arrhythmias of heart
DESCRIPTION
Arrhythmias of heart. Department of propede u ti c of internal medicine. Etiology. Violations of rhythm of cardiac activity cause such defeats of myocardium: organic: IHD,defects of heart, AH, m y o c arditi s, c ardiom y opat hy; · toxic: medicines, alcohol; - PowerPoint PPT PresentationTRANSCRIPT
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Arrhythmias of heart
Department of propedeutic of internal medicine
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Etiology.
• Violations of rhythm of cardiac activity cause such defeats of myocardium:
• organic: IHD,defects of heart, AH, myocarditis, cardiomyopathy;
• · toxic: medicines, alcohol;• · hormonal : thyrotoxicosis, myxedema,
pheochromocytoma, climax;• · functional : neurogenic, sporting;• · anomalies of development of heart - more
frequent all WPW.
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Pathogenesis
• Theories of origin of arrhythmias:
• Pathological automatism (presence of ectopic focuses)
• Mechanism of re-entry
• trigern (starting) activity.
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Classification of violations of rhythm and conductivity of
heart• I. Violations of formation of impulse• Sinus tachycardia
(more than 90 complexes are for a minute)
• Sinus bradycardia (less than 60 complexes are for a minute )
• Sinus arrhythmia • Stop (refuse) of sinus node• Migration of supraventricular driver of rhythm
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• Extrasystolia (a synonym is premature depolarization):
• auricle (atrial)
• auricle-ventrical (atrioventricular)
• ventrical
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• Tachycardia:
• supraventricular:
• sino - auricle
• (sinoatrial)
• auricle (atrial)
• auricle - ventrical
• (atrioventrical)
• ordinary (to 30 in a hour)
• frequent (30 and anymore in a hour)
• allorythmia (bi-, thre-, quadrigeminia)
• polymorphic
• twin
• early (R on T)
• chronic
• paroxysmal
• noudle
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• with the additional ways of leadthrough
• ortodromal
• antidromal
• ventrical:
• unsteady (from 3 ventrical complexes to 30)
• steady (more than 30 seconds)
• monomorphic
• polymorphic
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• ІІ. VIOLATION OF LEADTHROUGH OF IMPULSE
- Sinoauricular of blockade- Atrioventricular blockades:
• І st.
• ІІ st.
• ІІІ st.
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• III. COMBINED DYSPOIESISS AND LEADTHROUGHS OF IMPULSE
• · auricle
• Parasystole · from AV of connection
• · ventrical
• specified at possibility
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• IV. DISEASE, SYNDROMES And PHENOMENA
– Syndromes and ECG phenomena of pre-excitation of ventricles:
• syndrome of WPW
• syndrome of the shortened interval of PR (Launa - Ganonga - Livayna)
• syndrome of early repolarisation of ventricles
• innate
• purchased
• Syndrome of the prolonged interval of QT
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• Syndrome of weakness of sinus noudle
• Syndrome of Morganii-Adams-Stoks
• Syndrome of Frederic
• Cardiac sudden death (arrhythmical)
• asystole
• fibrilation of ventricles
• electromechanics dissociation
• Heart arest
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• V. ARRHYTHMIAS ARE AT NORMAL OR BROKEN FUNCTION OF CARDIOSTIMULATORS OF DIFFERENT TYPE
• Note: In a diagnosis it is needed to specify cardiac surgery interferences and devices, applied for treatment of arrhythmias and asequences heart (with pointing of method and to give interference) - kateterni (radio frequency et al) destruction, implantation of drivers of rhythm and cardioverters - defibrilyatoriv, leadthrough of cardioversion or defibrilyacii (the date of the last is specified) and others like that.
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Description of normal sinus rhythm
• correct rhythm with frequency of heartbeats 60-100 per 1 min.
• the P wave is positive in II, III, AVF leads, negative - in the AVR leads, permanent form of P wave
• a complex QRS follows by every P waveR (if there is not а-v-blockade).
