common pathologies of the integumentary system ©irene mueller, edd, rhia montana hospital...
TRANSCRIPT
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Common Pathologies Common Pathologies of the Integumentary Systemof the Integumentary System
©Irene Mueller, EdD, RHIA ©Irene Mueller, EdD, RHIA
Montana Hospital AssociationMontana Hospital AssociationJune 20, 2012June 20, 2012
http://etc.usf.edu/clipart
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Objectives Objectives • Review Integumentary System Pathologies
for ICD-10-CM/PCS Coding– ICD-10-CM
• Chapter 12 (L00-L99) – Diseases of Skin & SubQ• Chapter 19 (S00-T88) – Injury, Poisoning, & Other
consequences of External Causes• Chapter 2 (C00-D49) – Malignant Neoplasms
– ICD-10-PCS• 0 - Medical & Surgical Section• H – Skin and Breast• J – SubQ tissue and fascia
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Skin Chapter vs Other ChaptersSkin Chapter vs Other Chapters• Certain Skin Conditions in Other ICD-10
Chapters– Complications of Preg, Childbirth, Puerperium– Congenital– Infectious– Neoplasms– Parasitic– Perinatal– Systemic connective tissue disorders
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OutlineOutline• General Skin Lesions• Inflammations• Infections
– Abscess– Cellulitis– Furuncles/Carbuncles– Impetigo– Pilonidal cyst/sinus– Warts
• Lesions– Bullous– Cicatrix– Corns/Calluses– Other
• Abnormal Skin Pigmentations
• Pressure Ulcers, Ulcers• Scalded Skin Syndrome• Hair Pathologies– Alopecia– Folliculitis• Nail Pathologies– Paronychia• Tumors– B9, Malignant• Injuries– Burns, Wounds
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General Skin lesionsGeneral Skin lesions
• Bulla• Burrow• Comedo• Comedone• Crust• Cyst• Ecchymosis• Erosion• Eschar
• Excoriation• Fissure• Freckle• Induration• Keloid• Lichenification• Macule• Mole• Nodule
• Papule• Petechia• Plaque• Purpura • Pustule• Scale• Scleroma• Telangiectasia• Ulcer• Vesicle• Wheal
• WOW: Check a good medical dictionary for pictures of these;• Should know differences between them
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Patterns & Distribution of Lesions Patterns & Distribution of Lesions • Annular • Discrete• Clustered• Confluent• Dermatomal,
Zosteriform• Eczematoid• Follicular• Guttate• Iris/Target
• Koebner Phenomenon,
Isomorphic response• Linear Lesions• Morbilliform• Multiform• Reticular• Satellite lesions• Scarlatiniform• Serpiginous• Strawberry tongue• Universalis
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Skin Conditions and StressSkin Conditions and Stress
• Many conditions
– Aggravated by stress
– Cause of stress
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Infections of Skin/SQ (L00-L08)Infections of Skin/SQ (L00-L08)• Abscess/Furuncle/Carbuncle
• Scalded Skin Syndrome
• Impetigo
• Cellulitis and Acute Lymphangitis
• Acute Lymphadenitis
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Bullous Disorders (L10-L14)Bullous Disorders (L10-L14)
• Bulla = blister, bleb
• Latin bulla (pl. bullae), "bubble"
• Circumscribed elevation of skin
• 0.5 cm+ diameter containing a liquid– Vesicle = <0.5 cm in diameter
• Acantholytic disorders
• Pemphigoid
• Pemphigus
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Acantholytic DisordersAcantholytic Disorders
• Gk akantha, thorn or prickle, & lysis, loosening
• Primary disorder of skin (sometimes mucous membranes) – characterized by loss of cohesion between
epidermal cells, resulting in intraepidermal clefts, vesicles, and bullae
– Inflammatory, viral & autoimmune skin diseases, particularly pemphigus complex
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Dermatitis (L20-L30)Dermatitis (L20-L30)• Inflammation of skin• Many types/forms
– Acute– Subacute– Chronic
• Manifestations– Pruritus, Erythema– Cutaneous Lesions
• Most common– Atopic (Eczema)– Contact– Seborrheic
http://www.nlm.nih.gov/medlineplus/ency/imagepages/19323.htm
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Papulosquamous Disorders Papulosquamous Disorders (L40-45)(L40-45)
• Skin eruption – Both papular (pimple-like) AND scaly
• Psoriasis
• Parapsoriasis
• Pityriasis rosea
• Lichen planus
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Urticaria and Erythema (L50-L54)Urticaria and Erythema (L50-L54)
• Urticaria– Many types
• Erythema– Multiforme– Marginatum– Many other types
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Radiation-Related Disorders (L55-59)Radiation-Related Disorders (L55-59)
• Actinic keratosis– AKA Solar keratosis
• Radiodermatitis – AKA radiation dermatitis
• Sunburn– 1st
– 2nd – 3rd
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Disorders of Skin Appendages Disorders of Skin Appendages (L60-L75)(L60-L75)
• Nail Disorders– Ingrowing Nail– Onycholysis
• Hair Disorders– Acne– Rosacea
• Sweat Gland Disorders
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Hair PathologiesHair Pathologies
• Alopecia areata• Androgenic alopecia• Nonscarring hair loss• Cicatricial alopecia• Hair color/shaft
abnormalities• Hypertrichosis• Acne• Rosacea
• Follicular cysts• Eccrine sweat disorders• Apocrine sweat disorders
http://etc.usf.edu/clipart
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Nail PathologiesNail Pathologies
• Ingrowing nail
• Onychia/Paraonychia - Gk, onyx, nail
• Onycholysis
• Onychogryphosis
• Psoriasis
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Intraop/Postop Complications Intraop/Postop Complications of Skin (L76)of Skin (L76)
• Hemorrhage/hematoma of skin
• Accidental puncture/laceration of skin
• Postprocedural hemorrhage/hematoma
• Other information– Dermatologic procedure vs other procedure
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Other Disorders of Skin/SQ Other Disorders of Skin/SQ (L80-L99)(L80-L99)
• Vitiligo• Pigmentation
Disorders• Seborrheic keratosis• Pressure Ulcers• Atopic Disorders of
skin• Hypertrophic
Disorders of skin
• Lupus erythematosus• Vasculitis limited to
skin• Non-pressure chronic
ulcers• Keloid• Corns & Calluses
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Abscess/Furuncle/CarbuncleAbscess/Furuncle/Carbuncle
