introduction to movement dysfunction kinetic chain … · decreased gluteus maximus muscle...
TRANSCRIPT
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Introduction to Movement Dysfunction &
Kinetic Chain Assessment
Introduction to Movement Dysfunction &
Kinetic Chain Assessment
Jim Thornton, MA, ATC, NASM-PESHead Athletic Trainer – Clarion University of PA
2006 EATA Annual MeetingPhiladelphia, PA
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Objectives
1. Define common human movement imbalances2. Explain the evidence for common imbalances3. Define Corrective Exercise
a. What is it?b. Why is it used?c. How do you apply it?
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Paradigm Shift• Injury prevention is the primary goal of all
performance enhancement programs– The primary emphasis of traditional “Sports
Conditioning” programs has been on concentric force production (how much can you lift) primarily in the sagittal plane, in a controlled environment
• This may not be the best way to prevent injuries
– Also, many athletes have pre-existing injuries that must be addressed prior to advanced performance enhancement training
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Foot and Ankle Imbalances• Lateral ankle sprains are the most common
injury suffered in sports during sports participation (Safran, 1999)– Denegar, et al 2002 in a retrospective study
demonstrated altered arthrokinematic movement of the talus (decreased posterior glide) even though range of motion was restored
– This is very important because limited posterior glide of the talus may lead to decreased dorsiflexion
• Limited dorsiflexion may lead to other compensations
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Foot and Ankle Imbalances• Bullock-Saxton et al 1994 demonstrated
decreased gluteus maximus muscle activation post ankle sprain
• Beckman et al 1995 demonstrated decreased gluteus medius muscle activation post ankle sprain– If an athlete begins an integrated training program
and has muscle imbalances in the hip complex secondary to an ankle sprain, then further compensations and possible injury may occur
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Knee and Hip Imbalances• It has been recognized that the patellofemoral
joint may be influenced by the segmental interactions of the lower extremity (Fredericson, Powers)– Abnormal motions of the tibia and femur in the
transverse and frontal planes are believed to have an effect on the patellofemoral joint (Ford, Nyland)
– This abnormal motion may be caused by weakness in the hip abductors and external rotators (Ireland)
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Knee and Hip Imbalance• Recent kinetic analysis of running reveals that,
although the knee joint primarily moves in the sagittal plane, the knee is also subject to significant frontal and transverse plane moments (McClay)– In the absence of sufficient proximal hip strength, the femur
may adduct and internally rotate, further increasing the lateral patellar contact pressure (Lee)
– (Fredericson) demonstrated that distance runners with ITB Syndrome had weaker hip abduction strength than the control group and their unaffected leg
– (Ford and Hewett) demonstrated that female athletes landed with greater total valgus (femur adduction and tibia abduction) than male athletes and may lead to ACL Tears
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Lumbo-Pelvic-Hip Complex Imbalance• Low back pain is very common in the active
population (Nadler)– In a cross-sectional study of 100 patients (Cibulka)
demonstrated unilateral hip rotation ROM asymmetry in patients with SI joint regional pain
– Hodges and Richardson 1996 reported that slow speed of contraction of the transverse abdominus during arm and leg movements was well correlated with LBP
– O’Sullivan et al 1997 found that synergist substitution of the rectus abdominus for the agonist transverse abdominusduring the abdominal drawing-in maneuver suggesting less efficient intersegmental stabilizing mechanisms and greater shear forces at the intervertebral joints
– Hides et al 1994 demonstrated unilateral atrophy of the multifidus in patients with low back pain
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Current Concepts in Human Movement Science
• Two distinct yet interdependent muscle systems– Stabilization System (Stabilizers)
• Primarily involved in joint support• Broad spectrum of attachments• Prone to inhibition and weakness
– Movement System (Mobilizers)• Superficial muscles associated with extremity movement• Prone to overactivity and tightness• Categorized into four common sub-systems
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Understanding Muscle Function
