ecg workshop
TRANSCRIPT
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ECG Workshop
Dr. Zohair Alaseri, MD
FRCPc, Emergency Medicine
FRCPc, Critical Care Medicine
Assistant Professor
Chairman, Department of Emergency Medicine
King Saud University Hospitals, Riyadh, KSA
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60 Y. O. F. presented with sever palpitation and waeaknessArrested in 3 minutes from arrival to your department
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Severe hyperkalemiaprolonged PR interval and
widened QRS complex
Arrest ECG
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60 year old with sever crushing, heavy chest pain started 45 m. agowith BP of 85/45
Family want to know what is going on & what do you want to do for their relative??
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Elderly man with this rhythm and with previously normal ECG??State your intervention please??
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Electrical Alternans
Spot DX please????D/D???
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Wolff-Parkinson-White pattern
Explain what do you see????
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The short PR interval is due to a bypass track, also known as the Kent pathway. By bypassing the AV node the PR shortens .
The delta wave represents early activation of the ventricles from the bypass tract. The fusion QRS is the result of two activation sequences, one from the bypass tract and one
from the AV node .The ST-T changes are secondary to changes in the ventricular activation sequence .
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54 year old lady history of hyperthyroidism presented with palpitation and sweating
Med inderal and digoxin & carbimazol
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50 y.o. m. with sever submental pain,redness and tenderness ass with chest pain
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50 y.o. m. with sever submental pain,redness and tenderness ass with chest pain
“danger” (post to retropharyngeal space from skull base to diagphragm)
Peritonsillar, Parapharyngeal (contains carotids, jug vein, cerv symp chain and CN IX XII)
Retropharyngeal (skull base to mediastinum – T2)
Prevertebral spaces (skull ,coccyx)
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Prolonged QT
Elderly bed ridden man with increased weakness & fatigabulity with BP of 90/50
What do you want to give??
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The deep S waves in the inferior leads and a left axis deviation indicates Left Anterior hemiblock+ poor R wave progression .
70 y o m sever chest typical ischemic pain for 30 minutes
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The rhythm is atrial flutter sawtoothed pattern.
The conduction ratio is variable between 12:1 and 6:1
60 year old lady with suden onset of Palpitation
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PR interval is short at about 100ms. QRS is broad even running into the P wave in lead II. Initial slurring of the QRS is evident. This is a case of WPW, and it seems to be intermittent with a recovery to normal in the last 3 beats of the rhythm strip.
18 year old with sudden onset of sever dizziness for 15 minutes
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Anteroseptal InfarctionThe elevated S-T segements in leads
V1,V2,V3
69 y o f with sever pleuritc chest pain and diaphoresis for 30 minutes PMH DM IHD HTN
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acute anterolateral myocardial infarctio & left anterior hemiblock
65 year old male with sever chest pain radiating to his back similar to his pain when he had MI 1 year agoHis bp is 90/30
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Complete heart block third degree AV block & PVC QS complexes are commonly found in leads V1, and V2 as a normal variant.
45 year o. f. head of emergency department with sever headache, nausea & vomiting
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Mobitz type II AV block
80 y. o. 2 hours after streptokinase for anterior wall MI
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The ventricular rate is 75. The atrial rate is 150SVT with a 2:1 block. P waves are inverted in the inferior limb leadsThis points towards a low atrial location as the site of the ectopy. This is most likely a paroxysmal atrial tachycardia with block
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Right bundle branch block & left anterior hemiblock
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Wide QRS complex tachycardia. ventricular tachycardia
The QRS complex is wider than 140 ms. The R:S ratio in lead V6 is less than 1.
77 y o m with palpitation history of MI 3 years ago
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Nodal idiojunctional
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30 year old male sudden loc No PMHHis brother died at age of 39
Brugada
down-sloping Coved type or less commonly saddle-back type ST segment elevation followed by inverted T waves
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Second-degree AV block
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Mobitz type I AV block
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Ventricular fibrillation
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Multifocal atrial tachycardia
different P wave morphologies
45 y. o. m. heavy smoker with worsening SOB
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Atrial flutter with 2:1 AV conduction.
Atrial flutter with 4:1 AV conduction.
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Atrial fibrillation with ventricular preexcitation
S. rhythm with ventricular preexcitation.
17 year old with sudden palpitation
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Sinus arrhythmia.
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Multifocal atrial tachycardia.
The presence of at least three distinct P-wave morphologies and varying P-P, R-R, and PR intervals
MAT
7 year old with diarrhea and BP of 50/29
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65 YEAR OLD WITH SYNCOPE AND HYPOTENSION
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WPW Type Preexcitation
19 year old with on/off palpitation
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There are ectopic P wave before them; therefore these are PAC's with RBBB aberration
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LBBB and 2nd degree AV Block, Mobitz Type II
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RBBB + LAFB = Bifascicular block-KH
The LAFB is recognized by the marked left axis deviation (-75 degrees) in the frontal plane, rS complexes in II, III, aVF, and the tiny q-wave in aVL.