• Abscess - collection of pus & infected material in or on skin – anywhere on body
• Furuncle (Boil)– Abscess involving hair follicle and Subq tissue
• Carbuncle– Very large boil OR multiple furuncles joined by
many drainage canals
• Carbunculosis– Many carbuncles
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Acantholytic DisordersAcantholytic Disorders
• Acquired keratosis follicularis– NOT congenital– NOT Darier Disease
• Transient acantholytic dermatosis– Etiology – Unknown– Itchy red spots on trunk– Usu. older men– Usu. Lasts 6-8 mos,
can be much longer– AKA Grover disease– Tx: Cortisone cream,
Accutane, Tetracycline, Phototx, antifungals, Cortisone Injections
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Actinic Keratosis (L57.0) Actinic Keratosis (L57.0)
• Small, rough, raised area found on skin– In sun for long period of time – can become SCC
• RF: Fair skin, Blue/green eyes, Blond or red hair
• Kidney or other transplant
• Take medicines to suppress immune system
• Lot of time each day in sun
• Many, severe sunburns early in life
• Older
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Actinic KeratosisActinic Keratosis
• Face, scalp, back of hands, chest, or other sun-exposed areas– Flat, scaly areas – Color -gray, pink, red, or same color as skin – Often, white/yellow crusty "scale" on top– Later – develops hard & wart-like or gritty,
rough, surface– May be easier to feel than see
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Acne vulagisAcne vulagis
• L aknas, misreading of Gk. akmas, "points, dots” L. vulgaris “common”
• Inflammatory disease of sebaceous glands & hair follicles– Overproduction of sebum, faster shedding of skin
cells = plug and growth of bacteria in follicles
• Papules, putules, comedones, nodules, possible scars– Face, neck, shoulders, chest, back
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Acne vulgarisAcne vulgaris• Unknown etiology
• Hormonal changes
• Hereditary tendencies
• Any age, most common in Adolescents – 14-17 Girls– Late teens Boys
• Precipitating factors– Food allergies, carbonated drinks, endocrine
disorders, psychological factors, fatigue, steroid drugs
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Acute LymphangitisAcute Lymphangitis
• Bacterial infection in lymphatic vessels– Streptococcus pyogenes most often; Staph
• “Flesh-eating” bacterium
– Characterized by painful, red streaks below skin
– Potentially serious/fatal infection• Can rapidly spread to bloodstream
– Cut, scratch, insect bite, surgical wound, other skin injury
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Acute Lymphadenitis Acute Lymphadenitis
• Inflammation of lymph node– Neck, arms, or legs; chest or abdomen– Often complication of bacterial wound infection
• Hemolytic Streptococci or Staphylococci
– Caused by viruses/other disease agents also• Viruses, protozoa, rickettsiae, fungi, tuberculosis
– Generalized: number of lymph nodes– Localized: limited to few nodes in infection area– Sometimes accompanied by lymphangitis
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AlopeciaAlopecia
• Loss of hair• 2 main types • Alopecia areata = body's immune system
mistakenly attacks hair follicles Androgenetic alopecia = inherited– Head hair thins & falls out
• Men = male pattern hair loss• Women = female diffuse hair loss
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Carbuncle (L02)Carbuncle (L02)• Usu. caused by Staphylococcus aureus
– Contagious– Back, Nape of Neck, Anywhere– Friction from clothing, shaving– More common in Men than Women
• Composed of several skin boils (furuncles)
• Infected mass filled w/ fluid, pus, & dead tissue – up to golf ball size– May or may not drain
• RF: DM, dermatitis, weak immune system
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Cellulitis (L03)Cellulitis (L03)• Acute, diffuse, bacterial infection
– Infection spreads over 2 days– Deep Skin and SubQ– Lower extremities most often, anywhere
• Signs– Erythema, pitting edema– Skin is tender and hot– Red lines/streaks PROXIMAL
• Along lymph vessels to lymph glands
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CellulitisCellulitis• Streptococcus or Staphylococcus
– Small cut/lesion– Enzymes from bacteria
• Break down skin cells• Prevent body responses
• Can lead to – Abscesses– Bacteremia
• Tx: Antibiotics (IV)
http://www.nlm.nih.gov/medlineplus/ency/imagepages/8743.htm
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Corns & Calluses (L84)Corns & Calluses (L84)
• Localized, hyperplastic areas – Stratum Corneum of
Epidermis
• Calluses AKA Tylosis– Larger (up to 1 inch) – Ball of foot, palms
• Corns AKA Clavus– Smaller, on toes – Glassy core– More painful
• Pressure/friction– Shoes– Orthopedic deformity– Faulty weight-bearing– Repeated trauma
• (Musicians)
• Impaired Circulation– Foot– Peripheral Neuropathy– DM
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Dermatitis, Atopic (Eczema) (L24)Dermatitis, Atopic (Eczema) (L24)• Inflammation of skin
– Patients w/ family hx of allergic conditions– Vesicular eruptions
• Exudative in children• Dry, Leathery in adults
– Characteristic pattern• Face, neck, elbows & knees, upper trunk
– Pruritis
• Idiopathic• Allergic connection assumed
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Dermatitis, Atopic (Eczema) Dermatitis, Atopic (Eczema) Gk, ekzein, to boil overGk, ekzein, to boil over
• Infants– Sensitivity to milk, OJ, other foods
• Flare-up Triggered by– Stress, anxiety, conflict– Wool clothing, blankets– Frequent bathing/handwashing
• May improve in summer
• Tends to improve with time
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Dermatitis, Contact (L23-24)Dermatitis, Contact (L23-24)
• Acute inflammation of skin– Irritants on surface– Allergic reaction– Erythema, edema, small vesicles, itching, stinging
• Causes: Poison Ivy, Oak, Sumac; Dyes, Latex, Furs, Preservatives, Drugs, Detergents, Cleaners, Cosmetics, Chemicals, Acids, Metals, Sunshine, Tanning Beds (ICD-10-CM specific codes)
