assigment final end stage renal failure

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    NURSING MANAGEMENT FOR PATIENT UNDERGOING HEMODIALYSIS

    1. INTRODUCTION

    Nowadays people of Malaysia are threatened with a variety of illnesses caused by life style

    among the diseases that involve the kidneys of "renal failure". We know that the kidney is one of

    the most important organs in the human body functions. Renal is derived from the Latin word

    renes, meaning kidneys. Can be defined is a pair of kidneys are located behind the abdominal

    organs. Each kidney is about 4 or 5 inches long. It is about a fist size. Right kidney is located

    right below the diaphragm and behind the heart. While the left kidney is located below the

    diaphragm and spleen organ behind. Above each kidney is adrenal gland also known as

    (suprarenal glands), and each kidney contains about one million units, which is referred to

    neurons. Irregular position in the abdominal area due to the liver causing the right kidney is

    slightly lower than the left kidney.

    Retro peritoneum and kidneys is approximately 9 to 13 cm in diameter, which is slightly larger

    left than the right. It was around the level of T12 to L13 vertebral level. Some parts of the

    kidney is protected by the bones of the eleventh rib and two twelve and each adrenal gland is

    fully protected by two layers of fat (per renal and Para renal fat) and renal fascia which helps

    cushion it..

    1.1 KIDNEY FUNCTION

    The bladder holds about 400 ml (1 / 4 pints) of urine when full. Normal urine normally produced

    is around 2 liters per day. The main function of the kidney is responsible for the generation of

    waste from the body, regulating electrolyte balance and blood pressure and stimulates production

    of red blood cells. It cleanses the blood and makes urine from waste and they produce them.

    Blood is pumped by the heart to the kidneys. Their kidneys have a drainage system that takes

    urine from the kidneys to the bladder. Drainage system as a channel with a tube (ureter) that

    connects the kidney to the bladder. Urine down ureter (for each kidney) into the bladder. Urine is

    stored in the bladder prior to discharge from the body through another tube called the ureter

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    Another kidney function helps blood pressure. Fine blood pressure is controlled by a healthy

    kidney. When the kidneys fail one of their blood pressure usually rises. Although it is not really

    known why. Pressure high blood pressure will not cause symptoms except for very high

    pressure, but it increases the risk of stroke or heart attack.

    The kidneys also help control the actual manufacture of blood cells. The kidneys help regulate

    blood cells, the actual manufacturing in the bone marrow. Floating in the liquid if the blood

    (plasma). Their job is to carry oxygen around the body.

    The kidneys also produce certain hormones that have important functions in the body, including

    the following:

    Active form of vitamin D (calcitriol or 1.25 dihydroxy-vitamin D), which controls the

    absorption of calcium and phosphorus from foods, promoting formation of strong bones.

    Erythropoietin (EPO), which stimulates bone marrow to produce red blood cells.

    Renin, which regulates blood volume and blood pressure

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    2. END STAGE RENAL FAILURE

    As we know, kidney failure is in various stages. Among them are the "acute renal failure",

    "chronic renal failure" and the final stage is the "end stage renal failure". Based on studies in theUnited State is created by the National Kidney Foundation in 2001, a total of 370,000 person end

    stage renal failure have a number of cases increased approximately 8% each year.

    End-stage kidney failure (ESRF) defined by Abassi et all 2010 as a reduction in the

    irreversible in kidney function , which is severe enough to be fatal in the absence of

    dialysis or transplantation.

    .

    "End stage renal failure" can also be defined as an irreversible decline in kidney function that is

    severe enough to be fatal in the absence of dialysis or transplantation. In general, end stage renal

    failure occurs when there is less than 10% of the remaining kidney function. In 2000 the final

    stage of kidney failure than 375,000 American affected aspects of MRE increased to 651,000 by

    2010.

    Stage 5 chronic kidney disease can be described as kidney failure, also known as end stage renal

    failure and chronic kidney disease has developed. Renal replacement therapy, dialysis or kidney

    transplant may be needed to sustain life.

    2.2CAUSES OF END STAGE RENAL FAILURE

    Although chronic kidney disease sometimes results from the major diseases of the kidney itself,

    the main reasons for diabetes and high blood pressure.

    1. Type 1 and type 2 diabetes mellitus can cause a condition called diabetic nephropathy,which is the main cause of kidney disease in the United States.

    2. High blood pressure (hypertension), if uncontrolled, can damage the kidneys, from timeto time.

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    3. Glomerulonephritis inflammation and damage to the kidney filtration system, which canlead to kidney failure. Post infectious conditions and lupus which is one source of many

    Glomerulonephritis.