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RBBB plus Mobitz II 2nd Degree AV Block
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In this rhythm strip of sinus arrhythmia, the faster rates have a LBBB morphology. In some patients with a diseased left bundle branch, the onset of LBBB usually occurs initially as a rate-dependent block; i.e., the left bundle fails to conduct at the faster rate because of prolonged refractoriness
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Marked widenening of the QRS duration combined with tall, peaked T waves are suggestive of advanced hyperkalemia. Note the absence of P waves, suggesting a junctional rhythm, but in hyperkalemia the atrial muscle may be paralyzed while still in sinus rhythm. The sinus impulse conducts to the AV node through internodal tracts without activating the atrial muscle
Advanced Hyperkalemia
50 year old with pulmonary edema
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Lateral Myocardial Infarction
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•A23-year-old female was brought to the emergency department by ambulance after a "fainting spell."
•She felt acutely light-headed, weak and thought she was "going to pass out."
•She felt better approximately 5 minutes later, after lying down.
•There was no loss of consciousness and no evidence of seizure activity.
•She thought her heart might have been racing during the episode.
•She had never experienced any similar episodes in the past.
Case
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PMH revealed a 3-year history of anorexia nervosa.
Over the past 4 weeks she had significantly reduced her oral intake and had lost approximately 20 lbs. Her present weight was 90 lbs.
She smoked half a pack of cigarettes a day and denied recent alcohol intake.
Case
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Examination revealed a thin, pale woman in minimal distress.
Lying HR 86 beats/min BP 102/60 mm Hg.Standing HR 95 beats/min BP 90/58 mm Hg.
She was quite thin and had decreased muscle mass, No other pertinent physical findings were noted.
Case
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BUN 9.8 mmol/LCr 110 µmol/LCa 0.7 mmol/LMg 2.09 mmol/Lphosphate 1.8 mmol/L.
LabHGB of 110 g/L Na130 mmol/LCl 91 mmol/LHCO3 28 mmol/LK 3.0 mmol/L
Case
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During her ED stay the patient experienced another brief episode of "light-headedness" with palpitations. A rhythm strip was obtained
Case
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The most likely cause of the patient's symptoms is:
1. Orthostatic hypotension 2. Wolff-Parkinson-White syndrome 3. Sinus bradycardia 4. Ventricular arrhythmia secondary to prolonged QT
syndrome
Case
Prolonged QT syndromeProlonged QT syndrome
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60 year old female presented with sever dizziness and sweating started 2 hours ago
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20 minute later C/O C_P
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65 y. o. m. p. with typical CP.
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(TCA ingestionsinus tachycardia widened QRS complex, deep S wave in lead I,and prominent R wave in lead aVR.
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Ventricular paced rhythm with underlying complete heart block
infero-postero-lateral MI.
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A 68-year-old man presented with chest pain radiating to the left arm of 3 hours’ duration .
He was diaphoretic and pale.
ST-segment depression, prominent R wave, and upright T waves in leads V1 to V3 in addition, ST-segment depression was seen in the inferior and lateral leads
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V7, posterior axillary line;
V8, posterior scapular line;
V9, left border of spine.
Posterior Leads
All in the same horizontal plane of V4
to V6
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ST-segment elevation in leads V8 and V9 minimal ST-segment depression with large R waves in the right
precordial leads (V1 to V3), confirming posterior wall AMI .
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•Inferior STE & reciprocal STD •STE in lead V1 is typical for RV AMI • widespread STE
Inferior & RV MI
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V1R is same as standard V2,
V2R is the same as standard V1,
V3R is halfway between V2R
V4R, V4R is fifth intercostals space at right midclavicular line, V5R is same level as V4R in right anterior axillary line
V6R is same level in right midaxillary line
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• A. fib with subtle signs of acute inferior (MI) with ST elevations intermittently apparent in leads II, II and aVF.
• The right-sided chest leads show evidence of concomitant (RV) MI with slight ST elevations in leads V4R to V6R.
70-yr-old man presents with severe chest pain, nausea, vomiting and diaphoresis. Right-sided chest leads are shown (i.e., V1R to V6R).
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SVT
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Multifocal atrial tachycardia.
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Wide complex tachycardia: rate-related BBB.
After adenosine became
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VT
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Ventricular Tachycardia.
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Ventricular tachycardia with AV dissociation. (arrows denote regular P waves, although some are lost in the intervening complexes).
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Wide complex tachycardia. A fusion beat (arrow) is seen in the rhythm strip;
its presence confirms VT.
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Accelerated idioventricular rhythm.
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Wolff Parkinson White syndrome and atrial fibrillation.
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(A) Hypercalcemia shortened QT interval.prominent U waves (arrows) (B) Normocalcemia. This is baseline ECG from the same patient as in (A) when her
serum calcium level was normalized (7.8 mg/dL).
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Hypocalcemia. prolonged QT interval. The deeply inverted T waves were present on the baseline ECG.
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(A) Hyperkalemia. B) Resolving hyperkalemia..
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Sine wave rhythm of hyperkalemia. The patient was administered IV calcium gluconate, causing an immediate narrowing of the QRS complex.
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Hyperkalemia. minimal narrowed peaking of the T waves.
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Hypokalemia. demonstrating T-wave flattening and prominent U waves (arrows)
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Thank You
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