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Dematitis, Seborrheic (L21)Dematitis, Seborrheic (L21)
• One of most common skin conditions
• Inflammation of Oil glands in skin– Areas with greatest # of glands– Scalp, eyebrows, eyelids, sides of nose,
behind ears, middle of chest– Skin is reddened, covered with scales
(yellowish, greasy-looking)– May be itching, usu. mild
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Dermatitis, SeborrheicDermatitis, Seborrheic
• Any age; most common during infancy– Cradle cap
• Clears w/in 8-12 months (no tx)
• Adults w/ CNS disorders (ex. Parkinson)• Adults w/ stress
– AMI, long LOS in hospital/nursing home– Immune system disorders (AIDS)– Psoriasis
• Idiopathic; heredity predisposition• Tx: Corticosteroids; dandruff shampoos
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Furuncle (L02)Furuncle (L02)
• Usu. caused by Staphylococcus aureus – Other bacteria or fungi found on skin surface
• Face, neck, armpit, buttocks, thighs, anywhere
• One OR many boils
http://upload.wikimedia.org/wikipedia/commons/9/95/Furuncle-MIN-IMG_2589.jpg
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Impetigo (L01)Impetigo (L01)
• Superficial skin infection– Staphylococcus aureus or Streptococcus– Insect bites, scabies, poor hygiene, anemia, malnutrition,
eczema (loss of skin integrity)– Highly contagious, Warm weather– Face, arms/legs, anywhere
• Vesicular/Pustular lesions, – Rupture -Form thick, yellow crusts
• Children, 2 – 6 yo
MedlinePlus
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Ingrowing nail (L60.0)Ingrowing nail (L60.0)
• Edge of nail grows down/into skin of toe– Can be pain, redness, and swelling around nail– Cause: usu. Poorly fitting shoes & improperly
trimmed nails
• Dangerous for DM
http://www.nlm.nih.gov/medlineplus/ency/article/001237.htm
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Lupus erythematosus (L93.0)Lupus erythematosus (L93.0)
• AKA Local, Discoid, Chronic Cutaneous Lupus (CCLE), Discoid Lupus (DLE)
• Chronic skin disease – Primarily women 20 – 40– Characterized by eruption of red lesions over
cheeks & bridge of nose– Scarring w/ altered pigmentation
• Exacerbated by trauma & sunlight• Tx: Sunscreen, Topical Corticosteroids
– Systemic Agents
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Onychia/Paraonychia (L03.0)Onychia/Paraonychia (L03.0)• Onychia
– AKA onychitis – Inflammation of finger/toenail matrix; may lose
• Paraonychia– Superficial infection of epithelium lateral to nail plate– Acute More painful (usu. Bacterial)– Chronic (often several nails) (usu. Fungal)
• Culture of exudate to determine etiology
– RF – Hands in water for long periods
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Onycholysis (L60.1)Onycholysis (L60.1)• Spontaneous separation of nail plate
– Starting at distal free margin & progressing proximally– Beginning at proximal nail & extending to free
• Most often in psoriasis of nails (onychomadesis)
– Just at lateral borders (Rare)
• Etiology– Endogenous, exogenous, hereditary, &idiopathic
Contact irritants, trauma, & moisture– Associated with many other diseases
• Tx: Depends on Cause
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Onychogryphosis (L60.2)Onychogryphosis (L60.2)
• Gk, onyx + gryphein, to curve
• Abnormal hypertrophy & curving of nails
• Gives a claw-like appearance
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Parapsoriasis (L41)Parapsoriasis (L41)
• 2 general forms– Small plaque type, usu. B9 – Tx: emollients, topical tar, steroids, phototx– Large plaque type, precursor of cutaneous T-
cell lymphoma (CTCL). – Tx: phototherapy or topical corticosteroids
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Parapsoriasis (L41.x)Parapsoriasis (L41.x)
• No uniform, accepted definition• Group of skin diseases characterized by
– Scaly patches OR– Slightly elevated papules and/or plaques – Resemblance to psoriasis
• BUT includes several inflammatory cutaneous diseases unrelated in pathogenesis, histopathology, & tx response
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Pemphigoid vs PemphigusPemphigoid vs Pemphigus
• Separate autoimmune diseases
• Both have sub types
• Both cause burn-like blisters
• Both very rare; can’t be cured
• PemphigoiD = Deep PemphiguS = Superficial
• Pemphigoid more common, less dangerous – Tends to affect older people, usually 60+
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Pemphigoid (12.9)Pemphigoid (12.9)
• Gk pemphix, blister
• Autoantibodies – – Against basement membrane of epidermis – Not against epidermal cell junctions
• Intact, tense bullae up to 3 cm– Trunk, arms, & legs– Rare in mouth
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Pemphigus (L10)Pemphigus (L10)
• Group of disorders caused by auto-antibodies against some part of epidermis – Including Oral mucosa– Lead to disruption of intercellular junctions and
blisters– Ruptured bullae covered with scabs– Tx: Immunosuppressive agents– Prognosis is variable– Many patients - higher than normal mortality rate
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Pemphigus Risk FactorsPemphigus Risk Factors
• Possible triggers
• Non-steroidal anti-inflammatory drugs such as ibuprofen
• Drugs derived from penicillamine
• Herpes simplex virus
• Exposure to sunlight
• Long-term stress
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Pilonodal Cyst/SinusPilonodal Cyst/Sinus• Latin - pilus (hair); nidus (nest) nest
• Abnormal pocket in skin, usu. contains hair, skin debris & other abnormal tissue
• Almost always located near tailbone at top of buttocks cleft (small pit)
• Affect men more often; most commonly in young adults
• When becomes infected (abscess)– causes pains, inflammation & usu. drainage of
fluids
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Pilonidal Cyst (L05)Pilonidal Cyst (L05)
• Risk Factors– Obesity, Inactive lifestyle– Occupation or sports requiring prolonged sitting– Excess body hair; Stiff or coarse hair– Poor hygiene; Excess sweating
• When chronically infected pilonidal cyst isn't treated properly– Increased risk of developing squamous cell
carcinoma.
