devin peterson, md, frcsc, dip sport med associate professor, mcmaster university mcmaster...
TRANSCRIPT
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THE LIMPING CHILDAN ORTHOPEDIC
PERSPECTIVE
Devin Peterson, MD, FRCSC, Dip Sport Med Associate Professor, McMaster University
McMaster University Medical Centre
David Braley Sport Medicine & Rehabilitation Centre
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Faculty/Presenter Disclosure
• Faculty: Devin Peterson• Program: 51st Annual Scientific Assembly
• Relationships with commercial interests:• None
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Disclosure of Commercial Support
• This program has received no financial support • This program has received no in-kind support
• Potential for conflict(s) of interest:– None
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Mitigating Potential Bias
• N/A
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OBJECTIVES
1. To assess and diagnose common causes of childhood limping
2. To understand the management principles of the limping child including timely referral
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NORMAL GAIT
Smooth energy-efficient transfer of the body through space
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Limp: “to walk with a halting or irregular step”
Funk & Wagnall's
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PATHOLOGICAL GAITS
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Antalgic Gait: body’s effort to compensate for pain or instability in the stance-phase limb by minimizing the duration and magnitude of loading
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Trendelenburg Gait: leaning of the head and trunk toward the lower extremity affected by the pathologyPainWeakness in the hip muscles
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COMMON CAUSES OF LIMPING SEEN IN EARLY CHILDHOOD
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19 month old female referred because of limping
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Fifth born Normal delivery/presentation
Walking at 14 monthsAlways limped
No pain
Healthy Negative Family history
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Differential diagnosisTop three:
Hip dysplasiaNeuromuscular diseaseLeg length discrepancy
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DEVELOPMENTAL DYSPLASIA OF THE HIP
(DDH)
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Dislocated: the femoral head is not in contact with the
acetabulum
Dislocatable the femoral head is within the acetabulum but can be
forced out
Subluxed the femoral head is within the acetabulum but not in
its proper position
Subluxable the femoral head can be moved beyond its physiologic
limits within the acetabulum
Dysplastic although the femoral head is in the proper position the
acetabulum or head are abnormally developed
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HistoryRisk Factors
FemaleFirstbornBreechLarge babyLow amniotic fluidFamily history
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PhysicalAssociated
Conditionso Foot deformity,
Torticolliso Neuromuscular
disorderso SyndromesHip Examinationo Trendelenburg gaito Skin foldso Galeazzi signo Abductiono Provocative
maneuvers- Ortolani, Barlow
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ImagingUltrasound
< 6 monthsX-ray
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Treatment URGENT REFERRAL Pavlik Harness Closed Reduction
Safe Zone Open reduction
Extra-articular blocks Intra-articular blocks
Osteotomies Pelvic Femoral + Shortening
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Most common cause of hip pain in childhood 3% childhood risk
Idiopathic Frequently associated with
concurrent or antecedent illness Right hip = left hip
NEVER BILATERAL 2:1 male:female
TRANSIENT SYNOVITIS
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HistoryAge varies (9 months to
adolescence)Most between 3 and 8 years old
Unilateral hip painCan present with knee or thigh pain
Limp vs non-weight bearing
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PhysicalMay have a low grade temperatureAntalgic or Trendelenburg gaitFlexed and externally rotated
positionDecreased ROM
Especially abduction and internal rotation
Laboratory testsNon-specific
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ImagingRadiographs usually normalUltrasound may show effusion
Diagnosis of exclusion
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TreatmentURGENT REFERRAL
Differential includes a septic jointSelf limiting disorder
May have symptoms for up to 10 days or longer
Bed rest until full ROM, no pain, limp freeCrutches for older patients
NSAIDSGradual return to activity
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LEGG-CALVE-PERTHES DISEASE(PERTHES)
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Self limiting hip disorder Caused by ischemia and subsequent
necrosis of the femoral head Usually 4 to 8 years of age Male to female ratio:
4-5 to 1 Usually unilateral (88%) Age and lateral head involvement are
the key to prognosis 8 years of age seems to be the
watershed <50% of lateral pillar involvement
better prognosis
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DIFFERENTIAL DIAGNOSIS
Unilateral Perthes: septic arthritis sickle cell disease spondyloepiphyseal dysplasia
tarda Gaucher’s disease
Bilateral Perthes: Hypothyroidism Multiple epiphyseal dysplasia spondyloepiphyseal dysplasia
tarda sickle cell disease