• Interval of P-Q>0.12 (if there are not additional ways of leadthrough).
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Sinus tachycardia
• ECG is criteria:• correct rhythm• sinus P waves are ordinary configuration.• 100-180 beats per 1 min.• gradual beginning and completion• Reasons: physical and emotional loading, pain,
fever, hypovolumia, hypotension, anaemia, thyrotoxicosis, action of certain matters (coffeine, alcohol)
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Treatment
• liquidation of etiologic factor
• B-adrenoblockers (propranolol 10-40mg, oksprenolol 20mg 3 times per days, pindolol 5 mg 3 times on days, nadolol 20 mg 2 times per days, metaprolol 50 mg 2 times per days)
• sedative facilities
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Sinus bradycardia
• ECG is criteria:
• correct rhythm
• less than 60 beats per 1 min
• sinus P waves
• interval of PQ >0,12 sec.
• Reasons: increase of parasympatic tonus, myocarditis, myxedema, hypothermia, mechanical icterus, syndrome of weakness of sinus knot.
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Treatment
• to the atropine sulfate of 0,5-1 mg or 0,1% solution 1-2 times per day
• plathyphylinum hydrotartratum 0,5-1 mg or 0,2% solution 1-2 times per day
• extract of beladonnae -0,02 gr. 3-4 times per days
• izadrin 5-10 mg sublingval
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The first aid is at the attacks of Morgani-Adams-Stoks
• to the atropine sulfate of 0,5-1 mg or 0,1% solution intravenously
• izadrin 5-10 mg sublingval (at a necessity to repeat)
• alupent for 1-5-10 mg on 500 ml of izotonic soluble-sodium chloride
• electro-cardiostimulation (temporal or permanent)
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Sinus arrhythmia
• ECG is criteria:• P wave is sinus origin• interval of PQ >0,12 s• 45-100 beats per 1 min• the rhythm of heart is wrong with the
difference of the longest and shortest interval of R-R 0.16 and anymore
• Treatment: does not require
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• a-normal sinus rhythm• б- sinus tachycardia• в- sinus bradycardia• г- sinus arrhythmia
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Arest of sinus knot
• EKG is criteria:
• complete asystole (absence of P and QRST)
• the pause of R-R does not make two ordinary intervals, and anymore.
• Treatment: how to the syndrome of Morgani-Adams-Stoks
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Syndrome of weakness of sinus knot
• EKG is criteria:• sinus bradycardia is expressed• refuse of sinus knot with periods asystoles• a duty of sinus bradycardia is with the attacks
of auricle tachycardia, fibrilation (syndrome of «tachy-brady»)
• a sino-atrial blockade is with extra contractions heart beats
• slow renewal of function of sine knot.
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Treatment
• effective treatment does not exist
• medical therapy is at the attacks of Morgani-Adams-Stoks
• implantation of permanent electro-cardyostimulator
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Extrasystolia
premature excitation and reduction of heart or his separate parts is as a result of increase of activity of hearths of ectopic automatism.
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Auricle extrasystolia
• EKG of sign:• premature reduction after which incomplete
scray pause• the P wave is changed, negative• a complex QRS is not changed or aberrant.• Treatment: it is not needed, at presence of
clinical symptoms of b-blockers, verapamil, digitalis.
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• a- from the overhead departments of auricle
• б- from the middle departments of auricle
• в- from the lower departments of auricle
• г- is blocked auricle extrasystole
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А-V- extrasystoles
• with simultaneous excitation of atriums and ventricles.
• EKG-signs:
• the P wave is not determined
• an extraordinary complex QRS is not extended
• incomplete scray pause
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• with previous excitation of ventricles
• EKG-signs:
• an extraordinary complex QRS is not extended
• P wave is after QRS
• complete scray pause
• Treatment: like, as at auricle extrasystolia.