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Pilonidal SinusPilonidal Sinus• Hair may protrude from tract below skin’s
surface that connects infected pilonidal cyst to opening on skin's surface – More than one sinus tract may form
• Tx: I&D, currettage – May be left open to heal
MedlinePlus
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Psoriasis, Psoriasis, Gk - itchGk - itch• Non-infective
• Thick, flaky, various-sized red patches– Covered with white, silvery scales
– Scales progress to plaques, sometimes pustules
• Chronic condition, most common bet. 10-30 yo
• Scalp, outer arms/legs, trunk, palms/soles, nail beds (nails thicken, crumble)
• Genetic basis
• Maybe autoimmune
• Whites - more common
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Psoriasis, Arthropathic (L40.50)Psoriasis, Arthropathic (L40.50)
• Several different forms – Small distal joints, fingers & toes– Joints on one side of body only– Bilateral larger joints – Back and spine– psoriatic arthritis mutilans (rare) - joints & bones
destroyed (gnarled,club-like hands & feet) (L40.52)
• Symptoms of psoriasis usu. Precede arthritis– Clue to possible joint disease is pitting and other
changes in fingernails
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Psoriasis (L40)Psoriasis (L40)
• Precipitating factors– Hormonal changes, climate changes,
emotional stress, poor health
• Remissions and Exacerabations
• Guttate psoriasis – Rare form– Frequently follows Strep infection– L, drop
MedlinePlus
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Psoriasis and NailsPsoriasis and Nails• Nail changes occur in
– Up to 50 % of psoriasis patients– Up to 80 % psoriatic arthritis patients
• Most common Nail problems in psoriasis– Pitting—shallow or deep holes in nail– Deformation—alterations in normal shape of nail– Thickening of nail– Onycholysis—separation of nail from nail bed– Discoloration—unusual nail coloration, such as
yellow-brown
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Radiodermatitis (L58)Radiodermatitis (L58)
• Cutaneous inflammatory reaction– Acute or Chronic– Exposure to biologically effective levels of ionizing
radiation
• Most common example of normal tissue damage as a result of radiotherapy – About 95% of radiation oncology patients – 87% moderate to severe radiodermatitis
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RadiodermatitisRadiodermatitis• Includes
– Localized erythema & edema Grade 1– Skin shedding (desquamation)– Hair loss (epilation)– Fibrosis– Necrosis (ulcers/hemorrhages) Grade 4
• Late effects (90 days to years after tx)– Result of permanent damage to dermis – Atrophy, fibrosis, telangiectasias, & pigmentation
changes
• Tx: Depends on Grade
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Rosacea Rosacea L, rosaceus, rosyL, rosaceus, rosy
• Facial skin redness, where blushing occurs
• Insidious onset– Mistaken for sunburn or acnes
• Becomes more noticeable /does not go away
• Dryness, pimples (pustular)
• Small blood vessels dilate
• Knobby bumps on nose, looks swollen– Usu. Male patients
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Rosacea (L71)Rosacea (L71)
• Unknown etiology– May be inherited
• Lighter complexions – higher risk• Not infectious/contagious• Triggers vary from person to person
– Sunlight, hard exercise, extreme heat/cold, stress, spicy foods, hot drinks, alcohol, hot/cold weather, wind
• Tx: topical antibiotics, Finacea, laser surgery, sunscreens
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Scalded Skin Syndrome (L00)Scalded Skin Syndrome (L00)
• AKA - Ritter disease; Staphylococcal scalded skin syndrome (SSS)
• Infection - skin becomes damaged & sheds– certain strains of Staphylococcus– Toxin causes skin damage– Blisters as if skin were scalded
• Most commonly in infants & children under 5
• Tx: IV antibiotics and fluids
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Seborrheic Keratosis (L82)Seborrheic Keratosis (L82)• AKA Seborrheic
Wart• B9 skin tumor• Usu. After age 40• Runs in families• S&S• Often waxy surface• Round-to-oval• “Pasted-on"
appearance
• S&S, cont.• Located on face, chest, shoulders,
back, or other areas• Usu. painless, but may irritate &
itch• Yellow, brown, black, or other
colors• Slightly raised, flat surface; rough
or wart-like texture• May be single, but usually many
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Sunburn (L55)Sunburn (L55)• Severity determined by depth of burn• 1st
– Damage to epidermis– Erythema (Pain, Redness, Swelling)
• 2nd
– Damage to Dermis; Erythema, Blisters
• 3rd degree– Damage to Hypodermis & Nerve supply– Numbness; then scars– Loss of sensation/function possible
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Burn SeverityBurn Severity
http://www.nlm.nih.gov/medlineplus/ency/imagepages/1078.htm
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UlcersUlcers
• L, ulcus, a sore
• Circumscribed, craterlike lesion of he skin or mucous membrane
• Resulting from necrosis accompanying some inflammatory, infectious, or malignant processes
• Produced by sloughing of necrotic inflammatory tissue
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Ulcers, Non-Pressure (L97)Ulcers, Non-Pressure (L97)
• Arterial ulcers
• Venous stasis ulcers
• Diabetic ulcers
• Neurotrophic ulcers
• NOT pressure ulcers
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Arterial ulcersArterial ulcers• Complete or partial arterial blockage
– Causes tissue necrosis and/or ulceration of extremity
• AKA Ischemic ulcer
• Large/small vessel; toes, ankles, fingers
• Risk Factors– Peripheral vascular disease (PVD)
– Diabetes mellitus
– Advanced Age
• Tx: Surgical or Medical to increase circulation
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S&S of Arterial UlcerS&S of Arterial Ulcer• No pulse in area of extremity
• Painful ulceration Cool or Cold skin
• Small, punctate ulcers; usually well defined
• Delayed capillary return time– Briefly push on end of toe and release– Normal color should return to toe in <3 seconds
• Atrophic appearing skin (shiny, thin, dry)
• Loss of digital & pedal hair
• Frequently on dorsal foot; occur anywhere
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Venous stasis ulcersVenous stasis ulcers
• AKA Varicose Ulcers
• Venous Stasis– Cessation or impairment of venous flow– AKA phlebostasis, venostasis
• Location – Below knee – usu. Inner part of leg, just above
ankle. Can be uni/bilateral
• Tx: Bed rest, elevation, & pressure bandages– Antibiotics if needed for infection
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Venous stasis ulcers (I83)Venous stasis ulcers (I83)
• Common in patients– w/Hx of leg swelling– Varicose veins– Hx of blood clots in superficial OR deep leg veins
• Affect 500,000 - 600,000 people in US yearly
• Account for 80 to 90 % of all leg ulcers
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Diabetic ulcers (E08-E11)Diabetic ulcers (E08-E11)
• Most common foot injuries leading to lower extremity amputation– Risk of LE amputation - 15 - 46 times higher in
diabetics– Early detection/appropriate treatment of diabetic
ulcers may prevent up to 85 % of amputations