    4. Polycystic kidney disease is an example for the children of chronic kidney disease inwhich both kidneys have multiple cysts.

    5. The use of painkillers such as acetaminophen (Tylenol) and ibuprofen (Motrin, Advil)fixed on a long period of time can lead to analgesic nephropathy, another cause of kidney

    disease. Some other drugs can also damage the kidneys.

    6. clogging and hardening of the arteries (atherosclerosis) that lead to the kidneys causes acondition called ischemic nephropathy, which is another cause of progressive kidney

    damage.

    7. Obstruction of urine flow by the rocks, the restrictions (narrowing), or cancer can alsocause kidney disease.

    8. Other causes of chronic kidney disease, including HIV infection, the cells sickle disease,heroin abuse, amyloidosis, kidney stones, chronic kidney infections and certain cancers.

    3.0 CLINICAL FINDINGS AND EFFECTS ON PATIENTS

    Patient with end stage renal failure have a variety of signs or symptoms that can be seen and

    different. It can be explicit or hidden. Based on the laboratory results obtained from Madam

    Wong her serum creatinine is higher than normal readings, the normal creatinine are in range

    less than 97. Creatinine blood test is to find proper kidney function and is used to

    determine glomerular filtration rate, by the result found her had to undergo hemodialysis

    immediately.

    Based on the symptoms shown, she suffered "Shortness of breath '. It is caused by fluid

    accumulation in the lungs. Other signs that patient also experience fatigue and

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    confusion in which the patient is in a state of uremia syndrome. In this situation can be seen

    where the level of creatinine in the blood are increase. This syndrome can affect body systems

    such as the intestines, nerves and heart. Another cause of fatigue maybe patient have a mild

    anemia due to hemoglobin are 10.1 g/ dl. For the patient who need to go to hemodialysis

    ,hemoglobin should be maintain at 11 g/dl.

    "Pitting edema" is one of the symptoms listed for renal patients which it occur when kidney

    function has deteriorated it could no longer filter the liquid and keep the balance of salt. When

    this happens, fluid and salt from the body fluid build up in body tissues and cause edema.

    Other symptoms that may be identified as the patient's skin will become dark or bright, unusual

    skin than the original and will be a change in the nails.

    Others laboratory examination done such as taking blood for "blood urea nitrogen," in which to

    look at kidney function. Urea is the main product of protein metabolism. Its formed in liver

    and excreted mainly by kidney in urine. If the kidneys are unable to remove the urea in the

    blood it will rise.

    Besides taking full blood count is one of examination must do in patient with renal failure,

    its for detect renal anemia which full blood count is a component, such as hemoglobin,

    hematocrit .

    Urine output are also one of symptom that can be seen in end stage renal failure was caused

    when 90% of nephrons almost fails it can cause "blood urea nitrogen" and "creatinine"

    increases so that it causes "polyuria" changed to "oliguria . "

    Chronic disease have characteristic that lead to patient fatigue and symptoms of

    depression. End stage renal failure in terms of influencing an individual's physical,

    psychological, and emotional health. The impacts can be seen is that according to Morsch,

    Concalus & Banis, 2006 end stage renal failure is Associated with poor quality of life,

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    depression (Kimmel & Peterson, 2006), reduced physical functioning Ability (Cleary & Diennan

    2005; O'Sullivan 2007 ), poor sleep quality (Elder et all, 2007) and fatigue (Mc Cann & Boore,

    2008; O'Sullivan & Mc Carthy, 2007).

    Having chronic condition such end stage renal disease is likely to have a impact on

    patient everyday life. The concerns commonly identified among of end stage renal failure

    patient are disruption of lifestyle (Devins et all.2001).Whereas Blaschek 2003 said that

    suffering from continuing symptom , stress and uncertainly of life. Intervention aimed at

    modifying know mortality risk factors are expected to have considerable impact in this

    patient population in survival and hopefully, Qol (Lindquist& Syoden 1998, Golper 2001,

    Loss- Ayau et all- 2008 ).

    Other symptoms, if untreated, can cause life-threatening condition and can be formed.

    1. metabolic acidosis, or increased acidity of the body due to the inability to manufacturebicarbonate, will alter enzyme and oxygen metabolism, causing organ failure.

    2. Unable to remove potassium and increases in serum potassium levels (hyperkalemia) isAssociated with a fatal heart rhythm disturbances (arrhythmias), including ventricular

    tachycardia and ventricular fibrillation.