• Stabilizers– Gluteus Medius– Transverse Abdominus– Internal Oblique– Multifidus– Lower Trapezius– Serratus Anterior– Rotator Cuff– Deep Neck Flexors
• Mobilizers– Gastrocnemius– Quadriceps– Hamstrings– Adductors– Hip Flexors– Rectus Abdominus– Erector Spinae– Latissimus Dorsi
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Understanding Muscle Function
• Stabilizers– Delayed recruitment– Reacts to pain and
pathology with inhibition– Loss of joint
stabilizations– Leads to synergistic
dominance
• Mobilizers– Become overactive– Reacts to pain and
pathology with spasm– Develops myofascial
adhesions which alter (LTR, ATK)
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Lateral Sub-System• Muscles Involved
– Gluteus Medius– Tensor Fascia Latae– Adductor Complex– Quadratus Lumborum
• Function– Frontal plane stability
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Posterior-Oblique Sub-System• Muscles Involved
– Latissimus Dorsi– Thoracolumbar Fascia– Gluteus Maximus
• Function– Transverse plane
stabilization
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Anterior-Oblique Sub-System• Muscles Involved
– Internal Oblique and Hip Adductor Complex
– External Oblique and Hip External Rotators
• Function– Transverse plane
stabilization
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Deep Longitudinal Sub-System• Muscles Involved
– Erector Spinae– Thoracolumbar Fascia– Gluteus Maximus– Biceps Femoris– Peroneals
• Function– Force transmission from
the ground to the trunk
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Kinesiopathological Model
• If one component of the human movement system is not functioning optimally, than PREDICTABLE PATTERNS of dysfunction will develop and initiate the cumulative injury cycle
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Myofascial
Neuromuscular Control
Sensorimotor Integration
Articular Neural
Kinetic Chain
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Length-tension Relationships• There is a direct
relationship between tension development in a muscle and the length of the muscle– There is an optimum
length at which a muscle can generate maximum tension
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Force-couple Relationships
• Muscles work synergistically to reduce force, dynamically stabilize and concentrically produce force in all three planes of motion
• The CNS is designed to optimize the selection of muscle synergies
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Arthrokinematics• Articular partners have
predictable movement patterns– Roll– Slide– Glide– Translation
• These patterns are controlled by the CNS and the surrounding muscles of the kinetic chain
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Altered Length-tension Relationships
Initiation of theCumulativeInjury Cycle
Tissue Fatigue
Altered SensorimotorIntegration
Altered Force-Couple Relationships
AlteredArthrokinematics
DYSFUNCTION
Altered NeuromuscularEfficiency
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Results from Human Movement System Impairment
• Reciprocal Inhibition• Synergistic Dominance• Arthrokinetic Inhibition• Relative Flexibility• Pattern Overload
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Reciprocal Inhibition
• Increased neural drive or decreased extensibility of an agonist will decrease the neural drive to the antagonist– Leads to synergistic
dominance
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Synergistic Dominance
• The NMS phenomenon that occurs when synergists and stabilizers compensate for prime movers during functional movement patterns
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Arthrokinetic Inhibition
• The process of inhibition that occurs from lack of proper joint arthrokinematics
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Relative Flexibility
• The Human Movement System will take the path of least resistance
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Pattern Overload
• Repetitive recruitment of the same muscle fibers, in the same ROM/Plane of motion and at the same speed creates tissue overload and eventually injury
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Kinesiopatholocigal Model
Low Back Extension
Lengthened Shortened Joint Dysfunction
Gluteus MaximusAbdominal Complex
Erector SpinaeHip Flexors
LumbarSI-Joint
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Integrated Training
General Guidelines:• Identify all kinetic-chain imbalances.
• Correct all kinetic chain imbalances
• Develop proper structural integrity of the kinetic chain before activity-specific training.
• Integrate functional movements in the plane of motion, range of motion and speed of motion that replicates the training activity
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What is the Solution?