• Ulcers almost always form in patients with neuropathy– Typically painless
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Neurotrophic ulcersNeurotrophic ulcers
• Occur primarily in Diabetics – see Diabetic ulcer– 60 - 70 % of Diabetics - mild to severe forms of
nervous system damage
• Can affect anyone w/ impaired sensation in feet – Predominantly sensory neuropathy, Sensory-
motor neuropathy, or Autonomic neuropathy– Essential cryoglobulinemic vasculitis
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Ulcers, Pressure (L 89)Ulcers, Pressure (L 89)• Localized injury to skin and/or underlying
tissue– Usually over bony prominence– Result of pressure, or– Pressure in combination w/shear and/or friction
• 6 stages– NPUAP, 2007
• Tx: Decrease pressure, friction and shear
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Ulcers, PressureUlcers, Pressure
• AKA • Bed Sore• Decubitus ulcer• Plaster ulcer• Pressure area• Pressure sore• Slough [sluf] Layer/mass of dead tissue
separated from surrounding living tissue• Eschar - hard crust or scab
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Pressure Ulcer StagesPressure Ulcer Stages
• Suspected Deep Tissue Injury– Purple or maroon
localized area of discolored intact skin or blood-filled blister
– Due to damage of underlying soft tissue from pressure and/or shear
• Stage I– Intact skin w/ non-
blanchable redness of localized area
– Usu. Over bony prominence
– Darkly pigmented skin may not visibly blanch
– Color may differ from surrounding area
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Pressure Ulcer StagesPressure Ulcer Stages
• Stage II– Partial-thickness loss
of dermis– Shallow open ulcer w/
red pink wound bed, w/o slough
– May also present as an intact or open/ruptured serum-filled blister
• Stage III– Full-thickness tissue loss– SQ fat may be visible but
bone, tendon or muscle are NOT exposed
– Slough may be present but does not obscure depth of tissue loss
– May include undermining and tunneling
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Pressure Ulcer StagesPressure Ulcer Stages
• Stage IV– Full thickness tissue
loss w/ exposed bone, tendon or muscle
– Slough or eschar may be present on some parts of wound bed
– Often includes undermining & tunneling
• Unstageable– Full thickness tissue
loss in which base of ulcer is covered by slough (yellow, tan, gray, green or brown)
– and/or eschar (tan, brown or black) in wound bed
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http://www.nlm.nih.gov/medlineplus/ency/imagepages/19092.htm
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Risk Factors for Pressure Risk Factors for Pressure UlcersUlcers
• Age – Reduced skin cell turnover, drier skin, reduction of
collagen & elastin, & reduced vascularity at dermal/epidermal junction cause• Skin less resistant to shear forces• Increased surface area (potential fluid loss
increase)• Increased traumatic injury risk (e.g. skin tears)• Decreased homeostasis• Reduction of barrier function
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RF for Pressure UlcersRF for Pressure Ulcers• Thin Body
– More prone to develop pressure ulcers over bony prominences
• Nutrition & Hydration– hypoalbuminemia, weight loss, cachexias,
dehydration, and malnutrition– commonly reported risk factors predisposing persons
to pressure ulcer.– Pressure ulcers can generate stress response, which
further adds to nutritional needs
• Lower dietary protein intake
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RF for Pressure UlcersRF for Pressure Ulcers• Inability to feed oneself• Particular Medical Conditions
– Diabetes Malnutrition– Altered mental status– Spinal cord injury Orthopedic injury– Depression Vascular disease
• Anaerobic waste products – believed to accumulate due to pressure-induced
occlusion of lymphatic vessels & contribute to tissue necrosis
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PU Assessment ToolsPU Assessment Tools• Braden Scale
– Most researched, validated, & nationally recognized tool
– Comprehensive – May not have same
predictive value & accuracy in LTC
– Not readily used in outpatient clinic setting
– View tool at• www.bradenscale.com/
images/bradenscale.pdf
• Norton Scale– Short, straightforward,
easy to complete– Does NOT address
nutrition – View tool at
• http://www.ruralfamilymedicine.org/educationalstrategies/braden_scale_for_predicting_pres.htm
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Unavoidable Pressure UlcersUnavoidable Pressure Ulcers• CMS definition in LTC• “An unavoidable pressure ulcer occurs when
facility staff evaluated resident’s clinical condition and pressure ulcer risk factors, defined and implemented interventions consistent with resident needs, goals, and recognized standards of practice, monitored and evaluated impact of interventions, and revised approaches as appropriate.”
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Urticaria Urticaria L, urtica, nettleL, urtica, nettle
• AKA – Hives
• Severe itching, then redness, then wheal
• Localized, various sizes
• Can include – GI tract – abdominal colic– Pharynx – can cause asphyxiation– Deep tissues = angioedema – more serious
• Usu. Short duration, can become chronic
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Urticaria (L50)Urticaria (L50)
• Dermis• Acute hypersensitivity • Release of histamine• Local inflammation, vasodilation, edema• Allergic reaction
– Shellfish, strawberries, peanuts, etc.– Insect stings
• Infection, Sunlight, Temperature extremes• Tx: epinephrine, prednisone
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Skin Injuries & NeoplasmsSkin Injuries & Neoplasms
• Chapter 19 (S00-T88) – Injury, Poisoning, & Other consequences of External Causes
• Chapter 2 (C00-D49) – Malignant Neoplasms
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Skin Injuries in ICD-10-CMSkin Injuries in ICD-10-CM
• Abrasion– Excoriation
• Avulsion– Wound, Open
• Bite (nonvenomous)– Superficial– Open
• Blister (nonthermal)• Burn• Contusion• Corrosion
• Exfoliation– Desquamation
• External Constriction• Foreign Body• Frostbite• Incision• Laceration• Wound
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Abrasion Abrasion • L: abradere, to scrape off
• Rubbing away of skin surface by friction against rough surface, usu. Not deeper than epidermis, can bleed
• Mild abrasion (Graze) does NOT scar
• Deep abrasion may scar
• AKA – Scrape, Scratch, Excoriation
• Types– Rope burn, friction burn, road rash, rug burn
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Avulsion of SkinAvulsion of Skin
• L, avulsio, a pulling away
• Avulsion injury - removal of all layers of skin– Chunk of tissue removed from body– Includes all 3 layers of skin at minimum
• More than simple lacerations; less than amputations
• Etiology: Animal bites, industrial equipment, or MVAs
• Skin flap or Degloving
• Higher Risk of Infection
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Bite (Non-venomous)Bite (Non-venomous)
• Wound or puncture made by living organism
• Superficial = Skin
• Open = Communicates directly with atmosphere
• Pets, Humans, Insects, Snakes, etc.