    3. Increased level of urea in the blood (uremia) can affect the function of various organs ofthe brain (encephalopathy) with alteration of thinking, inflammation of the lining of the

    heart (pericarditis), decreased muscle function because of low calcium levels

    (hypocalcemia).

    4. The weakness may be caused by anemia in which red blood cell count decreased, because of lower levels of erythropoietin produced by the kidneys failed be able to

    stimulate the bone marrow. The decrease in red cells with a decrease in the ability to

    carry oxygen in the blood, resulting in decreased oxygen delivery to the cells for them to

    do the work, therefore, the body tires quickly. And, with less oxygen, the cells are easier

    to use anaerobic metabolism (= without aerobic = oxygen). This can lead to the total

    increase in acid production which can not be handled by the kidneys that have failed

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    5. As waste products build in the blood, loss of appetite, lethargy and fatigue was verystrong. This can lead to decreased mental function, and coma may occur.

    4. .MANAGEMENT OF THE PATIENT UNDERGOING HEMODIALYSIS

    Dialysis is a treatment that takes over the task performed by the kidneys. Once for the

    last stage kidney failure (end stage renal failure), most patient requiring dialysis treatment.

    This treatment is likely for life except for kidney transplants.

    According to Wong et all 2007 there are approximately 3000 patient on dialysis of

    whom 80% are on continuous ambulatory peritoneal dialysis and remaining 20% are

    receive hemodialysis.. Hemodialysis is done where an artificial kidney (haemodialyzer)connected to a machine that filters work to remove waste impurities and excess fluid from the

    body. This procedure is carried out in a special unit (hemodialysis unit) in the hospital and takes

    around four hours per session to three sessions a week. The patient will require fistula of blood

    from the body to flow into the artificial kidney. The surgeon will make this fistula by connecting

    an artery to the venous blood vessels to become larger blood vessels. During dialysis, wasteproducts in the bloodstream are carried out of the body. At the same time, electrolytes and other

    chemicals are added to the blood. The purified, chemically-balanced blood is then returned to the

    body.

    In the hemodialysis treatment of several aspects of care should be taken into consideration,

    including the vascular access. Patients who undergo dialysis vascular access and so requires

    access sites must be protected from damage or infection. Malovrh 2005 suggests a long-term

    survival and quality of life of patients on hemodialysis depend on dialysis adequacy through the

    appropriate vascular access. Ateriovenous fistula and graft require subcutaneous access and, to protect their access sites from unnecessary pressure or injury, venepuncture, cannulation,

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    arterial-line insertion and the use of blood-pressure cuffs must be avoided (Richard and

    Engebretson 2010).

    In addition of vascular access, care diet is also important for end stage renal failure patients.

    Among are fluid intake and other food. The volume of urine passed by patients undergoingdialysis is too small to remove all the fluid from their food and drink. Fluid retention leads to

    increased blood volume, distended blood vessels and high blood pressure, which can in turn

    lead to pulmonary edema (Agar 2010).

    According to Kalanter-Zadeh (2009)in a study of fluid gain in patients undergoing dialysis

    found that the average patient gained between 1.5 and 2.0kg of fluid between dialysis sessions,

    and that a weight gain of at least 4kg over two consecutive dialysis sessions was associated with

    a 28 per cent increased risk of death. Optimal fluid management is a determinant of survival in

    patients undergoing renal replacement therapy (Collins and Mujais 2002) and,Blair 2009 states

    that patients with chronic kidney disease, morbidity and mortality are increased by excessive

    fluid intake.

    Most patients undergoing dialysis are restricting to 1 liter fluid a day and if they are given extra

    fluids intravenously, they must be monitored constantly.

    Hyperkalaemia in most people, about 90 per cent of potassium is excreted by the kidneys and

    patients undergoing dialysis who fail to adhere to dietary advice may be at risk of

    hyperkalaemia due to decreased potassium excretion (Challinor 2008). Hyperkalaemia affectsthe ability of cardiac muscle tissue to conduct electrical impulses, potentially resulting in

    ventricular fibrillation and a systolic arrest (Ahee and Alexander 2000),

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    NURSING PROCESS

    PROBLEM GOAL INTERVENTION EVALUATION

    High risk of

    Arterivenous Fistula

    thrombosis and

    Aneurysm due to

    knowledge deficit.