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HistoryMay be painless at first
present with a limp onlysymptoms occur with subchondral
collapse/fractureCan present with knee or thigh pain
Positive family history 1.6% – 20%1% - 3% of patients with transient
synovitis will develop Perthes
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Physicalgait:
Trendelenburgdecreased
abduction/ internal hip rotation
thigh, calf, and buttock atrophy
LLD
ImagingX-ray, bone scan,
MRI
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TreatmentTIMELY REFERRALPrinciples of treatment are
maintenance of ROM & containment (good coverage of the head by the acetabulum) of the femoral head through the evolution of healing
May be obtained by non-operative means
o relative restopain controlophysiotherapyo tractionoabduction splinting at night
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OPERATIVE TREATMENTS
Containable Hipadductor releaseFemoral
varus/pelvic osteotomies
Non-Containable HipHip/Late-presenting
patient with deformity
Hinge abduction Chiari/lateral shelf Cheilectomy Femoral
abduction/extension osteotomy
OCD, non-operative, revascularization, removal, ORIF
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DISCOID MENISCUS First description: Young 1889 3-5% in general population have a
larger than normal meniscus Almost all in lateral, but reported in
medial 20% bilateral and 10% associated
with OCD of lateral femoral condyle
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ANATOMY
THREE SEGMENTS• Anterior horn• Body• Posterior horn
Attached to tibial plateau, primarily through Coronary Ligament
Attached to the capsule except at popliteal hiatus
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DIAGNOSIS
Clinical PresentationHistory:
Asymptomatic“Snapping knee syndrome”Meniscal tear symptoms
Physical: Snapping knee with gaitMeniscal signs
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X-rayWidened lateral joint
space, squaring of lateral femoral condyle, cupping of lateral tibial plateau
MRIVerify diagnosis and
assess damage
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TREATMENT OPTIONS Asymptomatic: observe Symptomatic:
TIMELY REFERRAL UNLESS LOCKED KNEE THEN URGENT
Non-operative: restricted activity, bracing, physiotherapy
Operative:Partial meniscal “saucerization” Repair of tear
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COMMON CAUSES OF LIMPING SEEN IN LATE CHILDHOOD
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APOPHYSEAL CONDITIONS
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Apophysitis of the Hip and Pelvis Sinding-Larsen-Johansson: inferior
pole of patella Osgood-Schlatter Disease: tibial
tuberosity disturbance Sever Disease: calcaneal apophysitis Iselin Disease: apophysitis of the
fifth metatarsal
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OSGOOD SCHLATTER DISEASE
Tibial tuberosity disturbancePartial avulsion (microscopic
fractures) of the ossification center and overlying hyaline cartilage
Epidemiology10 – 15 years oldBoys > girls> 10% of teenagers
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HistoryPain localized to tubercleWorse with direct blows to the are
and activity
PhysicalAntalgic gait may be presentProminent tubercle + local swellingTenderness localized to tubercle
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Lovell and Winter’s Pediatric Orthopaedics 5th edition
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TreatmentSpontaneous resolution at maturity
20% may have pain with kneelingsurgery for loose ossicles
ReassuranceSymptomatic treatment/activity
modificationNSAIDS, stretching, knee
pads/braces, foot orthosis, castsTIMELY REFERRAL
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OSTEOCHONDRITIS DISSECANS(OCD)
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INTRODUCTION
Acquired potentially reversible lesion of subchondral bone resulting in delamination and sequestration with or without articular cartilage involvement and instability
Juvenile and Adult formsAdult form is typically progressive
and unremittingMay occur in almost any joint in
upper or lower extremityVery common in the knee
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EPIDEMIOLOGY
15-29 per 100,000May be bilateral in 25% of casesMale: female ratio
5:3>70% are in the classical area
Posterolateral aspect of the medial femoral condyle
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ETIOLOGY
IdiopathicTheories include:
Genetics Inflammation IschemiaOssificationRepetitive trauma (stress reaction
causing a stress fracture in the underlying subchondral bone)
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CLINICAL PRESENTATION
History Juvenile
Poorly localized pain Exacerbated by
exercise May present with
symptoms of instability (swelling, stiffness, catching, locking)
Limp
PhysicalAntalgic gaitEffusionCrepitusPainful ROMQuads atrophyMaximum tenderness
usually anteromedial knee
Wilson sign Pain with internal
tibial rotation
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DIAGNOSTIC STUDIES
X-raysAPLateralNotch
MRI + gadoliniumLesion sizeStatus of the cartilage and subchondral boneBone edema and high signal zone beneath
fragmentLoose bodies
Technetium bone scan
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MANAGEMENTNonoperative
Open physis = good prognosis?Activity modification
Immobilization?RehabilitationLocal and systemic pain
managementReview every 3-6 months or sooner if
symptoms worseningRepeat MRI every 6 months?TIMELY REFERRAL if no improvement