• Can be heavily contaminated
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Blister (Non-thermal)Blister (Non-thermal)
• Friction (New Shoes)– Abrasion Blister
• Electricity
• Chemicals
• Radiation from sun
• Cold
• Pinched skin (blood blister)
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Burn (T20-T25)Burn (T20-T25)
• Electrical heating appliances
• Electricity• Flame• Friction• Hot air and hot gases• Hot objects
• Leading cause of injury in US
• Lightning• Radiation• Scalds
• Chemical Burn (Corrosion)
• NOT Sunburn
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BurnsBurns
• 85%–90% caused by heat
• 10%–15% frostbite, chemical, & electrical damage
• 1.5 - 2.0 million burns each year in U.S
• 60,000 to 80,000 burn victims hospitalized
• 5,000 people die
• 35,000 to 50,000 people - temporary or permanent disability 2ndary to burns
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Degrees of BurnsDegrees of Burns• Older Terminology • 1st degree; outer layers of
epidermis injured, erythema, but no blistering
• 2nd degree; epidermis into dermis but basal layer remains, blistering
• 3rd degree; all epidermis & dermis destroyed; only white eschar remains
• 4th degree; muscle, nerve and bone damaged
• Newer terminology • Superficial partial
thickness; epidermis and upper part of dermis injured
• Deep partial thickness; epidermis & large upper portion of dermis injured
• Full thickness; all layers destroyed
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Degrees of BurnsDegrees of Burns
• 4th degree burns extend to tissue
• 5th degree extends to bone
• 6th degree burns are through bone
• Old classification system
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http://www.ncbi.nlm.nih.gov/books/NBK27295/
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Rule of Nines for Burn Extent• Total Body Surface Area (BSA)
(Adults)
• Age Infants, children, & elderly survival
• Total Body Surface Area – greater area of involvement ~
prognosis
• Depth of Burn— More Total BSA ~ full thickness, prognosis
• Other Injuries - Prognosis as concurrent injuries increase
http://www.ncbi.nlm.nih.gov/books/NBK27295/
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Burn Treatment Burn Treatment
• Determined by Degree and Extent of Burns
• Table of Treatment Modalities by Burn Degree– Rehabilitation of Burn Injuries– http://www.ncbi.nlm.nih.gov/books/NBK27295/
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ContusionContusion
• L, contundere, to bruise
• AKA Bruise
• Injury that does not disrupt integrity of skin– Caused by blow to body– Characterized by
swelling, discoloration, & pain
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Corrosion (T20-T25)Corrosion (T20-T25)
• L, corrodere, to gnaw away
• Tissue damage due to exposure to strong alkali or acid
• Main types of irritant and/or corrosive – Acids, bases, oxidizers, solvents, reducing
agents and alkylants– Types of chemical weapons
• Vesicants or Urticants
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External ConstrictionExternal Constriction• Classified as superficial injury• Ex: Hair tourniquet syndrome
– AKA hair strangulation
• External constriction of finger, toe, or external genitalia by hair – OR thread, ring, rubber band, other object that can
go around a body part– Relatively common in infants– Can be emergency situation
• Object NOT removed; infection or amputation
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Foreign BodyForeign Body
• Any object or substance found in organ or tissue in which it does NOT belong under normal circumstances
• Superficial• Open Wound w/FB
http://upload.wikimedia.org/wikipedia/commons/7/74/Knee_puncture.JPG
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Foreign BodyForeign Body• Iatrogenic
– Sutures, sponges, instruments left during surgery
– Metals & plastics replacing/enhancing failing/non-functioning body parts
• Accidental/unintentional– From abrasions & open
wounds in various accidents or GSWs,
– May elicit FB-type granuloma formationr
• Intentional– Introduced in context of
sexual deviancy– Anorectum or vagina -
vibrators, bottles, light bulbs, eggs, etc.
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FrostbiteFrostbite• Superficial
– Damage to skin caused by extreme cold– Red, painful, Blisters, Gangrene – Hands, feet, nose, & ears most common areas
• RF:– Beta-blockers– Peripheral vascular disease– Smoke, Diabetes– Raynaud phenomenon
• IF blood vessels NOT damaged– Complete Recovery Possible
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IncisionIncision
• Neat, Clean cut– Surgery– Sharp instrument, such as knife
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WoundWound
• Laceration– Dull, Blunt instrument– Jagged, irregular wound– Some tearing, breaking of tissue
• Puncture wound– Sharp, pointed object pierces skin– Can be deep; look insignificant– Anaerobic infection risk
• W/Without FB
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LacerationsLacerations
• From shallow cuts to deep gashes penetrating thru muscle to internal organs/bone– Superficial laceration involves only skin– Deeper laceration may penetrate veins or arteries– Blood vessel may require repair
• Causes - include accidents at home & work, motor vehicle crashes, & violence
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Open Wound vs SuperficialOpen Wound vs Superficial
• Open wounds– Animal bites, avulsions, cuts, lacerations,
puncture wounds, traumatic amputations
• Superficial injuries– Animal bites that do NOT break all skin layers– Abrasions or contusions– Skin tears (Elderly, etc.)
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Gunshot woundsGunshot wounds• Skin Damage = external, surface wounds
caused by a bullet• Usually, entrance wound smaller/tidier than exit
site• Amount of injury at entrance/exit (if present) sites
related to– Caliber of bullet– Angle at which bullet traverses tissues– Distance from gun– Type of bullet
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Integumentary System NeoplasmsIntegumentary System Neoplasms
• Benign usu. Need ONLY cosmetic tx • Most common cancers in US• C43-44 Melanoma and Other malignant Neoplasms
of Skin– BCC– SCC– Melanoma– Merkel Cell Ca
• C50 Malignant Neoplasm of Breast• C79.2 2ndary Malignant Neoplasm of Skin• C84.0 Cutaneous Lymphoma
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Benign Neoplasms of SkinBenign Neoplasms of Skin• Acrochordon
– Skin tag
• Cherry angioma • Dermatofibroma • Dermatosis papulosa
nigra• Ephilides
– Freckles
• Epidermal inclusion cyst
• Lentigines– Liver spots
• Lipoma• Milium• Nevus
– Mole
• Pyogenic granuloma• Sebaceous hyperplasia• Seborrheic keratosis
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Keratinocyte cancers
• Most common non-melanoma skin cancers– Cells share some features of keratinocytes– Most abundant cell type of normal skin