Identify Causative Factors
Correct Weak Links
ReconditionBigger Engines or Better
Brakes
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ICE: Integrated Corrective Exercise• Identify the kinetic chain Imbalance responsible for the
movement inefficiency and the biomechanical overload
• Correct the Imbalance– Inhibit the overactive
• Self-Myofascial Release– Lengthen the overactive
• Static Stretching– Activate the under-active
• Active-Isolated Muscle Activation – Integrate functional movement patterns
• Integrated Isolation
• Empower your client– Give your client an individualized corrective exercise plan– Give your client an individualized Fitness and/or Performance
Enhancement Program
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Identifying the Weak Link:• Posture• Gait• Flexibility assessment• Neuromuscular assessment• Overhead-squat test• Single-leg balance excursion• Single-leg squat test• Multiplanar lunge test• Multiplanar step-up test• Push-up test• Overhead medicine-ball
throw
• Multiplanar vertical jump/hop
• Multiplanar horizontal jump/hop
• Shark skill test• Multiplanar cone jump/hop
test• Speed tests
– Straight-ahead speed – Lateral speed and agility – Sport-specific – Speed endurance
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Transitional Movement Assessment
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Single-leg Squat Assessment
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The Overhead Squat
1. Feet
2. Knees
3. Hips (Lumbar Spine)
4. Shoulders
5. Head
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Normal Movement Assessment
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Can You Guess the Chief Complaint?
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What are Contributing Factors?
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Foot & Ankle: Foot Turns Out
Normal Abnormal
Foot Turns Out: Note the 1st MTP Joint in relation to the medial malleolus. In a normal foot the 1st MTP joint will appear along the same plane as the medial malleolus. However in a foot that is turned out the 1st MTP joint will appear lateral to the medial malleolus.
StraightExternal Rotation
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Foot & Ankle: Foot Flattens
Normal Abnormal
Foot Flattens: Note the height of the longitudinal arch of the foot. It should be in a neutral position with a slight curve distinguishable and if the foot flattens it will appear to be flat along the floor. Another indicator of the foot flattening is the Achilles tendon. Note in the neutral picture how the tendon is straight, however when the foot flattens note the lateral angle that is produced by the Achilles tendon.
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Foot & Ankle: Heel of Foot Rises
Normal Abnormal
Heel of Foot Rises: Note the heel of the foot rising off of the floor. If the heel stays firmly planted on the floor then there is no abnormality. However any rise of the foot from the floor indicates an abnormal movement pattern.
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Foot & Ankle: Single-Leg Foot Flattens
Normal Abnormal
Foot Flattens Single-leg: Note the height of the longitudinal arch of the foot. It should be in a neutral position with a slight curve distinguishable and if the foot flattens it will appear to be flat along the floor. Another indicator of the foot flattening is the medial malleolus will appear larger than the lateral malleolus. In the neutral picture both medial and lateral malleolus are even.
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Knee: Moves Inward
Normal Abnormal
Knee Moves Inward: Note a line drawn from the patellar tendon which bisects the ankle. This line should be perpendicular to the ground. If the line is leaning toward the midline of the body then the knee is moving inward.
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Knee: Moves Outward
Normal Abnormal
Knee Moves Outward: Note a line drawn from the patellar tendon which bisects the ankle. This line should be perpendicular to the ground. If the line is leaning away from the midline of the body then the knee is moving outward.
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Knee: Single-leg Moves Inward
Normal Abnormal
Single-leg Knee Moves Inward: Note a line drawn from the patellar tendon which bisects the ankle. This line should be perpendicular to the ground. If the line is leaning toward the midline of the body then the knee is moving inward.
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Knee: Single-leg Moves Outward
Normal Abnormal
Single-leg Knee Moves Outward: Note a line drawn from the patellar tendon which bisects the ankle. This line should be perpendicular to the ground. If the line is leaning away from the midline of the body then the knee is moving outward.
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LPHC: Low Back Rounds
Normal Abnormal
Low Back Rounds: Take notice of the area from approximately the mid back through the Sacral Complex. If the area is rounding then this area will appear as a thoracic or convex curve.
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LPHC: Low Back Arches
Normal Abnormal
Low Back Arches: Take notice of the area from approximately the mid back through the Sacral Complex. If the area is arched then this area will appear with an excessive lumbar or convex curve.
