous right hemicolectomy.
Intussusception:
Children with no peritonitis contrast enema reduction. If failed or with signs of peritonitis surgery Surgery is indicated in adult intussusception why?Recurrence: 3% after contrast & 1%after surgery.
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Hartmann's procedure
Hartmann's procedure is the surgical resection of the rectosigmoid colon with closure of the rectal stump and formation of an end colostomy.
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Colonic masses and strictures: Endoscopic dilatation and stenting of obstructed colon:
Palliative- high risk patients with unresectable tumor preparation for surgery:
Relieve the acute obstruction. Allow time for resuscitation and bowel preparation
Surgery Right colon right hemicolectomy Left colon Hartmann procedure
Diverticular disease: Conservative followed by elective surgery If failed surgery (same principles of Ca tx)
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Prognosis
Depends on:Patient’s factors Underlying disease Management timing and procedures Development of complications
Mortality:Mechanical obst.: 20%-40% (with perforation) ACPO 15%-36% (with ischemia, perforation)
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Colostomies
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Colostomy
Colostomy: a surgical procedure where a portion of the large intestine is brought through the abdominal wall to carry stool out of the body.
Permanent or temporary.
End or loop colostomy
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Indications in Adults
Colorectal carcinoma Colonic Obstruction Traumatic perineal injury Colonic, High anal fistulae Protect a distal anastomosis Diverticular disease Ischemia IBD
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Indications in children
Hirschsprung disease Meconium ileus Imperforate anus Complex hindgut anomalies Volvulus Trauma
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End colostomy
The working end is brought through the abdomen to the skin surface after the damaged /diseased distal bowel is removed
End colostomy can be temporary to allow bowel rest or heal, following tumor resection, traumatic injury or inflammation of the bowel.
End colostomy can be permanent when the distal colon is resected or unresectable
Hartmann procedure involves leaving the distal portion of the colon in place,which is closed to create a Hartmann’s pouch.
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Loop colostomy
A loop of the bowel is brought through the abdomen to the skin surface,
temporarily supported by a plastic bridge/ rod. A communicating wall
remains between the proximal and the distal bowel.
Created in transverse colon (transverse loop colostomy) or in sigmoid
colon (sigmoid loop colostomy)
Typically an emergency procedure to relieve an intestinal obstruction or
perforation.
Opened at the time of surgery or a few days later.
Has two openings through the stoma – the proximal end drains stool
while the distal portion drains mucus.
Loop colostomies are typically temporary.
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Loop colostomy
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Double-barrel colostomy
Both ends of the bowel are brought through the abdomen to the skin surface as two separate sections.
The proximal stoma (colostomy) , diverts feces through the abdominal wall.
The distal stoma (mucous fistula), expels mucus from the distal colon
A double-barrel colostomy may be created because of trauma, tumors, or inflammation, and it may be temporary or permanent.
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Complications
Excessive bleeding
Ischemic stoma
Surgical wound infection
Retraction
Prolapse
Stenosis
Parastomal hernia
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