    To reduce

    Arteriovenous Fistula

    Complication

    1.Educate her on the

    care of Arteriovenous

    Fistula

    2.Avoid repeated

    cannulation at the

    same site

    3.Treat infection if

    Her arteriovenousFistula is still in good

    condition andfuctioning well

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    present

    4.Cannulate carefully

    try not to puncture the

    fistula

    5.Refer to Doctor if

    there is bad aneurysm

    6.Keep fistula clean

    and dry

    NURSING

    PROBLEM

    OBJECTIVE NURSING

    INTERVENTION

    EVALUATION

    Fluid volume excess

    related to failure or

    comprised renal

    regulatory mechanism

    Patient will not show

    signs and symptoms

    of excess fluid.

    1. Assist in dialysis

    and monitor patient

    progress.

    2 .Administer

    intravenous or oral

    fluids as prescribed.

    3. Monitor intake and

    output chart .Strict

    fluid intake to patient.

    4. Use small cup to

    drink ,so that patient

    cannot take a lot of

    water.

    5. Do daily weight

    and chart.

    Patient not show sign

    and symptoms of

    excess fluid.

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    6. Monitor vital sign

    .Sign and symptom

    Short of breath(SOB)

    Early sign of fluid

    overload

    7. Offer ice chips or

    flavored ice pops.

    Record intake. Helps

    minimizes thirst.

    Provide frequent

    mouth care. Helps

    minimize thirst.

    NURSING

    PROBLEM

    OBJECTIVE NURSING

    INTERVENTION

    EVALUATION

    Pedal edema To reduce pedaledema

    1 .Evaluate the

    extended of edema

    and its location ,so

    that action could be

    taken to relief the

    condition.

    2.Advice patient to

    elevate his leg during

    lying down to reduce

    Pedal edema reduce

    after intervention

    given.

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    swelling

    3. Restricted fluid 500

    ml POD (Previous

    Day Output) and take

    low salt diet to avoid

    fluid retention.

    4.Medication strictly

    follow Doctors

    ordered

    5. Monitor the

    condition of patient

    during treatment and

    inform doctor about

    the changes

    NURSING

    PROBLEM

    OBJECTIVE NURSING

    INTERVENTION

    EVALUATION

    Impaired skin

    integrity related to

    pruritus and dry skin

    secondary to uremia

    and edema

    Patient's skin remains

    intact and free of

    erythema and

    abrasions.

    1. Encourage use of

    prescribed phosphate

    binders when serum

    phosphorus level is

    elevated. Excess

    phosphorus binds with

    free calcium in the

    serum. The resulting

    calcium-phosphate

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    complex is deposited

    in soft tissues and can

    cause necrotic patches

    in the skin. In

    addition, elevation in

    calcium-phosphate

    product is associated

    with increased risk of

    death, aortic

    calcification, mitral

    valve calcification,

    and coronary artery

    calcification.

    Phosphate binders

    help prevent elevation

    in calcium-phosphate

    product.

    2. Administer

    antihistamines as prescribed if needed.

    Because accumulating

    nitrogenous wastes

    are excreted through

    the skin, pruritus is

    common in patients

    with uremia and

    causes frequent and

    intense scratching.

    Pruritus often

    decreases with a

    reduction in BUN and

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    improved phosphorus

    control. Decreases

    itching and

    subsequent scratching

    that can result in

    abrasions and/or

    infection.

    3. Keep patient's

    fingernails cut short.

    Prevents skin

    abrasions when

    scratching.

    4. Teach patient

    to monitor scratches

    for evidence of

    infection and to seek

    medical attention

    early if signs and

    symptoms of infectionappear. Uremia

    retards wound healing

    and thus increases the

    risk for infection.

    5. Encourage use

    of skin emollients and

    soaps with high fat

    content. Uremic skin

    is often dry and scaly

    because of reduction

    in oil gland activity.

    Skin emollients and

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    soaps with high fat

    content replace oils

    and help reduce

    itching from dryness.

    6. Advise patient to

    bathe every other day

    and to apply skin

    lotion immediately

    upon exiting

    bath/shower. Patients

    should avoid harsh

    soaps, soaps or skin

    products containing

    alcohol, and excessive

    bathing, to prevent

    skin dryness and

    itching.

    7. Advise patient and

    significant othersabout increased risk

    for bruising. Clotting

    abnormalities and

    capillary fragility

    place patient with

    uremia at increased

    risk for bruising.

    8. Provide

    scheduled skin care

    and position changes

    for patients with

    edema. Helps

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    prevents skin

    breakdown from

    pressure.

    PROBLEM OBJECTIVE INTERVENTION EVALUATION

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