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Ventrical extrasystolia
• EKG of sign:
• complex QRS wide without a previous P wave
• complete scray pause
• Treatments need frequent monotopic, politopic, group and early as R/T extrasystoles.
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• a- sinistroventrical extrasystole
• b- dextraventrical extrasystole
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Paroxysmal tachycardia Supraventricular
tachycardia• EKG is signs:
• frequency of reductions of atriums - 120-250 per 1 min.
• auricle complexes are preceded the complexes of QRS
• the complexes of QRS are not changed
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Atrioventricular tachycardia
• EKG-signs:
• 150-200 heart beats per 1 min.
• retrograde P wave (negative) after QRS or accumulates on him
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• a- auricle paroxysmal tachycardia• б - atrioventricular tachycardia with previous excitation of
ventricles• в - atrioventricular tachycardia with simultaneous excitation of
atriums and ventricles
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Treatment of paroxysmal supraventricular tachycardia
• at leadthrough to the impulse through an a-v knot:
• massage of sinus carotic• izoptin 0,25%-4ml• obzіdan 0,1% - 5ml• dіgoxin 0,025% - 1ml v/v• cordaron 5% - 6 ml
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Ventrical tachycardia
• EKG is criteria: - >140 hearts beats per 1 min.
- the complexes of QRS are extended
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EKG is at paroxysmal ventrical tachycardia
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Trembling and fibrilation of atriums
• EKG is criteria of trembling:
• frequency of auricle waves 250-350 per 1 min. ( waves of f)
• EKG is criteria of fibrilation:
• waves of f
• wrong rhythm (different R-R)
• absence of P wave
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Trembling and fibrilation of ventricles
• EKG is criteria:• Sinus wave curve with frequent, rhythmic, wide
and high waves, excitation of ventricles with frequency 200-300 per 1 min.
• it is not possible to distinguish the elements of ventrical complex
• Treatment:• electric cardioversion 200-300 Dzh. At unefficiency
the repeated cardioversion
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• a- trembling of ventricles
• b- blinking and fibrilation of ventricles
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Sinoauricular blockade
• EKG is criteria:
• periodic fall of cardiac cycles
• increase of pause between the waves of Р-Р in 2 times
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Inwardly auricle blockade
• EKG is criteria:
• increase of duration and breaking up of wave Р
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Atrioventricular blockade Іst
• permanent lengthening of interval of P-Q anymore as on 0,20s• a- auricle form• б- key form• в- distal form blockade
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А-v blockade ІІ ст • the periodic stopping of leadthrough of impulse is
from an auricle to the ventricles. There are three types:
• I type (Mobit I) is the gradual lengthening of interval of P-Q with the subsequent fall of QRST ( periods of Samoylov-Venkenbach)
• ІІ type (Mobit ІІ) is a fall of complexes of QRST without the gradual lengthening of interval of P-Q
• ІІІ type (Mobit ІІІ) of fall every second, or 2 and more complexes successively
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А-v blockade ІІІ ст
complete autonomy of reduction of auricle and ventricles.The intervals of P-P and R-R are permanent, but R-R>P-P.
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EKG is at the complete blockade of right bundle of bunch of Hiss
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EKG is at the complete blockade of left bundle of bunch of Hiss
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Syndrome of Wolf-Parkinson-White
• reduction of interval of P-Q (R)• a presence in composition a complex QRS
of additional wave of excitation is d-wave• deformation and increase of duration of
complex QRS• discordant displacement of RS-T and
change of polarity of wave T (inconstant sign)
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EKG is at the syndrome of WPW
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Conclusions• It is necessary to know for successful diagnostics
and treatment of arrhythmias:• basic nosotropic mechanisms of their development• · to own the methods of diagnostics of
arrhythmias• · to distinguish of high quality, potentially
malignant and malignant arrhythmias• · to understand algorithms diagnostic at the
therapeutic going near the different types of arrhythmias