• Most common types of keratinocyte cancer – Basal cell carcinoma– Squamous cell carcinoma
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Other Skin CancersOther Skin Cancers
• Other non-melanoma skin cancers include – Merkel cell carcinoma– Kaposi sarcoma– Cutaneous (skin) lymphoma– Skin adnexal tumors– Various types of sarcomas
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Basal Cell CarcinomaBasal Cell Carcinoma• About 80% of skin cancers
• Sun-exposed areas, especially head & neck
• Slow growing, seldom spread
• BUT IF left untxed; can invade & metastasize
• Can recur at same place
• Increases chance of getting BCC elsewhere
• Up to 50% of people will get new BCC w/in 5 years
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Breast CancerBreast Cancer
• 2 main types
• Ductal carcinoma – Begins in tubes (ducts) that move milk from
breast to nipple– Most breast cancers
• Lobular carcinoma– Begins in lobules that produce milk
• Rarely, can begin in other breast areas
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Breast CancerBreast Cancer• Invasive
– Spread from milk duct or lobule to other breast tissues
• Noninvasive– NOT yet invaded other breast tissue– “In situ“
• Women - 1 in 8 will be DX w/breast cancer• Men - <1% of all cancers in men
– < 1% of all diagnosed breast cancers
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Breast Ca Risk FactorsBreast Ca Risk Factors• Age and gender
– 50 +– Women - 100 times more likely to get than men
• Family Hx of breast cancer – Close relative had breast, uterine, ovarian, or colon cancer– 20 - 30% of women w/ breast cancer have family hx
• Genes – BRCA1 and BRCA2 genes
• Menstrual cycle– Early periods (before age 12) – Menopause late (after age 55)
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Breast Ca Risk FactorsBreast Ca Risk Factors
• Alcohol use– 1 - 2 glasses + daily
• Childbirth – NO children or after age
30– Being pregnant 2+ times
reduces risk– Pregnancy at early age
reduces risk
• Dense Breast tissue• Being White
• DES – – Increased risk of breast
cancer after age 40
• HRT –– Estrogen for several years
• Obesity – – Link is controversial
• Radiation Tx– Child or young adult – Treat ca of chest area
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Breast Cancer TxBreast Cancer Tx
• Six types of standard treatment – Surgery– Sentinel lymph node biopsy followed by surgery– Adjuvant Tx
• Radiation therapy• Chemotherapy• Hormone therapy
– Targeted therapy
• Various combinations based on Pt and Stage
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Breast Cancer SurgeryBreast Cancer Surgery• Breast-conserving surgery
– Remove cancer but not breast– Lumpectomy: Surgery to remove a tumor (lump)
& small amount of normal surrounding tissue– Partial mastectomy: Remove part of breast
w/cancer & some normal tissue• Lining over chest muscles below cancer may be
removed • AKA segmental mastectomy
– Lymph Node Dissection (separate incision)
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Breast Ca SurgeryBreast Ca Surgery
• Total (Simple) Mastectomy– All breast tissues
• Modified Radical Mastectomy– Whole breast, many of lymph nodes, lining
over chest muscles; sometimes, part of chest wall muscles
• Breast Reconstruction– At same time OR later
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Radiation for Breast CaRadiation for Breast Ca
• External radiation therapy– Machine outside body sends radiation to
cancer
• Internal radiation therapy – Radioactive substance sealed in needles,
seeds, wires, or catheters– Placed directly into or near cancer
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Chemotx for Breast CancerChemotx for Breast Cancer
• Systemic chemotherapy – Chemotherapy taken by mouth or injected into
vein or muscle– Drugs enter bloodstream, & can reach cancer
cells throughout body
• Regional chemotherapy− Chemotherapy placed directly into CSF, organ,
body cavity (such as abdomen)− Drugs mainly affect cancer cells in those areas
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Hormone Tx for Breast CaHormone Tx for Breast Ca
• Hormone therapy – Removes hormones OR blocks action– Stops cancer cells from growing
• Some hormones can cause certain Ca
• When tests show cancer cells have hormone receptors– Drugs, surgery, or radiation tx used to reduce
production of hormones or block them from working
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Targeted Tx for Breast CaTargeted Tx for Breast Ca
• Uses drugs or other substances– ID & attack specific cancer cells without
harming normal cells– Monoclonal antibodies and Tyrosine kinase
• Used to treat Breast Ca
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Cutaneous (skin) lymphomaCutaneous (skin) lymphoma
• Cancers of lymphocytes (white blood cells)– Primarily involving skin
• Classification is based on lymphocyte type– B-lymphocytes (B-cell) – T-lymphocytes (T-cell) (CTCL)
• Most common• Red, scaly patches or thickened plaques of skin
– Often mimic eczema or chronic dermatitis• More common in men; after age 50
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CTCLCTCL
• Most common type of Skin Lymphoma– Red, scaly patches or
thickened plaques of skin – Often mimics eczema or
chronic dermatitis• More common in men; after
age 50
• Two most common types– Mycosis fungoides &
Sezary syndrome
• Treatment – Topical
• Corticosteroids, Topical Chemo, Retinoids
– Systemic• IV Steroids• IV Retinoids• IV/ Oral Chemo
– Light Therapy– Immunotherapy– Radiation Therapy
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MelanomaMelanoma• Less than 5% of skin cancer cases • BUT causes large majority of skin ca deaths • 2012 - About 76,250 new melanomas dx
– 44,250 men & 32,000 women– Incidence rates for melanoma rising for 30+ yrs
• 9,180 are expected to die of melanoma– 6,060 men & 3,120 women– 2004 to 2008 - Death rate in whites
• Dropped those < 50 • Stable in women • Rising in men > 50
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MelanomaMelanoma
• Lifetime risk– 2% (1 in 50) for whites– 0.1% (1 in 1,000) for blacks– 0.5% (1 in 200) for Hispanics
• Risk increase with age– One of more common cas in young adults
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Melanoma Risk FactorsMelanoma Risk Factors• Ultraviolet (UV) light
exposure – Sun and Tanning beds
• Dysplastic nevi• Congenital melanocytic
nevi • Fair skin, freckling, &
light hair • Personal Hx of
melanoma
• Immune suppression • Age• Gender (Male)• Xeroderma
pigmentosum
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S&S of MelanomaS&S of Melanoma
• ABCDE rule– Asymmetry One half of mole or birthmark does not
match other half
– Border Edges irregular, ragged, notched, or blurred
– Color NOT same all over & may include shades of brown/black, sometimes patches of pink, red, white, or blue.