or worsening, URGENT IF LOCKED KNEE
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Operative Indications
Lesions not responding to nonoperative management
Unstable lesions?Detached lesions
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THE PAINFUL FLATFOOT:TARSAL COALITION
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HISTORY
Tarsal coalition is an abnormal connection between some of the tarsal bones May be painful Can be associated with increased
ankle sprains
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PHYSICAL
GaitAntalgic
FlexibilityToe standingSitting/supine
Subtalar ROM
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Flexible Flatfoot Arch returns with
sitting or tiptoe standing
Normal subtalar and midtarsal motion
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Tarsal Coalition Arch may not
return with sitting or tiptoe standing
May be painful to move or palpate subtalar joint or other tarsal bones
Subtalar motion often decreased
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IMAGING
Normal in flexible flatfoot
Oblique views and Harris view may help view a coalition
May need an MRI or CT to make diagnosis
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TREATMENT Tarsal Coalition
TIMELY REFERRALRest/activity modificationAntiinflammatoriesPhysiotherapy?OrthoticsCastsSurgery: resection or fusion
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SLIPPED CAPITAL FEMORAL EPIPHYSIS(SCFE)
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The slip normally occurs during adolescent growth phase
Mechanical or systemic factors may be presentCommonly obeseEndocrinopathies
(eg. 1o & 2o hypothyroidism, panhypopituitarism, GH, hypogonadal conditions, & renal osteodystrophy
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Male > female
Left > right
Bilateral involvement may occurSecond slip presents within 18 months
in 88%
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HistoryChronic and/or acute
Limp
May present with knee or thigh pain instead of hip/groin pain
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PhysicalGait: TrendelenburgShortened/external rotationDecreased abduction/internal hip
rotationPassive flexion leads to thigh
abduction and external rotation
ImagingX-ray, CT, MRI
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X-rays Physeal plate
widening & irregularity Decrease in
epiphyseal height Blanch sign of Steel
Crescent-shaped area of increased density in the proximal femoral neck
Femoral metaphysis appears laterally displaced Klein’s line
Southwick angles
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Imaging
Frog-leg lateral avoid in acute
situationCross-table lateral
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Treatment - Acute EMERGENT REFERRAL Immediate bed rest Insertion of one or
more screws in situ fixation
Designed to fuse the epiphysis on the metaphysis to prevent further slipping
Prophylactic Pinning
Known metabolic/endocrine disorders?
Inability to follow-up
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STRESS FRACTURES
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STRESS FRACTURES
Stress Fractures in Skeletally Immature Patients
Walker et. al.: JPO 1996
34 stress fractures Tibia (47%), fibula, femur, radius,
humerus, MT
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HistoryPain often associated with an
increase in activityBe wary of female triad
PhysicalAntalgic gait may be presentTenderness localized
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RadiographsRapid bony response
may be present Bone Scan
Helpful in questionable situations
Treatment (depends on causative factors)URGENT REFERRALModification of activitiesImmobilization
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COMMON CAUSES OF LIMPING SEEN AT VARIOUS AGES
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BONE AND SOFT TISSUE TUMORS
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BONE AND SOFT TISSUE TUMORS History
PainNight painHistory of trauma may delay
diagnosisOsteoid Osteoma pain relieved by
NSAIDsConstitutional Symptoms
Fever, night sweats, anorexia, weight losseg. Ewing sarcoma
Soft tissue mass may not be symptomatic
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Physical ExamGait disturbanceMuscle atrophyNeurovascular examRange of motionMass
Size, tenderness, pulsation, mobility, bruits, tenderness, erythema, consistency
Lymph nodes
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InvestigationsBloodwork
CBC, ESR, CRP, serum alkaline phosphatase, serum and urine calcium & phosphorus, LDH
ImagingX-rayBone ScanCT/MRI
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ManagementReferral
Urgency dependent on tumor type
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SEPTIC JOINTS
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HistoryPain
Refusal to bear weightLimping
Recent illnessDecreased immunity
eg. chickenpoxTrauma
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Physical ExamTemperatureAntalgic gaitDisuse of a partErythema/swellingTendernessDecreased ROM
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Laboratory tests CBC
WBCCRPESRBlood culturesAspirates (Gram
stain, Culture)
ImagingX-raysUltrasoundBone ScanCTMRI
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TreatmentEMERGENT REFERRALStop tissue destruction ASAP
Decrease bacterial load and irrigation of the joint
Identify the OrganismSelect appropriate antibiotic
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