eating disorders
DESCRIPTION
Eating DisordersTRANSCRIPT
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EATING DISORDERS
A. CRITERIA FOR ANOREXIA NERVOSAAccording to the latest Diagnostic and Statistical Manual of Mental Disorders Fourth Edition (DSM-IV) criteria:
• Refusal to maintain body at or above a minimally normal weight for age and height.
• Intense fear of gaining weight• The central concern of weight and shape in the evaluation of the self, in
addition to a reference to the denial of the serious consequence of weight loss.
• Amenorrhea.
SUBTYPES:• The restrictor type • The binge purger type
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B. CRITERIA FOR BULIMIA NERVOSADiagnostic criteria for Bulimia Nervosa:• Recurrent episodes of binge eating . • Recurrent inappropriate compensatory behavior to prevent weight gain.• The binge eating and inappropriate compensatory behaviors both occur,
on average, at least twice a week for 3 months.• Self evaluation is unduly influenced by body shape and weight.• The disturbance does not occur exclusively during episodes of anorexia
nervosa.• SUBTYPES:• Purging• Non purging
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C. ORAL MANIFESTATIONS
• Severe erosion of the enamel on the lingual surfaces of the maxillary teeth - cardinal oral
manifestation of eating disorders, due to acids from chronic vomiting.
• Parotid enlargement - as a result of starvation• Mandibular teeth may be affected.• Palatal ulcerations
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D. Management
• Refer to other practitioners.• Support of the patient physically, by treatment
of tooth desensitization and esthetics.• Support of the patient psychologically, by
demonstrating a caring and compassionate attitude.
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PLASMA CELL NEOPLASIA
• I. DEFINITION • Is a group of related malignant disorders of terminally differentiated B
lymphocytes (plasma cells), of which plasma cell myeloma or multiple myeloma is the most common (90% of cases).
• This condition is characterized by bone marrow multifocal infiltration by malignant plasma cells. There are typically multiple destructive lesions or diffuse demineralization of bone. The tumour cells secrete a homogenous, complete or partial, immunoglobulin molecule, an M component or para protein, most commonly IgG or IgA.
II. ETIOLOGY• Genetic predisposition• Ionizing radiation • Chronic antigenic
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III. ORAL MANIFESTATION
• Maxillary or mandibular lesions • Root resorption and loss of lamina dura may
be found.• Amyloid like deposition in the gingiva and
tongue.
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IV. DENTAL MANAGEMENT
• Patients are prone to postoperative hemorrhage due to thrombocytopenia.
• Development of dental amyloidosis.• Dental hygiene care should focus on controlling infections
associated with a compromised immune system.• Patients being treated with bisphophonates must be
monitored closely for the development of osteonecrosis of the jaw.
• Educate patient about the necessity of reporting unusual sores or painful areas in the mouth.