– Diameter Spot >6 mms across– size of pencil eraser, BUT melanomas can be smaller
– Evolution Any changes in skin lesions, new skin lesions, or growths that look different from moles
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S&S of MelanomaS&S of Melanoma
• Other warning signs– Sore doesn’t heal– Pigment spreads from border of spot to
surrounding skin– Redness or new swelling beyond border– Change in sensation
• itchiness, tenderness, or pain
– Change in surface of a mole• scaliness, oozing, bleeding, or bump/nodule appears
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Melanoma StagingMelanoma Staging
• The American Joint Committee on Cancer (AJCC) TNM system
• T ~ Tumor 0-4 a, b
• N ~ Node 0-3 a, b, c
• M ~ Metastasis
• Tx: Based on Stage
• Surgery Chemotherapy
• Immunotherapy Radiation therapy
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Melanoma Survival RatesMelanoma Survival Rates
Stage IA
5-year ~ 97%
10-year ~ 95%
Stage IB
5-year ~ 92%
10-year ~ 86%
• Stage IIIA
• 5-year ~ 78%
• 10-year ~ 68%
• Stage IIIB
– 5-year ~ 59%
– 10-year ~ 43%
• Stage IIIC
– 5-year ~ 40%
– 10-year ~ 24%
• Stage IV
– 5-year ~ 15%-20%
– 10-year ~ 10%-15%
Stage IIA 5-year ~ 81% 10-year ~ 67%Stage IIB 5-year ~ 70% 10-year ~ 57%Stage IIC 5-year ~ 53% 10-year ~ 40%
http://www.ncbi.nlm.nih.gov/pubmedhealth/PMH0001853/figure/A000850.B3200/?report=objectonly
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Merkel Cell Ca Merkel Cell Ca • Rare skin ca, but incidence increasing world-wide• Metastasizes quickly• Merkel cells at base of epidermis• Nodule
– flesh-colored OR bluish-red, shiny, painless• Usu. on face, head, neck; also on limbs or trunk• Risk Factors
– Age, sun exposure, weak immune system, other skin ca, light skin
– 80% of 10 Merkel cell cas are thought to be related to MCV (Merkel Cell Polyomavirus)
• AKA– Neuroendocrine carcinoma of skin
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Merkel Cell CaMerkel Cell Ca
• Metastasis– Liver, Bones, Lungs, Brain
• Prognosis– 33% fatality rate
• Diagnosis– PE, Bx, Sentinel Node Bx, x-rays, CT, PET,
octreotide scan
• Tx– Surgery (Mohs), Radiation, Chemo
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Squamous Cell CarcinomaSquamous Cell Carcinoma
• About 20% of skin cas are SCC
• Sun-exposed areas of body – Face, ears, neck, lips,
& backs of hands
• Scars or skin ulcers elsewhere
• Sometimes begin in actinic keratoses
• Genital area skin
http://www.nlm.nih.gov/medlineplus/ency/imagepages/2475.htm
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Squamous Cell CaSquamous Cell Ca
• More aggressive than BCC– More likely to invade fatty tissues just under
skin– More likely to spread to lymph nodes and/or
metastasize
• Squamous cell carcinoma in situ – AKA Bowen disease– Still w/in the epidermis
http://www.nlm.nih.gov/medlineplus/ency/imagepages/1516.htm
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2ndary Skin Cancers2ndary Skin Cancers
• Cancer cells can grow in skin– NOT skin cancer, melanoma, or cutaneous T cell
lymphoma – Secondary cancer may start to grow
• On/ near operation scar where primary cancer was removed
• In other parts of body
• Secondary skin cancer looks like a pink/red raised lump – These nodules can be treated
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Questions from Last SessionQuestions from Last Session
• ICD-10-CM/PCS Implementation date– Proposed Rule – 10-1-2014
• Federal Register on April 17, • Proposed rule available for comment for 30 days
– Announcement in June?
• http://www.cms.gov/Medicare/Coding/ICD10/index.html
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Questions from Last SessionQuestions from Last Session• Timeline for Required ICD-10-CM/PCS CEs?
– “CCHIIM will send out a communiqué within 30 days of final decision by HHS on ICD-10 implementation date to address ICD-10 CEU requirements and launch date for ICD-10 compliant exams. Certified professionals are still encouraged to obtain their ICD-10 required CEU’s for submission by December 31, 2013.”
– Please be aware that delay is not finalized until official notice is send out from HHS
• Linda DeViller, AHIMA 6/18/2012
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Query ExampleQuery Example
• Liposuction of lipomas
• ICD-10-CM– Location of lipomas
• ICD-10-PCS– INTENT of Root operation
• Medical reason ~ Extraction• Cosmetic reason ~ Alteration
What is missing from above statement?What would we need to query for?
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HomeworkHomework• Pressure Ulcer Online Training. The National
Database of Nursing Quality Indicators®– https://www.nursingquality.org/
NDNQIPressureUlcerTraining/
• Four Modules– Pressure Ulcers and Staging– Other Wound Types and Skin Injuries– Pressure Ulcer Survey Guide– Community vs. Hospital/Unit Acquired Pressure
Ulcers
• CE certificate when all completed w/tests
CEs
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General ResourcesGeneral Resources
• Cohen, B. A., Lehmann, C. U. Dermatology Image Atlas. Johns Hopkins University, 2000-2012.– http://dermatlas.med.jhmi.edu/derm/
• Dermatology A to Z. AAD. – http://www.aad.org/skin-conditions/dermatology-a-to-z
• MedlinePlus• Swanson, J. R. & Melton, J. L. Dermatology Atlas.
Chicago: Loyola University Dermatology Medical Education Website– http://www.meddean.luc.edu/lumen/MedEd/medicine/
dermatology/melton/title.htm
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ResourcesResources
• About Cutaneous Lymphoma. Cutaneous Lymphoma Foundation.– http://www.clfoundation.org/about-cutaneous-lymphoma
• Acute Lymphangitis. Medical Dictionary Article.– http://medical-dictionary.thefreedictionary.com/
Acute+Lymphangitis
• Armstrong, D. G. & Lavery. L. A. Diabetic Foot Ulcers: Prevention, Diagnosis and Classification. AAFP.– http://www.aafp.org/afp/1998/0315/p1325.html
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Resources Resources • Breast Cancer Treatment (PDQ®). National Cancer
Institute.– http://www.cancer.gov/cancertopics/pdq/treatment/breast/
Patient/page1
• Characteristics of Arterial Ulcers. Wound Educators. YouTube.– http://www.youtube.com/watch?
v~QDbknssi5H4&feature~related
• Common Benign Growths. Dermatology. Cleveland Clinic.– http://www.clevelandclinicmeded.com/medicalpubs/
diseasemanagement/dermatology/common-benign-growths/
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ResourcesResources• Cutaneous Lupus Erythematosus. Family Practice
Notebook.– http://www.fpnotebook.com/rheum/Derm/
CtnsLpsErythmts.htm
• Evaluation of an Acute Wound. Chapter 6.– http://practicalplasticsurgery.org/docs/Practical_06.pdf
• Gunshot Wounds. Chapter 7.– http://practicalplasticsurgery.org/docs/Practical_07.pdf
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ResourcesResources
• Gun Shot Wounds. Wheeless' Textbook of Orthopaedics. Duke Orthopedics.– http://www.wheelessonline.com/ortho/gun_shot_wounds
• Lymphadenitis. Medical Dictionary Article.– http://medical-dictionary.thefreedictionary.com/lymphadenitis
• Practical Plastic Surgery.– http://practicalplasticsurgery.org/the-book/
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ResourcesResources
• Peripheral Neuropathy Fact Sheet. NINDS.– http://www.ninds.nih.gov/disorders/
peripheralneuropathy/detail_peripheralneuropathy.htm
• Rashes: MedlinePlus Medical Encyclopedia. – http://www.nlm.nih.gov/medlineplus/ency/article/
003220.htm
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ResourcesResources• Rose, L. C. Recognizing Neoplastic Skin Lesions:
A Photo Guide. Am Fam Physician. 1998 Sep 15: 58 (4): 873-884.– http://www.aafp.org/afp/1998/0915/p873.html
• Skin Lesions. Article. Medical Dictionary.– http://medical-dictionary.thefreedictionary.com/
Skin+Lesions
• ICD-9-CM Coordination and Maintenance Committee Meeting December 6, 2002 – http://www.cdc.gov/nchs/data/icd9/agendadec02.pdf