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LPHC: Excessive Forward Lean
Normal Abnormal
Excessive Forward Lean: Imaginary lines that are created by the shins and torso of the client if extended out should remain parallel. If these lines would cross immediately or shortly after extending them then the person does have excessive forward lean.
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LPHC: Weight Shift
Normal Abnormal
Weight Shift: Taking a line extending from the cervical spine through the thoracic and lumbar spine that is parallel to the ground should bisect the LPHC resulting in equal parts falling on either side of the line. If the LPHC is split unevenly resulting in a larger percentage on one side of the line then there is a weight shift on the side of the line that has the larger percentage of the LPHC.
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LPHC: Single-leg Lateral Hip Shift
Normal Abnormal
Single-leg Lateral Hip Shift: Taking a line originating from the patellar tendon and bisecting the quadriceps should be parallel to the ground. If the line moves away from the midline then there is a lateral hip shift.
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Upper Body: Arms Fall Forward
Normal Abnormal
Arms Fall Forward: A line bisecting the torso and head should be noted. If this line travels parallel along the arms and the arms cover the ears of the subject then there are no abnormalities present. If the line does not parallel the arms and you can see the ears then the arms have fallen forward.
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Dynamic Assessments
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Valid and Reliable Tests• Orthopedic Assessment
(Magee)• Muscle length
assessment with Goniometer (Brosseau, Norkin)
• Single-leg Balance Excursion Test (Olmsted)
• Overhead Medicine Ball Throw (Stockburger)
• Vertical Jump (Manske)• Single-leg Vertical Hop
(Manske)• Horizontal Jump
(Manske)• Single-leg horizontal
hop (Manske)
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So, once you find it…how will you address it?
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ICE: Integrated Corrective Exercise• Identify the kinetic chain Imbalance responsible for the
movement inefficiency and the biomechanical overload
• Correct the Imbalance– Inhibit the overactive
• Self-Myofascial Release– Lengthen the overactive
• Static Stretching– Activate the under-active
• Active-Isolated Muscle Activation – Integrate functional movement patterns
• Integrated Isolation
• Empower your client– Give your client an individualized corrective exercise plan– Give your client an individualized Fitness and/or Performance
Enhancement Program
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A Deeper Look at Dysfunction
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ICE: Integrated Corrective Exercise• Identify the kinetic chain Imbalance responsible for the
movement inefficiency and the biomechanical overload
• Correct the Imbalance– Inhibit the overactive
• Self-Myofascial Release– Lengthen the overactive
• Static Stretching– Activate the under-active
• Active-Isolated Muscle Activation – Integrate functional movement patterns
• Integrated Isolation
• Empower your client– Give your client an individualized corrective exercise plan– Give your client an individualized Fitness and/or Performance
Enhancement Program
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Where are they going now?• A comprehensive training approach that strives
to improve all components necessary to allow each individual to achieve optimum performance (Clark 2000, Kraemer 2004)– Flexibility– Core Strength– Neuromuscular Efficiency– Power– Strength– Cardiorespiratory Efficiency
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Summary• Must appreciate movement as a whole when looking at postural
& functional assessments• Identify the weak link• Provide isolated exercise to increase motor unit recruitment
– Can only recruit motor units to the degree of dynamic joint stabilization • Integrate to CNS by:
– Multiplanar exercise selection– Variable speeds– ROM– Resistance (mode, frequency)– Acute variables (reps, sets, intensity, tempo, rest interval)
• Progress in training systematically to safely achieve goals
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Further Reading Suggestions• Professional Development
– Kinesiology of the Musculoskeletal System (Neumann 2002)– Diagnosis and Treatment of Movement Impairment Syndromes
(Sahrmann 2001)– Foundations of Clinical Research, 2nd ed (Portney & Watkins 1999)– Evidence-Based Medicine, 2nd ed (Sacket 2000)– Performance Enhancement Specialist (NASM 2001)– California University of Pennsylvania’s online, 12-month MS degree
• Personal Development– How Full is Your Bucket (Rath 2004)– The Leadership Challenge (Kouzes & Posner 2002)– Patch Adams (1998: 1 hr 56 min)